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Chapter 6
Burns
Topics
Introduction to Burn Injuries
Anatomy and Physiology of the Skin
Pathophysiology of Burns
Assessment of Thermal Burns
Management of Thermal Burns
Assessment and Management of Electrical,
Chemical, and Radiation Burns
Bla bla bla bla
Introduction to Burn Injuries
Introduction to Burn Injuries
1.25–2 million Americans treated for burns
annually
– 45,000 require hospitalization
– 90% of burns can be prevented
– 3–5% considered life threatening
– 2nd leading cause of death for children <12
– Half of all tap-water burns occur to children <5
Introduction to Burn Injuries
Greatest Risk
– Very young and very old
– Infirm
– Firefighters
– Metal smelters
– Chemical workers
Drugs and alcohol play major role
Alcohol plays a major role
Introduction to Burn Injuries
Reducing burn injuries
– Improved building codes
– Safer construction techniques
– Sprinkler systems
– Use of smoke detectors
– Educational campaigns aimed primarily at school
children
Anatomy and Physiology
of the Skin
Anatomy and Physiology
of the Skin
Layers
– Epidermis
– Dermis
– Subcutaneous
– Underlying
structures
Fascia
Nerves
Tendons
Ligaments
Muscles
Organs
Anatomy and Physiology
of the Skin
Functions of the Skin
– Protection from infection
– Sensory organ
Temperature
Touch
Pain
– Controls loss and movement of fluids
– Temperature regulation
– Insulation from trauma
– Flexible to accommodate free body movement
Pathophysiology of Burns
Pathophysiology of Burns
Disruption of proteins in the cell membranes
Causes
– Thermal
– Electrical
– Chemical
– Radiation energies
Thermal Burns
Molecular structure changed by heat
Extent of burn damage depends on:
– Temperature of agent
– Concentration of heat
– Duration of contact
Thermal Burns
Jackson’s Theory of Thermal Wounds
– Zone of Coagulation
Area in a burn nearest the heat source
Suffers the most damage as evidenced by clotted blood and
thrombosed blood vessels
– Zone of Stasis
Characterized by decreased blood flow
– Zone of Hyperemia
Increased blood flow
Jackson’s Theory of
Thermal Wounds
Zone of Hyperemia
Zone of Stasis
Zone of Coagulation
Thermal Burns
Emergent Phase (Stage 1)
– Pain response
– Catecholamine release
Tachycardia
Tachypnea
Mild hypertension
Mild anxiety
Thermal Burns
Fluid Shift Phase (Stage 2)
– Length 18–24 hours
– Begins after emergent
phase
Reaches peak in 6–8 hours
– Damaged cells initiate
inflammatory response:
Vasodilation
Increased capillary permeability
Intravascular hypovolemia
Extravascular edema
Thermal Burns
Hypermetabolic Phase (Stage 3)
– Large increase in the body’s need for nutrients as
it repairs itself
– Fluid and electrolytes begin to move back into the
vasculature
– Influx of fluid within vascular space causes the
GFR to increase, leading to diuresis
– Fluid shifts may lead to hypernatremia and
hypokalemia
– Cardiac workload and O2 consumption increase
Thermal Burns
Resolution Phase (Stage 4)
– Scar formation
– General rehabilitation; progression to normal function
Electrical Burns
Terminology
– Voltage – the pressure
Difference of electrical potential between two points
Different concentrations of electrons
– Amperes – the velocity
Strength of electrical current
– Resistance (Ohms) – the friction
Opposition to electrical flow
Electrical Burns
Greatest heat occurs at the points of
resistance:
– Entrance and exit wounds
– Dry skin = greater resistance
– Wet Skin = less resistance
Longer the contact, the greater the potential
of injury
– Increased damage inside body
Smaller the point of contact, the more
concentrated the energy, the greater the
injury
Electrical Burns
Electrical Current Flow
– Tissue of less resistance
Blood vessels
Nerve
– Tissue of greater resistance
Muscle
Bone
