Smoking affecting implants |Dental Implants and TobaccoDr. Rajat Sachdeva
Smoking has its influnce on general as well as oral health of an individual .
It enhances the risk of Periodontal diseases oral precancerous and cancerous lesion, root caries and Peri-implantitis.
Nicotine slower down healing and Immune defenses.
A sympathomimetic drugs which increases vasoconstriction, limits overall tissue perfusion.
Habit Cessation help in tissue recovery.
Call us regarding Dental Implants:-
Dr. Rajat Sachdeva
+919818894041,01142464041
drrajatsachdeva@gmail.com
Connect with us here:- • Google+ link: https://goo.gl/vqAmvr
• Facebook link: https://goo.gl/tui98A
• Youtube link: https://goo.gl/mk7jfm
• Linkedin link: https://goo.gl/PrPgpB
• Slideshare link : http://goo.gl/0HY6ep
• Twitter Page : https://goo.gl/tohkcI
• Instagram page : https://goo.gl/OOGVig
Learn More:-
• www.sachdevadentalcare.com
• www.dentalclinicindelhi.com
• www.dentalimplantindia.co.in
• www.dentalcoursesdelhi.com
• www.facialaestheticsdelhi.com
Smoking has significant negative effects on periodontal health and outcomes of periodontal treatment. Smoking increases the prevalence and severity of periodontal disease by reducing gingival inflammation and bleeding, increasing attachment loss and bone loss, and elevating levels of periodontal pathogens. Heavier smoking is associated with greater periodontal destruction. While smoking cessation decreases the risks, it does not fully reverse the effects of smoking on the periodontium.
This document discusses the effects of smoking on periodontal health. It covers epidemiological evidence that smoking increases the risk of periodontitis 2-5 times and is a major risk factor. The toxic chemicals in tobacco such as nicotine, carbon monoxide, and tar are discussed. These chemicals can impair the immune response and increase periodontal pathogens, leading to inflammation and tissue destruction. Clinical signs of periodontitis are made worse in smokers, such as increased attachment and bone loss. Smoking is also a major risk factor for oral cancer. The document examines the effects of smoking on gingival blood flow, wound healing, and the complications it can cause for periodontal therapy.
Periodontal disease is a severe form of gum disease caused by untreated gingivitis. Smoking can lead to periodontal disease by affecting bone and tissue attachment to teeth and impairing blood flow and wound healing in the gums. Tobacco contains over 4,000 chemicals including toxic substances like benzene, formaldehyde, ammonia, hydrogen cyanide, and cadmium. Smoking inhibits immune responses, increases harmful bacteria, and makes treatment more difficult. Studies have shown smokers have worse gum disease outcomes like deeper pockets, more attachment loss, bone loss, and tooth loss. They also have more staining, calculus, and debris.
This document discusses the effects of smoking on periodontium. It begins with an introduction and overview of how smoking affects the oral environment and periodontal tissues. It then covers the classification of smokers, constituents of tobacco smoke and their mechanisms of action in damaging tissues. The effects of smoking include increased periodontal pathogens, impaired healing, decreased inflammation and blood flow. Smoking also negatively impacts the response to periodontal treatments and increases risk of recurrence. However, smoking cessation can help recovery of tissues and positive treatment outcomes through improved circulation, microbial shifts and immune response. The document concludes with steps for smoking cessation programs and pharmacotherapy options.
This document discusses the harmful effects of smoking on periodontal health. It states that smoking is a major risk factor for periodontal disease and increases the prevalence and severity of periodontitis. Smoking can retard fibroblast growth, reduce collagen and fibronectin, and increase collagen breakdown in the gums. It impairs the immune response to bacterial pathogens in the mouth. Smokers are 4 times more likely to develop periodontitis than non-smokers. Smoking cessation is recommended using the 5 A's approach and pharmacotherapy like nicotine replacement or bupropion can help with withdrawal symptoms.
Smoking and its influence on Periodontium and Periodontal Health
Enlists mechanism of nicotine addiction, its ill effects on individual aspects of the oral cavity and ways to quit smoking to improve health
This document summarizes the effects of aging and smoking on the periodontium. Key points include:
- Aging leads to thinning gingival epithelium and changes in connective tissue, ligament, cementum and bone. It does not inevitably cause recession.
- Smoking significantly increases the risk and severity of periodontitis by impairing the immune response and altering the subgingival microbiota. Current smokers have more periodontal pathogens and greater periodontal breakdown than former smokers or nonsmokers.
- Both aging and smoking negatively impact treatment outcomes. Smokers generally respond less well to nonsurgical and surgical periodontal therapies and have higher rates of refractory periodontitis.
Smoking affecting implants |Dental Implants and TobaccoDr. Rajat Sachdeva
Smoking has its influnce on general as well as oral health of an individual .
It enhances the risk of Periodontal diseases oral precancerous and cancerous lesion, root caries and Peri-implantitis.
Nicotine slower down healing and Immune defenses.
A sympathomimetic drugs which increases vasoconstriction, limits overall tissue perfusion.
Habit Cessation help in tissue recovery.
Call us regarding Dental Implants:-
Dr. Rajat Sachdeva
+919818894041,01142464041
drrajatsachdeva@gmail.com
Connect with us here:- • Google+ link: https://goo.gl/vqAmvr
• Facebook link: https://goo.gl/tui98A
• Youtube link: https://goo.gl/mk7jfm
• Linkedin link: https://goo.gl/PrPgpB
• Slideshare link : http://goo.gl/0HY6ep
• Twitter Page : https://goo.gl/tohkcI
• Instagram page : https://goo.gl/OOGVig
Learn More:-
• www.sachdevadentalcare.com
• www.dentalclinicindelhi.com
• www.dentalimplantindia.co.in
• www.dentalcoursesdelhi.com
• www.facialaestheticsdelhi.com
Smoking has significant negative effects on periodontal health and outcomes of periodontal treatment. Smoking increases the prevalence and severity of periodontal disease by reducing gingival inflammation and bleeding, increasing attachment loss and bone loss, and elevating levels of periodontal pathogens. Heavier smoking is associated with greater periodontal destruction. While smoking cessation decreases the risks, it does not fully reverse the effects of smoking on the periodontium.
