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Shoulder and knee
conditions in work-related
Injuries
______JAMES T MAZZARA, MD
ORTHOPEDIC ASSOCIATES OF HARTFORD, PC
MANCHESTER / ROCKY HILL
HARTFORD HOSPITAL GLASTONBURY SURGERY CENTER
CONNECTICUT JOINT REPLACEMENT INSTITUTE @ ST FRANCIS
HOSPITAL
ECHN
Contact Information
James T Mazzara, MD
Orthopedic Associates of Hartford, PC
29 Haynes Street
Manchester, CT 06040
_________________
150 Enterprise Drive
Rocky Hill, CT 06067
860-649-2267
www.HartfordSportsOrthopedics.com
Why Workers’ Comp Matters
 The workers’ compensation system is a
medical driven legal compromise described as
a “Grand Bargain” between labor and
employees
Causation
 The cause (injury or history of exposure)
is medically probable and occurred
while at work or with a specific event
 The effect is medically probable.
Condition is medically diagnosable &
consistent with the symptoms, history,
examination and clinical studies.
Causation
 Cause & effect are probably related
Causation
 Medical findings are compatible with
the effects of an injury
 Sufficient exposure is present to have
caused the condition
 The weight of evidence supports the
disease as having an occupational
rather than non-occupational cause.
The Method
 1. Identify evidence of disease
 2. Review and assess available epidemiologic
evidence for a causal relationship
 3. Obtain and assess evidence of exposure
 4. Consider other relevant factors
 5. Judge the validity of the facts (Plausibility)
 6. Form conclusions about the work-relatedness
of the condition
1. Identify the condition
 History
 Exam
 Clinical studies
 Make the correct diagnosis
2. Review & assess epidemiologic
evidence for causal relationship
 AMA Guides to Disease and Injury Causation
 Second edition
 Analyses the studies based on study design, quality
and value
 Strength of evidence
 From Very strong evidence to No Risk evidence
3. Risk of Exposure
 Evidence of exposure
What tasks. Quantified. Standardized
Personal measurement
Exposure of other workers
Defined job category or trade
Defined by employer
4. Other relevant factors
 How does the individual perform the job
 How long on the job, previous job
 Has the job changed
 Other jobs
 Modifications
 Previous restrictions or impairments
 Pre existing conditions
 Hobbies
5. Judge the facts
 Temporality
 Strength of association
 Dose response relationship
 Consistency
 Coherence to current science
 Plausibility
 Reversibility
Form a Conclusion
 Convert data from the whole to data for the individual
 May not be intuitively obvious
 Work relatedness involves the concept of medical and
legal causation
 These may be mutually exclusive
 Medical causation is scientific
 Legal causation from a desire for social justice
Justice v. Science
 The courts are not based in science
 Therefore, laws are not scientifically derived
 Accepted wisdom if justice and fair play that
underlie the concept of proximate cause are
subject to many interpretations in WC law.
 Each state may determine for itself what
constitutes proximate cause
 Judges and legislature can and will substitute
convenience for science
 The judges have the final say
Connecticut Law
 Threshold Standard
Reasonable degree of medical probability
(certainty)
 Proving causal relationship is on the claimant
 Can only be met by competent medical evidence
 At least one doctor must state RDOMC
Aggravation
 Permanent worsening of a prior, or underlying,
condition by an event or exposure
Exacerbation
 Temporary worsening of a prior, or underlying,
condition by an event or exposure that will or
has returned to baseline
Aggravation v. Exacerbation
The Knee
Age-related Cartilage Changes
 Older cartilage is less
cellular
 Cartilage cells do not
reproduce after
growth plates close
 Chondrocytes only in
lower layers
 Chondrocytes
maintain extracellular
matrix
 Bad news for aging
cartilage
 Water content
decreases
Grading Cartilage Wear
I II
III IV
Meniscus
 Cushion, shock absorber, stabilizer
 Compression results in hoop (circumferential) stress
 Laterally 70% load transmitted
 Medially 50% load transmitted
Meniscectomy
 Medial meniscectomy
 50-70% reduction in femoral
condyle contact w/ meniscus
 100% increase in contact
stress
 Lateral meniscectomy
 Total meniscectomy: 40-50%
decrease in contact area
 Increased contact stresses in
lateral compartment 200-
300%
 Contributes to articular
cartilage damage and
degeneration
Meniscectomy and Laxity
 In ACLR, additional MM resection increased whereas MM repair
preserved knee laxity in comparison with the ACLR knee with intact
menisci.
