3. DEFINITION
Emergency condition in which severe blood or other fluid loss
makes the unable to pump enough blood to the body.
A clinical manifestation of cellular function due to inadequate
perfusion and consequent cellular hypoxia.
Resulting from effective circulatory volume.
4. INCIDENCE
• Approximately 150 per hundred thousand in the USA
• Difficulty to determine in Ghana
• Due to variation in data recording
5. ETIOLOGY
Acute hemorrhage
Internal:Ruptured spleen, haemothorax
External: Open wound, crush injury
Loss of Plasma: extensive burns
Loss of extracellular fluid: diarrhea, intestinal obstruction
RAPTURED SPLEEN
6. PATHOPHYSIOLOGY
Average adult person has 4-5L
25%(100ml) must be loss for shock to occur
Reduction in venous return
Activation of compensatory mechanism
Vasoconstriction
More forceful contraction
If reduction continues compensatory mechanism fails.
Reduction in tissue perfusion
Worsened hypoxia
11. MANAGEMENT
History taking
Physical examination: colour of mucous membrane
mental state: restless, coma or anxiety
hydration: skin moist or dry
State of veins: collapsing or filled
Respiration : Rate and depth
Prescence of bleeding external wound.
Check vital signs: pulse , temperature, blood pressure
12. MANAGEMENT
Check vital signs: pulse , temperature, blood pressure
Oxygen therapy
Set a line with large bore cannula
Take blood for grouping and cross matching
start IV fluids ( Ringers Lactate)
Manage urine output
13. MANAGEMENT
• FLUID MANAGEMENT
Acute haemorrhage:
whole blood- 1.5 -2L
IV Fluid: Ringers lactate in first 30-45mins
followed by 5%dextrose in same period.
Plasma loss: Fresh Frozen Plasma (human albumin solution
dextran ,haemaccel)
Extracellular fluid: Ringers lactate
14. MANAGEMENT
INVESTIGATION
LABORATORY – Hemoglobin(reduced)
Electrolyte-Na+, k+
PH- Acidosis- Increased H+
Base deficit: low HCO3-
hematocrit- low
IMAGING – x-ray ,CT in acute haemorrhagic shock to check for
internal bleeds.