Chapter 2. Defibrillator
History
1849: Ludwigg and Hoffa – VF induced by electrical
stimuli
History
1899: Prevost and Batelli - while a weak stimulus can
produce fibrillation, a stimulus of higher strength
applied to the heart could arrest ventricular fibrillation
and restore normal sinus rhythm.
History
1947: First defibrillation on humans
History
1966: Belfast Ambulance transported physicians
performed first pre-hospital defibrillation.
1969: First pre-hospital defibrillation by non physicians.
1970’s: Diack, Wellborn and Rullman developed first
automated external defibrillator AED’s.
Chain of Survival
Early Recognition and Assessment
• Early Access
• Early CPR
• Early Defibrillation
• Early Advanced Cardiac Life Support
7
Normal ECG
8
Ventricular Fibrillation (VF)
•The ventricles Move in a fast and disorganized way instead of contracting normally.
•No blood is pumped out of the heart → this causes cardiac arrest.
•⚠️Medical emergency — needs CPR and defibrillation (electric shock) immediately.
No identifiable P waves,
QRS complexes, or T waves
Rate 150 to 500 per minute
9
Ventricular Tachycardia (VT)
•The heart beats very fast (usually >180 beats per minute), but the rhythm is still organized.
•Shows wide and regular QRS complexes (each heartbeat is wide and abnormal).
•Blood still flows, but not effectively.
•If not treated, it can turn into ventricular fibrillation.
10
Tachyarrhythmia
A
B
A tachyarrhythmia is any
abnormally fast heart rhythm
caused by a problem in the
heart’s electrical system.
It combines two ideas:
•“Tachy” = fast
•“Arrhythmia” = irregular or
abnormal rhythm
So, tachyarrhythmia = a fast
and abnormal heart rhythm.
11
Sinus bradycardia
Sinus bradycardia is a slow but regular heart rhythm that starts from the sinus node
“Sinus” → means the rhythm comes from the sinus node (normal origin).
•“Bradycardia” → means slow heart rate (less than 60 beats per minute).
So, sinus bradycardia = normal rhythm, just slower than usual.
Introduction
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13
DEFIBRILLATORS
Cardiac fibrillation is a condition where in the
individual myocardial cells contract asynchronously
with only very local patterns relating to the
contraction of one cell and that of the next. This
serious condition reduces the cardiac output to near
zero, and it must be corrected as soon as possible
to avoid damage to the patient and death. Electric
shock to the heart can be used to reestablish a more
normal cardiac rhythm. Electric machines that
produce the energy to carry out this function are
known as defibrillators.
Cardiac Fibrillation
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Why Cardiac Defibrillation?
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Cardiac Arrest
 Occulsion of the
coronary artery leads to
reduced blood flow
 reduced blood flow
leads to infarct which
causes interruption of
normal cardiac
conduction
 Infarct = VF/VT
Shockable Rhythms
Ventricular Fibrillation Ventricular Tachycardia
Sinoatrial (SA) Node
Atrioventricular (AV)
Node
What is cardiac defibrillator
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How to use?
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What is defibrillator
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So Defibrillation
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Defibrillator
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The longer the duration of fibrillation, the greater the deterioration
of the myocardium, because a fibrillating heart consumes a very large
amount of oxygen.
Defibrillators deliver a brief electric shock to the heart, which enables
the heart's natural pacemaker to regain control and establish a normal
sinus rhythm .
Need a Defibrillator
Types of defibrillators
Manual external defibrillator
Automated external defibrillator (AED)
Implantable cardiac defibrillator (ICD)
Manual external defibrillator
DC defibrillator
Clinician decides what charge
has to be set, depending on
prior knowledge and experience
Shock will be delivered through
paddles applied to the patient’s
chest.
Found in hospitals &
ambulances
Automated external
defibrillator
A unit based on computer technology and designed to analyze the
heart rhythm itself, and then advise whether a shock is required or
not.
 Designed to be used by lay persons, who require little training.
 Usually limited in the treatment of VF and VT rhythms.
Usually take time ( around 5-10 secs) in diagnosing the rhythm
Can be found in places like corporate and government offices,
shopping centers, airports, restaurants, sports stadiums, schools and
universities, community centers, fitness centers and health clubs.
