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GROUP 5 PRESENTATION
Group Members

1.   Andrew Moyo
2.   Alex Mbewe
3.   Mtisunge Wandale
4.   Monica Banda
5.   Jacqualine Ntaba
6.   Nelson Munthali
7.   Rosella Munyenyembe
PRESENTS



   ANGINA PECTORIS
BROAD OBJECTIVE

   At the end of this presentation, learners
    should be able to manage the patient with
    angina pectoris.
OUTLINE
1.   Define and describe the classification of angina pectoris
2.   Explain the etiology and pathophysiology of angina
     pectoris
3.   Describe the clinical manifestation of angina pectoris
4.   Describe the diagnostic evaluation of the angina
     pectoris
5.   Explain the nursing assessment and its diagnosis
6.   Explain the nursing management of angina pectoris
7.   Explain the medical/ surgical management of angina
     pectoris
8.   Outline the complications of angina pectoris
Definition

   It is commonly called “angina”
   It is the chest pain that is due to ischemia of
    the heart muscle, generally due to obstruction
    or spasm of the coronary arteries
TYPES OF ANGINA PECTORIS

   The most three types of angina include:
   Stable angina
-   It is triggered by a predictable degree of physical exertion
    or motion but can be improved after rest.
-   It is the most common type of angina
     Unstable angina
-   It is triggered by unpredictable degree which may occur at
    night
-   It is more dangerous and requires emergency treatment
    and is often a sign that heart attack can occur soon
   Variant angina
-   this is a rare form that is caused by a spasm in coronary
    artery
ETIOLOGY AND PATHOPHYSIOLOGY


   Angina pectoris is as a result of ischemia of
    the heart which is due to antherosclerosis of
    the coronary arteries which restricts blood
    flow to the myocardium.
   Due to myocardium ischemia, the myocardial
    tissues are deprived of oxygen and nutrients
    for the aerobic metabolism.
   As a result there is an inclusion of anaerobic
    metabolism which leads to accumulation of
    lactic acid
   Due to increase of lactic acid, myocardial
    nerve fibers are irritated and this transmit a
    pain message to the cardiac nerves and
    upper thoracic posterior nerve roots
   And all this leads to cardiac pain which is the
    angina pectoris
CLINICAL MANIFESTATION

   Dyspnea
   Sweating/ diaphoresis
   Faintness
   Palpitations
   Dizziness
   Indigestive disturbances
DIAGNOSTIC EVALUATION

   Electrocardiography (ECG)
-   an ECG recorded in the presence of pain may
    record transient ischemic attacks.
-   it also suggest coronary artery’s involvement and
    the extent of the myocardium affected by the
    ischemic event
   Exercise electrocardiography
-   This involves the client exercising on a treadmill or
    stationery bicycle until reaching a maximum of 85%
    heart rate.
    If any change in the ECG or vital signs may indicate
    the presence of ischemia
   Radioisotope
-   this involves the use of different nuclear imaging
    techniques to evaluate heart muscles.
-   regions of ischemia appear as areas of
    diminished activity
   Ultrafast computed tomography
-   it enables detection of the amount of calcium in
    the coronary arteries. High coronary calcium
    rate is associated with obstructive coronary
    disease.
   Coronary angiography
-   it detect the percentage of blockage in
    coronary artery
-   it remains the most accurate test
   Chest x-ray
-   it allows the detection of cardiomegaly and
    non cardiac causes of chest pain e.g
    pneumonia
NURSING ASSESSMENT
SUBJECTIVE DATA
Past health history
- previous history of cardiac artery
  disease, angina, myocardial infarction, anemia and heart
  failure
Medications
- use of aspirin, calcium channel blockers and
  antihypertensive
Family history
- history of heart disease, tobacco use
Nutritional history
- heart burn, nausea, vomiting
Coping stress mechanism
- anger, anxiety, depression and stressful life style
OBJECTIVE DATA
General
- anxiety, fear, restlessness
Integumentary
- cool, clammy, pale skin
Cardivascular
- tachycardia or bradycardia, dysrhythmias, low Bp or hypertension