Chemical Burns
Chemicals destroy tissue
– Acids
Form a thick, insoluble mass
where they contact tissue
Coagulation necrosis
Limits burn damage
– Alkalis
Destroy cell membrane
through liquefaction necrosis
Deeper tissue penetration and
deeper burns
© Roy Alson, PhD, MD, FACEP, FAAEM
Coagulation Necrosis
Alkali Burn
Radiation Injury
Radiation
– Transmission of energy
Nuclear energy
Ultraviolet light
Visible light
Heat
Sound
X-rays
Radioactive Substance
– Emits ionizing radiation
– Radionuclide or radioisotope
Types of Radiation
Alpha
– Very weak energy source
– Only significant if ingested
Beta
– Can travel 6 to 10 feet
– May penetrate clothing
Gamma
– Most powerful type of ionizing
radiation
– Penetrates entire body
Neutron
– Great penetrating power
Radiation Injury
Exposure can occur through two
mechanisms:
– Direct exposure to a strong radioactive source
– Contamination by dust, debris, or fluids that
contain very small particles of radioactive material
Radiation Injury
Three factors are important to remember:
– Duration
The longer exposed, the more absorption
– Distance
Travel farther from the source for safety
– Shielding
The more material between you and the source, the less
radioactive exposure you experience
Radiation Injury
Radiation exposure
is measured with a
Geiger counter
Cumulative
exposure is
recorded by a
device called a
dosimeter
© Ogunquit, Maine Fire-Rescue
© Jeff Forster
Radiation Injury
Different tissues are sensitive to different
levels of absorbed radiation
Signs and symptoms of exposure
– Nausea and fatigue
– Anorexia, vomiting, diarrhea, and malaise
– Erythema of the skin
– Confusion
– Watery diarrhea
– Physical collapse
Long-term effects include cancer and sterility
Inhalation Injury
Toxic Inhalation
– Synthetic resin
combustion
– More common than
thermal injury
Carbon Monoxide
Poisoning
– Colorless, odorless,
tasteless gas
– Byproduct of
incomplete combustion
of carbon products
Suspect with faulty
heating unit
Inhalation Injury
Airway Thermal Burn
– Supraglottic structures absorb heat and prevent
lower airway burns
Moist mucosa lining the upper airway
– Injury is common from superheated steam
– Symptoms:
Stridor or “crowing” inspiratory sounds
Singed facial and nasal hair
Black sputum or facial burns
Progressive respiratory obstruction and arrest due to
swelling
Depth of Burn
Depth of burn damage is normally
classified into three categories
Burn Depth
Superficial Burn
– Red
– Painful
– Dry (no blisters)
Burn Depth
Partial-Thickness
Burn
– Red or White
– Painful
– Blisters (wet)
Burn Depth
Full-Thickness Burn
– Leathery skin
White
Dark brown
Charred
– Minimally painful
– Dry
Body Surface Area
Rule of Nines
– Best used for large surface areas
– Expedient tool to measure extent of burn
Rule of Palms
– Irregular or splash burns
– Best used for burns <10% BSA
Rule of Nines
Rule of Palms
Systemic Complications
Hypothermia
– Disruption of skin and its ability to thermoregulate
Hypovolemia
– Shift in proteins, fluids, and electrolytes to the
burned tissue
Eschar
– Hard, leathery product of a deep full-thickness
burn
– Dead and denatured skin
Systemic Complications
Infection
– Greatest risk of burn is infection
– Carefully employ Standard Precautions
Organ Failure
– Release of myoglobin
Clogs the tubules of the kidneys
Special Factors
– Age and health
Systemic Complications
Physical Abuse
– Child or an elderly
and infirm adult
– MOI does not make
sense
Multiple circular burns
Dipping
“Stocking” burns
Branding
© Roy Alson. PhD, MD, FDCEP, FAAEM
Assessment of Thermal Burns
Assessment of Thermal Burns
Skin evaluation tells more about the body’s
condition than any other aspect of patient
assessment.
– Must be deliberate, careful, and complete
Assign burns the appropriate priority for care.
