Gastric & Duodenal Release of Cholecystokinin
•
1 like•356 views
Presented at the 2005 Conference of the International Society for Neuroscience.
![Gastric & Duodenal Determinants of Blunted Cholecystokinin Release in Patients with Bulimia Nervosa (BN) ,[object Object],[object Object],[object Object],[object Object],[object Object]](https://image.slidesharecdn.com/sfnguss2005-12740556034495-phpapp01/85/gastric-duodenal-release-of-cholecystokinin-1-320.jpg)
![Bulimia Nervosa (BN), Binge Eating & CCK ,[object Object],[object Object],[object Object]](data:image/gif;base64,R0lGODlhAQABAIAAAAAAAP///yH5BAEAAAAALAAAAAABAAEAAAIBRAA7)
Recommended
More Related Content
What's hot
What's hot (20)
Similar to Gastric & Duodenal Release of Cholecystokinin
Similar to Gastric & Duodenal Release of Cholecystokinin (20)
Gastric & Duodenal Release of Cholecystokinin
- 4. Significantly Blunted CCK Release in BN Devlin et al 1997
- 7. Study Design: 2x2 Repeat measures on treatment: delivery Group: Treatment: nutrient delivery
- 11. Oral CCK Release (+/- SE) * Indicates times at which plasma CCK differed signficantly between groups * * * *
- 12. Duodenal CCK Release (+/- SE) * Indicates times at which plasma CCK differed signficantly between groups * * *
- 13. CCK: 15 min into/after nutrient delivery Different letters above bars indicate significant differences between means
- 14. Plasma CCK: 60-90 min into/after delivery Different letters above bars indicate significant differences between means a a b b
Editor's Notes
- Meal patterns vary among individuals yet most individuals maintain stable body weights over long intervals. The explanation is that energy homeostasis is accomplished via control of meal size. A key concept regards the integration of signals generated acutely in response to food being eaten with signals that reflect the current state of energy balance (adipose stores). Meal-related (i.e., "satiety" signals) are typified by cholecystokinin (CCK), and adiposity signals are typified by leptin and insulin.