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Complications of
Chronic Otitis Media
- Dr. Alka Kapil
( Assistant Professor,
Dept. of ENT & Head Neck Surge
Relations of Mastoid Antrum
 Superiorly : Tegmen antri/Dural plate
 Posteriorly : Sinus plate
 Medially : Solid angle and posterior fossa dural plate
 Laterally : Skin ,subcutaneous tissue & Mac Ewan’s triangle
 Anteriorly : posterior wall of middle ear & facial canal
Pathways of spread of infection
INTRATEMPORAL COMPLICATIONS
Mastoiditis
 When infection spreads from the mucosal lining the mastoid air cells to
involve the bony walls of the mastoid air cell system it is called Mastoiditis.
 Can be :
Acute mastoiditis or
Chronic mastoiditis or
Coalescent mastoiditis or
Masked mastoiditis
 Acute inflammation of the mastoid bone
 Streptococcus pneumoniae > β haemolytic streptococcus
 Pathology :
1. Production of pus under tension
2. Hyperaemic decalcification and osteoclastic resorption of bony walls
Acute Mastoiditis
Accumulation of pus under tension
hyperaemia of mucosal lining
venous stasis
local acidosis and dissolution of calcium from the bone
hyperaemic decalcification
destruction of bony septa
Coalescent mastoiditis
Clinical presentations Signs
i. Mastoid tenderness
ii. Ear discharge
iii. Reservoir sign & Light house sign
iv. Tympanic membrane perforation
v. Swelling of mastoid/ ironed out appearance
vi. Hearing loss
vii. Fever
viii. Sagging of posterosuperior wall of EAC
Symptoms
i. Pain behind the ear
ii. Fever
iii. Ear discharge
BEZOLD ABSCESS
Abscesses in Relation to Mastoid Infection
(A) Abscesses in relation to mastoid: (1) postauricular, (2) zygomatic and (3) Bezold abscess
(B) Citelli, postauricular and Bezold abscesses seen from behind.
1. Blood culture to find causative organisms
2. Routine hematological investigations ( CBC & ESR)
3. X-ray mastoids
4. Imaging studies : CT scan Temporal bone - MRI
5. Culture and sensitivity of ear swab
Investigations
Treatment
1. Intravenous culture directed antibiotics
2. Myringotomy if TM is intact and bulging with pus in tension
3. ± Mastoid exploration
done if : (a) Subperiosteal abscess
(b) Sagging of posterosuperior meatal wall.
(c) Positive reservoir sign
(d) No change in condition of patient or it worsens in spite of adequate medical
treatment for 48 h.
(e) Mastoiditis, leading to complications
4. Drainage of neck abscesses in case they are present
Petrositis
 Spread of infection from middle ear and mastoid to the petrous part of temporal
bone is called petrositis
Clinical presentations
Gradenigo syndrome is the classical presentation, and consists of a triad of
(i) external rectus palsy (VIth nerve palsy)
(ii) deep-seated ear or retro-orbital pain (Vth nerve involvement) &
(iii) persistent ear discharge
1. Routine blood investigations
2. Blood culture to find the
causative organisms
3. X-ray mastoids
4. Imaging studies : CT scan & MRI
Investigations
1. Intravenous culture directed antibiotics
2. Mastoid exploration / Petrous apicectomy
Treatment
Facial palsy
Labyrinthitis
CIRCUMSCRIBED
LABYRINTHITIS
DIFFUSE SEROUS
LABYRINTHITIS
DIFFUSE SUPPURATIVE
LABYRINTHITIS
Thinning or erosion of bony capsule
of labyrinth
diffuse intralabyrinthine inflammation
without pus
formation
diffuse pyogenic infection of the
labyrinth
exposure
of a localised part of membranous
labyrinth due to
erosion of the overlying bone
toxins from middle ear
infection
complete destruction of
membranous labyrinth
(Labyrinthitis OSSIFICANS )
reversible condition if treated early permanent loss of vestibular and
cochlear functions
Vertigo + Vertigo + Vertigo & imbalance +
Partial SNHL + Parital SNHL + SNHL +
Fistula test + nystagmus is
towards the same side of lesion
nystagmus towards the normal side
Mastoid exploration with closure of
fistula
Antibiotics, Labyrinthine sedatives,
mastoid exploration
Same as serous
INTRACRANIAL COMPLICATIONS
Meningitis
 It is inflammation of leptomeninges (pia and arachnoid) usually with bacterial invasion of
