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CARDIOVASCULAR
EMERGENCIES
AKAMAH ASAA SOLOMON
RN ,CCN
Core concept
• Aspects of acute coronary syndrome (ACS)
• Conditions that may lead to a cardiac emergency
• Other cardiovascular emergencies
Topics
•Cardiac Anatomy and Physiology
•Acute Coronary Syndrome
•Causes of Cardiac Conditions
•Other Cardiovascular Emergencies
Cardiovascular system
•The cardiovascular system delivers oxygen
and nutrients to tissue and removes by-
products of metabolism and cellular waste
from tissue throughout the body.
•Consist mainly of the heart, blood vessels
and blood.
Cardiac A&P
Four Chambers of the Heart
Right Atrium
Right Ventricle
Left Atrium
Left Ventricle
Receives blood from
veins; pumps to right
ventricle.
Receives blood
from lungs; pumps
to left ventricle.
Pumps blood to
the lungs.
Pumps blood through
the aorta to the body.
Cardiac Conduction System
Blood Vessels
14-9
Coronary Circulation
• Right & left coronary
arteries branch to supply
heart muscle
• anterior & posterior
interventricular aa.
14-10
Arteries
• Tunica internal (intima)
• simple squamous epithelium
known as endothelium
• basement membrane
• internal elastic lamina
• Tunica media
• circular smooth muscle & elastic
fibers
• Tunica externa
• elastic & collagen fibers
Think About It
•How does the normal function of the heart
and blood vessels relate to blood pressure
and distal pulses?
•How is shock related to the function of the
heart and blood vessels?
Angina pectoris
•Refers to any time the heart may not be getting
enough oxygen
•Angina pectoris is chest pain caused by a temporary
imbalance between the coronary arteries' ability to
supply oxygen and the cardiac muscle's demand for
oxygen
continued
Angina
Conditions that present with Angina are:
1. Stable Angina
2. Unstable Angina
3. Myocardial Infarction
a. ST-segment elevation myocardial infarction (STEMI)
b. Non-ST-segment elevation myocardial infarction (NSTEMI
Angina Pectoris
•Typical STABLE ANGINA is exertional, and is relieved
promptly by rest or nitroglycerin.
•Unstable Angina consists of ischemic coronary artery
symptoms which are more frequent, severe, or prolonged
than stable angina,
• The angina
a. Occur at rest or with minimal exertion
b. Is more difficult to control with drugs
Acute Coronary Syndrome (ACS)
•Symptoms often mimic non-cardiac
conditions
•Treat all patients with ACS-like signs and
symptoms as though they are having a heart
problem
Symptoms of ACS
• Chest pain is best-known symptom
• Can be described as “crushing, dull, heavy, or
squeezing”
• Sometimes described only as pressure or discomfort
• Radiates to arms, upper abdomen, jaw
continued
Symptoms
• Dyspnea also found in
ACS
• May be the only finding in
some patients
Other Signs and Symptoms
• Anxiety, feeling of impending doom
• Nausea and pain or discomfort in upper abdomen (epigastric
pain)
• Sweating
• Abnormal pulse (tachycardia/bradycardia)
• Abnormal blood pressure
Assessment
•Perform primary assessment
•Obtain history and physical exam
•Use OPQRST to get history of present illness
•Obtain SAMPLE history
•Take baseline vital signs
Treatment
• Place patient in position of comfort
(typically sitting up)
• Apply high-concentration oxygen
• Transport
Treatment
•If trained, equipped, and authorized to do so,
obtain a 12-lead electrocardiogram (ECG)
•Follow local protocol as to whether to
transmit it to hospital for interpretation
•Employ O . N. A of MONA.
