Bill Marler discussed 20 years of foodborne illness litigation and how it has impacted food policy and behavior as part of a conference at the University of Washington.
How To Lobby in an Era of Divided Government with Bill MarlerBill Marler
Food safety attorney Bill Marler's presentation at the 2011 Guiding Lights Weekend in Seattle on how to lobby for your cause in an era of divided government.
Commercial disputes are certain issues that need to be addressed as soon as possible. Consult primedm.com.au for a commercial mediation, to sort out your problems.
These are the slides for my lecture, "Interview Skills and Contract Negotiation Advice for the Fellowship or Junior Faculty Applicant", presented at the 2015 Academic Assembly of the Council of Residency Directors in Emergency Medicine (CORD-EM), Phoenix, AZ, April 2015.
How To Lobby in an Era of Divided Government with Bill MarlerBill Marler
Food safety attorney Bill Marler's presentation at the 2011 Guiding Lights Weekend in Seattle on how to lobby for your cause in an era of divided government.
Commercial disputes are certain issues that need to be addressed as soon as possible. Consult primedm.com.au for a commercial mediation, to sort out your problems.
These are the slides for my lecture, "Interview Skills and Contract Negotiation Advice for the Fellowship or Junior Faculty Applicant", presented at the 2015 Academic Assembly of the Council of Residency Directors in Emergency Medicine (CORD-EM), Phoenix, AZ, April 2015.
Food Safety History with Expert & Attorney William MarlerBill Marler
From Upton Sinclair's The Jungle to the Food Safety Modernization Act (FSMA), foodborne illness attorney William Marler gives an overview of how food safety has evolved in the US in his 2012 speech at Washington State University.
This presentation provides a good insight on the emerging consumers trends and industry response to these trends globally.
You can have a complete learning experience from lms.agribusiness.academy
2011 GMA Conference: A Bit(e) of HistoryBill Marler
Food safety attorney Bill Marler's presentation at the 2011 Grocery Manufacturers Association Foodborne Illness Litigation Conference in Chicago in which he reviews the history of foodborne illness policy in the United States and how the culmination of numerous factors ultimately lead to the passage of The Food Safety Modernization Act in 2010.
Ms. Janet Riley - How Ongoing Negative Media Coverage Is Impacting Animal Agr...John Blue
How Ongoing Negative Media Coverage Is Impacting Animal Agriculture and What We Can Do About It - Ms. Janet Riley, Senior Vice President of Public Affairs and Member Services at North American Meat Institute, from the 2015 NIAA Annual Conference titled 'Water and the Future of Animal Agriculture', March 23 - March 26, 2015, Indianapolis, IN, USA.
More presentations at http://www.trufflemedia.com/agmedia/conference/2015_niaa_water_future_animal_ag
2011 IAFP Meeting Speech with Bill MarlerBill Marler
Food safety advocate and leading food poisoning attorney Bill Marler's presentation on the progression of food safety in America at the 100th annual 2011 IAFP conference in Milwaukee, WI.
What has Changed since Upton Sinclair? A contemporary view of food safetyBill Marler
What’s behind the shiny abattoir walls of contemporary slaughterhouses? After all the regulation, safety protocols, worker initiatives, and animal rights action, we still have millions of pounds of beef recalled every year due to contamination with deadly pathogens like E. coli O157:H7 and its toxic cousins. Hundreds are sickened, many are permanently injured, and there are still deaths. Why can’t we get it right?
Food safety attorney Bill Marler will address the many challenges facing the meat industry and the consumers who eat their product. Climate, industry pressures and protocols, regulatory successes and failures, and consumer behavior all play a part. In addition to the discouraging list of what isn’t working in the system, he will present a list of proactive steps that can be taken to improve the safety of the American meat supply.
20 years after Jack in the Box - from a lawyer’s perspectiveBill Marler
It's been 20 years since "E. coli" became a household word during the Jack in the Box E. coli outbreak. Attorney Bill Marler, who represented the most seriously injured survivor of the E. coli outbreak, presents at the 2013 Food Safety Summit: What's changed and what still needs to be done to improve the safety of our nation's food supply?
The Impact of Global Commerce on Food Safety and Liability in 2009 with Bill ...Bill Marler
Marler Clark's Bill Marler speaks on the impact of global commerce on food safety and liability to the Canadian Institute of Public Health Inspectors in 2009
Dr. Matthew J. Salois - One Health, Working together to safeguard agricultureJohn Blue
One Health, Working together to safeguard agriculture - Dr. Matthew J. Salois, Elanco Animal Health, from the 2017 NIAA Annual Conference, U.S. Animal Agriculture's Future Role In World Food Production - Obstacles & Opportunities, April 4 - 6, Columbus, OH, USA.