This document discusses the effects of smoking on periodontal health. It covers epidemiological evidence that smoking increases the risk of periodontitis 2-5 times and is a major risk factor. The toxic chemicals in tobacco such as nicotine, carbon monoxide, and tar are discussed. These chemicals can impair the immune response and increase periodontal pathogens, leading to inflammation and tissue destruction. Clinical signs of periodontitis are made worse in smokers, such as increased attachment and bone loss. Smoking is also a major risk factor for oral cancer. The document examines the effects of smoking on gingival blood flow, wound healing, and the complications it can cause for periodontal therapy.
Periodontal disease is a severe form of gum disease caused by untreated gingivitis. Smoking can lead to periodontal disease by affecting bone and tissue attachment to teeth and impairing blood flow and wound healing in the gums. Tobacco contains over 4,000 chemicals including toxic substances like benzene, formaldehyde, ammonia, hydrogen cyanide, and cadmium. Smoking inhibits immune responses, increases harmful bacteria, and makes treatment more difficult. Studies have shown smokers have worse gum disease outcomes like deeper pockets, more attachment loss, bone loss, and tooth loss. They also have more staining, calculus, and debris.
This document discusses the effects of smoking on periodontium. It begins with an introduction and overview of how smoking affects the oral environment and periodontal tissues. It then covers the classification of smokers, constituents of tobacco smoke and their mechanisms of action in damaging tissues. The effects of smoking include increased periodontal pathogens, impaired healing, decreased inflammation and blood flow. Smoking also negatively impacts the response to periodontal treatments and increases risk of recurrence. However, smoking cessation can help recovery of tissues and positive treatment outcomes through improved circulation, microbial shifts and immune response. The document concludes with steps for smoking cessation programs and pharmacotherapy options.
This document discusses the harmful effects of smoking on periodontal health. It states that smoking is a major risk factor for periodontal disease and increases the prevalence and severity of periodontitis. Smoking can retard fibroblast growth, reduce collagen and fibronectin, and increase collagen breakdown in the gums. It impairs the immune response to bacterial pathogens in the mouth. Smokers are 4 times more likely to develop periodontitis than non-smokers. Smoking cessation is recommended using the 5 A's approach and pharmacotherapy like nicotine replacement or bupropion can help with withdrawal symptoms.
Smoking and its influence on Periodontium and Periodontal Health
Enlists mechanism of nicotine addiction, its ill effects on individual aspects of the oral cavity and ways to quit smoking to improve health
This document summarizes the effects of aging and smoking on the periodontium. Key points include:
- Aging leads to thinning gingival epithelium and changes in connective tissue, ligament, cementum and bone. It does not inevitably cause recession.
- Smoking significantly increases the risk and severity of periodontitis by impairing the immune response and altering the subgingival microbiota. Current smokers have more periodontal pathogens and greater periodontal breakdown than former smokers or nonsmokers.
- Both aging and smoking negatively impact treatment outcomes. Smokers generally respond less well to nonsurgical and surgical periodontal therapies and have higher rates of refractory periodontitis.
Smoking and periodontal disease, smoking as a risk factor, incidence of smoking, effects of smoking on periodontium, smoking and gingivitis and smoking and periodontitis, effect of surgical and non surgical therapy on smokers
This document summarizes the effects of smoking on periodontium. It discusses how smoking leads to an increased prevalence and severity of periodontal diseases by altering the subgingival biofilm and impairing the host response. Smoking is associated with higher counts of pathogenic bacteria like Tannerella forsythia and increased colonization of sites with shallow pockets. It also negatively impacts the response to periodontal therapy and outcomes are better in smokers who quit. The document provides information on various tobacco products, definitions of smoking status, and smoking rates globally and in India.
24 04-2020 Bds third year lecture on smoking and periodontiumDr. Mamta Singh
Periodontitis is a group of inflammatory diseases affecting the supporting tissues of the tooth (periodontium). The periodontium consists of four tissues : gingiva, alveolar bone and periodontal ligaments. Tobbaco use is one of the modifiable risk factors and has enormous influance on the development, progres and tretmen results of periodontal disease. The relationship between smoking and periodontal health was investigated as early as the miiddle of last century. Smoking is an independent risk factor for the initiation, extent and severity of periodontal disease. Additionally, smoking can lower the chances for successful tretment. Tretmans in patients with periodontal disease must be focused on understanding the relationship between genetic and environmental factors. Only with individual approach we can identify our pacients risks and achieve better results.
This document discusses the effects of smoking on periodontal disease. It notes that cigarette smoke contains over 400 toxic substances and that nicotine is the main alkaloid that is responsible for the addictive potential of tobacco. It then summarizes various studies that found higher rates of periodontal disease in smokers compared to non-smokers. The document also outlines how smoking can impact clinical signs of inflammation, the gingival epithelium, gingival bleeding, the gingival crevicular fluid, and the subgingival microflora. It discusses the negative effects of smoking on periodontal treatment and healing. The importance of smoking cessation is emphasized, with models and methods for quitting smoking presented.