continued
Treatment
• Indications for administering nitroglycerin
•Chest pain
•History of cardiac problems and prescribed
nitroglycerin
•Patient has nitroglycerin
•Medical direction
authorizes administration
continued
Treatment
• Contraindications for administering nitroglycerin
•Systolic blood pressure less than 90–100
(consult local protocol)
•Patient has taken Viagra or similar drug for
erectile dysfunction within 48–72 hours
continued
Treatment
•Indications for administering aspirin
• Chest pain
• Ability to safely swallow
• Medical control authorization
continued
Treatment
•Contraindications for administering aspirin
•Inability to swallow
•Allergy to aspirin
•History of asthma
•Patient already taking other anti-clotting
medications
Causes of Cardiac Conditions
• Heart problems are caused by a number of disorders
affecting condition and function of blood vessels and heart
Risk factors
• Age high cholesterol
• Gender diabetes
• Family history
• Smoking
• Poor diet
• High blood pressure
Coronary Artery Disease
• Conditions that narrow or block arteries of heart
• Often result from fatty deposit build-up on inner
walls of arteries
• Build-up narrows inner vessel diameter, restricts
flow of blood
Coronary Artery Disease
• Thrombus—occlusion of blood flow caused by
formation of a clot on rough inner surface of
diseased artery
• Thrombus can break loose and form an embolism
• Emboli can move to occlude flow of blood
downstream in a smaller artery
continued
Coronary Artery Disease
•Reduced blood supply to myocardium
causes emergency in majority of cardiac-
related medical emergencies
•Chest pain is most common symptom of
reduced blood supply
Aneurysm
•Weakened sections of
blood vessels begin to
dilate (balloon)
•Bursting can cause rapid,
life-threatening internal
bleeding
Aortic dissection
• A serious condition in which a tear occurs in the inner layer of the
aorta
• Blood rushes through the tear, causing the inner and middle
layers to split.
• There are 2 types: type A and type B
• Type A- most common and dangerous type, occurs at the area
where the aorta exits the heart,
• Type B- occurs at the descending aorta and may extend to the
abdomen
Signs and symptoms
• Sudden severe chest pain or upper back pain which is
described as tearing.
• Sudden severe abdominal pain
• Loss of consciousness
• Shortness of breath
• Symptoms of stroke
• Weak pulse in one arm or thigh
Management of aortic dissection
• Assessment
• High concentration of oxygen
• Transport
• ACLS intercept
Electrical
Malfunction of the Heart
• Malfunction of heart’s electrical system generally
results in dysrhythmia
• Dysrhythmias include bradycardia, tachycardia,
and rhythms that may be present when there is
no pulse
Mechanical
Malfunctions of the Heart
•Angina pectoris
•Acute myocardial infarction (AMI)
•Congestive Heart Failure (CHF)
Angina Pectoris
•Chest pain caused by insufficient blood flow to the
myocardium
•Typically due to narrowed arteries secondary to
coronary artery disease
•Pain usually during times of increased myocardial
oxygen demand, such as exertion or stress
Acute Myocardial Infarction (AMI)
•Death of a portion of the myocardium due to lack
of oxygen
•Coronary artery disease is usually the underlying
reason
Congestive Heart Failure (CHF)
• Inadequate pumping of the heart
• Often leads to excessive fluid build-up in lungs and/or body
• May be brought on by diseased heart valves, hypertension,
obstructive pulmonary disease
• Often a complication of AMI
Progression of CHF
• Patient sustains AMI
• Myocardium of left ventricle dies
• Because of damage to left ventricle, blood backs
up into pulmonary circulation and lungs
• If untreated, left heart failure commonly causes
right heart failure
Signs and Symptoms of CHF
• Tachycardia
• Dyspnea and cyanosis
• Normal or elevated blood pressure
• Diaphoresis
• Pulmonary edema
continued
continued
Signs and Symptoms of CHF
• Anxiety or confusion due to hypoxia
• Pedal edema
• Engorged, pulsating neck veins (late sign)
• Enlarged liver and spleen
Hypertensive crisis
• Hypertension –blood pressure of 140/90mmHg and above.
Hypertensive crisis
• Blood pressure reading of 180/120mmHg or greater. Usually due
to uncontrolled blood pressure.
• Occurs in two forms: urgency and emergency.
• Hypertensive urgency- extremely high blood pressure without
suspected end organ damage.
Hypertensive crisis
• Hypertensive emergency- extremely high blood pressure
associated with life-threathening complications.
• Signs and symptoms include; severe chest pain,severe
headache, confusion, blurr vision, severe anxiety, shortness of
breath, seizures , unresponsive
Management of Hypertensive crisis
• Perform primary survey
-correct any ABCDE problems
-high concentration of oxygen
-transport promptly to appropriate facility
• History taking and physical examination.