Assessment of Thermal Burns
Scene Size-up
– Do not enter until
the scene is made
safe
– Be wary of entering
enclosed spaces
– Stop the burning
process
– Consider the burn
mechanism
– Consider the need
for other resources
© Glen E. Ellman
Assessment of Thermal Burns
Initial Assessment
– Form a general impression of the patient
– Ensure the airway is patent
Look for the signs of any thermal or inhalation injury
Provide high-flow, high-concentration oxygen
– Ensure that the patient’s breathing is adequate
Assessment of Thermal Burns
Focused and Rapid Trauma Assessment
– Accurately approximate extent of burn injury:
Rule of Nines or Rule of Palms
Depth of burn
Area of body affected
Age of patient affected
Severity of Thermal Burns
Any partial- or full-thickness burn involving hands, feet, joints, face, or genitalia
>30% BSA
Partial Thickness
Inhalation Injury
>10% BSA
Full Thickness
Critical
>2% BSA
Full Thickness
>50% BSA
Superficial
<2% BSA
Full Thickness
<15% BSA
Partial Thickness
<50% BSA
Superficial
>15% BSA
Partial Thickness
Moderate
Minor
Burn Severity
Assessment of Thermal Burns
Ongoing Assessment
– Non-critical: Reassess Q 15 min
– Critical: Reassess Q 5 min
– Watch for early signs of hypovolemia and airway
problems
Be cautious of aggressive fluid therapy
– Carefully monitor distal circulation and sensation
with any circumferential burn
Management of Thermal
Burns
Management of Thermal Burns
Includes the prevention of shock,
hypothermia, and any further wound
contamination
Care is divided into two categories:
– Local and minor burn care
– Moderate and severe burn care
Management of Thermal Burns
Local and Minor Burns
– Local cooling
Partial thickness: <15% of BSA
Full thickness: <2% BSA
– Remove clothing
– Cool or cold water immersion
– Consider analgesics
Morphine sulfate
Fentanyl (Sublimaze)
Management of Thermal Burns
Moderate to Severe Burns
– Dry sterile dressings
– Maintain warmth
Prevent hypothermia
– Consider aggressive fluid therapy
– Burns over IV sites:
Place IV in partial-thickness burn site
– Consider analgesics
Morphine sulfate
Fentanyl (Sublimaze)
Management of Thermal Burns
Fluid resuscitation
– Parkland Formula
4 mL X weight X % burn
½ volume in first 8 hours
Second ½ over last 16 hours
– Where transport time is short (less than 1 hour)
0.25 mL X Patient weight in kg X BSA burned = Amount
of fluid
Management of Thermal Burns
Moderate to Severe Burns
– Caution for fluid overload
Frequent auscultation of breath sounds
– Consider analgesic for pain
Morphine
Fentanyl
– Prevent infection
Management of Thermal Burns
Inhalation Injury
– Provide high-flow O2
by NRB
Consider intubation if
swelling
Consider hyperbaric
oxygen therapy
Management of Thermal Burns
Cyanide exposure
– Sodium nitrite, amyl
nitrite, sodium
thiosulfate
Forms methemoglobin
which binds cyanide
Excreted in urine
– Cyanokit®
Binds cyanide by freeing it
from the cytochromea3
enzyme
Excreted in urine
Courtesy © Dey, L.P.
© Jeff Forster
Assessment and Management
of Electrical, Chemical, and
Radiation Burns
Assessment and Management
of Electrical, Chemical, and
Radiation Burns
Electrical Injuries
– Safety
Turn off power
Energized lines act as whips
Establish a safety zone
– Lightning strikes
High voltage, high current, high energy
Lasts fraction of a second
No danger of electrical shock to EMS
Electrical Injuries
Entrance and exit wounds
Remove clothing, jewelry, and leather items
Treat any visible injuries
ECG monitoring
Consider fluid bolus for serious burns.
– 20 ml/kg
Consider sodium bicarbonate
– 1 mEq/kg
Consider mannitol:
– 10 g
Chemical Burns
Chemical Burns
– Scene size-up
Hazardous materials team
Establish hot, warm, and cold zones
Prevent personnel exposure from chemical
– Specific Chemicals
Phenol
Dry lime
Sodium
Riot control agents
Chemical Burns
Specific Chemicals
– Phenol
Industrial cleaner
Alcohol dissolves phenol
Irrigate with copious amounts of water
– Dry Lime
Strong corrosive that reacts with water
Brush off dry substance
Irrigate with copious amounts of cool water
Prevents reaction with patient tissues
Chemical Burns
Sodium
– Unstable metal
– Reacts vigorously with water
– Decontaminate:
Brush off dry chemical
– Cover the wound with oil substance used to
store metal
Chemical Burns
Riot Control Agents
– Agents
CS, CN (Mace), Oleoresin, Capsicum (OC, pepper
