5. Fxgmv l H,Ā» Microglossia. Abnormally small tongue associated
with constricted mandibular arch.
6. m.ā .mmmā
Flāhāullā I I Microglossia. Associated constriction of the maxi
Iary arch in he same patient shown in Figure 1716.
7. Figure 1-19 ' Ankyloglossia. Abnormal attachment of the lingual
frenum, limitlng tongue mobility.
8. w
Fugum I ~22 Fissured tongue. Moderate ļ¬ssuring of the dorsal
tongue. (From Allen CM, Camisa C: Diseases of the mouth and
lipsā In Sams WM, Lynch P, editors: Principles ofdermatology, New
York, 1990, Churchill Livingstone.)
9. Figure 1724 H tongue. Eiongated. black-ļ¬aming ļ¬hform
papillae on the posterior dorsai tongue.
21. Figure 1-78 Hemihyperplasia. Same patient as depicted in
Figure 1777, with associated enlargement of the right half of the
tongue. (Courtesy of Dr. George Blozis.)
22. Figure 2-1 > Environmental enamel hypoplas . Bilaterally
symmetric pattern of horizontal enamel hypoplasia of the anterior
dentition. Maxillary central incisors have been restored previously
(From Neville BW, Damm DD, White DK: Color atlas ofclinicul oral
pathology. ed 2, Baltimore, 1999. Williams & Wi ins.)
23. Figure 2-2 Environmental enamel hypoplasia. Same patient
as depicted in Figure 271ā Note the lack of enamel damage on
bicuspids, (From Neville BW, Damm DD, White DK: Color atlas of
clinical oral pathology, ed 2, Baltimore, 1999, Williams & Wilkins.)
24. Turnerās hypoplasia. Extensive enamel hypoplasia
of mandibular ļ¬rst bicuspid secondary to previous inļ¬ammatory
process associated with overlying ļ¬rst deciduous molar. (From
Halstead CL, Blozis CG, Drinnan AJ, et al: Physical evaluation ofthe
dental patient, St Louis, 1982, Mosbyt)
25. F ure 2-6 - Turner's hypopla 3. Extensive corona! hypopfasia
of permanent maxillary left (entral incisor secondary to previous
trauma to deciduous ce (ral incisor,
27. Figure 2-9 ' Dental ļ¬uoros White opaque alteration of the
bicuspids and second molars in a patient who also exhibits
discoloration of the teeth secondary to (etracycline use. Patient
moved to area of endemic ļ¬uorosis at 3 years of age.
28. Figure 2-10 ~ Attrition. Extensive loss of coronal tooth height
WIthout pulp exposure in patient with anterior edge-to-edge
occlusionv
29. Figure 2-11 * Abrasion Horizontal cervical nokhes on the
anterior mandibular dentition
30. Figure 2-12 Abrasion. Extensive recession and loss of buccal
radlculardentin. Note visible pulp Canals that have been ļ¬lled
wllh lertiary dentin.
31. Abrasion. Notching of the right central incisor
caused by improper use of bobby pins. The patient aiso exhibits
environmental enamel hypopiasia of the anterior dentition.
(Courtesy of Dr. Robert]. Corlin.)
32. Figure 2 14 Abra on. Notching ofthe ante or dentition on
the right side caused by long-term use of tobacco pipe.
33. Erosion Extensive loss of buccal and occlusalh 'rnz
tooth structure. Note that the amalgam margins are above the
surface of the dentin.
34. mum
Figure 2716 Erosion. Occlusal surface of the mandibular
dentition exhibiting concave dentin depressions surrounded by
eievated rims of enamel.
35. Erosion. Extensive loss of enamel and dentin on
the buccal surface of the maxillary bicuspids. The patient had
sucked chronically on tamarinds (an acidic fruit).