Automated external
defibrillator
Require self-adhesive
electrodes(pads) instead of
handheld paddles
The ECG signal acquired from
self-adhesive electrodes usually
contains less noise and has higher
quality allows faster and more
⇒
accurate analysis of the ECG
better shock decisions
⇒
“Hands off” defibrillation - safer
procedure for the operator,
especially if the operator has little
or no training
Implantable cardiac
defibrillator
An electronic device that constantly monitors heart rate and rhythm.
When it detects a very fast, abnormal rhythm, it delivers energy to the
heart muscle. This causes the heart to beat in a normal rhythm again.
Used for cardioversion, defibrillation, anti-tachycardia pacing &
bradycardia pacing.
2 parts :
a)The leads
b)The pulse generator
What are fibrillator
components?
02/03/2026 MEDICAL INSTRUMENTATION 31
How does it work?
Electronic counter-shock between to paddles or pads
Depolarises all cardiac cells and interrupts arrhythmia
Allows Sinoatrial (SA) node to recommence its
dominant role
Defibrillation is the most time critical intervention in a patient with a
shockable rhythms
How does it work?
Thoracic Impedance
Impedance is the natural resistance to the flow of
electrical current, measured in Ohms.
Impedance is determined by a number of factors, such
as:
◦ Underlying structures and pathology
◦ Paddle or adhesive pad position
How does it work?
Monophasic Defibrillation
Delivers ‘shock’ in one phase
Adult: 200J, 300J, 360J, all subsequent shocks at 360J
Child: 2J/Kg, 2J/Kg, 4J/Kg, all subsequent shocks at 4J/Kg
How does it work?
Biphasic Defibrillation
Two phases to the delivery of the ‘shock’
Adjusts ‘shock’ according to thoracic impedance
Adult: 150J, 150J, 150J
Child: 1– 2J/Kg
How does it work?
Monophasic v Biphasic Defibrillation
Peak current decreased resulting in less myocardial
damage
Simplifying electricity
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Simplifying electricity
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Simplifying electricity
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Defibrillator
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Electronics of the defibrillator
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Electronics of the defibrillator
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Power supply/ Voltage source
Step-up transformers are transformers that increase voltage
Allow the doctor to choose among different amounts of charge
This output voltage is then fed to a capacitor, which stores the
high voltage charge.
As an additional energy source, many defibrillators also have internal
rechargeable batteries.
Capacitors
Capacitors store a large amount of energy in the form of
electric charge
This stored energy is released over a short period of time
“Capacitance” describes a capacitor quantitatively
C = Q/V
A capacitor has 1 farad of capacitance if a potential difference of 1 volt
is present across its plates, when they hold a charge of 1 coulomb.
Capacitors typically have values of microfarads
Capacitors
Capacitance is directly proportional to area and indirectly proportional to the
distance between plates
Inductors
Coils of wire that produce a magnetic field
when current flows through them, prolong the
duration of current flow
Inductors generate electricity that opposes the
motion of current passing through it
This opposition is called “inductance (L)”.
 Inductors typically have values of microhenries
(µH).
02/03/2026 MEDICAL INSTRUMENTATION 47
(a) Basic circuit diagram for a capacitive–discharge type of cardiac defibrillator. (b) A
typical waveform of the discharge pulse. The actual wave shape is strongly dependent
on the values of L, C, and the torso resistance RL.
48
Defibrillators
With a circuit such as this, 50 to 100 J (W.s) is required for defibrillation,
using electrodes applied directly to the heart. When external electrodes
are used, energies as high as 400 J may be required.
The energy stored in the capacitor is given by the well-known equation
where C is the capacitance and is the voltage to which the capacitor
is charged. Capacitors used in defibrillators range from 10 to 50 μF
in capacitance. Thus we see that the voltage for a maximal energy of
400 J ranges from 2 to 9 KV, depending on the size of the capacitor.
2
2

C
E 

Defibrillator electrodes
The electrodes for external defibrillation
are metal discs about 3-5 cm in diameter
(or rectangular flat paddles 5x10 cm ) and
attached to highly insulated handle.
The size of electrodes plays an important
part in determining the chest wall
impedance which influence the efficiency
of defibrillation.
The capacitor is discharged only when the
electrodes make a good and firm contact
with the chest of the patient.
Defibrillator electrodes
For internal defibrillation
when the chest is open,
large spoon- shaped
electrodes are used.