spray)
– Irritation of the eyes, mucous membranes, and
respiratory tract
– No permanent damage
– General signs and symptoms
Coughing, gagging, and vomiting
Eye pain, tearing, temporary blindness
– Management
Irrigate eyes with normal saline
Tar
Hot Asphalt
Phenol
Lime Burn
Riot Agents
Radiation Burns
Radiation Burns
– Notify hazardous materials team
– Establish safety zones
Hot, warm, and cold
– Personnel positioned upwind and uphill
– Use older rescuers for recovery
– Decontaminate ALL rescuers, equipment, and
patients
Assessment and Management
of Electrical, Chemical, and
Radiation Burns
Ongoing Assessment
– Re-evaluate initial assessment
– Re-evaluate all interventions
Summary
Introduction to Burn Injuries
Anatomy and Physiology of the Skin
Pathophysiology of Burns
Assessment of Thermal Burns
Management of Thermal Burns
Assessment and Management of Electrical,
Chemical, and Radiation Burns

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Burn suza

  • 2. Topics Introduction to Burn Injuries Anatomy and Physiology of the Skin Pathophysiology of Burns Assessment of Thermal Burns Management of Thermal Burns Assessment and Management of Electrical, Chemical, and Radiation Burns Bla bla bla bla
  • 4. Introduction to Burn Injuries 1.25–2 million Americans treated for burns annually – 45,000 require hospitalization – 90% of burns can be prevented – 3–5% considered life threatening – 2nd leading cause of death for children <12 – Half of all tap-water burns occur to children <5
  • 5. Introduction to Burn Injuries Greatest Risk – Very young and very old – Infirm – Firefighters – Metal smelters – Chemical workers Drugs and alcohol play major role
  • 6. Alcohol plays a major role
  • 7. Introduction to Burn Injuries Reducing burn injuries – Improved building codes – Safer construction techniques – Sprinkler systems – Use of smoke detectors – Educational campaigns aimed primarily at school children
  • 9. Anatomy and Physiology of the Skin Layers – Epidermis – Dermis – Subcutaneous – Underlying structures Fascia Nerves Tendons Ligaments Muscles Organs
  • 10. Anatomy and Physiology of the Skin Functions of the Skin – Protection from infection – Sensory organ Temperature Touch Pain – Controls loss and movement of fluids – Temperature regulation – Insulation from trauma – Flexible to accommodate free body movement
  • 12. Pathophysiology of Burns Disruption of proteins in the cell membranes Causes – Thermal – Electrical – Chemical – Radiation energies
  • 13. Thermal Burns Molecular structure changed by heat Extent of burn damage depends on: – Temperature of agent – Concentration of heat – Duration of contact
  • 14. Thermal Burns Jackson’s Theory of Thermal Wounds – Zone of Coagulation Area in a burn nearest the heat source Suffers the most damage as evidenced by clotted blood and thrombosed blood vessels – Zone of Stasis Characterized by decreased blood flow – Zone of Hyperemia Increased blood flow
  • 15. Jackson’s Theory of Thermal Wounds Zone of Hyperemia Zone of Stasis Zone of Coagulation
  • 16. Thermal Burns Emergent Phase (Stage 1) – Pain response – Catecholamine release Tachycardia Tachypnea Mild hypertension Mild anxiety
  • 17. Thermal Burns Fluid Shift Phase (Stage 2) – Length 18–24 hours – Begins after emergent phase Reaches peak in 6–8 hours – Damaged cells initiate inflammatory response: Vasodilation Increased capillary permeability Intravascular hypovolemia Extravascular edema
  • 18. Thermal Burns Hypermetabolic Phase (Stage 3) – Large increase in the body’s need for nutrients as it repairs itself – Fluid and electrolytes begin to move back into the vasculature – Influx of fluid within vascular space causes the GFR to increase, leading to diuresis – Fluid shifts may lead to hypernatremia and hypokalemia – Cardiac workload and O2 consumption increase
  • 19. Thermal Burns Resolution Phase (Stage 4) – Scar formation – General rehabilitation; progression to normal function
  • 20. Electrical Burns Terminology – Voltage – the pressure Difference of electrical potential between two points Different concentrations of electrons – Amperes – the velocity Strength of electrical current – Resistance (Ohms) – the friction Opposition to electrical flow