36. Figure 2-18 Erosion. Palatal surfaces of the maxillary dentition
in which the exposed dentin exhibits a concave surface and a
peripheral white line ofenamel. The patient suffered from bulimia
37. Figure 2-19 Abfraction. Deep and rrow enamel cervncal
defects on the facial surface of the mandibular dentition (From
Neville BW. Damm DD. White DK: Coloratlus of clinical omāl
pathology. ed 2, Baltimore, 1999, Williams & Wilkinsl)
38. m mĀ»
, Internal resorption (pink tooth of Mummery).
Pmk disrolomh'on of H19 maxillary (Pntml indsor,
39. Exgm'e 1 21 Internal resorption. Same patient as depicted in
Figure 2-21ā Note extensive resorption of both maxil|ary central
incisors,
41. Flgure 2-24 0 External resorption. Extensive Irregular destrucā
tion of both roots of1he mandibular second molar associated
wixh chronic periodontitis. (Courtesy of Dr. Tommy Shimer)
42. w
Figure 2<25 ā External resorp Ion. āMotheatenā radiolucent
alteration of the maxillary left central incisor The tooth had been
reimplanted after traumatic avulsion. (Courtesy of Drā Harry Meyers)
43. Figure 227 ~ External resorption. Diffuse external resorption of
radicular dentin of maxillary dentition. This prOCESS arose after
initiation of orthodontics.
44. Figure 2-40 Hypodont . MultIple developmentally mlssmgpel
manent teeth and several retained deciduous teeth in a female adult
45. Figure 2742 Hypodontia, Developmentally missing maxillary
lateral incisors. Radiographs revealed no underlying teeth, and
there was no history of trauma or extraction.
46. Figure 2-43 * Hyperdon a (mesiodens). Erupted superr
numerary, rudl'memary tooth of the anterior maxilla
47. Figure 2-44 Hyperdon a (mesiodens). U teral :uper-
numerary tooth of the anterior maxilla, which has altered the
eruption path of the maxil|ary right permanent central incisor.
48. supernumerary teeth of the anterior maxilla.
Figure 2ā45 % Hyperdontia (meslodens). Bulateral Inverted
49. Figure 2ā46 '-Ā» Hyperdontia. Right mandibular dentition
exhibiting four erupted bicuspids.
50. Figure 2ā47 v> Paramolar. Rudimentary tooth situated palatal to a
maxiilary meter in a patient who also exhibits hypodontia.
51. Figure 2-48 + Paramolar. Radiograph of the same patient
depicted in Figure 2-4.7 Note the fully formed tooth overlying
crown of the adjacent molar.
52. Flgure 2749 Natal teeth. Mandihu|ar rentral I'n(i<ors (hat were
erupted at birth.
53. Figure Z~50 ' Diffuse microdontla. Dentition in which the teeth
are Smaller than normal and widely spaced within the arch
54. Figure 2ā51 Ā» Isolated microdon Ia (peg lateral). Small, cone,
shaped right maxillary lateral incisor.
55. Figure 2-52 āv Bilateral gemination. Two double teeth, The
moth count was normal when each anomalous tooth was counted
axoneā
60. #
Figure 2761 Concrescence. Cross photograph of the same
teeth depicted in Figure 260. Histopathologir examination
revealed that union occurred in the area of cemental repair
previously damaged by a periapical inļ¬ammatory lesion.
61. Figure 2 3 Talon cusp. Acce ory cusp present on the palatal
surface of H19 maxill y loft rpntral incisor: Note the (hreer
pronged pattern, which rezemlļ¬e: an eagle talon.
62. Figure 2-70 Ā« Coronal den vag atus, type II. Mandibular
lateral incisor exhibiting lingual bulbous enlargement at the site of
(clonal opening of enamel invagination.
63. F gure 2-7] x Coronal dens vag natus, type II. Radiograph of
the mandibular lateral incisor depicted in Figure 2-70. Note the
radiopaque and enamel-lined invagination extending below the
level of the cementoenamel junction.
64. Figure 2ā72 Coronal dens invaginatus, type M. Cross photo-
graph ofa sectioned tooth. Note the dilated invagination with
apical accumulation of dystrophic enamel.