Possible findings
- increase in white blood cell count, positive exercise stress test, ST
   segment and T wave abnormalities on ECG, cardiac
   enlargement, pulmonary congestion on chest x-ray and positive
   coronary angiography.
NURSING DIAGNOSIS
   Acute pain related to myocardial ischemia as
    evidenced by severe chest pain and tightness
   Ineffective tissue perfusion related to myocardial
    injury as evidenced by dyspnea, decrease in Bp.
   Anxiety related to perceived or actual threat of death,
    pain and possible life style changes as manifested by
    restlessness and patient asking too much questions
   Activity intolerance related to fatigue secondary to
    decreased cardiac output and poor lung and tissue
    perfusion as manifested by increased heart rate
   Ineffective therapeutic regimen management
    related to lack of knowledge of disease
    process as evidenced by frequent
    questioning about the illness and
    management after discharge
NURSING MANAGEMENT

-   evaluate chest pain e.g location in order to
    accurately evaluate, treat and prevent further
    ischemia
-   promote bed rest in semi fowlers position, it
    relaxes the muscles of the chest cavity
-   monitor vital signs after every 5 minutes on
    each attack to review the effectiveness of the
    care given
-   Monitor fluid balance (e.g. monitor input and
    output, daily weight ) to monitor renal
    perfusion and observe for fluid retention
-   Provide factual information concerning the
    disease process to decrease fear of the
    unknown
-   administer prescribed medication to relieve
    pain and ischemia to decrease anxiety and
    cardiac workload e.g. Nitroglycerin-which
    dilates the coronary arteries and increases
    the supply of oxygen to the myocardium
MEDICAL MANAGEMENT
    It focuses on three goals
1.     Relief of the acute pain. The major drugs
       used to reduce acute pain are opioid
       analgesics e.g nitroglycerin
2.     Restore coronary blood flow. Administer
       vasodilators e.g propanolol, calcium channel
       blockers such as felodipine.
3.     Prevent further attacks-this is done through
       education and counselling regarding
       modification of risk factors of the disease.
This reduce the progression of coronary heart
  disease.
Complications of Angina Pectoris

   Angina pectoris can lead to:-
•   Heart failure
•   Acute renal failure
•   Myocardial infarction
•   Cardiac arrest
•   Death
References

1.   Black J. M. and Hawks J (2009) Medical –
     surgical Nursing, Clinical Management for
     positive outcomes (8th edition) saunders,
     Elsevier
2.   Lewis S. M., Heitkemper M. M and Dirksen S.
     R (2007) Medical /Surgical Nursing.
     Assessment and management of clinical
     problems. (7th edition) St Louis: C.V. Mosby
3.   http://www.servier.co.uk
   A BENGO TADZUKANI!!!!!!!!