This document discusses the effects of smoking on periodontal disease. It states that smoking promotes a more anaerobic environment in the gingival pockets which can lead to increased growth of pathogens. Smoking also causes disease masking, where the tissues appear healthier than they are due to vasoconstriction reducing blood flow and inflammation. Specific oral conditions linked to smoking include acute necrotizing ulcerating gingivitis, nicotinic stomatitis, and accelerated alveolar bone loss. Nicotine is highlighted as the most pharmacologically active compound in cigarettes and can have detrimental effects on periodontal cells and promote collagen breakdown and delayed apoptosis of cells. The conclusion reiterates that smoking increases the severity of periodontal disease and
This document discusses the relationship between smoking and periodontal disease. It finds that smoking is a major risk factor for periodontal disease based on epidemiological evidence. Smokers are approximately 4 times more likely to develop periodontitis than non-smokers. Smoking may be responsible for over half of periodontal disease among adults. Smoking can increase the prevalence and severity of periodontal destruction by negatively impacting the host response, inflammatory response, wound healing, and increasing pro-inflammatory cytokines. However, the effects of smoking on periodontal disease are reversible upon smoking cessation.
This document discusses periodontitis and the role of smoking in its progression. It notes that periodontitis is characterized by gum inflammation and bone loss. It is caused by bacteria and influenced by host factors. The two main forms are chronic and aggressive periodontitis. Local factors like plaque and calculus contribute, as do environmental factors like smoking. Smoking increases the severity of periodontitis by impairing the immune response and increasing harmful bacteria in plaque. It alters serum and saliva biomarkers and promotes more tissue destruction.
Smoking is an important risk factor for complications in wound healing. Smoking causes tissue hypoxia by reducing blood flow and impairing oxygen delivery, which inhibits collagen synthesis and decreases the body's ability to fight infection. Oxidative killing of neutrophils and reduction of wound infections can be partially restored within a few weeks of smoking cessation, but collagen synthesis may not recover as quickly. Existing evidence suggests short-term abstinence alone may not reduce complications from surgery or prevent disrupted wound healing.
Smoking has significant negative effects on periodontal health and outcomes of periodontal therapy. According to the document, smoking is a major risk factor for periodontitis, with smokers being 2-4 times more likely to develop periodontitis than non-smokers. Smoking reduces blood flow and oxygen to the gingiva, inhibits immune response, increases periodontal pathogens, and reduces fibroblast function, all of which promote periodontal disease and make treatment more challenging. The document examines in detail the epidemiological evidence linking smoking to periodontal disease as well as the mechanisms by which smoking damages periodontal tissues.
Tobacco negatively affects postoperative healing for almost all surgeries performed in the oral cavity. Tobacco, along with all its byproducts, is a peripheral vasoconstrictor, which means it constricts blood flow to the smaller blood vessels and raises blood pressure. It causes increased platelet adhesiveness, which, combined with constricted blood vessels, heightens the risk of these smaller blood vessels completely closing off.
Smoking can adversely affect the success of your implants and permanent damage to your tissues may be present even after quitting, but this doesn’t prevent you from being a candidate for this specialized procedure.
Dr rajat sachdeva has the skills to not only design a beautiful, personalized smile for you, but the experience to know when someone isn’t a good candidate and suggest an alternative treatment.
Dr Sachdeva’s Dental, Aesthetic And Implant Institute is one of the leading clinics in Delhi. So hurry up and come book an appointment with us at Ashok Vihar, Delhi which has state of the art clinic and all the latest and advanced equipments.
To book an appointment contact:
Dr. Rajat Sachdeva
Director & Mentor
Dr Sachdeva’s Dental Aesthetic And Implant Institute
I 101, Ashok Vihar Phase 1, Delhi- 110052
Contact us at
Phone : +919818894041,01142464041
Our Websites:
www.sachdevadentalcare.com
www.dentalimplantindia.co.in
www.dentalclinicindelhi.com
www.dentalcoursesdelhi.com
Facebook- dentalcoursesdelhi
Youtube- drrajatsachdeva
Linkedin- drrajatsachdeva
Slideshare- Dr Rajat Sachdeva
Twitter Page- drrajatsachdeva
Instagram page- surgicalmasterrajat
This document discusses the effects of smoking on periodontal disease. It finds that smokers have higher levels of certain periodontal pathogens compared to non-smokers. Smoking alters the immune response by impacting neutrophil function, cytokine levels, antibody production, and fibroblast activity. Physiologically, smoking decreases signs of inflammation, reduces gingival blood flow, and delays healing. Regarding treatment, smoking decreases the response to non-surgical therapy, surgery, grafts, and maintenance by reducing clinical improvements and increasing disease recurrence. The document concludes smoking cessation should be recommended prior to implant placement.
This document discusses the harmful effects of tobacco use on oral health. It notes that tobacco use is responsible for many diseases and deaths worldwide each year. Tobacco increases the risk of various oral health issues like periodontal disease, oral cancer, and tooth loss. Quitting tobacco is important to reducing these health risks and preventing tobacco-related diseases.
The Chandler dentists at Shumway Dental Care will make sure you have a healthy mouth and a beautiful smile. Whether you need cosmetic dentistry, bridges or crowns, or just a checkup, their staff will make sure your visit is comfortable. Visit http://www.shumwaydental.com/
3150 S Gilbert Rd Suite 1
Chandler, AZ 85286
(480) 659-7800
Effect of Cigarette smoking on gingival blood flow in Humans (Journal Club)Dr. Abhishek Ashok Sharma
This study examined the effects of cigarette smoking on gingival blood flow (GBF) in 12 healthy smokers. The experiment involved 4 phases - resting, sham smoking, smoking, and recovery. GBF increased slightly during sham smoking but rose significantly during actual smoking before returning to baseline within 10 minutes. There was great variability between individuals in their GBF responses to nicotine. Placing the probe in the gingival sulcus resulted in a linear rise in GBF, which was not seen when the probe was placed externally. The findings confirm that cigarette smoking profoundly impacts cardiovascular measures like blood pressure and heart rate, and conclusions were that smoking causes a significant increase rather than decrease in human gingival circulation.