65. Figure 2773 Coronal dens nva atus, type III. Parulis overr
lying vital maxillary cuspid and lateral incisor. The Luspld contained
a dens Invaginatus that perforated t mesidl >urlace ofits root.
66. Figure 2-74 Coronal dens invaginatus, type III. Maxillary
cuspid exhibiting an enamel invagination that parallels the pulp
canal and perforates the lateral root surface (Courlesy of Dr.
Brian Blocher.)
67. Figure 2ā76 ' Enamel pearl. Mass of ectopic enamel located in
the furcation area ofa molar tooth. (Courtesy of Drā Joseph
Beard.)
68. m āĀ«w
Ā§Ā§
Fegure 2779 Taurodontism. Mandibular molar teeth exhibiting
increased pulpal apicoocclusal height With apically positioned
pulpal floor and bifurcation. (Courtesy of Dr. Michael Kahn,)
69. Figure 2-81 O Hypercemento Mandibular ļ¬rst molar
exhibiting thickening and blunting of the roots.
70. Figure 2-83 Ilacerahon. Maxlllary molar exhlbltlng sharp
angulation of the rootsā Note the interradicular bone.
71. Figure 2-87 v Supernumerary root. A, Cross photograph exhibiting a maxillary molar with a small
supernumerary root. B, MeSIal-to-distal radiographic view exhibiting the a(cessory root with central
pulp canal. lfa buccalitoJingual mdiographic view had been taken (as would be necessary in
patient (are), the additional root would not have been evident.
72. Figure 2789 Hypoplastic amelogenesu Imperfecta, general-
ized pitted pattern. Note the numerous pinpoint pits scattered
across the surface of the teeth. The enamel between the pits is of
normal thickness. hardness. and coloration, (From Stewart RE.
Prescott CH: Oml facial genetics, St Louis, 1976, Mosby.)
73. Figure 2 95 Hypomaluration amelogenesis imperfecta.
Denlition thibiting mottled, opaque white enamel with scattered
areas 0{ brown discoloration.
74. Figure 2 ~96 Hypomaturation amelogenesis imperfecta,
snowcapped pattern. Dentition exhibiting zone of white opaque
enamei in the incisal and occlusal one fourth of the enamel
surface. (Courtesy of DLā Heddie O. Sedano.)
75. Fugure 2-97 ' Hypocalciļ¬ed amelogenesis imperfecta. DentiĀ»
lion exhibiting diffuse yellow-brown discoloration, Note numerous
teeth wiļ¬l loss of coronaI enamel except for the cervical portion.
77. Figure Z-IOZ ~ā Dentinogenesis imperfecta. Dentition exhibiting
grayish discoloration with Signiļ¬cant enamel loss and attrition.
78. Flgulāe 3-1 Chm Ic hyperplastlc pulpltls. Erythematous
granulation tissue extruding from the pulp chamber of the
mandibular first molar:
79. Figure 3716 Periapical granuloma. WEN-deļ¬ned radiolucency
associated with the apex of the maxillary ļ¬rst bicuspid, (Courizsy
of Drā Frank Beylotātel)
80. Figure 3-] er plcal granuloma. Large, well-defined radiolui
cency associated with the apices of the mandibular ļ¬rst molarā
(Courtesy of Drā Robert E. Loy.)
81. *
Figure 3-24 Ā« Periap al cyst. Wechircumscribed radiolucency
intimater associated with the apex of the mandibular central
incisor. Note the loss oflamina dura in the area of the lesion
82. . . NāN,Ā§
Figure 3-25 ~ Periapical cyst. Radiolucency associated with the
maxillary central incisor, which exhibits signiļ¬cant root resorption.
83. Figure 3-36 ' Periapical abscess. Bilateral soft-tissue swelling of
the anterior palate.
84. ļ¬gure 3-42 - Parulls. Asymptomatic yellowush nodule of the
anurior mandibular alveolar ridge. Adjacent teeth were normal
clinically and also asymptomatic
85. Figure 3-43 * Periapical abscess. Same patient as depicted in
Figure 3742ā Periapical radiolucency associated with the nonvital
mandibular lateral incisor.