02/03/2026 MEDICAL INSTRUMENTATION 52
Electrodes used in cardiac defibrillation
(a) A spoon-shaped internal electrode that is applied directly to the heart.
(b) A paddle-type electrode that is applied against the anterior chest wall.
Excellent contact: pulse reaches the heart=firmly placed against the patient
Well insulated =Safe to use
Defibrillator mechanism
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Defibrillator mechanism
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Defibrillator mechanism
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Defibrillator mechanism
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Defibrillator mechanism
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Annex timing circuitry
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Theory behind
defibrillator
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Defibrillator mechanism
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Defibrillator mechanism
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Defibrillator Safety
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Synchronization
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Bloc diagram of a
defibrillator
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Benchmark
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After defibrillation
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CARDIOVERTER
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1.The Problem: A patient has a fast heart rhythm caused by a problem in
the upper chambers (atria). A controlled electric shock can fix this.
2.The Danger: The heart's lower chambers (ventricles) have a brief, vulnerable
period during their resetting phase (represented by the T-wave on an ECG). If a
shock is delivered during this T-wave, it can throw the ventricles into a deadly,
chaotic rhythm (ventricular fibrillation).
3.The Solution: A synchronized defibrillator (cardioveter) is used. It has a
special safety circuit that:
1. Monitors the patient's heartbeat.
2. Identifies the safe, contracting phase of the ventricles (represented by
the R-wave on an ECG).
3. Forces the shock to be delivered only during this safe R-wave,
ensuring it occurs well before the dangerous T-wave and preventing
ventricular fibrillation.
Cardioversion
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• Cardioversion is the medical procedure (the action).
• Cardioverter is the device used to perform that procedure.
The implantable cardioverter-
defibrillator
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A cardioverter The defibrillation pulse in this case must be synchronized with the R
wave of the ECG so that it is applied to a patient shortly after the occurrence of the R
wave.
74
Cardioverters
The signal from the electrodes passes through a
switch that is normally closed. The operator can
observe the patient’s ECG to see whether the
cardio version was successful. The output is
also filtered and passed through a threshold
detector that detects the R wave. This activates
a delay circuit that delays the signal by 30 ms
and then activates a trigger circuit that opens the
switch connecting the ECG electrodes to the
amplifier. At the same time, it closes a switch
that discharges the defibrillator capacitor.
75
Defibrillator Vs. Cardioverter
Defibrillation Cardioversion
76
Defibrillator Vs. Cardioverter
Defibrillator maintenance
policy
First, The daily test procedure - 30 J self test
‑ : is a low energy
test to check the charging circuits & the integrity of cables.
Second, a weekly check - is carried out to test at higher energy
level using ECG simulator.
Third, the detailed half yearly test procedure-
‑ should be
performed by the biomedical department in a hospital
Daily low energy test
Step 1 : Put the defibrillator on Battery mode and ensure
machine
is disconnected from the AC power supply .
Turn the selector switch to ON and select Manual mode
Select leads to PADDLES/PADS
Step 2 : Ensure the universal cable is connected to the paddles
Place paddles in paddle wells
Step 3 : Select the ENERGY to 30 J
Step 4 : Press the CHARGE button
Step 5 : The unit charges to 30J, then the red LED charge
indicator illuminates and the charge tone sounds
Step 6 : Ensure DEFIB 30J READY displays on screen
Step 7 : Press and hold both paddles SHOCK buttons
Step 8 : The unit discharges. The TEST OK message displays and
the red LED turns off
Step 9 : The above TEST OK message conforms that low energy
circuits are in proper working condition
Weekly test: Defibrillator
internal discharge test
Repeat the steps from 1 to 9
Step 10 : Select ENERGY button to maximum energy level 200J displays
Step 11 : The unit charges to 200J, then the red LED charge indicator
illuminates and the charge tone sounds
Step 12 : Ensure DEFIB 200J READY displays on screen
Step 13 : Ensure the machine holds the charge for 50 seconds by giving a long
continuous sound
Step 14 : This confirms the unit is fully functional
1) Duration of VF
- the longer VF lasts, the harder it is to cure
- the quicker the better
- shock early, shock often
- likelihood of resuscitation decrease by 7-10% with every
passing minute (Ann Emerg Med. 1993;22:1652–1658 )
Factors to consider during defibrillation
2) Myocardial environment / condition
Hypoxia, acidosis, hypothermia, electrolyte imbalance,
drug toxicity – impede conversion.