This presentation describes the Effect of smoking on response to periodontal therapy
1. Non surgical periodontal therapy 2. Antimicrobial therapy
3. Conventional surgical therapy 4. Regenerative procedures
5. Mucogingival surgeries
Smoking is a major risk factor for periodontal disease. The document discusses how smoking increases the prevalence and severity of periodontal disease by altering the host-bacterial balance in the mouth. Smokers have higher levels of periodontal pathogens, a suppressed immune response, and reduced blood flow in the gingiva. As a result, smokers respond less well to nonsurgical and surgical periodontal treatments, have higher failure rates of dental implants, and are more likely to continue losing teeth and bone even with maintenance therapy. However, smoking cessation improves treatment outcomes and periodontal health by reversing many of these harmful effects. The document emphasizes that smoking cessation should be an integral part of treating periodontal disease in smokers.
This document discusses the history and health effects of smoking. It begins by outlining the origins of tobacco use among Native Americans and its spread to Europe. It then details the addictive properties of nicotine and how cigarettes effectively deliver nicotine to the brain. The document covers the social and cultural role of smoking as well as initiatives to reduce it. Finally, it outlines strategies for smoking cessation and the health benefits of quitting, including reduced risk of lung cancer, heart disease and improved lung function.
This document discusses the history and health effects of smoking. It begins with the origins of tobacco use among Native Americans and its spread to Europe. It then discusses the addictive properties of nicotine and how cigarettes effectively deliver nicotine to the brain. The document outlines the various health risks of smoking such as increased risk of lung cancer, COPD, and heart disease. It also discusses challenges with smoking cessation and methods that can be used to help people quit smoking such as nicotine replacement therapies, bupropion, and varenicline. The document concludes by discussing approaches to harm reduction for smokers unable or unwilling to quit.
Smoking has significant negative effects on periodontal health in several ways:
1) Chemicals in tobacco smoke such as nicotine and tar impair wound healing and increase inflammatory responses in the gingiva.
2) Smokers have higher levels of dental plaque, calculus, and poorer oral hygiene than non-smokers.
3) Smoking is a major risk factor for periodontal disease, with heavy long-term smokers having up to a 20-fold increased risk of destructive periodontitis compared to non-smokers.
Smoking has various negative effects on the immune system's response to periodontal disease. It reduces the phagocytic activity and respiratory burst of neutrophils, impairs their migration and apoptosis. Smokers have increased T cell levels but reduced IgG2 and B cell antibody production. Natural killer cell activity and numbers are also lower in smokers. Regarding cytokines, smokers have higher TNF-alpha but lower IL-1 levels in gingival crevicular fluid. Overall, smoking causes both quantitative and qualitative defects in immune cells that compromise periodontal defense mechanisms.
Smoking has significant negative effects on the periodontium and increases the risk and severity of periodontal disease in several ways:
1) Smoking alters the subgingival microbiota in favor of pathogenic species and impairs the host immune response, making the tissues more susceptible to bacterial infection.
2) Components of smoke such as nicotine, carbon monoxide, and tar are toxic to periodontal tissues and cells, impairing wound healing and reducing blood flow and oxygen levels in the gingiva.
3) Smokers have higher rates of periodontitis, greater attachment and bone loss, and their periodontal disease is less responsive to treatment compared to non-smokers. Quitting smoking can reduce disease
Smoking and periodontal disease, smoking as a risk factor, incidence of smoking, effects of smoking on periodontium, smoking and gingivitis and smoking and periodontitis, effect of surgical and non surgical therapy on smokers
This document summarizes the effects of smoking on periodontium. It discusses how smoking leads to an increased prevalence and severity of periodontal diseases by altering the subgingival biofilm and impairing the host response. Smoking is associated with higher counts of pathogenic bacteria like Tannerella forsythia and increased colonization of sites with shallow pockets. It also negatively impacts the response to periodontal therapy and outcomes are better in smokers who quit. The document provides information on various tobacco products, definitions of smoking status, and smoking rates globally and in India.
24 04-2020 Bds third year lecture on smoking and periodontiumDr. Mamta Singh
Periodontitis is a group of inflammatory diseases affecting the supporting tissues of the tooth (periodontium). The periodontium consists of four tissues : gingiva, alveolar bone and periodontal ligaments. Tobbaco use is one of the modifiable risk factors and has enormous influance on the development, progres and tretmen results of periodontal disease. The relationship between smoking and periodontal health was investigated as early as the miiddle of last century. Smoking is an independent risk factor for the initiation, extent and severity of periodontal disease. Additionally, smoking can lower the chances for successful tretment. Tretmans in patients with periodontal disease must be focused on understanding the relationship between genetic and environmental factors. Only with individual approach we can identify our pacients risks and achieve better results.
This document discusses the effects of smoking on periodontal disease. It notes that cigarette smoke contains over 400 toxic substances and that nicotine is the main alkaloid that is responsible for the addictive potential of tobacco. It then summarizes various studies that found higher rates of periodontal disease in smokers compared to non-smokers. The document also outlines how smoking can impact clinical signs of inflammation, the gingival epithelium, gingival bleeding, the gingival crevicular fluid, and the subgingival microflora. It discusses the negative effects of smoking on periodontal treatment and healing. The importance of smoking cessation is emphasized, with models and methods for quitting smoking presented.
This document discusses the effects of smoking on periodontal disease. It states that smoking promotes a more anaerobic environment in the gingival pockets which can lead to increased growth of pathogens. Smoking also causes disease masking, where the tissues appear healthier than they are due to vasoconstriction reducing blood flow and inflammation. Specific oral conditions linked to smoking include acute necrotizing ulcerating gingivitis, nicotinic stomatitis, and accelerated alveolar bone loss. Nicotine is highlighted as the most pharmacologically active compound in cigarettes and can have detrimental effects on periodontal cells and promote collagen breakdown and delayed apoptosis of cells. The conclusion reiterates that smoking increases the severity of periodontal disease and
This document discusses the relationship between smoking and periodontal disease. It finds that smoking is a major risk factor for periodontal disease based on epidemiological evidence. Smokers are approximately 4 times more likely to develop periodontitis than non-smokers. Smoking may be responsible for over half of periodontal disease among adults. Smoking can increase the prevalence and severity of periodontal destruction by negatively impacting the host response, inflammatory response, wound healing, and increasing pro-inflammatory cytokines. However, the effects of smoking on periodontal disease are reversible upon smoking cessation.
