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PERICARIAL DISEASES
OBJECTIVES
• Review of anatomy
• Define pericarditis
• Etiology & pathophysiology
• Clinical manifestations
• Diagnostic evaluation studies
• Medical and surgical management
• complications
• Nursing management
INTRODUCTION
• Pericardium • The pericardium is
a sac made up of outer fibrous
pericardium – inner serous
pericardium (parietal & visceral)
separated by a (potential) space,
the pericardial cavity.
• the pericardial cavity contains 15
to 50 mL of an ultra filtrate of
plasma.
Functions of Pericardium
1. Stabilization of the heart within the thoracic cavity --
limiting the heart’s motion.
2. Protection of the heart from mechanical trauma and
infection.
3. The pericardial fluid functions as a lubricant and
decreases friction of cardiac surface.
4. Prevention of excessive dilation of heart. (e.g. acute aortic
or mitral regurgitation).
PERI CARDIAL DISEASES
• Introduction
• Acute pericarditis
• Constrictive pericarditis
• Pericardial effusion
• Cardiac tamponade
DEFINITION
• Pericarditis: is a
condition caused by
inflammation of the
pericardial sac.
• Pericarditis can be
acute or chronic.
• Acute pericarditis occurs due to
complications of infections or even as a
result of heart attack.
• Chronic pericarditis is less common form of
constrictive pericarditis. It can also develop
as primary illness / as a result of medical
and surgical disorders.
CHRONIC PERICARDITIS
Chronic pericarditis occurs when the layers
adhere to each other causing fibrosis of the
pericardial sac.
It restricts movement of the heart. So the
fibrosis pericardium tightens the heart
decreasing cardiac filling and output.
It leads the patient reports the symptoms of
heart failure.
ETIOLOGY
ETIOLOGICAL CLASSIFICATION - TUMOR
• T = Trauma, Tumour , Neoplasm
• U = Uraemia
• M = Myocardial infarction (acute)
• Medications (hydralazine)
• O = Other infections (viral, bacterial, fungal, TB)
• R = Rheumatoid arthritis, autoimmune disorder/
hypersensitivity
PATHOPHYSIOLOGY
Etiology
Inflammatory
response
Influx of neutrophils & other
chemical mediators
Change the
permeability of
pericardial vascularity
Pericardial
inflammation &
edema
Restriction of heart
motion and pain
with breathing
CLINICAL MANIFESTATIONS
• Chest pain beneath the clavicle, in the neck
region worsens with deep inspiration, relieved
with sitting or leaning forward.
• Mild fever, chills and night sweats.
• Malaise, myalgia
• Dyspnea due to constriction or cardiac
tamponade
DIAGNOSTIC FINDINGS
• Patient history
• Physical examination ( pericardial friction
rub by auscultation).
• ECG (ST changes)
• Chest X – ray
• ECHO
• Blood test – Elevated WBC,CRP,ESR
• Pericardial biopsy - analysis
MANAGEMENT
• Treat underlying cause
• Analgesic agents – codeine 15 – 30 mg
• Anti –inflammatory agents – Aspirin,
NSAID – indomethacin 25 -50 mg
• Corticosteroids – (Prednisolone)
symptomatically effective.
CONSTRICTIVE
PERICARDITIS
• Constrictive pericarditis is an another type
which caused thickened pericardium.
ETIOLOGY
• Idiopathic
• Post cardiac surgery
• Post radiation therapy
• Post infectious – tuberculus /purulent
pericarditis
• Others – malignancy, trauma, uremic
pericarditis, asbestosis.
PATHOPHYSIOLOGY
CONSTRICTIVE PERICARDITIS
A thickened, fibrotic pericardium forms a
non-compliant shell around the heart.
Ventricles are encased by thick
pericardium
It prevents the heart from expanding when
blood enters in it.
It interferes ventricular filling.
CONSTRICTIVE PERICARDITIS
• Due to impaired ventricular filling –
fatigue, muscle wasting, weight loss,
reduced cardiac output.