More presentations at http://www.trufflemedia.com/agmedia/conference/2017_niaa_us_animal_ag_future_role_world_food_production
Historical overview of disease patterns- pg 83 in Pretoruis:
• Defining health and disease and illness - pg 106-116 in Pretoruis
• Stages of illness experience- pg 118 in Pretoruis
Acute hemolytic uremic syndrome (HUS).
Post-diarrheal hemolytic uremic syndrome (D+HUS) is a severe, life-threatening complication that occurs in about 10 percent of those infected with E. coli O157:H7 or other Shiga toxin-producing (Stx) E. coli (STEC).
The cascade of events leading to HUS begins with ingestion of Stx-producing E. coli (e.g., E. coli O157: H7) in contaminated food, beverages, animal to person, or person-to-person transmission. The bacteria rapidly multiply in the gut, causing inflammation and diarrhea (colitis) as they tightly bind to cells that line the large intestine. This snug attachment becomes a route for the toxin to travel from the gut into the bloodstream, where it attaches to weak receptors on white blood cells (WBCs). From there, WBCs carry the toxin to the kidneys and other organs.
To induce toxicity in target cells, Shiga toxins must first bind to specific receptors on their surface (Gb3 receptors). Organ injury is primarily a function of Gb3 receptor location and density. They are found on epithelial, endothelial, mesangial, and glomerular cells of the kidney, as well as microvascular endothelial cells of the brain and intestine. Because this attachment causes these organs to be susceptible to the toxicity of Shiga toxins, this distribution explains the involvement of the gut, kidney, and brain in STEC-associated hemolytic uremic syndrome (HUS).
Within the target organ, Shiga toxins disrupt the cellular machinery, resulting in cell injury and/or death. Within the intestine, infectious bacterial lesions cause derangements in the intestinal lining, disrupting the structure of the villi, affecting absorption in the gut, and eventually leading to watery diarrhea. Damage to the intestinal endothelium also causes mucosal/submucosal edema and, hemorrhage, introducing blood into the diarrhea.
Within the circulatory system, Shiga toxins are directly involved in platelet activation and aggregation (clot formation). The thrombotic microangiopathy that characterizes hemolytic uremic syndrome (HUS) occurs when platelet microthrombi (tiny clots) form in the walls of small blood vessels (arterioles and capillaries) causing platelet consumption. This pathologic reduction in platelets is called thrombocytopenia and is one of the hallmarks of HUS. Within the microvasculature of the kidney these clots disturb blood flow to the organ, causing acute kidney injury and kidney failure.
Hemolytic Uremic Syndrome (HUS) chris edits 7.31.23.pptxBill Marler
Acute hemolytic uremic syndrome (HUS).
Post-diarrheal hemolytic uremic syndrome (D+HUS) is a severe, life-threatening complication that occurs in about 10 percent of those infected with E. coli O157:H7 or other Shiga toxin-producing (Stx) E. coli (STEC).
The cascade of events leading to HUS begins with ingestion of Stx-producing E. coli (e.g., E. coli O157: H7) in contaminated food, beverages, animal to person, or person-to-person transmission. The bacteria rapidly multiply in the gut, causing inflammation and diarrhea (colitis) as they tightly bind to cells that line the large intestine. This snug attachment becomes a route for the toxin to travel from the gut into the bloodstream, where it attaches to weak receptors on white blood cells (WBCs). From there, WBCs carry the toxin to the kidneys and other organs.
To induce toxicity in target cells, Shiga toxins must first bind to specific receptors on their surface (Gb3 receptors). Organ injury is primarily a function of Gb3 receptor location and density. They are found on epithelial, endothelial, mesangial, and glomerular cells of the kidney, as well as microvascular endothelial cells of the brain and intestine. Because this attachment causes these organs to be susceptible to the toxicity of Shiga toxins, this distribution explains the involvement of the gut, kidney, and brain in STEC-associated hemolytic uremic syndrome (HUS).
Within the target organ, Shiga toxins disrupt the cellular machinery, resulting in cell injury and/or death. Within the intestine, infectious bacterial lesions cause derangements in the intestinal lining, disrupting the structure of the villi, affecting absorption in the gut, and eventually leading to watery diarrhea. Damage to the intestinal endothelium also causes mucosal/submucosal edema and, hemorrhage, introducing blood into the diarrhea.
