1. RV vs LV Myocardial Infarction
By Wendy Cooley RN BSN
2. RVMI and Coronary Circulation
The majority of Right Ventricular Myocardial Infarction (RVMI) result from 100%
occlusion of the proximal Right Coronary Artery (RCA)
RVMI is also associated with acute ST-elevation MI of the inferior wall of the LeftVentricle
as left dominant systems feed the RV in 15% of patients
Therefore, in the presence of hypotension or cardiogenic shock without signs of LV
failure, a diagnosis of RVMI is highly probable
Chronic lung disease and right ventricular hypertrophy are considered significant risk
factors for RVMI
3. Importance of Identification & Differentiation
RVMI occur in about ~50% of all inferior MIs
RVMI has a mortality rate of 23-30% BUT those who survive have a recovery
rate of 62-82% in the first few months
LeadV4R is 90% specific to RVMI
Therefore, literature suggests that every patient with ST ↑ in leads II, III & AVF
should have right-sided precordial lead ECG
Because, proper identification leads to proper treatment which leads to tissue &
patient survival
6. Coronary
Arteries as
they associate
with the ECG
Inferior: RCA, PDA, PLV (Leads
II, III, aVF) Suspect RVMI
Lateral: Circumflex (Leads I, aVL,
V5-V6*)
Anterior: LAD, Diagonals, Septal
(Leads V1-V6*) Suspect LVMI
*Leads V5-6 can appear as lateral or anterior
depending on coronary anatomy. Often referred
to as an “Antero-lateral MI”
7. Blood pressure basics
Injured muscle from an MI can’t pump well. Depending on which ventricle is affected
will determine your treatment.
Pre-load-Volume of blood at end diastolic in the RV
After-load- Forward resistance that LV has to pump against
StrokeVolume- Amount of blood pumped out with each beat
Contractility- How well the heart contracts with each beat
Cardiac Output- How much volume the heart ejects per minute
Volume- Amount of blood in the system
8. Symptoms:
Chest Pain is still the most frequent symptom of MI in
addition to nausea/vomiting, diaphoresis, dizziness & anxiety
LVMI
LV has lost pumpability & cannot push to body
therefore blood will back up in the lungs:
Pulmonary congestion
New MV murmur
CHF
Flash pulmonary edema
Cardiogenic shock
RVMI
RV has lost pumpability & cannot push to lungs
therefore blood will back up in the body:
Clear lungs sounds
Hypotension
Jugular distention
Bradycardia, possible vagal response
Kussmaul’s respirations
Pulsus paradoxus
9. MI and Arrhythmias
LVMI
Ventricular tachycardia
Ventricular fibrillation
Tachycardia
Be ready to defibrillate
RVMI
Third Degree Heart Block
A.fib/flutter
Bradycardia or Tachycardia
Be ready to transcutaneous pace
10. MI Mimics
LVMI
Pericarditis
LV Hypertrophy
Early repolarization
Hyperkalemia
RVMI
Pulmonary Embolism
Anterior- Septal MI
Pericarditis
11. Emergent PCI & Specific Treatment
LVMI
1. Nitroglycerin
2. Morphine
3. Diuretics
4. Inotropes
5. Amiodarone/Lidocaine
6. Ventricular Support (IABP or LV
Impella)
Defibrillation if needed
RVMI
1. Fluids for Pre-load
2. Fluids for Pre-load (get the point here?)
3. Inotropes
4. Atropine
5. Ventricular Support (IABP or Impella both LV
or RV)
Temporary pacer if needed