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LIFE
STYLE
RELATED
HEALTH
HAZARDS
DIFFERENCE???
Varicose Veins
 Varicose Veins
 Are dilated, protruding,
tortuous saccular superficial
veins in the subcutaneous
tissues
ANATOMY
 The veins in the leg are
divided into two systems –
the deep veins and the
superficial veins. The two
systems are linked
periodically by
perforating veins.
A superficial vein can
become varicose because a
perforating vein is
allowing blood to flow the
wrong way (outwards).
 Normal Venous Pump Mechanism
The contraction of muscles compressing veins helps
push blood up through the leg veins back to the
heart. The valves allow the blood to flow towards
the heart only.
 Descending valvular incompetence
 If the valve at the top of a vein becomes
‘incompetent’ and stops working properly,
this allows a head of pressure to distend the
section of vein below it. This stretches the
vein’s wall, making it varicose, and this makes
the next valve down incompetent, and so on
down the leg.
 Descending valvular incompetence
 Descending valvular incompetence
 Occupation and posture (MAJOR)
 A number of studies have found that varicose
veins are more common in people who stand
up at work – particularly those who stand still
for long periods.
 Age
 More people develop varicose veins as they
get older – above 40
 Heredity
 It is not unusual for varicose veins to ‘run in
the family’ to some extent, but there is no
well-proven genetic basis for varicose veins
 Height and weight
 Although very obese people and very tall
people sometimes have particularly
troublesome varicose veins, no significant
correlation has ever been shown between
height and varicose veins, and the evidence
about obesity and varicose veins is
inconsistent.
 Pregnancy
 Varicose veins are more common in women
who have had children, and the more
pregnancies women have, the more likely
they are to develop varicose veins. Varicose
veins that develop in pregnancy are said to
result partly from the pressure of the womb
on the veins, but the evidence for this is poor,
and relaxation of the vein walls by hormones
may be more important.
 Diet and bowel habit
 It has been suggested that lack of fibre in the
diet and sitting straining on the lavatory
(rather than squatting briefly to pass a bulky
stool) might predispose to varicose veins. This
idea has given rise to a lot of debate, but there
is no real evidence to support it.
 Standing for long time
 Positive family history.
 Pregnancy.
 Abdominal tumors.
 Use of OCP(Oral Contraceptive Pills).
 Physical inactivity.
 Obesity.
 Increased age.

 Asymptomatic “early disease”
 Cosmetic
 Dull aching discomfort in lower extremities
 Exacerbated with standing and hot weather
 Itching and tingling
 Dry and hard skin
 Ulcers
 Dilation and tortuosity of superficial veins
 Pigmented skin at site of varicosity
 Ulceration
 Edema can be present
 Thrombophlebitis
 Ulcer
 Marjolin`s ulcer
 Talipes Equinovarus
 Hyperpigmentation &
Lipodermatosclerosis
 Bleeding
 Part One
 Get rid of the reflux
 Part Two
 Get rid of the varicose veins
 Compression stockings
 Surgical, vein stripping
 Endoluminal
 Laser
 Radiofrequency ablation
 Sclerotherapy
 Ultrasound guided
 Catheter delivered
Compression
 Typically requires general anesthesia
 Two incisions are need
 Can be painful post-operatively
 Requires 4-7 days off work
 Ankle-to-groin
 Segmental (groin to knee)
 Started in 1985
 For active or healed
ulcers.
Thank
You

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varicose_veins N10.pptx

  • 4.  Varicose Veins  Are dilated, protruding, tortuous saccular superficial veins in the subcutaneous tissues
  • 5. ANATOMY  The veins in the leg are divided into two systems – the deep veins and the superficial veins. The two systems are linked periodically by perforating veins. A superficial vein can become varicose because a perforating vein is allowing blood to flow the wrong way (outwards).
  • 6.
  • 7.  Normal Venous Pump Mechanism The contraction of muscles compressing veins helps push blood up through the leg veins back to the heart. The valves allow the blood to flow towards the heart only.
  • 8.  Descending valvular incompetence  If the valve at the top of a vein becomes ‘incompetent’ and stops working properly, this allows a head of pressure to distend the section of vein below it. This stretches the vein’s wall, making it varicose, and this makes the next valve down incompetent, and so on down the leg.
  • 9.  Descending valvular incompetence
  • 10.  Descending valvular incompetence
  • 11.  Occupation and posture (MAJOR)  A number of studies have found that varicose veins are more common in people who stand up at work – particularly those who stand still for long periods.
  • 12.  Age  More people develop varicose veins as they get older – above 40
  • 13.  Heredity  It is not unusual for varicose veins to ‘run in the family’ to some extent, but there is no well-proven genetic basis for varicose veins
  • 14.  Height and weight  Although very obese people and very tall people sometimes have particularly troublesome varicose veins, no significant correlation has ever been shown between height and varicose veins, and the evidence about obesity and varicose veins is inconsistent.
  • 15.  Pregnancy  Varicose veins are more common in women who have had children, and the more pregnancies women have, the more likely they are to develop varicose veins. Varicose veins that develop in pregnancy are said to result partly from the pressure of the womb on the veins, but the evidence for this is poor, and relaxation of the vein walls by hormones may be more important.
  • 16.  Diet and bowel habit  It has been suggested that lack of fibre in the diet and sitting straining on the lavatory (rather than squatting briefly to pass a bulky stool) might predispose to varicose veins. This idea has given rise to a lot of debate, but there is no real evidence to support it.
  • 17.  Standing for long time  Positive family history.  Pregnancy.  Abdominal tumors.  Use of OCP(Oral Contraceptive Pills).  Physical inactivity.  Obesity.  Increased age.
  • 18.   Asymptomatic “early disease”  Cosmetic  Dull aching discomfort in lower extremities  Exacerbated with standing and hot weather  Itching and tingling  Dry and hard skin  Ulcers
  • 19.  Dilation and tortuosity of superficial veins  Pigmented skin at site of varicosity  Ulceration  Edema can be present
  • 22.  Talipes Equinovarus  Hyperpigmentation & Lipodermatosclerosis  Bleeding
  • 23.
  • 24.
  • 25.
  • 26.
  • 27.
  • 28.
  • 29.  Part One  Get rid of the reflux  Part Two  Get rid of the varicose veins
  • 30.  Compression stockings  Surgical, vein stripping  Endoluminal  Laser  Radiofrequency ablation  Sclerotherapy  Ultrasound guided  Catheter delivered
  • 32.  Typically requires general anesthesia  Two incisions are need  Can be painful post-operatively  Requires 4-7 days off work
  • 34.  Started in 1985  For active or healed ulcers.
  • 35.
  • 36.