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Angina pectoris

  • 2. Group Members 1. Andrew Moyo 2. Alex Mbewe 3. Mtisunge Wandale 4. Monica Banda 5. Jacqualine Ntaba 6. Nelson Munthali 7. Rosella Munyenyembe
  • 3. PRESENTS ANGINA PECTORIS
  • 4. BROAD OBJECTIVE  At the end of this presentation, learners should be able to manage the patient with angina pectoris.
  • 5. OUTLINE 1. Define and describe the classification of angina pectoris 2. Explain the etiology and pathophysiology of angina pectoris 3. Describe the clinical manifestation of angina pectoris 4. Describe the diagnostic evaluation of the angina pectoris 5. Explain the nursing assessment and its diagnosis 6. Explain the nursing management of angina pectoris 7. Explain the medical/ surgical management of angina pectoris 8. Outline the complications of angina pectoris
  • 6. Definition  It is commonly called “angina”  It is the chest pain that is due to ischemia of the heart muscle, generally due to obstruction or spasm of the coronary arteries
  • 7. TYPES OF ANGINA PECTORIS  The most three types of angina include:  Stable angina - It is triggered by a predictable degree of physical exertion or motion but can be improved after rest. - It is the most common type of angina  Unstable angina - It is triggered by unpredictable degree which may occur at night - It is more dangerous and requires emergency treatment and is often a sign that heart attack can occur soon  Variant angina - this is a rare form that is caused by a spasm in coronary artery
  • 8. ETIOLOGY AND PATHOPHYSIOLOGY  Angina pectoris is as a result of ischemia of the heart which is due to antherosclerosis of the coronary arteries which restricts blood flow to the myocardium.  Due to myocardium ischemia, the myocardial tissues are deprived of oxygen and nutrients for the aerobic metabolism.  As a result there is an inclusion of anaerobic metabolism which leads to accumulation of lactic acid
  • 9. Due to increase of lactic acid, myocardial nerve fibers are irritated and this transmit a pain message to the cardiac nerves and upper thoracic posterior nerve roots  And all this leads to cardiac pain which is the angina pectoris
  • 10. CLINICAL MANIFESTATION  Dyspnea  Sweating/ diaphoresis  Faintness  Palpitations  Dizziness  Indigestive disturbances
  • 11. DIAGNOSTIC EVALUATION  Electrocardiography (ECG) - an ECG recorded in the presence of pain may record transient ischemic attacks. - it also suggest coronary artery’s involvement and the extent of the myocardium affected by the ischemic event  Exercise electrocardiography - This involves the client exercising on a treadmill or stationery bicycle until reaching a maximum of 85% heart rate. If any change in the ECG or vital signs may indicate the presence of ischemia
  • 12. Radioisotope - this involves the use of different nuclear imaging techniques to evaluate heart muscles. - regions of ischemia appear as areas of diminished activity  Ultrafast computed tomography - it enables detection of the amount of calcium in the coronary arteries. High coronary calcium rate is associated with obstructive coronary disease.
  • 13. Coronary angiography - it detect the percentage of blockage in coronary artery - it remains the most accurate test  Chest x-ray - it allows the detection of cardiomegaly and non cardiac causes of chest pain e.g pneumonia
  • 14. NURSING ASSESSMENT SUBJECTIVE DATA Past health history - previous history of cardiac artery disease, angina, myocardial infarction, anemia and heart failure Medications - use of aspirin, calcium channel blockers and antihypertensive Family history - history of heart disease, tobacco use Nutritional history - heart burn, nausea, vomiting Coping stress mechanism - anger, anxiety, depression and stressful life style
  • 15. OBJECTIVE DATA General - anxiety, fear, restlessness Integumentary - cool, clammy, pale skin Cardivascular - tachycardia or bradycardia, dysrhythmias, low Bp or hypertension Possible findings - increase in white blood cell count, positive exercise stress test, ST segment and T wave abnormalities on ECG, cardiac enlargement, pulmonary congestion on chest x-ray and positive coronary angiography.
  • 16. NURSING DIAGNOSIS  Acute pain related to myocardial ischemia as evidenced by severe chest pain and tightness  Ineffective tissue perfusion related to myocardial injury as evidenced by dyspnea, decrease in Bp.  Anxiety related to perceived or actual threat of death, pain and possible life style changes as manifested by restlessness and patient asking too much questions  Activity intolerance related to fatigue secondary to decreased cardiac output and poor lung and tissue perfusion as manifested by increased heart rate
  • 17. Ineffective therapeutic regimen management related to lack of knowledge of disease process as evidenced by frequent questioning about the illness and management after discharge
  • 18. NURSING MANAGEMENT - evaluate chest pain e.g location in order to accurately evaluate, treat and prevent further ischemia - promote bed rest in semi fowlers position, it relaxes the muscles of the chest cavity - monitor vital signs after every 5 minutes on each attack to review the effectiveness of the care given
  • 19. - Monitor fluid balance (e.g. monitor input and output, daily weight ) to monitor renal perfusion and observe for fluid retention - Provide factual information concerning the disease process to decrease fear of the unknown - administer prescribed medication to relieve pain and ischemia to decrease anxiety and cardiac workload e.g. Nitroglycerin-which dilates the coronary arteries and increases the supply of oxygen to the myocardium
  • 20. MEDICAL MANAGEMENT  It focuses on three goals 1. Relief of the acute pain. The major drugs used to reduce acute pain are opioid analgesics e.g nitroglycerin 2. Restore coronary blood flow. Administer vasodilators e.g propanolol, calcium channel blockers such as felodipine. 3. Prevent further attacks-this is done through education and counselling regarding modification of risk factors of the disease.
  • 21. This reduce the progression of coronary heart disease.
  • 22. Complications of Angina Pectoris  Angina pectoris can lead to:- • Heart failure • Acute renal failure • Myocardial infarction • Cardiac arrest • Death
  • 23. References 1. Black J. M. and Hawks J (2009) Medical – surgical Nursing, Clinical Management for positive outcomes (8th edition) saunders, Elsevier 2. Lewis S. M., Heitkemper M. M and Dirksen S. R (2007) Medical /Surgical Nursing. Assessment and management of clinical problems. (7th edition) St Louis: C.V. Mosby 3. http://www.servier.co.uk
  • 24. A BENGO TADZUKANI!!!!!!!!