• Reduced cardiac output
• Hepatic congestion, peripheral edema
• Ascitis, anasarca and
• Cardiac cirrhosis
DIAGNOSTIC EVALUATION
• ECG – Flattened / inverted T waves
• Chest X – Ray (Pericardial
calcification)
• ECHO cardiogram ( Pericardial
thickening)
PERICARDIAL EFFUSION
Accumulation of fluid between the
visceral and parietal layers of serous
pericardium.
•Serous – Transudative (CHF, Renal failure)
•Suppurative – Pyogenic infection.
•Hemorrhagic – occurs with any type of
pericarditis – especially with infections
and malignancies.
ETIOLOGY
• Inflammation from infection, immunologic
process.
• Trauma causing bleeding in pericardial space.
• Noninfectious conditions such as:
a. increase in hydrostatic pressure e.g.
congestive heart failure.
b. increase in capillary permeability e.g.
hypothyroidism.
ETIOLOGY…………
c. decrease in plasma oncotic pressure
e.g. cirrhosis
• Decreased drainage of pericardial fluid
due to obstruction of thoracic duct as a
result of malignancy or damage during
surgery.
CLINICAL FEATURES
• Usually asymptomatic
• Can have signs of compression -
dyspnoea, dysphagia, hoarseness of
voice, hiccoughs, nausea
• Signs : muffled heart sounds paradoxically
reduced intensity of rub
• Chest x ray - distinguish
between pericardial
effusion and cardiomegaly.
– Water bottle sign
Electrocardiogram
Echocardiogram
MANAGEMENT
• Diuretics & salt restriction
• Digoxin for sinus tachycardia to slow the
ventricular rate.
• Beta blockers & calcium channel blockers
Surgical management
• Pericardiectomy
NURSING ASSESMENT
• Physical assessment to obtain the base
line data.
• Vital signs.
• Assess patient in various positions in order
to detect pain whether it is influenced by
respiratory movements, coughing and
swallowing.
NURSING ….
• Assess Heart and Lung Sounds
May hear a pericardial friction rub,
muffled heart sounds, or extra sounds because of
the pressure being placed on the hear
• Assess and Address Oral Hygiene
There is a significant connection between
oral health and pericarditis. Bacteria can travel to
the heart easily from the oral cavity. Patients
should brush their teeth twice daily to prevent
complications.
NURSING INTERVENTIONS
• Pain management – elevate head end to
45 deg.
• Anti inflammatory drugs as prescribed.
• Monitor complications
NURSING DIAGNOSIS
• Ineffective breathing pattern related to chest pain.
• Altered thermoregulation , hyperthermia, related to infection
and inflammation as evidenced by temp.
• Pain related to inflammation of the pericardium.
• Risk for cardiogenic shock related to decreased cardiac
output.
• Ineffective tissue perfusion related to decrease blood flow.
• Anxiety related to therapeutic interventions and uncertainty
of prognosis.
NURSING MANAGEMENT
• Explain every procedure to the patient in order to
get consent, cooperation and array anxiety.
• Position patient in semi fowlers to relieve pain
and allow expansion of chest for effective
breathing.
• Explain the pathophysiology to the patient.
• Encourage gradual increase activity.
• Monitor vital signs closely to detect infections.
NURSING MANAGEMENT
• Educate patient on signs and symptoms of
infection.
• Educate patient to inform the need
prophylactic antibiotics.
• No dental procedures for at least 6 months.
COMPLICATIONS
• Pericarditis progresses to heart failure.
• Pericardial effusion fluid collection in
pericardial space.
• Cardiac tamponade - it develops when
pericardial effusion increases.
REFERENCES
• Smeltzer S.C, Bare B.G. & Hinke J.L
(1999). Brunner & Suddarth’s textbook of
medical surgical nursing. (9th E.D).
Philadelphia: J.B. Lippincott
• Linton A.D (2007) Introduction to medical
– surgical nursing. (4th ed) Saunders
Elsevier.