CSF in subarachnoid space
 MC organism causing otogenic meningitis is Streptococcus pneumoniae > H. influenzae
1.There is rise in temperature (102-104 °F) often with chills and rigors
2. Headache
3. Neck rigidity
4. Photophobia and mental irritability
5. Nausea and vomiting (sometimes projectile)
6. Drowsiness which may progress to delirium or coma
7. Cranial nerve palsies and hemiplegia
Clinical presentations
 Signs :
(i) Neck rigidity
(ii) positive Brudzinski’s sign (flexion of neck causes flexion of hip and knee)
(iii) positive Kernig’s sign (extension of leg with thigh flexed on abdomen causing pain)
(iv) tendon reflexes exaggerated initially but later sluggish or absent
(v) papilloedema
Investigations
1. CT scan & MRI
2. Lumber Puncture & CSF examination
CSF finding - turbid
- cell count is raised
- predominance of polymorphs
- protein level is raised
- sugar & chlorides reduced
Treatment
Mainly medical management
Lateral Sinus Thrombophlebitis
 Also called as Sigmoid Sinus Thrombophlebitis
 It is an inflammation of inner wall of lateral venous sinus with formation of intrasinus thrombus
 Pathology :
Pathology
Clinical presentations
i. Picket Fence fever pattern (diurnal temperature spikes that exceed 103°F/39.4°C)
ii. Headache
iii. Progressive anemia and emaciation
iv. Griesinger’s sign : oedema over mastoid d/t thrombosis of mastoid emissary vein
v. Papilloedema
vi. Tobey-ayer test
vii. Crowe-beck test
viii. Tenderness along jugular vein
Investigations
1. Blood smear to rule out malaria
2. Blood culture to find causative organisms
3. CSF examination
4. X-ray mastoids
5. Imaging studies : CT scan & MRI
Delta sign / Empty Delta sign
6. Culture and sensitivity of ear swab
The pathognomonic "delta" sign is seen (2 small arrows)
The contralateral sigmoid sinus is patent (single large arrow)
Treatment
1. Intravenous culture directed antibiotics
2. Mastoid exploration
3. ± Anticoagulation therapy
Otogenic brain abscess
Clinical features
fever
headache
vomiting
focal neurological
signs
Mostly silent
Common : LOC - Lethargy & confusion
Nausea and vomiting
Papilloedema
Slow pulse and subnormal temperature
headache
Spontaneous nystagmus
I/L hypotonia &
weakness
Pastpointing &
intentional tremors
Dysdiadochokinesia
Nominal aphasia
Homonymous
hemianopia
C/L motor
paralysis
Epileptic fits
1. Blood smear & Haemogram
2. Blood culture to find causative organisms
3. CSF examination
4. X-ray mastoids
5. Imaging studies : CT scan & MRI
6. Culture and sensitivity of ear swab
1. High doses of Antibiotics
2. Corticosteroids 4mg iv QID & mannitol 20% @ 0.5 g/kg
3. I &D of abscess & aural toileting
Investigations
Treatment
Otitic Hydrocephalus
 It is characterized by raised intracranial pressure with normal CSF findings
Encephalocele
 Erosion leading to defects of tegmen tympani or tegmen mastoideum can lead to encephalocele or even
CSF leakage
 Investigation – CT scan & MRI
 Surgical repair
Clinicals
1. Given is the CECT of a patient of foul smelling ear discharge with
convulsions. He should be managed :
a. Abscess drainage followed by MRM
b. MRM followed by abscess drainage
c. Myringoplasty
d. Only MRM
2. Treatment of Brain fungus in mastoid cavity is:
a. Amphotericin therapy
b. Miconazole powder application
c. Surgical repair
d. Syringing
3. Treatment of choice for CSOM with vertigo and facial palsy is :
a. Antibiotics and labyrinthine sedatives
b. Myringoplasty
c. Immediate mastoid exploration
d. Labyrinthectomy
Thank You

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Complications of Chronic Otitis Media.pptx

  • 1. Complications of Chronic Otitis Media - Dr. Alka Kapil ( Assistant Professor, Dept. of ENT & Head Neck Surge
  • 2. Relations of Mastoid Antrum  Superiorly : Tegmen antri/Dural plate  Posteriorly : Sinus plate  Medially : Solid angle and posterior fossa dural plate  Laterally : Skin ,subcutaneous tissue & Mac Ewan’s triangle  Anteriorly : posterior wall of middle ear & facial canal
  • 3.