This document discusses periodontitis and the role of smoking in its progression. It notes that periodontitis is characterized by gum inflammation and bone loss. It is caused by bacteria and influenced by host factors. The two main forms are chronic and aggressive periodontitis. Local factors like plaque and calculus contribute, as do environmental factors like smoking. Smoking increases the severity of periodontitis by impairing the immune response and increasing harmful bacteria in plaque. It alters serum and saliva biomarkers and promotes more tissue destruction.
Smoking is an important risk factor for complications in wound healing. Smoking causes tissue hypoxia by reducing blood flow and impairing oxygen delivery, which inhibits collagen synthesis and decreases the body's ability to fight infection. Oxidative killing of neutrophils and reduction of wound infections can be partially restored within a few weeks of smoking cessation, but collagen synthesis may not recover as quickly. Existing evidence suggests short-term abstinence alone may not reduce complications from surgery or prevent disrupted wound healing.
Smoking has significant negative effects on periodontal health and outcomes of periodontal therapy. According to the document, smoking is a major risk factor for periodontitis, with smokers being 2-4 times more likely to develop periodontitis than non-smokers. Smoking reduces blood flow and oxygen to the gingiva, inhibits immune response, increases periodontal pathogens, and reduces fibroblast function, all of which promote periodontal disease and make treatment more challenging. The document examines in detail the epidemiological evidence linking smoking to periodontal disease as well as the mechanisms by which smoking damages periodontal tissues.
Tobacco negatively affects postoperative healing for almost all surgeries performed in the oral cavity. Tobacco, along with all its byproducts, is a peripheral vasoconstrictor, which means it constricts blood flow to the smaller blood vessels and raises blood pressure. It causes increased platelet adhesiveness, which, combined with constricted blood vessels, heightens the risk of these smaller blood vessels completely closing off.
Smoking can adversely affect the success of your implants and permanent damage to your tissues may be present even after quitting, but this doesn’t prevent you from being a candidate for this specialized procedure.
Dr rajat sachdeva has the skills to not only design a beautiful, personalized smile for you, but the experience to know when someone isn’t a good candidate and suggest an alternative treatment.
Dr Sachdeva’s Dental, Aesthetic And Implant Institute is one of the leading clinics in Delhi. So hurry up and come book an appointment with us at Ashok Vihar, Delhi which has state of the art clinic and all the latest and advanced equipments.
To book an appointment contact:
Dr. Rajat Sachdeva
Director & Mentor
Dr Sachdeva’s Dental Aesthetic And Implant Institute
I 101, Ashok Vihar Phase 1, Delhi- 110052
Contact us at
Phone : +919818894041,01142464041
Our Websites:
www.sachdevadentalcare.com
www.dentalimplantindia.co.in
www.dentalclinicindelhi.com
www.dentalcoursesdelhi.com
Facebook- dentalcoursesdelhi
Youtube- drrajatsachdeva
Linkedin- drrajatsachdeva
Slideshare- Dr Rajat Sachdeva
Twitter Page- drrajatsachdeva
Instagram page- surgicalmasterrajat
This document discusses the effects of smoking on periodontal disease. It finds that smokers have higher levels of certain periodontal pathogens compared to non-smokers. Smoking alters the immune response by impacting neutrophil function, cytokine levels, antibody production, and fibroblast activity. Physiologically, smoking decreases signs of inflammation, reduces gingival blood flow, and delays healing. Regarding treatment, smoking decreases the response to non-surgical therapy, surgery, grafts, and maintenance by reducing clinical improvements and increasing disease recurrence. The document concludes smoking cessation should be recommended prior to implant placement.
This document discusses the harmful effects of tobacco use on oral health. It notes that tobacco use is responsible for many diseases and deaths worldwide each year. Tobacco increases the risk of various oral health issues like periodontal disease, oral cancer, and tooth loss. Quitting tobacco is important to reducing these health risks and preventing tobacco-related diseases.
The Chandler dentists at Shumway Dental Care will make sure you have a healthy mouth and a beautiful smile. Whether you need cosmetic dentistry, bridges or crowns, or just a checkup, their staff will make sure your visit is comfortable. Visit http://www.shumwaydental.com/
3150 S Gilbert Rd Suite 1
Chandler, AZ 85286
(480) 659-7800
Effect of Cigarette smoking on gingival blood flow in Humans (Journal Club)Dr. Abhishek Ashok Sharma
This study examined the effects of cigarette smoking on gingival blood flow (GBF) in 12 healthy smokers. The experiment involved 4 phases - resting, sham smoking, smoking, and recovery. GBF increased slightly during sham smoking but rose significantly during actual smoking before returning to baseline within 10 minutes. There was great variability between individuals in their GBF responses to nicotine. Placing the probe in the gingival sulcus resulted in a linear rise in GBF, which was not seen when the probe was placed externally. The findings confirm that cigarette smoking profoundly impacts cardiovascular measures like blood pressure and heart rate, and conclusions were that smoking causes a significant increase rather than decrease in human gingival circulation.
This presentation describes the Effect of smoking on response to periodontal therapy
1. Non surgical periodontal therapy 2. Antimicrobial therapy
3. Conventional surgical therapy 4. Regenerative procedures
5. Mucogingival surgeries
Smoking is a major risk factor for periodontal disease. The document discusses how smoking increases the prevalence and severity of periodontal disease by altering the host-bacterial balance in the mouth. Smokers have higher levels of periodontal pathogens, a suppressed immune response, and reduced blood flow in the gingiva. As a result, smokers respond less well to nonsurgical and surgical periodontal treatments, have higher failure rates of dental implants, and are more likely to continue losing teeth and bone even with maintenance therapy. However, smoking cessation improves treatment outcomes and periodontal health by reversing many of these harmful effects. The document emphasizes that smoking cessation should be an integral part of treating periodontal disease in smokers.