Within the circulatory system, Shiga toxins are directly involved in platelet activation and aggregation (clot formation). The thrombotic microangiopathy that characterizes hemolytic uremic syndrome (HUS) occurs when platelet microthrombi (tiny clots) form in the walls of small blood vessels (arterioles and capillaries) causing platelet consumption. This pathologic reduction in platelets is called thrombocytopenia and is one of the hallmarks of HUS. Within the microvasculature of the kidney these clots disturb blood flow to the organ, causing acute kidney injury and kidney failure.
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Food Safety History with Expert & Attorney William MarlerBill Marler
From Upton Sinclair's The Jungle to the Food Safety Modernization Act (FSMA), foodborne illness attorney William Marler gives an overview of how food safety has evolved in the US in his 2012 speech at Washington State University.
This presentation provides a good insight on the emerging consumers trends and industry response to these trends globally.
You can have a complete learning experience from lms.agribusiness.academy
2011 GMA Conference: A Bit(e) of HistoryBill Marler
Food safety attorney Bill Marler's presentation at the 2011 Grocery Manufacturers Association Foodborne Illness Litigation Conference in Chicago in which he reviews the history of foodborne illness policy in the United States and how the culmination of numerous factors ultimately lead to the passage of The Food Safety Modernization Act in 2010.
Ms. Janet Riley - How Ongoing Negative Media Coverage Is Impacting Animal Agr...John Blue
How Ongoing Negative Media Coverage Is Impacting Animal Agriculture and What We Can Do About It - Ms. Janet Riley, Senior Vice President of Public Affairs and Member Services at North American Meat Institute, from the 2015 NIAA Annual Conference titled 'Water and the Future of Animal Agriculture', March 23 - March 26, 2015, Indianapolis, IN, USA.
More presentations at http://www.trufflemedia.com/agmedia/conference/2015_niaa_water_future_animal_ag
2011 IAFP Meeting Speech with Bill MarlerBill Marler
Food safety advocate and leading food poisoning attorney Bill Marler's presentation on the progression of food safety in America at the 100th annual 2011 IAFP conference in Milwaukee, WI.
What has Changed since Upton Sinclair? A contemporary view of food safetyBill Marler
What’s behind the shiny abattoir walls of contemporary slaughterhouses? After all the regulation, safety protocols, worker initiatives, and animal rights action, we still have millions of pounds of beef recalled every year due to contamination with deadly pathogens like E. coli O157:H7 and its toxic cousins. Hundreds are sickened, many are permanently injured, and there are still deaths. Why can’t we get it right?
Food safety attorney Bill Marler will address the many challenges facing the meat industry and the consumers who eat their product. Climate, industry pressures and protocols, regulatory successes and failures, and consumer behavior all play a part. In addition to the discouraging list of what isn’t working in the system, he will present a list of proactive steps that can be taken to improve the safety of the American meat supply.
20 years after Jack in the Box - from a lawyer’s perspectiveBill Marler
It's been 20 years since "E. coli" became a household word during the Jack in the Box E. coli outbreak. Attorney Bill Marler, who represented the most seriously injured survivor of the E. coli outbreak, presents at the 2013 Food Safety Summit: What's changed and what still needs to be done to improve the safety of our nation's food supply?
The Impact of Global Commerce on Food Safety and Liability in 2009 with Bill ...Bill Marler
Marler Clark's Bill Marler speaks on the impact of global commerce on food safety and liability to the Canadian Institute of Public Health Inspectors in 2009
Dr. Matthew J. Salois - One Health, Working together to safeguard agricultureJohn Blue
One Health, Working together to safeguard agriculture - Dr. Matthew J. Salois, Elanco Animal Health, from the 2017 NIAA Annual Conference, U.S. Animal Agriculture's Future Role In World Food Production - Obstacles & Opportunities, April 4 - 6, Columbus, OH, USA.
More presentations at http://www.trufflemedia.com/agmedia/conference/2017_niaa_us_animal_ag_future_role_world_food_production
Historical overview of disease patterns- pg 83 in Pretoruis:
• Defining health and disease and illness - pg 106-116 in Pretoruis
• Stages of illness experience- pg 118 in Pretoruis
Acute hemolytic uremic syndrome (HUS).
Post-diarrheal hemolytic uremic syndrome (D+HUS) is a severe, life-threatening complication that occurs in about 10 percent of those infected with E. coli O157:H7 or other Shiga toxin-producing (Stx) E. coli (STEC).