DO NOT DELAY SHOCK trying to correct these problems.
Factors to consider during defibrillation
3) Heart size / Body type
Pediatric requirement lower than adult
2J /kg initial shock
4J /kg repeat shock
Higher dose (up to 10J/kg)
Or adult maximum dose
Direct size / energy relationship in adults unknown
Factors to consider during defibrillation
4) Use largest size paddles
- completely contact chest without paddles touching each
other
- In pediatric minimum of 3cm distance between pads.
NOTE :-
- Small paddles : concentrate current, burn heart.
- Large paddles : reduces current density
Factors to consider during defibrillation
5) Previous counter shock
- repeated shocks lower resistance
- give one shock at a time & then continue CPR
- subesquent shock either equal or higher energy
6) Paddle size
( as discussed before)
Factors to consider during defibrillation
7) Paddle placement
- In pacemaker / ICD
at least 12cm from generator
90 degree toAICD electrode
avoid placing pads directly over
no delay in defibrillation
- for other as described before…….
Factors to consider during defibrillation
8) Paddles – Skin interface
- only gel should be used (ECG gelly)
- cream, paste, saline pads etc.- not recommended
- gel decreases resistance to the flow of current
- never use alcohol
9) Paddle contact pressure
- firm pressure of 25 pounds
- in child <10kgs --- 3kg pressure
- in large children >10kgs --- 5kg pressure
- deflate lung, shortens the path of current
- do not lean on paddles : they slip
Factors to consider during defibrillation
Thank you!!
02/03/2026 MEDICAL INSTRUMENTATION 88

Chapter 2- Defibrillator.pptx...........

  • 1.
  • 2.
    History 1849: Ludwigg andHoffa – VF induced by electrical stimuli
  • 3.
    History 1899: Prevost andBatelli - while a weak stimulus can produce fibrillation, a stimulus of higher strength applied to the heart could arrest ventricular fibrillation and restore normal sinus rhythm.
  • 4.
  • 5.
    History 1966: Belfast Ambulancetransported physicians performed first pre-hospital defibrillation. 1969: First pre-hospital defibrillation by non physicians. 1970’s: Diack, Wellborn and Rullman developed first automated external defibrillator AED’s.
  • 6.
    Chain of Survival EarlyRecognition and Assessment • Early Access • Early CPR • Early Defibrillation • Early Advanced Cardiac Life Support
  • 7.
  • 8.
    8 Ventricular Fibrillation (VF) •Theventricles Move in a fast and disorganized way instead of contracting normally. •No blood is pumped out of the heart → this causes cardiac arrest. •⚠️Medical emergency — needs CPR and defibrillation (electric shock) immediately. No identifiable P waves, QRS complexes, or T waves Rate 150 to 500 per minute
  • 9.
    9 Ventricular Tachycardia (VT) •Theheart beats very fast (usually >180 beats per minute), but the rhythm is still organized. •Shows wide and regular QRS complexes (each heartbeat is wide and abnormal). •Blood still flows, but not effectively. •If not treated, it can turn into ventricular fibrillation.
  • 10.
    10 Tachyarrhythmia A B A tachyarrhythmia isany abnormally fast heart rhythm caused by a problem in the heart’s electrical system. It combines two ideas: •“Tachy” = fast •“Arrhythmia” = irregular or abnormal rhythm So, tachyarrhythmia = a fast and abnormal heart rhythm.
  • 11.
    11 Sinus bradycardia Sinus bradycardiais a slow but regular heart rhythm that starts from the sinus node “Sinus” → means the rhythm comes from the sinus node (normal origin). •“Bradycardia” → means slow heart rate (less than 60 beats per minute). So, sinus bradycardia = normal rhythm, just slower than usual.
  • 12.
  • 13.
    13 DEFIBRILLATORS Cardiac fibrillation isa condition where in the individual myocardial cells contract asynchronously with only very local patterns relating to the contraction of one cell and that of the next. This serious condition reduces the cardiac output to near zero, and it must be corrected as soon as possible to avoid damage to the patient and death. Electric shock to the heart can be used to reestablish a more normal cardiac rhythm. Electric machines that produce the energy to carry out this function are known as defibrillators.