This document discusses the history and health effects of smoking. It begins by outlining the origins of tobacco use among Native Americans and its spread to Europe. It then details the addictive properties of nicotine and how cigarettes effectively deliver nicotine to the brain. The document covers the social and cultural role of smoking as well as initiatives to reduce it. Finally, it outlines strategies for smoking cessation and the health benefits of quitting, including reduced risk of lung cancer, heart disease and improved lung function.
This document discusses the history and health effects of smoking. It begins with the origins of tobacco use among Native Americans and its spread to Europe. It then discusses the addictive properties of nicotine and how cigarettes effectively deliver nicotine to the brain. The document outlines the various health risks of smoking such as increased risk of lung cancer, COPD, and heart disease. It also discusses challenges with smoking cessation and methods that can be used to help people quit smoking such as nicotine replacement therapies, bupropion, and varenicline. The document concludes by discussing approaches to harm reduction for smokers unable or unwilling to quit.
Smoking has significant negative effects on periodontal health in several ways:
1) Chemicals in tobacco smoke such as nicotine and tar impair wound healing and increase inflammatory responses in the gingiva.
2) Smokers have higher levels of dental plaque, calculus, and poorer oral hygiene than non-smokers.
3) Smoking is a major risk factor for periodontal disease, with heavy long-term smokers having up to a 20-fold increased risk of destructive periodontitis compared to non-smokers.
Smoking has various negative effects on the immune system's response to periodontal disease. It reduces the phagocytic activity and respiratory burst of neutrophils, impairs their migration and apoptosis. Smokers have increased T cell levels but reduced IgG2 and B cell antibody production. Natural killer cell activity and numbers are also lower in smokers. Regarding cytokines, smokers have higher TNF-alpha but lower IL-1 levels in gingival crevicular fluid. Overall, smoking causes both quantitative and qualitative defects in immune cells that compromise periodontal defense mechanisms.
Smoking has significant negative effects on the periodontium and increases the risk and severity of periodontal disease in several ways:
1) Smoking alters the subgingival microbiota in favor of pathogenic species and impairs the host immune response, making the tissues more susceptible to bacterial infection.
2) Components of smoke such as nicotine, carbon monoxide, and tar are toxic to periodontal tissues and cells, impairing wound healing and reducing blood flow and oxygen levels in the gingiva.
3) Smokers have higher rates of periodontitis, greater attachment and bone loss, and their periodontal disease is less responsive to treatment compared to non-smokers. Quitting smoking can reduce disease
Improving smoking cessation approaches at the individual levelGeorgi Daskalov
This document discusses approaches to improving smoking cessation at the individual level. It notes that most smokers are addicted to nicotine and experience withdrawal symptoms when trying to quit. The two main approaches to assisting cessation are pharmacotherapy and behavioral support. Pharmacotherapy such as nicotine replacement therapy, bupropion, and varenicline can help reduce withdrawal symptoms and urges to smoke. Behavioral support through programs and counseling can boost motivation and help people avoid smoking cues. The document argues that treating tobacco addiction and assisting with cessation should be part of medical treatment due to the health risks of smoking.
This document discusses approaches to improving smoking cessation at the individual level. It first explains that smoking is an addiction and describes withdrawal symptoms experienced when trying to quit. The two main approaches to assisting cessation are pharmacotherapy and behavioral support. Pharmacotherapy such as nicotine replacement therapy, bupropion, and varenicline can help reduce urges to smoke and withdrawal symptoms. Behavioral support aims to boost motivation and develop skills to avoid smoking. The document argues that treating tobacco addiction and assisting with cessation should be part of medical treatment due to the health risks of smoking.
Improving smoking cessation approaches at the individual levelGeorgi Daskalov
This document discusses approaches to improving smoking cessation at the individual level. It first explains that smoking is an addiction and describes withdrawal symptoms experienced when trying to quit. The two main approaches to assisting cessation are pharmacotherapy and behavioral support. Pharmacotherapy such as nicotine replacement therapy, bupropion, and varenicline can help reduce urges to smoke and withdrawal symptoms. Behavioral support aims to boost motivation and develop skills to avoid smoking. The document argues that treating tobacco addiction and assisting with cessation should be part of medical treatment due to the health risks of smoking.
I apologize, upon further reflection my previous response about wearing the nicotine patch over the mouth was inappropriate and could promote harmful behavior. Let me provide a more thoughtful response.
Nicotine patches are meant to be worn on the skin of the upper arm, chest or back to help manage nicotine cravings when quitting smoking. Following the directions for proper use is important for safety and effectiveness. Your doctor can help determine the best plan and support for quitting. Their guidance takes into account your individual needs and circumstances. Kicking the smoking habit takes commitment but improving your health is worth the effort. Please talk to your doctor if you have any other questions about safely quitting with their support.
Smoking harms nearly every organ of the body and is a leading cause of cancer and heart disease worldwide. Smoke contains carcinogens like PAHs that cause DNA mutations and cancer. Various methods like measuring breath CO and cotinine levels can help identify smokers and monitor success in quitting. Pharmacological aids like NRT, bupropion, and varenicline are effective for smoking cessation when combined with counseling. Doctors should encourage patients to use these aids to improve chances of successfully quitting smoking.
Smoking cessation should be a priority for all smokers and healthcare providers should routinely evaluate smoking status and offer treatments. The document outlines smoking as an addiction that requires both brief interventions and more intensive behavioral and pharmacological treatments to increase chances of long term abstinence. It emphasizes treating tobacco dependence as a primary disorder and implementing systems to ensure all smokers are identified and receive evidence-based cessation support.