86. Figure 3 Ā»45 Ludwig's angina. Soft-tissue swelling of the rig?ā
submandibular region (Courtesy of Drā Brian Blocher.)
87. Figure 3-46 Cellu 5 involving canine space. Erythematous
and edematous enlargement of the left side of the face with
involvement of the eyelids and conjunctiva. Patients with odonto-
genic infections involving the canine space are at risk for
cavernous sinus thrombosis. (Courtesy of Dr. Richard Ziegle )
88. Figure 348 . Acute osteomyelitis with sequestruml Radior
lucency of the right body of the mandible with central radiopaque
mass of necrotic bone. (Courtesy of Dr. Michael Meyrowitz.)
89. Figure 3-52 t lefuse sclerosmg osteomyelitis. Duffuse area of
increased mdiodensity of the right body of the mandible in the
tooth-bearing area. No other quadrants were involved (Courtesy
of Dr. Louis M. Beta)
90. Figure 3753 : Condensing osteitis. Increased areas of radioā
density surrounding the apices of the nonvital mandibular ļ¬rst
molar.
91. Figure 4718 Nifedipine-related gingival hyperp|asia. Diffuse,
fibrotic gingival hyperplasia after 1 month ofintensive oral
hygiene. Significant erythema, edema, and increased enlargement
were present before intervention,
92. Figure 4-22 Phenytoin-related ginglval hyperplasia.
Signiļ¬cant gingival hyperplasia almost totally covers the crowns of
the posterior maxillary dentition (Courtesy of Dr. Ann Drummond
and Dr. Timothy Johnsonā)
93. 'i-āguāyo 1: 3 , s. Painful erythematous enlargement
of the soll tissues overlying the crown of the partially elupted
right mandlbular third molar.
94. Flgulāe 5"ā Hutchinson's incisors of congenital syphilis.Den
tition exhibiting crowns tapering toward the incisal edges. (me
Halstead CL, Blozis CG, Drinnan A], Cier RE: Physicai evaiuntionq'
the dental patient. St Louis, 1982, Mosby.)
95. Figure 7-31 Mumps. Bilateral parotid enlargement. (From
Neville BW. Damm DD, White DK: Color atlas ofclim'ral oml
pathology, ed 2, Baltimore, 1999, Williams & Wilkinsā)
96. ļ¬guraļ¬ S Asp rin bur . Extensive area of white epithelial
mosisulthe left buccal muwsa caused by aspirin placement in
maltempl to alleviate dental pain,
97. Figure 8719 Cotton roll burn. Zone ofwhite epithelial ne(rosis
and erythema of the maxillary alveolar mucosa.
98.
99. F ure 8727 Osteoradionecrosis. Same patient as depicted in
Figure 8725. Note fistula formation of the left submandibular area
resulting from osteoradionecrosis of the mandibular body.
100. K 3'! Amalgam tattoo. Area of mucosal discoloralioml
the mandibular alveolar ridge immediately below the bridge
politic
101. Lg. . 3 Minor aphthous ulcerations. Two ulcerations of
different Sizes located on the maxillary labial mucosi
102. Figure 9-7 Major aphthous ulcerat on. A, Large ulceration of the left anterior buccal mucosa.
B, Same lesion after 5 days of therapy with betamethasone syrup used in a SWIshāand-swallow
method. The patient was free of pain by the second day of therapy. The ulcerahoh healed com-
pletely during the foHowmg weekā
103. _ Allergic mucosal reaction to systemic drug
administration. Large irregular erosion ofātlāle right ventral surr
fare of the tongue. The lesion arose secondary to use of
oxapmzin, a nonsteroidal antHnļ¬ammatory drug.
104. Figure 9-33 0 Contact stomatitis from cinnamon ļ¬avoring.
Oblong area of sensitive erythema with overlying shaggy
hyperkeratosis.