• Baseline vital signs.
• Prompt transport.
Shock
4 main etiologies of shock include:
Hypovolemic: Low cardiac output due to low intravascular volume.
Cardiogenic: Low cardiac output despite adequate intravascular
volume.
Distributive: Low total peripheral or systemic vascular resistance,
usually septic.
Obstructive: Low cardiac output due to outflow obstruction.
Hypovolemic shock
1. Hemorrhage
a. Trauma
b. Gastrointestinal hemorrhage
c. Postsurgical, post procedural bleeding
d. Intra-abdominal (e.g., abdominal aortic
aneurysm, ruptured ectopic pregnancy)
2. Volume depletion
a. Vomiting
b. Diarrhea
c. Excessive diuresis (from diuretics or
uncontrolled diabetes). D: Burns
Classes of Hypovolemic Shock
Class I Class II Class III Class IV
Blood Loss < 750 750-1500 1500-2000 > 2000
% Blood Vol. < 15% 15 – 30% 30 – 40% > 40%
Pulse < 100 > 100 > 120 > 140
Blood Pressure Normal Normal Decreased Decreased
Pulse Pressure Normal Decreased Decreased Decreased
Resp. Rate 14 – 20 20 – 30 30 – 40 > 40
UOP > 30 20 – 30 5 – 15 negligible
Mental Status sl. Anxious mildly anx confused lethargic
Fluid crystalloid crystalloid blood blood
Clinical Presentation of Hypovolemic Shock
• Tachycardia and tachypnea
• Weak, thready pulses
• Hypotension
• Skin cool & clammy
• Mental status changes
• Decreased urine output: dark & concentrated
Cardiogenic shock
Poor contractility:
1. Myocardial infarction (MI)
2. Myocarditis
3. Arrythmias (severe bradycardia and tachycardia
4. Valve problems
Obstructive shock
Outflow Obstruction:
a. Aortic stenosis
b. Hypertrophic cardiomyopathy
c. Pulmonary embolism (PE)
Reduced filling
a. Constrictive pericarditis
b. Tension pneumothorax
c. Mitral stenosis
Distributive shock
1. Septic shock; as a result of severe infection in the blood.
2. Anaphylactic shock; as a result of severe allergic reaction.
3. Neurogenic shock; as a result of injury to the spinal cord.
Cellular Response to Shock
Blood
Loss Inadequate
Perfusion
Cellular
Hypoxia
Aerobic
Metabolism
Anaerobic
Metabolism
Lactic
Acid
Cellular
Edema
Acidosis
Stages of shock
•Initial stage- tissues are under perfused, decreased cardiac
output without signs and symptoms.
•Compensatory/reversible stage- activation of sympathetic
nervous system and renin-angiotensin system.
Stages of shock
Progressive stage- if there is no interventions or if
interventions fails, compensatory mechanisms worsen cardiac
state leading to anerobic metabolic metabolism and acidosis.
Cardiologist care required for cardiogenic shock.
Refractory/irreversible stage- survival is extremely limited,
complications such as cardiopulmonary arrest, dysrhythmias,
multiple organ failure, stroke and death.
Sympatho-Adrenal Response to Shock
• Most immediate of compensatory mechanisms are those of
Sympathetic Nervous System and Renin Angiotensin Mechanism
•Sympathetic Nervous System
• Epinephrine, NE and Cortisol released
•Cause vasoconstriction, increase in HR, and increase of
Cardiac Contractility (Cardiac Output)
•Renin-Angiotensin Axis
• Water and Sodium Conservation and Vasoconstriction
• Increase in Blood Volume and Blood Pressure 55
Shock Index (SI)
• A bedside assessment defined as heart rate divided by systolic blood
pressure, with a normal range of 0.5 to 0.7 in healthy adults.
• Has been suggested as such a marker that can be used to predict severity of
hypovolemic shock.
• Classification of hypovolemic shock based on the SI enables a fast and
reliable assessment of hypovolemic shock in the emergency department.
• SI <0.6 (no shock), SI ≥0.6 to <1.0 (mild shock), SI ≥1.0 to <1.4 (moderate
shock) and SI ≥1.4 (severe shock).