  • 21. Electrical Burns Greatest heat occurs at the points of resistance: – Entrance and exit wounds – Dry skin = greater resistance – Wet Skin = less resistance Longer the contact, the greater the potential of injury – Increased damage inside body Smaller the point of contact, the more concentrated the energy, the greater the injury
  • 22. Electrical Burns Electrical Current Flow – Tissue of less resistance Blood vessels Nerve – Tissue of greater resistance Muscle Bone
  • 23. Chemical Burns Chemicals destroy tissue – Acids Form a thick, insoluble mass where they contact tissue Coagulation necrosis Limits burn damage – Alkalis Destroy cell membrane through liquefaction necrosis Deeper tissue penetration and deeper burns © Roy Alson, PhD, MD, FACEP, FAAEM
  • 26. Radiation Injury Radiation – Transmission of energy Nuclear energy Ultraviolet light Visible light Heat Sound X-rays Radioactive Substance – Emits ionizing radiation – Radionuclide or radioisotope
  • 27. Types of Radiation Alpha – Very weak energy source – Only significant if ingested Beta – Can travel 6 to 10 feet – May penetrate clothing Gamma – Most powerful type of ionizing radiation – Penetrates entire body Neutron – Great penetrating power
  • 28. Radiation Injury Exposure can occur through two mechanisms: – Direct exposure to a strong radioactive source – Contamination by dust, debris, or fluids that contain very small particles of radioactive material
  • 29. Radiation Injury Three factors are important to remember: – Duration The longer exposed, the more absorption – Distance Travel farther from the source for safety – Shielding The more material between you and the source, the less radioactive exposure you experience
  • 30. Radiation Injury Radiation exposure is measured with a Geiger counter Cumulative exposure is recorded by a device called a dosimeter © Ogunquit, Maine Fire-Rescue © Jeff Forster
  • 31. Radiation Injury Different tissues are sensitive to different levels of absorbed radiation Signs and symptoms of exposure – Nausea and fatigue – Anorexia, vomiting, diarrhea, and malaise – Erythema of the skin – Confusion – Watery diarrhea – Physical collapse Long-term effects include cancer and sterility
  • 32. Inhalation Injury Toxic Inhalation – Synthetic resin combustion – More common than thermal injury Carbon Monoxide Poisoning – Colorless, odorless, tasteless gas – Byproduct of incomplete combustion of carbon products Suspect with faulty heating unit
  • 33. Inhalation Injury Airway Thermal Burn – Supraglottic structures absorb heat and prevent lower airway burns Moist mucosa lining the upper airway – Injury is common from superheated steam – Symptoms: Stridor or “crowing” inspiratory sounds Singed facial and nasal hair Black sputum or facial burns Progressive respiratory obstruction and arrest due to swelling
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  • 39. Depth of Burn Depth of burn damage is normally classified into three categories
  • 40. Burn Depth Superficial Burn – Red – Painful – Dry (no blisters)
  • 41. Burn Depth Partial-Thickness Burn – Red or White – Painful – Blisters (wet)
  • 42. Burn Depth Full-Thickness Burn – Leathery skin White Dark brown Charred – Minimally painful – Dry
  • 43. Body Surface Area Rule of Nines – Best used for large surface areas – Expedient tool to measure extent of burn Rule of Palms – Irregular or splash burns – Best used for burns <10% BSA
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  • 47. Systemic Complications Hypothermia – Disruption of skin and its ability to thermoregulate Hypovolemia – Shift in proteins, fluids, and electrolytes to the burned tissue Eschar – Hard, leathery product of a deep full-thickness burn – Dead and denatured skin
  • 48. Systemic Complications Infection – Greatest risk of burn is infection – Carefully employ Standard Precautions Organ Failure – Release of myoglobin Clogs the tubules of the kidneys Special Factors – Age and health
  • 49. Systemic Complications Physical Abuse – Child or an elderly and infirm adult – MOI does not make sense Multiple circular burns Dipping “Stocking” burns Branding © Roy Alson. PhD, MD, FDCEP, FAAEM
  • 51. Assessment of Thermal Burns Skin evaluation tells more about the body’s condition than any other aspect of patient assessment. – Must be deliberate, careful, and complete Assign burns the appropriate priority for care.