  • 14.
  • 15.
    Why Cardiac Defibrillation? 02/03/2026MEDICAL INSTRUMENTATION 15
  • 16.
    Cardiac Arrest  Occulsionof the coronary artery leads to reduced blood flow  reduced blood flow leads to infarct which causes interruption of normal cardiac conduction  Infarct = VF/VT
  • 17.
    Shockable Rhythms Ventricular FibrillationVentricular Tachycardia Sinoatrial (SA) Node Atrioventricular (AV) Node
  • 18.
    What is cardiacdefibrillator 02/03/2026 MEDICAL INSTRUMENTATION 18
  • 19.
    How to use? 02/03/2026MEDICAL INSTRUMENTATION 19
  • 20.
    What is defibrillator 02/03/2026MEDICAL INSTRUMENTATION 20
  • 21.
  • 22.
  • 23.
    The longer theduration of fibrillation, the greater the deterioration of the myocardium, because a fibrillating heart consumes a very large amount of oxygen. Defibrillators deliver a brief electric shock to the heart, which enables the heart's natural pacemaker to regain control and establish a normal sinus rhythm . Need a Defibrillator
  • 24.
    Types of defibrillators Manualexternal defibrillator Automated external defibrillator (AED) Implantable cardiac defibrillator (ICD)
  • 25.
    Manual external defibrillator DCdefibrillator Clinician decides what charge has to be set, depending on prior knowledge and experience Shock will be delivered through paddles applied to the patient’s chest. Found in hospitals & ambulances
  • 26.
    Automated external defibrillator A unitbased on computer technology and designed to analyze the heart rhythm itself, and then advise whether a shock is required or not.  Designed to be used by lay persons, who require little training.  Usually limited in the treatment of VF and VT rhythms. Usually take time ( around 5-10 secs) in diagnosing the rhythm Can be found in places like corporate and government offices, shopping centers, airports, restaurants, sports stadiums, schools and universities, community centers, fitness centers and health clubs.
  • 28.
    Automated external defibrillator Require self-adhesive electrodes(pads)instead of handheld paddles The ECG signal acquired from self-adhesive electrodes usually contains less noise and has higher quality allows faster and more ⇒ accurate analysis of the ECG better shock decisions ⇒ “Hands off” defibrillation - safer procedure for the operator, especially if the operator has little or no training
  • 29.
    Implantable cardiac defibrillator An electronicdevice that constantly monitors heart rate and rhythm. When it detects a very fast, abnormal rhythm, it delivers energy to the heart muscle. This causes the heart to beat in a normal rhythm again. Used for cardioversion, defibrillation, anti-tachycardia pacing & bradycardia pacing. 2 parts : a)The leads b)The pulse generator
  • 31.
  • 32.
    How does itwork? Electronic counter-shock between to paddles or pads Depolarises all cardiac cells and interrupts arrhythmia Allows Sinoatrial (SA) node to recommence its dominant role Defibrillation is the most time critical intervention in a patient with a shockable rhythms
  • 33.
    How does itwork? Thoracic Impedance Impedance is the natural resistance to the flow of electrical current, measured in Ohms. Impedance is determined by a number of factors, such as: ◦ Underlying structures and pathology ◦ Paddle or adhesive pad position
  • 34.
    How does itwork? Monophasic Defibrillation Delivers ‘shock’ in one phase Adult: 200J, 300J, 360J, all subsequent shocks at 360J Child: 2J/Kg, 2J/Kg, 4J/Kg, all subsequent shocks at 4J/Kg
  • 35.
    How does itwork? Biphasic Defibrillation Two phases to the delivery of the ‘shock’ Adjusts ‘shock’ according to thoracic impedance Adult: 150J, 150J, 150J Child: 1– 2J/Kg
  • 36.
    How does itwork? Monophasic v Biphasic Defibrillation Peak current decreased resulting in less myocardial damage
  • 37.
  • 38.
  • 39.
  • 40.
  • 41.
    Electronics of thedefibrillator 02/03/2026 MEDICAL INSTRUMENTATION 41
  • 42.
    Electronics of thedefibrillator 02/03/2026 MEDICAL INSTRUMENTATION 42
  • 43.
