Coronary Artery Disease Cvs 2 Es (1)

CORONARY ARTERY DISEASE ANGINA Pectoris MYOCARDIAL INFARCTION (MI) Presenter Erum S. kabani Sr. Instructor April 03,2008

Objectives
Coronary Artery Diseases
Angina
Types
Mechanism
Causes
Clinical manifestation
Complication
Nursing care
06/07/09 www.health-nurses-doctors.blogspot.com

Coronary Artery Disease Definition: CAD is a term that refers to the effect of the accumulation of atherosclerosis plaque in the coronary arteries that obstructs blood flow to the myocardium 06/07/09 www.health-nurses-doctors.blogspot.com

Cont.
conditions result from CAD
1 . Angina Pectoris
2. Myocardial Infarction

Angina Pectoris
Definition:
Angina: Choking or suffocation.
Pectoris: Chest.
Angina pectoris, is the medical term used to describe acute chest pain or discomfort.
Angina occurs when the heart’s need for oxygen increases beyond the level of oxygen available from the blood nourishing the heart.
It has 3 types
Stable Angina
Un stable angina &
Variant Angina (Prinzmetal’s or resting angina) :

Cont.
Types of Angina
Stable angina :
People with stable angina have episodes of chest discomfort that are usually predictable. That occur on exertion or under mental or emotional stress.
Normally the chest discomfort is relieved with rest,
nitroglycerin (GTN) or both.
It has a stable pattern of onset, duration and intensity of symptoms.

Cont.
Unstable angina:
It is triggered by an un predictable degree of exertion or emotion.
(progressive), more severe than stable. Characterized by increasing frequency & severity. Provoked by less than usual effort, occurring at rest &
interferes with pt lifestyle.

Cont.
Variant Angina (Prinzmetal’s or resting angina) :
occur spontaneously with no relationship to activity. Occurs at rest due to spasm. Pt discomfort that occurs rest usually of longer duration. Appears to by cyclic & often occurs at about the same time each day (usually at night). Thought to be caused by coronary artery spasm

Mechanism Of Angina

Causes
Coronary atherosclerosis (atheroma )
Factors increasing preload :
Hyperthyroidism
Exercise
Anemia
Factors increasing after load:
Hypertension
Aortic stenosis
Obstructive cardio myopathy
Coronary artery spasm

Clinical Manifestations
Characteristics: Squeezing, burning, pressing, choking, or bursting pressure.
Onset: Quickly or slowly
Location: Chest, right or left arms, shoulder, or neck, jaw.
Duration: Less then 5 minutes.
Associated: Dyspnea, Sweating, faintness, palpitation, dizziness ect.
Relieving: GTN and rest.
Aggravating: exertion, exercise, heavy meal, emotional upset, and anger.

Investigations
Electrocardiogram ( ECG)
Coronary angiography
Exercise Electrocardiogram (Stress test).
Complications:
Myocardial infarction
Cardiac Arrhythmias

Myocardium Infarction
Myo means muscle, “Cardiac” heart, infarction means “death of tissues due to lack of blood supply”.
It is also called heart attack. It occurs when coronary arteries become blocked and the part of myocardial muscles become dead due to prolonged lack of oxygen supply to the muscle cells.

PATHOPHYSIOLOGY
Coronary artery cannot supply enough blood to the heart in response to the demand due to CAD
Within 10 seconds myocardial cells experience ischemia
Ischemic cells cannot get enough oxygen or glucose
Ischemic myocardial cells may have decreased electrical & muscular function
Cells convert to anaerobic metabolism.
Cells produce lactic acid as waste
Pain develops from lactic acid accumulation
Pt feels anginal symptoms until receiving demand increase 02 requirements of myocardial cells

ECG changes in Angina & MI
Zone of Ischemia: T wave inversion
Zone of Injury: ST elevation
Zone of Necrosis: Abnormal Q wave

Sign and Symptom
Classic symptom of heart attack are chest pain radiating to neck, jaws, back of shoulder, or left arm
The pain can be felt like:
Squeezing or heavy pressure
A tight band on the chest
An elephant sitting on the chest

Cont
Other symptoms include:
Shortness of breath (SOB)
Weakness and tiredness
Anxiety
Lightheadedness
Dizziness
Nausea vomiting
Sweating, which may be profuse

Collaborative Management
Assessment:
History
Clinical manifestation
Cardiovascular assessment
Laboratory assessment
Troponin T & I
CK-MB

Radiographic Assessment
ECG
Stress Test
Myocardial perfusion imaging
MRI
Cardiac Catheterization

IMPORTANT INFORMATION TO REMEMBER Increase supply of Oxygen
Decreasing the demand of Oxygen:
Stop activity and lie down (CBR)
Take Tab. Angisid sublingually and wait till it dissolves.
If pain continues take up to 03 Tab. Angisid one every five minutes. If pain is not relieved yet take another tab. and rush to EMERGENCY services.

IMMEDIATE MANAGEMENT OF MI:
GOALS:
To prolong life.
Minimize infarct size.
Reverse ischemia.
Reduce cardiac work.
Prevent and treat complications.
A) INITIAL TREATMENT:
Rapid triage.
OMI (oxygen, monitor and I/V line).
Check vital signs and O2 saturation.
ECG within 10 minutes and repeat ECG.
Blood samples for enymes, CBC, lytes, and lipid profile.

Intervention
Medication:
Morphine Sulphate
Nitrates (GTN)
Beta blockers
Calcium Channel Blocker
Anti platelets / Anti coagulant
Thrombolytic therapy

Surgical management
PTCA (Percutaneous Transluminal Coronary Angioplasty

Coronary Artery Bypass Graft surgery (CABG) 06/07/09 www.health-nurses-doctors.blogspot.com

Nursing Diagnosis
Acute pain R/T imbalance between myocardial oxygen supply and demand
Ineffective tissue perfusion R/T interruption of arterial blood flow
Ineffective coping R/T effects of acute illness and major changes in life style
Impaired gas exchange related to ineffective breathing pattern and decreased systemic tissue perfusion.
Anxiety related to present status and unknown future, possible lifestyle changes, pain, and perceived threat of death.
Activity intolerance related to fatigue

Prevention
Recognize the symptoms
Reduce your risk factors:
Lose weight
Quit Smoking
Keep your cholesterol at a normal level.
Keep your blood pressure under control.
Use techniques to ease stress.
Control blood sugar level.
Eat Right
REGULER EXERCISE

COMPLICATIONS OF MI:
Arrhythmias
Atrial arrhythmias.
Ventricular arrhythmias.
Bradycardia and heart block.
Asystol.
Hypertension.
LV failure.
Cardiogenic shock.

CARDIAC REHABILITATION:
Cardiac rehabilitation provides a venue for continued education, re-enforcement of lifestyle modification, and adherence to a comprehensive prescription of therapies for recovery from MI, which includes exercise training
Goals of Rehabilitation program :
Develop a program for progressive physical activity
Lives as full, vital and productive life
Remain within the limits of the heart’s ability to respond to increases in activity and stress.

FOLLOW UP 06/07/09 www.health-nurses-doctors.blogspot.com

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Coronary Artery Disease Cvs 2 Es (1)

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