THANK YOU

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Pericardial diseases part i

  • 2. OBJECTIVES • Review of anatomy • Define pericarditis • Etiology & pathophysiology • Clinical manifestations • Diagnostic evaluation studies • Medical and surgical management • complications • Nursing management
  • 3. INTRODUCTION • Pericardium • The pericardium is a sac made up of outer fibrous pericardium – inner serous pericardium (parietal & visceral) separated by a (potential) space, the pericardial cavity. • the pericardial cavity contains 15 to 50 mL of an ultra filtrate of plasma.
  • 4. Functions of Pericardium 1. Stabilization of the heart within the thoracic cavity -- limiting the heart’s motion. 2. Protection of the heart from mechanical trauma and infection. 3. The pericardial fluid functions as a lubricant and decreases friction of cardiac surface. 4. Prevention of excessive dilation of heart. (e.g. acute aortic or mitral regurgitation).
  • 5. PERI CARDIAL DISEASES • Introduction • Acute pericarditis • Constrictive pericarditis • Pericardial effusion • Cardiac tamponade
  • 6. DEFINITION • Pericarditis: is a condition caused by inflammation of the pericardial sac. • Pericarditis can be acute or chronic.
  • 7. • Acute pericarditis occurs due to complications of infections or even as a result of heart attack. • Chronic pericarditis is less common form of constrictive pericarditis. It can also develop as primary illness / as a result of medical and surgical disorders.
  • 8. CHRONIC PERICARDITIS Chronic pericarditis occurs when the layers adhere to each other causing fibrosis of the pericardial sac. It restricts movement of the heart. So the fibrosis pericardium tightens the heart decreasing cardiac filling and output. It leads the patient reports the symptoms of heart failure.
  • 9. ETIOLOGY ETIOLOGICAL CLASSIFICATION - TUMOR • T = Trauma, Tumour , Neoplasm • U = Uraemia • M = Myocardial infarction (acute) • Medications (hydralazine) • O = Other infections (viral, bacterial, fungal, TB) • R = Rheumatoid arthritis, autoimmune disorder/ hypersensitivity
  • 10. PATHOPHYSIOLOGY Etiology Inflammatory response Influx of neutrophils & other chemical mediators Change the permeability of pericardial vascularity Pericardial inflammation & edema Restriction of heart motion and pain with breathing
  • 11. CLINICAL MANIFESTATIONS • Chest pain beneath the clavicle, in the neck region worsens with deep inspiration, relieved with sitting or leaning forward. • Mild fever, chills and night sweats. • Malaise, myalgia • Dyspnea due to constriction or cardiac tamponade
  • 12. DIAGNOSTIC FINDINGS • Patient history • Physical examination ( pericardial friction rub by auscultation). • ECG (ST changes) • Chest X – ray • ECHO • Blood test – Elevated WBC,CRP,ESR • Pericardial biopsy - analysis
  • 13. MANAGEMENT • Treat underlying cause • Analgesic agents – codeine 15 – 30 mg • Anti –inflammatory agents – Aspirin, NSAID – indomethacin 25 -50 mg • Corticosteroids – (Prednisolone) symptomatically effective.
  • 14. CONSTRICTIVE PERICARDITIS • Constrictive pericarditis is an another type which caused thickened pericardium.
  • 15. ETIOLOGY • Idiopathic • Post cardiac surgery • Post radiation therapy • Post infectious – tuberculus /purulent pericarditis • Others – malignancy, trauma, uremic pericarditis, asbestosis.
  • 16. PATHOPHYSIOLOGY CONSTRICTIVE PERICARDITIS A thickened, fibrotic pericardium forms a non-compliant shell around the heart. Ventricles are encased by thick pericardium It prevents the heart from expanding when blood enters in it. It interferes ventricular filling.
