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FUNCTIONAL ANATOMY
a. IDENTIFYTHE DIAGRAM.
a. WHO STUDIED ON
GOITRE ABOUT THYROID
DYSFUNCTION?
FUNCTIONAL ANATOMY
Ans:
a. HISTOLOGY OF
THYROID GLAND
b. EMIL THEODOR
KOCHER
FUNCTIONAL ANATOMY
 Which endocrine gland DERIVES max blood
supply per gramTISSUE ?
FUNCTIONAL ANATOMY
 Which endocrine gland revives max blood
supply per gram
Ans: ADRENAL ORTHYROID GLAND
BUZZER ROUND
 WHAT IS GLUCOSE FEVER ?
BUZZER ROUND
 WHAT IS GLUCOSE FEVER ?
Ans: GLUCOSE INFUSION IN ADRENAL
INSUFFICIENCY LEADSTO HIGH FEVER.
MCQ ROUND
6. WHICH OF THE FOLLOWING PITUITARY
HORMONES IS AN OPIOID PEPTIDE?
A. α- MELANOCYTE – STIMULATING HORMONE (α-
MSH)
B. ß-MSH
C. GROWTH HORMONE
D. ß- ENDORPHIN
MCQ ROUND
6. WHICH OF THE FOLLOWING PITUITARY
HORMONES IS AN OPIOID PEPTIDE?
A. α- MELANOCYTE – STIMULATING HORMONE (α-
MSH)
B. ß-MSH
C. GROWTH HORMONE
D. ß- ENDORPHIN
RECENT ADVANCES
 METATHYROID DIABETES
RECENT ADVANCES
 METATHYROID DIABETES
Ans:
IN SEVERE HYPOTHYROIDISM BETA CELLS
ARE DESTROYED DUE TO EXCESSIVE
STIMULATION RESULTING IN PERSISTENT
GLYCOSURIA
CASE SCENARIO
 IDENTIFYTHE DISEASE ?
 WHAT ISTHE CAUSE FOR
FLUID ACCUMULATION ?
CASE SCENARIO
 MYXEDEMA BEFOREAND AFTER
TREATMENT
 GLYCOAMINO GLYCANS
(MUCOPOLYSACCHARIDES)
CASE SCENARIO
 TESTTO DIFFERENTIATE BETWEEN
DIABETES MELLITUS AND INSIPIDUS BY
URINE EXAMINATION ?
 DEFINE CUSHINGOID STATE ?
CASE SVENARIO
 URINE SPECIFIC GRAVITY
 CHRONIC TREATMENTWITH
GLUCOCORTICOIDS
RUSH
 MOST POTENT STIMULI FOR CALCITONIN
RELEASE ?
 GASTRIN
RELAX
 MASCULINIZATION OF FEMALE
 HIRSUTISM
 ENLARGED CLITORIS
 ABSENCE OF MENSTRUAL CYCLE
IDENTIFYTHE CONDTION ?
 ADRENOGENITAL SYNDROME (21-
HYDRROXYLASE DEFICENCY )
HISTORY ROUND
 HORMONEWAS FIRST INTRODUCD BY -
Ans: STARLING
HISTORY ROUND
 WHO DISCRIBED THE NOXIOUS STIMULI
INCREASEACTH SECRETIONAS STRESSORS
 WHO DISCOVERED PROINSULIN
HISTORY ROUND
 WHO DISCRIBEDTHE NOXIOUS STIMULI
INCREASEACTH SECRETIONAS
STRESSORS
Ans :-HANS SELYE
 WHO DISCOVERED PROINSULIN
Ans :-STEINER
JUMBLED WORDS
GLAD SAINTPR SON
 LOCAL HORMONES
 PROSTAGLANDINSARE DERIVED FROM ?
 WHICH PG INHIBITS PLATELET
AGGREGATION ?
 MEMBRANE PHOSPHOLIPIDS
 PgE1

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