  • 4. Pathways of spread of infection
  • 6. Mastoiditis  When infection spreads from the mucosal lining the mastoid air cells to involve the bony walls of the mastoid air cell system it is called Mastoiditis.  Can be : Acute mastoiditis or Chronic mastoiditis or Coalescent mastoiditis or Masked mastoiditis
  • 7.  Acute inflammation of the mastoid bone  Streptococcus pneumoniae > β haemolytic streptococcus  Pathology : 1. Production of pus under tension 2. Hyperaemic decalcification and osteoclastic resorption of bony walls Acute Mastoiditis
  • 8. Accumulation of pus under tension hyperaemia of mucosal lining venous stasis local acidosis and dissolution of calcium from the bone hyperaemic decalcification destruction of bony septa Coalescent mastoiditis
  • 9. Clinical presentations Signs i. Mastoid tenderness ii. Ear discharge iii. Reservoir sign & Light house sign iv. Tympanic membrane perforation v. Swelling of mastoid/ ironed out appearance vi. Hearing loss vii. Fever viii. Sagging of posterosuperior wall of EAC Symptoms i. Pain behind the ear ii. Fever iii. Ear discharge
  • 10. BEZOLD ABSCESS Abscesses in Relation to Mastoid Infection
  • 11. (A) Abscesses in relation to mastoid: (1) postauricular, (2) zygomatic and (3) Bezold abscess (B) Citelli, postauricular and Bezold abscesses seen from behind.
  • 12. 1. Blood culture to find causative organisms 2. Routine hematological investigations ( CBC & ESR) 3. X-ray mastoids 4. Imaging studies : CT scan Temporal bone - MRI 5. Culture and sensitivity of ear swab Investigations
  • 13. Treatment 1. Intravenous culture directed antibiotics 2. Myringotomy if TM is intact and bulging with pus in tension 3. ± Mastoid exploration done if : (a) Subperiosteal abscess (b) Sagging of posterosuperior meatal wall. (c) Positive reservoir sign (d) No change in condition of patient or it worsens in spite of adequate medical treatment for 48 h. (e) Mastoiditis, leading to complications 4. Drainage of neck abscesses in case they are present
  • 14. Petrositis  Spread of infection from middle ear and mastoid to the petrous part of temporal bone is called petrositis
  • 15. Clinical presentations Gradenigo syndrome is the classical presentation, and consists of a triad of (i) external rectus palsy (VIth nerve palsy) (ii) deep-seated ear or retro-orbital pain (Vth nerve involvement) & (iii) persistent ear discharge
  • 16. 1. Routine blood investigations 2. Blood culture to find the causative organisms 3. X-ray mastoids 4. Imaging studies : CT scan & MRI Investigations
  • 17. 1. Intravenous culture directed antibiotics 2. Mastoid exploration / Petrous apicectomy Treatment
  • 19. Labyrinthitis CIRCUMSCRIBED LABYRINTHITIS DIFFUSE SEROUS LABYRINTHITIS DIFFUSE SUPPURATIVE LABYRINTHITIS Thinning or erosion of bony capsule of labyrinth diffuse intralabyrinthine inflammation without pus formation diffuse pyogenic infection of the labyrinth exposure of a localised part of membranous labyrinth due to erosion of the overlying bone toxins from middle ear infection complete destruction of membranous labyrinth (Labyrinthitis OSSIFICANS ) reversible condition if treated early permanent loss of vestibular and cochlear functions Vertigo + Vertigo + Vertigo & imbalance + Partial SNHL + Parital SNHL + SNHL + Fistula test + nystagmus is towards the same side of lesion nystagmus towards the normal side Mastoid exploration with closure of fistula Antibiotics, Labyrinthine sedatives, mastoid exploration Same as serous