Mgt of shock
• Assessment- ABCDE approach
• High concentration of oxygen
• Keep patient warm
• Specific treatment dependind on cause of shock.
• Transport

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Cardiovascular emergencies - solo.pptx

  • 2. Core concept • Aspects of acute coronary syndrome (ACS) • Conditions that may lead to a cardiac emergency • Other cardiovascular emergencies
  • 3. Topics •Cardiac Anatomy and Physiology •Acute Coronary Syndrome •Causes of Cardiac Conditions •Other Cardiovascular Emergencies
  • 4. Cardiovascular system •The cardiovascular system delivers oxygen and nutrients to tissue and removes by- products of metabolism and cellular waste from tissue throughout the body. •Consist mainly of the heart, blood vessels and blood.
  • 6. Four Chambers of the Heart Right Atrium Right Ventricle Left Atrium Left Ventricle Receives blood from veins; pumps to right ventricle. Receives blood from lungs; pumps to left ventricle. Pumps blood to the lungs. Pumps blood through the aorta to the body.
  • 9. 14-9 Coronary Circulation • Right & left coronary arteries branch to supply heart muscle • anterior & posterior interventricular aa.
  • 10. 14-10 Arteries • Tunica internal (intima) • simple squamous epithelium known as endothelium • basement membrane • internal elastic lamina • Tunica media • circular smooth muscle & elastic fibers • Tunica externa • elastic & collagen fibers
  • 11. Think About It •How does the normal function of the heart and blood vessels relate to blood pressure and distal pulses? •How is shock related to the function of the heart and blood vessels?
  • 12. Angina pectoris •Refers to any time the heart may not be getting enough oxygen •Angina pectoris is chest pain caused by a temporary imbalance between the coronary arteries' ability to supply oxygen and the cardiac muscle's demand for oxygen continued
  • 13. Angina Conditions that present with Angina are: 1. Stable Angina 2. Unstable Angina 3. Myocardial Infarction a. ST-segment elevation myocardial infarction (STEMI) b. Non-ST-segment elevation myocardial infarction (NSTEMI
  • 14. Angina Pectoris •Typical STABLE ANGINA is exertional, and is relieved promptly by rest or nitroglycerin. •Unstable Angina consists of ischemic coronary artery symptoms which are more frequent, severe, or prolonged than stable angina, • The angina a. Occur at rest or with minimal exertion b. Is more difficult to control with drugs
  • 15. Acute Coronary Syndrome (ACS) •Symptoms often mimic non-cardiac conditions •Treat all patients with ACS-like signs and symptoms as though they are having a heart problem
  • 16. Symptoms of ACS • Chest pain is best-known symptom • Can be described as “crushing, dull, heavy, or squeezing” • Sometimes described only as pressure or discomfort • Radiates to arms, upper abdomen, jaw continued
  • 17. Symptoms • Dyspnea also found in ACS • May be the only finding in some patients
  • 18. Other Signs and Symptoms • Anxiety, feeling of impending doom • Nausea and pain or discomfort in upper abdomen (epigastric pain) • Sweating • Abnormal pulse (tachycardia/bradycardia) • Abnormal blood pressure
  • 19. Assessment •Perform primary assessment •Obtain history and physical exam •Use OPQRST to get history of present illness •Obtain SAMPLE history •Take baseline vital signs
  • 20. Treatment • Place patient in position of comfort (typically sitting up) • Apply high-concentration oxygen • Transport
  • 21. Treatment •If trained, equipped, and authorized to do so, obtain a 12-lead electrocardiogram (ECG) •Follow local protocol as to whether to transmit it to hospital for interpretation •Employ O . N. A of MONA. continued
  • 22. Treatment • Indications for administering nitroglycerin •Chest pain •History of cardiac problems and prescribed nitroglycerin •Patient has nitroglycerin •Medical direction authorizes administration continued
  • 23. Treatment • Contraindications for administering nitroglycerin •Systolic blood pressure less than 90–100 (consult local protocol) •Patient has taken Viagra or similar drug for erectile dysfunction within 48–72 hours continued
  • 24. Treatment •Indications for administering aspirin • Chest pain • Ability to safely swallow • Medical control authorization continued
  • 25. Treatment •Contraindications for administering aspirin •Inability to swallow •Allergy to aspirin •History of asthma •Patient already taking other anti-clotting medications