    Power supply/ Voltagesource Step-up transformers are transformers that increase voltage Allow the doctor to choose among different amounts of charge This output voltage is then fed to a capacitor, which stores the high voltage charge. As an additional energy source, many defibrillators also have internal rechargeable batteries.
  • 44.
    Capacitors Capacitors store alarge amount of energy in the form of electric charge This stored energy is released over a short period of time “Capacitance” describes a capacitor quantitatively C = Q/V A capacitor has 1 farad of capacitance if a potential difference of 1 volt is present across its plates, when they hold a charge of 1 coulomb. Capacitors typically have values of microfarads
  • 45.
    Capacitors Capacitance is directlyproportional to area and indirectly proportional to the distance between plates
  • 46.
    Inductors Coils of wirethat produce a magnetic field when current flows through them, prolong the duration of current flow Inductors generate electricity that opposes the motion of current passing through it This opposition is called “inductance (L)”.  Inductors typically have values of microhenries (µH).
  • 47.
    02/03/2026 MEDICAL INSTRUMENTATION47 (a) Basic circuit diagram for a capacitive–discharge type of cardiac defibrillator. (b) A typical waveform of the discharge pulse. The actual wave shape is strongly dependent on the values of L, C, and the torso resistance RL.
  • 48.
    48 Defibrillators With a circuitsuch as this, 50 to 100 J (W.s) is required for defibrillation, using electrodes applied directly to the heart. When external electrodes are used, energies as high as 400 J may be required. The energy stored in the capacitor is given by the well-known equation where C is the capacitance and is the voltage to which the capacitor is charged. Capacitors used in defibrillators range from 10 to 50 μF in capacitance. Thus we see that the voltage for a maximal energy of 400 J ranges from 2 to 9 KV, depending on the size of the capacitor. 2 2  C E  
  • 49.
    Defibrillator electrodes The electrodesfor external defibrillation are metal discs about 3-5 cm in diameter (or rectangular flat paddles 5x10 cm ) and attached to highly insulated handle. The size of electrodes plays an important part in determining the chest wall impedance which influence the efficiency of defibrillation. The capacitor is discharged only when the electrodes make a good and firm contact with the chest of the patient.
  • 51.
    Defibrillator electrodes For internaldefibrillation when the chest is open, large spoon- shaped electrodes are used.
  • 52.
    02/03/2026 MEDICAL INSTRUMENTATION52 Electrodes used in cardiac defibrillation (a) A spoon-shaped internal electrode that is applied directly to the heart. (b) A paddle-type electrode that is applied against the anterior chest wall. Excellent contact: pulse reaches the heart=firmly placed against the patient Well insulated =Safe to use
  • 53.
  • 54.
  • 55.
  • 56.
  • 57.
  • 58.
    Annex timing circuitry 02/03/2026MEDICAL INSTRUMENTATION 58
  • 59.
  • 60.
  • 61.
  • 62.
  • 63.
  • 64.
    Bloc diagram ofa defibrillator 02/03/2026 MEDICAL INSTRUMENTATION 64
  • 65.
  • 66.
  • 67.
  • 68.
  • 69.
    CARDIOVERTER 02/03/2026 MEDICAL INSTRUMENTATION70 1.The Problem: A patient has a fast heart rhythm caused by a problem in the upper chambers (atria). A controlled electric shock can fix this. 2.The Danger: The heart's lower chambers (ventricles) have a brief, vulnerable period during their resetting phase (represented by the T-wave on an ECG). If a shock is delivered during this T-wave, it can throw the ventricles into a deadly, chaotic rhythm (ventricular fibrillation). 3.The Solution: A synchronized defibrillator (cardioveter) is used. It has a special safety circuit that: 1. Monitors the patient's heartbeat. 2. Identifies the safe, contracting phase of the ventricles (represented by the R-wave on an ECG). 3. Forces the shock to be delivered only during this safe R-wave, ensuring it occurs well before the dangerous T-wave and preventing ventricular fibrillation.
  • 70.
    Cardioversion 02/03/2026 MEDICAL INSTRUMENTATION71 • Cardioversion is the medical procedure (the action). • Cardioverter is the device used to perform that procedure.
  • 71.
  • 72.
    02/03/2026 MEDICAL INSTRUMENTATION73 A cardioverter The defibrillation pulse in this case must be synchronized with the R wave of the ECG so that it is applied to a patient shortly after the occurrence of the R wave.
  • 73.