  • 17. CONSTRICTIVE PERICARDITIS • Due to impaired ventricular filling – fatigue, muscle wasting, weight loss, reduced cardiac output. • Reduced cardiac output • Hepatic congestion, peripheral edema • Ascitis, anasarca and • Cardiac cirrhosis
  • 18. DIAGNOSTIC EVALUATION • ECG – Flattened / inverted T waves • Chest X – Ray (Pericardial calcification) • ECHO cardiogram ( Pericardial thickening)
  • 19. PERICARDIAL EFFUSION Accumulation of fluid between the visceral and parietal layers of serous pericardium. •Serous – Transudative (CHF, Renal failure) •Suppurative – Pyogenic infection. •Hemorrhagic – occurs with any type of pericarditis – especially with infections and malignancies.
  • 20. ETIOLOGY • Inflammation from infection, immunologic process. • Trauma causing bleeding in pericardial space. • Noninfectious conditions such as: a. increase in hydrostatic pressure e.g. congestive heart failure. b. increase in capillary permeability e.g. hypothyroidism.
  • 21. ETIOLOGY………… c. decrease in plasma oncotic pressure e.g. cirrhosis • Decreased drainage of pericardial fluid due to obstruction of thoracic duct as a result of malignancy or damage during surgery.
  • 22. CLINICAL FEATURES • Usually asymptomatic • Can have signs of compression - dyspnoea, dysphagia, hoarseness of voice, hiccoughs, nausea • Signs : muffled heart sounds paradoxically reduced intensity of rub
  • 23. • Chest x ray - distinguish between pericardial effusion and cardiomegaly. – Water bottle sign Electrocardiogram Echocardiogram
  • 24. MANAGEMENT • Diuretics & salt restriction • Digoxin for sinus tachycardia to slow the ventricular rate. • Beta blockers & calcium channel blockers Surgical management • Pericardiectomy
  • 25. NURSING ASSESMENT • Physical assessment to obtain the base line data. • Vital signs. • Assess patient in various positions in order to detect pain whether it is influenced by respiratory movements, coughing and swallowing.
  • 26. NURSING …. • Assess Heart and Lung Sounds May hear a pericardial friction rub, muffled heart sounds, or extra sounds because of the pressure being placed on the hear • Assess and Address Oral Hygiene There is a significant connection between oral health and pericarditis. Bacteria can travel to the heart easily from the oral cavity. Patients should brush their teeth twice daily to prevent complications.
  • 27. NURSING INTERVENTIONS • Pain management – elevate head end to 45 deg. • Anti inflammatory drugs as prescribed. • Monitor complications
  • 28. NURSING DIAGNOSIS • Ineffective breathing pattern related to chest pain. • Altered thermoregulation , hyperthermia, related to infection and inflammation as evidenced by temp. • Pain related to inflammation of the pericardium. • Risk for cardiogenic shock related to decreased cardiac output. • Ineffective tissue perfusion related to decrease blood flow. • Anxiety related to therapeutic interventions and uncertainty of prognosis.
  • 29. NURSING MANAGEMENT • Explain every procedure to the patient in order to get consent, cooperation and array anxiety. • Position patient in semi fowlers to relieve pain and allow expansion of chest for effective breathing. • Explain the pathophysiology to the patient. • Encourage gradual increase activity. • Monitor vital signs closely to detect infections.
  • 30. NURSING MANAGEMENT • Educate patient on signs and symptoms of infection. • Educate patient to inform the need prophylactic antibiotics. • No dental procedures for at least 6 months.
  • 31. COMPLICATIONS • Pericarditis progresses to heart failure. • Pericardial effusion fluid collection in pericardial space. • Cardiac tamponade - it develops when pericardial effusion increases.
  • 32. REFERENCES • Smeltzer S.C, Bare B.G. & Hinke J.L (1999). Brunner & Suddarth’s textbook of medical surgical nursing. (9th E.D). Philadelphia: J.B. Lippincott • Linton A.D (2007) Introduction to medical – surgical nursing. (4th ed) Saunders Elsevier.