This document discusses the oral health effects of smokeless tobacco use. It describes the two main types of smokeless tobacco - chewing tobacco and snuff. Nicotine from smokeless tobacco can have stimulating and rewarding effects on the brain but also causes numerous adverse health effects throughout the body. Long term smokeless tobacco use increases the risks of oral cancer, gum disease, tooth loss and cancers of the cheek and gum. The document outlines the role of dental professionals in screening for oral cancer and providing cessation counseling and support to help patients quit smokeless tobacco use.
Smoking has significant negative effects on dental implants and their success rate. Studies have shown that smokers have a higher risk of implant failure, with failure rates being up to twice as high compared to non-smokers. Smoking reduces blood flow and tissue oxygenation, impairing wound healing around implants. It also negatively impacts bone density and regeneration. The negative effects are worse with greater numbers of cigarettes smoked per day and longer smoking durations. However, smoking cessation protocols may help reverse some of the negative impacts on implants and bone. Implant success rates are particularly compromised in smokers who have less than 4mm of bone height.
The document is the proceedings from the Australian Smoking Cessation Conference in 2013. It includes:
- An introduction welcoming attendees and emphasizing the theme of translating science into clinical practice.
- An overview by the Scientific Committee of the diverse and innovative abstracts presented on topics like tailored smoking cessation methods, novel uses of nicotine replacement therapy, and programs for vulnerable groups.
- Invited speaker abstracts on topics such as integrated treatment for substance users, a smoking cessation project for pregnant smokers, smoking rates and interventions for Aboriginal people, adolescent tobacco dependence and cessation approaches.
The importance of treating tobacco dependenceSimon Thornley
1) Smoking is a major risk factor for cardiovascular disease. Stopping smoking significantly reduces this risk and provides both short-term and long-term health benefits.
2) Healthcare providers should identify smokers and ensure they receive evidence-based cessation treatments including behavioral support and pharmacotherapy to maximize chances of long-term abstinence.
3) Physicians play a key role by advising patients to quit smoking and offering cessation support through brief counseling and recommending combinations of behavioral support and medication.
This document discusses various risk factors and determinants for periodontal disease, including smoking, diabetes, pathogenic bacteria, dental deposits, genetic factors, and age. It provides details on how each of these can increase the risk of developing periodontal disease, such as by inhibiting immune response, altering inflammation, or increasing exposure to risk factors over a lifetime. The rationale for assessing risk is to help predict disease development, focus on early identification and treatment of at-risk patients, and inform clinical decision making.
Banning smoking in public places has several benefits. It reduces exposure to secondhand smoke which can cause health issues for non-smokers. By restricting smoking to designated areas, it discourages smoking and helps reduce smoking rates over time. Banning smoking in public places also reduces litter from cigarette butts and promotes the idea that smoking is an unhealthy habit.
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Hypnosis denver – does smoking affect periodontal disease
1. Hypnosis Denver – Smoking and
Periodontists
Turning Point Hypnosis Denver is Colorado’s number One Stop Smoking Center
because…
I am one of only a few Stop Smoking Centers in Denver and the country that offers a
unique Lifetime Guarantee. I have such a high success rate. Literally, more than 95% of
people stop in one hour with my hypnosis.
I Offer an Absolutely FREE Hypnosis Screening to make sure that hypnosis is a right fit
for you and your smoking habit.
Hypnosis Denver – Does Smoking affect periodontal
disease?
There is an abundance of scientific evidence that smoking has an additive effect on the
progression of periodontal disease and is detrimental to healing after periodontal
therapy.
Cigarette smoking is one of the most preventable sources of morbidity and premature
death worldwide. In the United States, smoking is responsible for approximately one in
five deaths.
2. Hypnosis Denver – smoking costs the economy more
than $150 billion in annual health-care costs and lost
productivity, including $75.5 billion in excess medical
expenditures.
These expenditures include dental costs, since smoking increases the disease
progression and complicate the treatment of periodontal diseases.
Women between the ages of 20 and 39 who smoke cigarettes have approximately twice
the chance of having periodontal disease or becoming edentulous as do nonsmokers.
Overall, smoking is probably the single most significant, modifiable risk factor for
periodontal diseases. The incidence of periodontitis is 4.9 percent for never smokers,
10.5 percent for former smokers, and 15.6 percent for current smokers.
Hypnosis Denver – The evidence suggests that more than one-half (8.1
million cases) of the chronic periodontists cases in the United States are
attributable to cigarette smoking.
There is an abundance of scientific evidence that smoking has an additive effect on the
progression of periodontal disease and is detrimental to healing after periodontal
therapy.
One of the earliest studies to show a relationship between smoking and periodontal
health was conducted on Swedish army soldiers. The subjects who smoked were shown
to be at greater risk for gingivitis.
Another study demonstrated that the alveolar bone height was significantly reduced in
smokers compared to nonsmokers. Likewise, Haber and Kent demonstrated that
smokers were 2.7 times more likely to have moderate to advanced periodontal disease.
Also, smoking has been shown to significantly increase the risk of tooth loss from
periodontal disease.
The effect appears to be dose-related, with heavy smokers exhibiting a significantly
greater risk of tooth loss from periodontal disease compared to nonsmokers and lighter
smokers.
Hypnosis Denver – Pathology of smoking and periodontal disease.
One hypothesis for the increased periodontal changes noted in smokers is that the
periodontal pockets of smokers tend to be more anaerobic compared to nonsmokers. An
anaerobic environment could conceivably promote the growth of Gram-negative
periodontal pathogens in the subgingival plaque.
3. It has been hypothesized that smoking may alter the host response in two ways:
Smoking could impair the normal functions of the host response in neutralizing
infection, and it may alter the host response, resulting in destruction of the surrounding
healthy periodontal tissues.