The cascade of events leading to HUS begins with ingestion of Stx-producing E. coli (e.g., E. coli O157: H7) in contaminated food, beverages, animal to person, or person-to-person transmission. The bacteria rapidly multiply in the gut, causing inflammation and diarrhea (colitis) as they tightly bind to cells that line the large intestine. This snug attachment becomes a route for the toxin to travel from the gut into the bloodstream, where it attaches to weak receptors on white blood cells (WBCs). From there, WBCs carry the toxin to the kidneys and other organs.
To induce toxicity in target cells, Shiga toxins must first bind to specific receptors on their surface (Gb3 receptors). Organ injury is primarily a function of Gb3 receptor location and density. They are found on epithelial, endothelial, mesangial, and glomerular cells of the kidney, as well as microvascular endothelial cells of the brain and intestine. Because this attachment causes these organs to be susceptible to the toxicity of Shiga toxins, this distribution explains the involvement of the gut, kidney, and brain in STEC-associated hemolytic uremic syndrome (HUS).
Within the target organ, Shiga toxins disrupt the cellular machinery, resulting in cell injury and/or death. Within the intestine, infectious bacterial lesions cause derangements in the intestinal lining, disrupting the structure of the villi, affecting absorption in the gut, and eventually leading to watery diarrhea. Damage to the intestinal endothelium also causes mucosal/submucosal edema and, hemorrhage, introducing blood into the diarrhea.
Within the circulatory system, Shiga toxins are directly involved in platelet activation and aggregation (clot formation). The thrombotic microangiopathy that characterizes hemolytic uremic syndrome (HUS) occurs when platelet microthrombi (tiny clots) form in the walls of small blood vessels (arterioles and capillaries) causing platelet consumption. This pathologic reduction in platelets is called thrombocytopenia and is one of the hallmarks of HUS. Within the microvasculature of the kidney these clots disturb blood flow to the organ, causing acute kidney injury and kidney failure.
Hemolytic Uremic Syndrome (HUS) chris edits 7.31.23.pptxBill Marler
Acute hemolytic uremic syndrome (HUS).
Post-diarrheal hemolytic uremic syndrome (D+HUS) is a severe, life-threatening complication that occurs in about 10 percent of those infected with E. coli O157:H7 or other Shiga toxin-producing (Stx) E. coli (STEC).
The cascade of events leading to HUS begins with ingestion of Stx-producing E. coli (e.g., E. coli O157: H7) in contaminated food, beverages, animal to person, or person-to-person transmission. The bacteria rapidly multiply in the gut, causing inflammation and diarrhea (colitis) as they tightly bind to cells that line the large intestine. This snug attachment becomes a route for the toxin to travel from the gut into the bloodstream, where it attaches to weak receptors on white blood cells (WBCs). From there, WBCs carry the toxin to the kidneys and other organs.
To induce toxicity in target cells, Shiga toxins must first bind to specific receptors on their surface (Gb3 receptors). Organ injury is primarily a function of Gb3 receptor location and density. They are found on epithelial, endothelial, mesangial, and glomerular cells of the kidney, as well as microvascular endothelial cells of the brain and intestine. Because this attachment causes these organs to be susceptible to the toxicity of Shiga toxins, this distribution explains the involvement of the gut, kidney, and brain in STEC-associated hemolytic uremic syndrome (HUS).
Within the target organ, Shiga toxins disrupt the cellular machinery, resulting in cell injury and/or death. Within the intestine, infectious bacterial lesions cause derangements in the intestinal lining, disrupting the structure of the villi, affecting absorption in the gut, and eventually leading to watery diarrhea. Damage to the intestinal endothelium also causes mucosal/submucosal edema and, hemorrhage, introducing blood into the diarrhea.
Within the circulatory system, Shiga toxins are directly involved in platelet activation and aggregation (clot formation). The thrombotic microangiopathy that characterizes hemolytic uremic syndrome (HUS) occurs when platelet microthrombi (tiny clots) form in the walls of small blood vessels (arterioles and capillaries) causing platelet consumption. This pathologic reduction in platelets is called thrombocytopenia and is one of the hallmarks of HUS. Within the microvasculature of the kidney these clots disturb blood flow to the organ, causing acute kidney injury and kidney failure.
31 of the Biggest Recalls in Food Safety HistoryBill Marler
In November 2018 Cheyenne Buckingham and John Harrington wrote “31 Food Recalls That Poisoned the Most People.” Here is the Marler Clark view of the same list.
