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Cortical Anatomy & Function.pptx
- 1. Cortical Anatomy &Function -AIL-
- 2. Outline • Cortical Anatomy •Broadman Area • Regional Brain Syndrome
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- 4. M-around sign &T-sign PTR POP POR hr vr PRG
- 5. Broca Area • Broadmannarea 44 (pars opercularis) • Broadmann area 45 (separuh pars triangularis) • Heschl’s gyrus/transverse temporal gyrus posterior STG Wernicke Area Language Area
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- 8. Omega sign, L-sign,Thickness sign, Bracket sign Primary motor & sensory cortex
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- 13. Lateral - Lapisan ke-5mengandung sel piramidalis Betz - Asal gerakan volunter Persepsi nyeri dan suhu Sensasi somatik dan propioseptif dari 1/2 tubuh dan wajah kontralateral - Menerima impuls dari radiasi optika - Korteks visual kanan – separuh lapang pandang kiri - Menerima impuls dari korpus genikulatum medial - Masing-masing sisi mengolah impuls dari kedua telinga (bilateral) - Impuls awal diolah di nukleus rostralis tr solitarius VPM thalamus kapsula interna genu posterior pars opercularis - Area 2v dan 3a integrasi informasi somatosensorik, sensorik khusus, motorik
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- 16. Frontal lobe syndrome •Prefrontal lobe: • Prefontal korteks: penyimpanan cepat dan analisis informasi objektif • Prefrontal korteks dorsolateral (9,10,46): perencanaan, motivasi dan berpikir • Prefrontal korteks orbitalis (11,47): kontrol pada perilaku, hubungan sosial • Area olfaktori Foster Kennedy Syndrome 1. Ipsilateral anosmia 2. Ipsilateral central scotoma (dengan optik atropi e.c kompresi n.optikus) 3. Contralateral papiledema
- 17. Parietal lobe syndrome •Kedua sisi: parietal anterior mengatur persepsi taktil (kontralateral) bersamaan dengan sensasi visual dan auditori • Sisi dominan: kalkulasi • Non dominan: body images & awareness of external environment
- 18. Temporal lobe syndrome 1.Korteks auditori: • Cortical deafness • Amusia • Halusinasi auditori 2. MTG & ITG: gangguan memori/proses belajar 3. Limbic lobe: • Halusinasi olfaktori • Kepribadian agresif atau anti sosial • Tidak dpt membuat memori baru 4. Radiasi optika kuadranopsia homonim
- 19. Occipital lobe syndrome •Visual korteks: korteks striata dan parastriata • Anton syndrome: opasien tidak sadar dan men-deny visual loss • Prosopagnosia: otidak dapat naming keluarga • Ilusi visual: oMikropsia oMakropsia Kortex Striata Kortex parastriata
Editor's Notes
- #5 Pars opercularis berhubungan dg gyrus precentral dan sulcus sentralis The inferior frontal sulcus and the precentral sulcus form a capital T tilted 90° clockwise (T-sign). The top of the T marks the precentral sulcus and the precentral sulcus identifies the precen- tral gyrus. The central sulcus is located immediately parallel and posterior to it
- #14 Area 43 secondary gustatorik
- #17 Mengapa terjadi kontralateral papiledema?
- #20 Cortical deafness: Bilateral lesions are rare but may result in complete deafness of which the patient may be unaware. Lesions which involve surrounding association areas may result in difficulty in hearing spoken words (dominant) or difficulty in appreciating rhythm/music (non-dominant) – AMUSIA. Auditory hallucinations may occur in temporal lobe disease.
- #21 Anton’s syndrome Involvement of both the striate and the parastriate cortices affects the interpretation of vision. The patient is unaware of his visual loss and denies its presence. This denial in the presence of obvious blindness characterizes Anton’s syndrome. Cortical blindness occurs mainly in vascular disease (posterior cerebral artery), but also following hypoxia and hypertensive encephalopathy or after surviving tentorial herniation. Balint’s syndrome Inability to direct voluntary gaze, associated with visual agnosia (loss of visual recognition) due to bilateral parieto-occipital lesions Prosopagnosia: the patient, though able to see a familiar face, e.g. a member of the family, cannot name it. This is usually associated with other disturbances of ‘interpretation’ and naming with intact vision such as colour agnosia (recognition of colours and matching of pairs of colours). Bilateral lesions at occipito-temporal junction are responsible.