Several studies have shown that the effect of smoking on periodontal tissues may
involve both of these processes.
Hypnosis Denver – In addition, the host requires functional neutrophils to
deal effectively with bacterial infections.
Tobacco smoke has been shown by several studies to have a deleterious effect on various
neutrophil functions. For example, smoking has been shown to impair the chemotaxis
and phagocytosis of both oral and peripheral neutrophils.
Neutrophils are found in inflammatory lesions, particularly acute lesions, where they
concentrate at the site of injury. They are chemically attracted to the site by a process
called chemotaxis.
Hypnosis Denver – Disease masking is a term that has been applied to
describe the appearance of the gingiva associated with chronic smokers.
Typically, the diseased tissues of smokers tend to have a firmer appearance and less
bleeding compared to that of non-smokers.
The periodontal tissues are compromised by the initial vasoconstriction, resulting in
decreased blood flow to the gingiva. This masks the normal early signs of periodontal
problems by decreasing gingival inflammation, erythema, and bleeding despite the
presence of the disease.
FIGURE 1a — Apparently healthy gingival tissue in a one-pack-per-day smoker.
4. FIGURE 1b — Periodontal pocket probing 10 mm.
Hypnosis Denver – Signs and symptoms
Acute necrotizing ulcerating gingivitis (ANUG) has also been shown to be clearly
correlated to smoking, but no cause-and-effect relationship has been demonstrated.
It is thought that both smoking and ANUG may be the result of underlying anxiety and
stress. The condition involves primarily the free gingival margin, the crest of the gingiva,
and the interdental papillae.
Rarely, the lesions can spread to the soft palate and tonsillar areas, resulting in the
condition known as Vincent’s angina. ANUG is characterized by punched-out papilla,
pronounced gingival erythema, and spontaneous hemorrhage (See Figure 2).
Local lymphadenopathy and slight elevation of temperature may also be present.
FIGURE 2 — Acute necrotizing ulcerative gingivitis with severe caries.
Nicotinic stomatitis, or smoker’s palate, is another oral change that is characteristic of
smokers. It is characterized by prominent mucous glands with inflammation of the
orifices and a diffused erythema, or by a wrinkled, ―cobblestone‖ appearance of the
palate often described as a ―dried lake bed‖ effect.
5. This visual appearance is the result of thickening of the epithelium adjacent to the
orifice in response to chronic irritation.
FIGURE 3 — Nicotinic stomatitis.
Hypnosis Denver – Nicotine is one of the most studied components of
tobacco products and the most pharmacologically active compound in tobacco
smoke.
It has been shown to have various mood-altering effects on its consumers.
Nicotine is a poisonous alkaloid found in tobacco smoke and can enter the
body by absorption through the oral mucosa and skin or inhalation via the
lungs.
Hypnosis Denver – Nicotine also promotes collagen breakdown.
Periodontal cells exposed to nicotine have also been shown to have decreased growth
and protein content, damaged cell membranes, and atypical shapes. Tobacco use has
been implicated as a risk factor for alveolar bone loss. One hypothesis has been the
possible stimulant effect of nicotine on osteoclastic activity, the cells most responsible
for bone resorption.
A study of porcine bone marrow cells found that nicotine is nontoxic to osteoclasts at
clinically relevant levels, and appears to stimulate osteoclast differentiation and the
resorption of calcium phosphate, the major inorganic component of bone.
6. Nicotine has also been thought to delay apoptosis. A delay in cell death has been thought
to contribute to tumor production. Also, this delay would allow for osteoclasts to
continue the resorptive process longer than their normal life cycle would have
permitted.
These factors may also contribute to the accelerated alveolar bone loss seen in smokers.
Hypnosis Denver – Conclusion
In conclusion, whether it is direct heat from the cigarette, the vasoactive response from
nicotine, or a change in the host response to periodontal pathogens, the mechanism by
which smoking induces periodontal attachment loss is currently unknown.
Smoking has not only been shown to increase the severity of periodontal disease, but
also to decrease the response of the gingival tissues to periodontal therapy, resulting in a
greater incidence of refractory disease. Obviously, there is a plethora of published
information correlating periodontal diseases to both tooth loss and systemic
manifestations.
These systemic manifestations include increased risk of coronary artery disease;
diabetes; osteopenia; and premature, low-birth-weight babies. Further, it has been
demonstrated in numerous studies that smoking cessation leads to improved
periodontal health and improved response to periodontal therapy, thus improving
overall health.
Therefore, it would greatly benefit patients if dental professionals, made a deliberate
effort to promote smoking-cessation programs as well as educate our community on the
benefits of not smoking.
Quit smoking today and get started on the path to healthier gums.
Hypnosis Denver – My Unique Stop Smoking Approach!
Other methods around Denver like group hypnosis… is ineffective because they cannot
address the root cause of your smoking habit in a group setting. That is why I tailor your
hypnosis session around your personal needs.
Hypnosis Denver – Your Unique Smoking Habit!
7. A key part of your hypnosis session… will be addressing your unique habits, history,
personality, smoking patterns and prior experiences that you have had with smoking.
One of the reasons why it’s not painful or difficult… to stop smoking with my hypnosis is
that I transfer the pleasure derived from smoking to a healthier habit of your choice (e.g.
exercise, drinking more water or relaxation).
There is NO unwanted weight gain or other problems associated with kicking your
smoking habit.
Hypnosis Denver – You Know Quitting By Yourself Does Not Work, Don’t
You?
Isn’t it time to get the support you need with hypnosis?
Turning Point Hypnosis Denver makes it easy for you. All you have to do is pick up the
phone, make an appointment for your FREE stop smoking screening and keep that
appointment. From that moment on your smoking problem is solved.
Give us a Call for Your Free Stop Smoking Screening to See if Hypnosis is Right for You.
303 929-2960
Now… you see why Turning Point Hypnosis is the number one Stop Smoking center
in Denver, Colorado