  • 26. Causes of Cardiac Conditions • Heart problems are caused by a number of disorders affecting condition and function of blood vessels and heart Risk factors • Age high cholesterol • Gender diabetes • Family history • Smoking • Poor diet • High blood pressure
  • 27. Coronary Artery Disease • Conditions that narrow or block arteries of heart • Often result from fatty deposit build-up on inner walls of arteries • Build-up narrows inner vessel diameter, restricts flow of blood
  • 28. Coronary Artery Disease • Thrombus—occlusion of blood flow caused by formation of a clot on rough inner surface of diseased artery • Thrombus can break loose and form an embolism • Emboli can move to occlude flow of blood downstream in a smaller artery continued
  • 29. Coronary Artery Disease •Reduced blood supply to myocardium causes emergency in majority of cardiac- related medical emergencies •Chest pain is most common symptom of reduced blood supply
  • 30. Aneurysm •Weakened sections of blood vessels begin to dilate (balloon) •Bursting can cause rapid, life-threatening internal bleeding
  • 31. Aortic dissection • A serious condition in which a tear occurs in the inner layer of the aorta • Blood rushes through the tear, causing the inner and middle layers to split. • There are 2 types: type A and type B • Type A- most common and dangerous type, occurs at the area where the aorta exits the heart, • Type B- occurs at the descending aorta and may extend to the abdomen
  • 32. Signs and symptoms • Sudden severe chest pain or upper back pain which is described as tearing. • Sudden severe abdominal pain • Loss of consciousness • Shortness of breath • Symptoms of stroke • Weak pulse in one arm or thigh
  • 33. Management of aortic dissection • Assessment • High concentration of oxygen • Transport • ACLS intercept
  • 34. Electrical Malfunction of the Heart • Malfunction of heart’s electrical system generally results in dysrhythmia • Dysrhythmias include bradycardia, tachycardia, and rhythms that may be present when there is no pulse
  • 35. Mechanical Malfunctions of the Heart •Angina pectoris •Acute myocardial infarction (AMI) •Congestive Heart Failure (CHF)
  • 36. Angina Pectoris •Chest pain caused by insufficient blood flow to the myocardium •Typically due to narrowed arteries secondary to coronary artery disease •Pain usually during times of increased myocardial oxygen demand, such as exertion or stress
  • 37. Acute Myocardial Infarction (AMI) •Death of a portion of the myocardium due to lack of oxygen •Coronary artery disease is usually the underlying reason
  • 38. Congestive Heart Failure (CHF) • Inadequate pumping of the heart • Often leads to excessive fluid build-up in lungs and/or body • May be brought on by diseased heart valves, hypertension, obstructive pulmonary disease • Often a complication of AMI
  • 39. Progression of CHF • Patient sustains AMI • Myocardium of left ventricle dies • Because of damage to left ventricle, blood backs up into pulmonary circulation and lungs • If untreated, left heart failure commonly causes right heart failure
  • 40. Signs and Symptoms of CHF • Tachycardia • Dyspnea and cyanosis • Normal or elevated blood pressure • Diaphoresis • Pulmonary edema continued
  • 41. continued Signs and Symptoms of CHF • Anxiety or confusion due to hypoxia • Pedal edema • Engorged, pulsating neck veins (late sign) • Enlarged liver and spleen
  • 42. Hypertensive crisis • Hypertension –blood pressure of 140/90mmHg and above. Hypertensive crisis • Blood pressure reading of 180/120mmHg or greater. Usually due to uncontrolled blood pressure. • Occurs in two forms: urgency and emergency. • Hypertensive urgency- extremely high blood pressure without suspected end organ damage.
  • 43. Hypertensive crisis • Hypertensive emergency- extremely high blood pressure associated with life-threathening complications. • Signs and symptoms include; severe chest pain,severe headache, confusion, blurr vision, severe anxiety, shortness of breath, seizures , unresponsive
  • 44. Management of Hypertensive crisis • Perform primary survey -correct any ABCDE problems -high concentration of oxygen -transport promptly to appropriate facility • History taking and physical examination. • Baseline vital signs. • Prompt transport.
