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Abnormal valve
function
•Valve Stenosis
•Obstruction to valve flow during that phase of
the cardiac cycle when the valve is normally
open.
•Hemodynamic hallmark -“pressure gradient”
•Valve Regurgitation, insufficiency, incompetence
•Inadequate valve closure → reverse flow of the
blood, back leakage
•Combined – a single valve can be both stenotic
and
regurgitant; combinations of valve lesions can
coexist
•Single disease process
•Different disease processes
Diagnos
is
• History
• Physical examination
• ECG
• Chest x-ray
• Echocardiography
•Invasive evaluation, CT,
MRI
Histor
y
•Other known heart diseses
• Congenital heart disease,
bicuspid aortic valve
• Ischemic heart disease
• Rheumatic fever
• Hypertrophic/dilated
cardiomyopathy
• Aortic diseases –
Marfans,Ehlers-Danlos
Histor
y
• Generally: symptoms of heart failure
and low cardiac output
• Breathlessness
• Chest pain or dyscomfort
• Syncope
• Fatigue
• Peripheral or pulmonary oedema
• Palpitations
Physical
examination
• MURMURS!!!
• Periferal oedema
• Lung crackles
• Elevated JVP
• Displaced apex
beat, irregular
heart beat…
Hundreds of eponymous signs from
past millenium (↓importance in
daily routine, ↑importance for
passing exam)
EC
G
• Not specific
• Findings might be caused or altered by other
concomitant heart disease (hypertensive heart
disease, ischemic heart disease)
• Left ventricular hypertrophy (aortic valve
disease)
• Left atrial enlargement (mainy MS, but any left
heart valve disease)
• Atrial fibrilation
• Bundle branch block
• Arrytmias (atrial fibrilation, ectopic beats)
Chest x-ray in valvular
disease
• Different heart shapes in different valvular
heart diseses, ↓specificity, ↓significance
• Cardiomegaly, pulmonary congestion
• Widened mediastinum
• Valve calcifications, prosthetic valves
Echocardiogra
phy
• Mainstay of valve
disease diagnosis
and follow-up
• Allows real-time measurement
of chamber and wall
diameters, ejection fraction
assessment and functional
valve evaluation
• Easily avaiable and repeated
• Essential in acute valve
disease diagnosis
• No radiation harm
• Trans-esophageal
echocardiography avaiable for
Invasive evaluation, CT,
MRI
• Methods usualy used for uncertain cases or repeat
cardiac surgery / percutaneous inteventions planning
• Angiography to assess regurgitation severity – direct
transcatheter contrast medium administration into
heart chambers – aortography, ventriculography
• Hemodynamic measurment – measuring of
intracardial pressures and gradients
• CT aortography – method of choice in aortic
dissection diagnosis
• CMRI – very precise evaluation of cardiac tissues and
function, but expensive, low avaiability, long
examination time
drtoufiq19711@yahoo.c
drtoufiq19711@yahoo.c
Pathophysiolo
gy
Tricuspid valve
disesease
•Tricuspid regurgitation
• Is usually secondary – due to right ventricle dilation and failure as
a result of pumonary hypertension (the most common cause of
right heart failure is left heart failure) or volume overload (left-right
shunt abnormalities)
• Cor pulmonale – right ventricle failure in pulmonary disease
•Primary:
•Infective endocarditis – in i.v. abusers
•Other are rare - Carcinoid, rheumatic, Ebstein anomaly –
apical displacement of septal and posterior leaflet of
tricuspid valve →
„atrialization“ of a portion of the morphologic right ventricle
•Symptoms of right heart failure – periferal oedema, elevated
JVP,
hepatomegaly, ascites…
•Symptoms treated by diuretics, surgical treatment is only
Tricuspid stenosis is a
rare condition, whereas
tricuspid regurgitation
is more common,
especially in its
secondary form.
•For appropriate
management,
secondary tricuspid
regurgitation has to
be clearly
distinguished from
primary tricuspid
Similar to mitral regurgitation,
primary tricuspid regurgitation
requires intervention sufficiently
early to avoid secondary
damage of the RV, which is
associated with poor outcome.
•Secondary tricuspid
regurgitation should be liberally
treated at the time of left-sided
valve surgery.
•Consideration of isolated surgery of
secondary tricuspid regurgitation after
previous left-sided valve surgery
requires comprehensive assessment
of the underlying disease, pulmonary
haemodynamics and RV function.