  • 52. Assessment of Thermal Burns Scene Size-up – Do not enter until the scene is made safe – Be wary of entering enclosed spaces – Stop the burning process – Consider the burn mechanism – Consider the need for other resources © Glen E. Ellman
  • 53. Assessment of Thermal Burns Initial Assessment – Form a general impression of the patient – Ensure the airway is patent Look for the signs of any thermal or inhalation injury Provide high-flow, high-concentration oxygen – Ensure that the patient’s breathing is adequate
  • 54. Assessment of Thermal Burns Focused and Rapid Trauma Assessment – Accurately approximate extent of burn injury: Rule of Nines or Rule of Palms Depth of burn Area of body affected Age of patient affected
  • 55. Severity of Thermal Burns Any partial- or full-thickness burn involving hands, feet, joints, face, or genitalia >30% BSA Partial Thickness Inhalation Injury >10% BSA Full Thickness Critical >2% BSA Full Thickness >50% BSA Superficial <2% BSA Full Thickness <15% BSA Partial Thickness <50% BSA Superficial >15% BSA Partial Thickness Moderate Minor Burn Severity
  • 56. Assessment of Thermal Burns Ongoing Assessment – Non-critical: Reassess Q 15 min – Critical: Reassess Q 5 min – Watch for early signs of hypovolemia and airway problems Be cautious of aggressive fluid therapy – Carefully monitor distal circulation and sensation with any circumferential burn
  • 58. Management of Thermal Burns Includes the prevention of shock, hypothermia, and any further wound contamination Care is divided into two categories: – Local and minor burn care – Moderate and severe burn care
  • 59. Management of Thermal Burns Local and Minor Burns – Local cooling Partial thickness: <15% of BSA Full thickness: <2% BSA – Remove clothing – Cool or cold water immersion – Consider analgesics Morphine sulfate Fentanyl (Sublimaze)
  • 60. Management of Thermal Burns Moderate to Severe Burns – Dry sterile dressings – Maintain warmth Prevent hypothermia – Consider aggressive fluid therapy – Burns over IV sites: Place IV in partial-thickness burn site – Consider analgesics Morphine sulfate Fentanyl (Sublimaze)
  • 61. Management of Thermal Burns Fluid resuscitation – Parkland Formula 4 mL X weight X % burn ½ volume in first 8 hours Second ½ over last 16 hours – Where transport time is short (less than 1 hour) 0.25 mL X Patient weight in kg X BSA burned = Amount of fluid
  • 62. Management of Thermal Burns Moderate to Severe Burns – Caution for fluid overload Frequent auscultation of breath sounds – Consider analgesic for pain Morphine Fentanyl – Prevent infection
  • 63. Management of Thermal Burns Inhalation Injury – Provide high-flow O2 by NRB Consider intubation if swelling Consider hyperbaric oxygen therapy
  • 64. Management of Thermal Burns Cyanide exposure – Sodium nitrite, amyl nitrite, sodium thiosulfate Forms methemoglobin which binds cyanide Excreted in urine – Cyanokit® Binds cyanide by freeing it from the cytochromea3 enzyme Excreted in urine Courtesy © Dey, L.P. © Jeff Forster
  • 65. Assessment and Management of Electrical, Chemical, and Radiation Burns
  • 66. Assessment and Management of Electrical, Chemical, and Radiation Burns Electrical Injuries – Safety Turn off power Energized lines act as whips Establish a safety zone – Lightning strikes High voltage, high current, high energy Lasts fraction of a second No danger of electrical shock to EMS
  • 67. Electrical Injuries Entrance and exit wounds Remove clothing, jewelry, and leather items Treat any visible injuries ECG monitoring Consider fluid bolus for serious burns. – 20 ml/kg Consider sodium bicarbonate – 1 mEq/kg Consider mannitol: – 10 g
  • 68. Chemical Burns Chemical Burns – Scene size-up Hazardous materials team Establish hot, warm, and cold zones Prevent personnel exposure from chemical – Specific Chemicals Phenol Dry lime Sodium Riot control agents
  • 69. Chemical Burns Specific Chemicals – Phenol Industrial cleaner Alcohol dissolves phenol Irrigate with copious amounts of water – Dry Lime Strong corrosive that reacts with water Brush off dry substance Irrigate with copious amounts of cool water Prevents reaction with patient tissues
  • 70. Chemical Burns Sodium – Unstable metal – Reacts vigorously with water – Decontaminate: Brush off dry chemical – Cover the wound with oil substance used to store metal
  • 71. Chemical Burns Riot Control Agents – Agents CS, CN (Mace), Oleoresin, Capsicum (OC, pepper spray) – Irritation of the eyes, mucous membranes, and respiratory tract – No permanent damage – General signs and symptoms Coughing, gagging, and vomiting Eye pain, tearing, temporary blindness – Management Irrigate eyes with normal saline
  • 72. Tar
  • 77. Radiation Burns Radiation Burns – Notify hazardous materials team – Establish safety zones Hot, warm, and cold – Personnel positioned upwind and uphill – Use older rescuers for recovery – Decontaminate ALL rescuers, equipment, and patients
  • 78. Assessment and Management of Electrical, Chemical, and Radiation Burns Ongoing Assessment – Re-evaluate initial assessment – Re-evaluate all interventions
  • 79. Summary Introduction to Burn Injuries Anatomy and Physiology of the Skin Pathophysiology of Burns Assessment of Thermal Burns Management of Thermal Burns Assessment and Management of Electrical, Chemical, and Radiation Burns