    74 Cardioverters The signal fromthe electrodes passes through a switch that is normally closed. The operator can observe the patient’s ECG to see whether the cardio version was successful. The output is also filtered and passed through a threshold detector that detects the R wave. This activates a delay circuit that delays the signal by 30 ms and then activates a trigger circuit that opens the switch connecting the ECG electrodes to the amplifier. At the same time, it closes a switch that discharges the defibrillator capacitor.
  • 74.
  • 75.
  • 76.
    Defibrillator maintenance policy First, Thedaily test procedure - 30 J self test ‑ : is a low energy test to check the charging circuits & the integrity of cables. Second, a weekly check - is carried out to test at higher energy level using ECG simulator. Third, the detailed half yearly test procedure- ‑ should be performed by the biomedical department in a hospital
  • 77.
    Daily low energytest Step 1 : Put the defibrillator on Battery mode and ensure machine is disconnected from the AC power supply . Turn the selector switch to ON and select Manual mode Select leads to PADDLES/PADS Step 2 : Ensure the universal cable is connected to the paddles Place paddles in paddle wells Step 3 : Select the ENERGY to 30 J Step 4 : Press the CHARGE button Step 5 : The unit charges to 30J, then the red LED charge indicator illuminates and the charge tone sounds
  • 78.
    Step 6 :Ensure DEFIB 30J READY displays on screen Step 7 : Press and hold both paddles SHOCK buttons Step 8 : The unit discharges. The TEST OK message displays and the red LED turns off Step 9 : The above TEST OK message conforms that low energy circuits are in proper working condition
  • 79.
    Weekly test: Defibrillator internaldischarge test Repeat the steps from 1 to 9 Step 10 : Select ENERGY button to maximum energy level 200J displays Step 11 : The unit charges to 200J, then the red LED charge indicator illuminates and the charge tone sounds Step 12 : Ensure DEFIB 200J READY displays on screen Step 13 : Ensure the machine holds the charge for 50 seconds by giving a long continuous sound Step 14 : This confirms the unit is fully functional
  • 80.
    1) Duration ofVF - the longer VF lasts, the harder it is to cure - the quicker the better - shock early, shock often - likelihood of resuscitation decrease by 7-10% with every passing minute (Ann Emerg Med. 1993;22:1652–1658 ) Factors to consider during defibrillation
  • 81.
    2) Myocardial environment/ condition Hypoxia, acidosis, hypothermia, electrolyte imbalance, drug toxicity – impede conversion. DO NOT DELAY SHOCK trying to correct these problems. Factors to consider during defibrillation
  • 82.
    3) Heart size/ Body type Pediatric requirement lower than adult 2J /kg initial shock 4J /kg repeat shock Higher dose (up to 10J/kg) Or adult maximum dose Direct size / energy relationship in adults unknown Factors to consider during defibrillation
  • 83.
    4) Use largestsize paddles - completely contact chest without paddles touching each other - In pediatric minimum of 3cm distance between pads. NOTE :- - Small paddles : concentrate current, burn heart. - Large paddles : reduces current density Factors to consider during defibrillation
  • 84.
    5) Previous countershock - repeated shocks lower resistance - give one shock at a time & then continue CPR - subesquent shock either equal or higher energy 6) Paddle size ( as discussed before) Factors to consider during defibrillation
  • 85.
    7) Paddle placement -In pacemaker / ICD at least 12cm from generator 90 degree toAICD electrode avoid placing pads directly over no delay in defibrillation - for other as described before……. Factors to consider during defibrillation
  • 86.
    8) Paddles –Skin interface - only gel should be used (ECG gelly) - cream, paste, saline pads etc.- not recommended - gel decreases resistance to the flow of current - never use alcohol 9) Paddle contact pressure - firm pressure of 25 pounds - in child <10kgs --- 3kg pressure - in large children >10kgs --- 5kg pressure - deflate lung, shortens the path of current - do not lean on paddles : they slip Factors to consider during defibrillation
  • 87.

Editor's Notes

  • #2 1849: Written history of fibrillation and defibrillation goes back to the pioneering work of Carl Ludwig’s laboratory. In 1849, Ludwig’s student M. Hoffa was the first to witness and, most importantly, to document the onset of ventricular fibrillation, which he induced by electrical stimulus. This picture from their paper shows rapid contractions produced by electrical stimulation, which resulted in cardiac arrest.