  • 21. Meningitis  It is inflammation of leptomeninges (pia and arachnoid) usually with bacterial invasion of CSF in subarachnoid space  MC organism causing otogenic meningitis is Streptococcus pneumoniae > H. influenzae
  • 22. 1.There is rise in temperature (102-104 °F) often with chills and rigors 2. Headache 3. Neck rigidity 4. Photophobia and mental irritability 5. Nausea and vomiting (sometimes projectile) 6. Drowsiness which may progress to delirium or coma 7. Cranial nerve palsies and hemiplegia Clinical presentations
  • 23.  Signs : (i) Neck rigidity (ii) positive Brudzinski’s sign (flexion of neck causes flexion of hip and knee) (iii) positive Kernig’s sign (extension of leg with thigh flexed on abdomen causing pain) (iv) tendon reflexes exaggerated initially but later sluggish or absent (v) papilloedema
  • 24. Investigations 1. CT scan & MRI 2. Lumber Puncture & CSF examination CSF finding - turbid - cell count is raised - predominance of polymorphs - protein level is raised - sugar & chlorides reduced Treatment Mainly medical management
  • 25. Lateral Sinus Thrombophlebitis  Also called as Sigmoid Sinus Thrombophlebitis  It is an inflammation of inner wall of lateral venous sinus with formation of intrasinus thrombus  Pathology :
  • 27. Clinical presentations i. Picket Fence fever pattern (diurnal temperature spikes that exceed 103°F/39.4°C) ii. Headache iii. Progressive anemia and emaciation iv. Griesinger’s sign : oedema over mastoid d/t thrombosis of mastoid emissary vein v. Papilloedema vi. Tobey-ayer test vii. Crowe-beck test viii. Tenderness along jugular vein
  • 28.
  • 29. Investigations 1. Blood smear to rule out malaria 2. Blood culture to find causative organisms 3. CSF examination 4. X-ray mastoids 5. Imaging studies : CT scan & MRI Delta sign / Empty Delta sign 6. Culture and sensitivity of ear swab The pathognomonic "delta" sign is seen (2 small arrows) The contralateral sigmoid sinus is patent (single large arrow)
  • 30. Treatment 1. Intravenous culture directed antibiotics 2. Mastoid exploration 3. ± Anticoagulation therapy
  • 32. Clinical features fever headache vomiting focal neurological signs Mostly silent Common : LOC - Lethargy & confusion Nausea and vomiting Papilloedema Slow pulse and subnormal temperature headache Spontaneous nystagmus I/L hypotonia & weakness Pastpointing & intentional tremors Dysdiadochokinesia Nominal aphasia Homonymous hemianopia C/L motor paralysis Epileptic fits
  • 33. 1. Blood smear & Haemogram 2. Blood culture to find causative organisms 3. CSF examination 4. X-ray mastoids 5. Imaging studies : CT scan & MRI 6. Culture and sensitivity of ear swab 1. High doses of Antibiotics 2. Corticosteroids 4mg iv QID & mannitol 20% @ 0.5 g/kg 3. I &D of abscess & aural toileting Investigations Treatment
  • 34. Otitic Hydrocephalus  It is characterized by raised intracranial pressure with normal CSF findings
  • 35. Encephalocele  Erosion leading to defects of tegmen tympani or tegmen mastoideum can lead to encephalocele or even CSF leakage  Investigation – CT scan & MRI  Surgical repair
  • 36. Clinicals 1. Given is the CECT of a patient of foul smelling ear discharge with convulsions. He should be managed : a. Abscess drainage followed by MRM b. MRM followed by abscess drainage c. Myringoplasty d. Only MRM
  • 37. 2. Treatment of Brain fungus in mastoid cavity is: a. Amphotericin therapy b. Miconazole powder application c. Surgical repair d. Syringing
  • 38. 3. Treatment of choice for CSOM with vertigo and facial palsy is : a. Antibiotics and labyrinthine sedatives b. Myringoplasty c. Immediate mastoid exploration d. Labyrinthectomy