I will be giving a talk on the 2011 Listeria Outbreak in the US that sickened 147 and killed at least 33. Over 100 have died from Listeria in South Africa in the last year.
2018 Royal Society for Public Health SpeechBill Marler
Later this month I will be giving a talk to the Royal Society for Public Health. The talk will be a bit of history of E. coli and the Jack in the Box case.
The Fresh Produce Safety Centre (FPSC) provides open access to actionable information for fresh produce businesses and government to continually enhance the safety and quality of fruit and vegetables.
We aim to raise awareness and to increase the collaboration between stakeholders of the Australian and New Zealand fresh produce industry, as well as establish global partnerships to better utilise the information available.
Any outbreak of foodborne illness will impact the entire fresh produce sector, therefore fresh produce safety and traceability is of the utmost importance for all levels of the supply chain including growers, packers, processors, distributors, wholesalers and retailers.
The FPSC website covers all aspects of fresh produce safety by supplying valuable information and resources, providing research with practical application, and identifying knowledge gaps in fresh produce safety.
Horticulture New Zealand Incorporated, in conjunction with Pipfruit New Zealand Incorporated and industry association partners: Process Vegetables New Zealand, Tomatoes New Zealand Incorporated and Vegetables New Zealand Incorporated, are pleased to announce our joint conference in 2016. We are delighted to host the inaugural New Zealand Horticulture Conference at the Rutherford Hotel, Nelson from the 3rd to the 5th August 2016.
The theme "Innovate from seed to plate" aims to interest and benefit the widest range of growers and horticulturalists and will have an emphasis on technology and innovations that can assist us to further improve our growth and delivery of the world's best fruit and vegetables.
New Zealand horticulture leads the world in third party assurance and HortNZ invited a selection of high calibre experts to come and talk about where we can go next to continue to satisfy consumer and trade needs.
2016 Michigan State Speech on the Risk of Criminal ProsecutionsBill Marler
2016 Michigan State Speech on the Risk of Criminal Prosecutions in Food Production. This will be an overview of cases in the last several years when CEO's and others have been prosecuted for violations of the Federal Food Drug and Cosmetic Act
2. Food Production is a Risky Business
• Competitive Markets
• Stockholder Pressures
for Increasing Profits
over Long-term Safety
• Lack of Clear Reward
For Marketing and
Practicing Food Safety
• Brand Awareness Risks
4. To Put Things in Perspective
• Microbial pathogens
in food cause an
estimated 48 million
cases of human
illness annually in
the United States
• 125,000 hospitalized
• Cause up to 3,000
deaths
5. Estimates Differ From Actual Counts
• Annual E. coli O157:H7
Estimates:
– 62,000 illnesses
– 1,800 hospitalizations
– 52 deaths
7. Incubation Periods of
Common Foodborne Pathogens
PATHOGEN INCUBATION PERIOD
Staphylococcus aureus 1 to 8 hours, typically 2 to 4 hours.
Campylobacter 2 to 7 days, typically 3 to 5 days.
E. coli O157:H7 1 to 10 days, typically 2 to 5 days.
Salmonella 6 to 72 hours, typically 18-36 hours.
Shigella 12 hours to 7 days, typically 1-3 days.
Hepatitis A 15 to 50 days, typically 25-30 days.
Listeria 3 to 70 days, typically 21 days
Norovirus 24 to 72 hours, typically 36 hours.
19. Magic Moment?
• 2006 Spinach
205 sickened
and 5 deaths
• 2007 Peanut Butter
746 sickened
and 3 years
of product recalled
• House and Senate
party switch
20. Well, Not Quite So Fast
• 2007 Hamburger
paralyzed dancer –
Front Page of New
York Times and a
Pulitzer Prize
• 2009 Cookie Dough
mother of six
hospitalized 2 years –
Front Page Washington
Post
21. What About Industry?
• Tomato,
errr, Pepper
Outbreak
• Peanut Butter
Again - $1
Billion in
Recall and
Economic
Losses
22. 2009 – The Magic Moment
Consumers and Industry Coming Together
25. Well, Not Quite Yet
"I would not identify it as
something that will necessarily
be zeroed out, but it is quite
possible it will be scaled back if
it is significant overreach," said
Rep. Kingston, who is likely to
become chairman of the
subcommittee when Republicans
assume control of the House in
January.
"We still have a food supply
that's 99.99 percent safe," Rep.
Kingston said in an interview.
"No one wants anybody to get
sick, and we should always
strive to make sure food is safe.
But the case for a $1.4 billion
expenditure isn't there."