  • #3 1899: Further experiments with “faradization” of the heart were conducted by two physiologists from University of Geneva, Switzerland, J.-L. Prevost and F. Batelli. They discovered that, while a weak stimulus can produce fibrillation, a stimulus of higher strength applied to the heart could arrest ventricular fibrillation and restore normal sinus rhythm. This discovery was made in 1899. Unfortunately, unlike discovery of contemporary electrocardiogram, defibrillation did not enjoy similar attention and success. They did this using dogs!
  • #4 Work of Carl J. Wiggers in the Department of Physiology of Western Reserve University was well known to the thoracic surgeon Claude S. Beck from the University Hospitals in Cleveland, which are adjacent to the Western Reserve University. In 1947, Dr. Beck successfully applied defibrillation therapy and saved the first human life by this method (C.S. Beck, W.H. Pritchard, H.S. Feil, Ventricular fibrillation of long duration abolished by electric shock. Jour. Amer. Med. Assoc. 135: 985, 1947). His success triggered the immediate acceptance of this method by the clinical community and started a wide front of basic and clinical research of fibrillation and defibrillation.
  • #5 Work of Carl J. Wiggers in the Department of Physiology of Western Reserve University was well known to the thoracic surgeon Claude S. Beck from the University Hospitals in Cleveland, which are adjacent to the Western Reserve University. In 1947, Dr. Beck successfully applied defibrillation therapy and saved the first human life by this method (C.S. Beck, W.H. Pritchard, H.S. Feil, Ventricular fibrillation of long duration abolished by electric shock. Jour. Amer. Med. Assoc. 135: 985, 1947). His success triggered the immediate acceptance of this method by the clinical community and started a wide front of basic and clinical research of fibrillation and defibrillation.
  • #7 Normal ecg
  • #8 VF: Chaotic irregular deflections of varying amplitude No identifiable P waves, QRS complexes, or T waves Rate 150 to 500 per minute
  • #9 VT: Shows wide and regular QRS complexes (each heartbeat is wide and abnormal). The rhythm is fast but organized — you can still see a repeating pattern. Rate: usually between 150–250 beats per minute. Wide QRS complex Tachycardia Atrioventricular AV dissociation
  • #10 Tachyarrhythmia:- AF: absence P wave, irregular rhythm Atrial flutter: saw tooth
  • #11 Sinus bradycardia
  • #36 Peak current should be less then 30Ohms
  • #44 C = capacitance of the capacitor (in farads, F) Q = charge stored on one plate (in coulombs, C) V = potential difference or voltage across the plates (in volts, V)
  • #73 The diagram shows how an AED works: It listens to the heart (ECG Electrodes), cleans up the signal (Amplifier, Filter), analyzes it (Threshold Detector) to see if a shock is needed, and then waits for human confirmation (Operator Switch) before delivering the shock (Trigger, Defibrillator, Electrodes). The AND Gate is the crucial safety feature ensuring a shock can never be given automatically without the rescuer's consent.
  • #82 1. Hypoxia A condition where tissues receive insufficient oxygen to maintain normal cellular functions. ➡️ Causes: respiratory failure, anemia, circulatory issues. ➡️ Effect: reduces energy (ATP) production in cells. 2. Acidosis An abnormal increase in acidity (low pH) of blood or body tissues. ➡️ Causes: buildup of CO₂ (respiratory acidosis) or metabolic acids (metabolic acidosis). ➡️ Effect: interferes with enzyme activity and cellular metabolism. 3. Hypothermia A state in which body temperature drops below normal (< 35°C). ➡️ Causes: exposure to cold, anesthesia, shock. ➡️ Effect: slows metabolic reactions and enzyme function. 4. Electrolyte Imbalance An abnormal level of ions (like Na⁺, K⁺, Ca²⁺, Cl⁻) in the body. ➡️ Causes: dehydration, kidney problems, medications. ➡️ Effect: disrupts nerve conduction, muscle contraction, and enzyme activity. 5. Drug Toxicity A harmful effect of excessive or inappropriate drug concentration in the body. ➡️ Causes: overdose, impaired metabolism, drug interactions. ➡️ Effect: damages organs and interferes with normal biochemical processes.
  • #86 Automatic Implantable Cardioverter-Defibrillator.