  • 45. Shock 4 main etiologies of shock include: Hypovolemic: Low cardiac output due to low intravascular volume. Cardiogenic: Low cardiac output despite adequate intravascular volume. Distributive: Low total peripheral or systemic vascular resistance, usually septic. Obstructive: Low cardiac output due to outflow obstruction.
  • 46. Hypovolemic shock 1. Hemorrhage a. Trauma b. Gastrointestinal hemorrhage c. Postsurgical, post procedural bleeding d. Intra-abdominal (e.g., abdominal aortic aneurysm, ruptured ectopic pregnancy) 2. Volume depletion a. Vomiting b. Diarrhea c. Excessive diuresis (from diuretics or uncontrolled diabetes). D: Burns
  • 47. Classes of Hypovolemic Shock Class I Class II Class III Class IV Blood Loss < 750 750-1500 1500-2000 > 2000 % Blood Vol. < 15% 15 – 30% 30 – 40% > 40% Pulse < 100 > 100 > 120 > 140 Blood Pressure Normal Normal Decreased Decreased Pulse Pressure Normal Decreased Decreased Decreased Resp. Rate 14 – 20 20 – 30 30 – 40 > 40 UOP > 30 20 – 30 5 – 15 negligible Mental Status sl. Anxious mildly anx confused lethargic Fluid crystalloid crystalloid blood blood
  • 48. Clinical Presentation of Hypovolemic Shock • Tachycardia and tachypnea • Weak, thready pulses • Hypotension • Skin cool & clammy • Mental status changes • Decreased urine output: dark & concentrated
  • 49. Cardiogenic shock Poor contractility: 1. Myocardial infarction (MI) 2. Myocarditis 3. Arrythmias (severe bradycardia and tachycardia 4. Valve problems
  • 50. Obstructive shock Outflow Obstruction: a. Aortic stenosis b. Hypertrophic cardiomyopathy c. Pulmonary embolism (PE) Reduced filling a. Constrictive pericarditis b. Tension pneumothorax c. Mitral stenosis
  • 51. Distributive shock 1. Septic shock; as a result of severe infection in the blood. 2. Anaphylactic shock; as a result of severe allergic reaction. 3. Neurogenic shock; as a result of injury to the spinal cord.
  • 52. Cellular Response to Shock Blood Loss Inadequate Perfusion Cellular Hypoxia Aerobic Metabolism Anaerobic Metabolism Lactic Acid Cellular Edema Acidosis
  • 53. Stages of shock •Initial stage- tissues are under perfused, decreased cardiac output without signs and symptoms. •Compensatory/reversible stage- activation of sympathetic nervous system and renin-angiotensin system.
  • 54. Stages of shock Progressive stage- if there is no interventions or if interventions fails, compensatory mechanisms worsen cardiac state leading to anerobic metabolic metabolism and acidosis. Cardiologist care required for cardiogenic shock. Refractory/irreversible stage- survival is extremely limited, complications such as cardiopulmonary arrest, dysrhythmias, multiple organ failure, stroke and death.
  • 55. Sympatho-Adrenal Response to Shock • Most immediate of compensatory mechanisms are those of Sympathetic Nervous System and Renin Angiotensin Mechanism •Sympathetic Nervous System • Epinephrine, NE and Cortisol released •Cause vasoconstriction, increase in HR, and increase of Cardiac Contractility (Cardiac Output) •Renin-Angiotensin Axis • Water and Sodium Conservation and Vasoconstriction • Increase in Blood Volume and Blood Pressure 55
  • 56. Shock Index (SI) • A bedside assessment defined as heart rate divided by systolic blood pressure, with a normal range of 0.5 to 0.7 in healthy adults. • Has been suggested as such a marker that can be used to predict severity of hypovolemic shock. • Classification of hypovolemic shock based on the SI enables a fast and reliable assessment of hypovolemic shock in the emergency department. • SI <0.6 (no shock), SI ≥0.6 to <1.0 (mild shock), SI ≥1.0 to <1.4 (moderate shock) and SI ≥1.4 (severe shock).
  • 57. Mgt of shock • Assessment- ABCDE approach • High concentration of oxygen • Keep patient warm • Specific treatment dependind on cause of shock. • Transport