Tricuspid valve
disesease
drtoufiq19711@yahoo.co
tricuspid valve.pptx
tricuspid valve.pptx
tricuspid valve.pptx
tricuspid valve.pptx

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tricuspid valve.pptx

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  • 5. Abnormal valve function •Valve Stenosis •Obstruction to valve flow during that phase of the cardiac cycle when the valve is normally open. •Hemodynamic hallmark -“pressure gradient” •Valve Regurgitation, insufficiency, incompetence •Inadequate valve closure → reverse flow of the blood, back leakage •Combined – a single valve can be both stenotic and regurgitant; combinations of valve lesions can coexist •Single disease process •Different disease processes
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  • 11. Diagnos is • History • Physical examination • ECG • Chest x-ray • Echocardiography •Invasive evaluation, CT, MRI
  • 12. Histor y •Other known heart diseses • Congenital heart disease, bicuspid aortic valve • Ischemic heart disease • Rheumatic fever • Hypertrophic/dilated cardiomyopathy • Aortic diseases – Marfans,Ehlers-Danlos
  • 13. Histor y • Generally: symptoms of heart failure and low cardiac output • Breathlessness • Chest pain or dyscomfort • Syncope • Fatigue • Peripheral or pulmonary oedema • Palpitations
  • 14. Physical examination • MURMURS!!! • Periferal oedema • Lung crackles • Elevated JVP • Displaced apex beat, irregular heart beat… Hundreds of eponymous signs from past millenium (↓importance in daily routine, ↑importance for passing exam)
  • 15. EC G • Not specific • Findings might be caused or altered by other concomitant heart disease (hypertensive heart disease, ischemic heart disease) • Left ventricular hypertrophy (aortic valve disease) • Left atrial enlargement (mainy MS, but any left heart valve disease) • Atrial fibrilation • Bundle branch block • Arrytmias (atrial fibrilation, ectopic beats)
  • 16. Chest x-ray in valvular disease • Different heart shapes in different valvular heart diseses, ↓specificity, ↓significance • Cardiomegaly, pulmonary congestion • Widened mediastinum • Valve calcifications, prosthetic valves
  • 17. Echocardiogra phy • Mainstay of valve disease diagnosis and follow-up • Allows real-time measurement of chamber and wall diameters, ejection fraction assessment and functional valve evaluation • Easily avaiable and repeated • Essential in acute valve disease diagnosis • No radiation harm • Trans-esophageal echocardiography avaiable for
  • 18. Invasive evaluation, CT, MRI • Methods usualy used for uncertain cases or repeat cardiac surgery / percutaneous inteventions planning • Angiography to assess regurgitation severity – direct transcatheter contrast medium administration into heart chambers – aortography, ventriculography • Hemodynamic measurment – measuring of intracardial pressures and gradients • CT aortography – method of choice in aortic dissection diagnosis • CMRI – very precise evaluation of cardiac tissues and function, but expensive, low avaiability, long examination time
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  • 23. Tricuspid valve disesease •Tricuspid regurgitation • Is usually secondary – due to right ventricle dilation and failure as a result of pumonary hypertension (the most common cause of right heart failure is left heart failure) or volume overload (left-right shunt abnormalities) • Cor pulmonale – right ventricle failure in pulmonary disease •Primary: •Infective endocarditis – in i.v. abusers •Other are rare - Carcinoid, rheumatic, Ebstein anomaly – apical displacement of septal and posterior leaflet of tricuspid valve → „atrialization“ of a portion of the morphologic right ventricle •Symptoms of right heart failure – periferal oedema, elevated JVP, hepatomegaly, ascites… •Symptoms treated by diuretics, surgical treatment is only
  • 24. Tricuspid stenosis is a rare condition, whereas tricuspid regurgitation is more common, especially in its secondary form. •For appropriate management, secondary tricuspid regurgitation has to be clearly distinguished from primary tricuspid Similar to mitral regurgitation, primary tricuspid regurgitation requires intervention sufficiently early to avoid secondary damage of the RV, which is associated with poor outcome. •Secondary tricuspid regurgitation should be liberally treated at the time of left-sided valve surgery. •Consideration of isolated surgery of secondary tricuspid regurgitation after previous left-sided valve surgery requires comprehensive assessment of the underlying disease, pulmonary haemodynamics and RV function. Tricuspid valve disesease drtoufiq19711@yahoo.co