 Neither LM resection or LM repair showed a significant effect on
knee laxity.
 Surgeons should make every effort to repair the meniscus whenever
possible to avoid the residual postoperative laxity present in the
meniscus-deficient knee.
 Medial Meniscus Resection Increases and Medial Meniscus Repair Preserves Anterior Knee Laxity:
A Cohort Study of 4497 Patients With Primary Anterior Cruciate Ligament Reconstruction
 Riccardo Cristiani, MD*, Erik Rönnblad, MD, Björn Engström, MD, PhD, ...
 First Published October 24, 2017
Orthopedic Evaluation
 Activity level
 Expectations & Goals
 Symptoms
 Examination
 Alignment
 Hip exam
 Knee Exam
 ROM
 Meniscal signs
 Patellar mobility
Radiographic Evaluation
 Standing AP
 Standing 45o flexion PA
 Lateral
 Patellofemoral
 Each x-ray position tells us something different about
the location of problems and the alignment of the
knee.
X-Rays: Standing Films- AP
Standing x-rays are performed
with the knee in different
positions. Each position reveals
something about the location
and degree of arthritis or other
conditions that may lead to
knee pain.
This is a standing x-ray with
knees straight (extended).
X-Rays: Standing Films- PA w/ Flexion
This is an x-ray with the
knee bent (flexed) 45
degrees.
Properly positioned knee x-
rays can reveal a degree of
arthritis that may be missed
on non-weight bearing x-ray
views.
X-Rays
This weight bearing tunnel view can
reveal the degree of arthritis not seen
on other x-rays
This patellar view reveals the position of
the patella in mid flexion.
Knee
Arthroscopy:
Meniscectomy
Knee
Arthroscopy:
Meniscectomy
Knee
Arthroscopy
Knee
Arthroscopy
Knee
Arthroscopy
Knee Arthroscopy: Microfracture
 Full thickness joint cartilage
defects
 Unstable full thickness lesions
 Osteoarthritis with proper knee
alignment
 Not for partial thickness defects
Knee
Arthroscopy:
Microfracture
Knee Arthroscopy: Microfracture
Cartilage injury
Loose
cartilage
removed
After
microfracture
Knee
Arthroscopy:
Microfracture
Knee
Arthroscopy:
Microfracture
Microfracture: Predictors of Outcome
 Poorer results in certain
patients
Increased age
Malalignment
Higher BMI (obesity)
Rim height (condition of
adjacent cartilage)
Proper technique and
rehabilitation
Autogenous Cartilage Implantation
 Focal cartilage defects
 Intact meniscus
 Meniscus transplant
 Normal alignment
MACI: Autogenous Chondrocyte Implantation via
Collagen Membrane
 A sample of the
patient’s own cartilage
is sent to the lab,
regenerated and
implanted into a
collagen membrane
for reimplantation into
the patient’s knee.
Arthroscopy &
Microfracture in the
Degenerative Knee
 Different than in non-
degenerative knees
Surrounding cartilage is
thinned
Dense, sclerotic bone
Joint scarring and
contractures
Synovitis
Osteophytes (Bone Spurs)
Total Knee Replacement
 >50% of patients over 65 have at least one joint with OA
 Over 250,000 TKA performed annually
 Excellent survival and function in >90% at 10-20 years
Knee Osteoarthritis – What does it look
like?
Knee with OsteoarthritisHealthy Knee
Tibia
(shin bone)
Cartilage
Femur
(thigh bone)
Knee Replacement Surgery
 Considered when
nonsurgical
interventions aren’t
alleviating pain
 The only long-term
solution to knee pain
Total Knee Replacement
Total Knee Replacement
Total Knee Replacement
The
Shoulder
The Shoulder Complex
Deltoid Muscle
Pectoralis
Clavicle (Collar Bone)
The Shoulder Complex
Deltoid muscle
removed
The Shoulder Complex
Rotator Cuff
 Supraspinatus
 Active in any elevation of
the arm
 Stabilizes the shoulder
joint
Rotator Cuff
 Infraspinatus
 Depressor of the humeral
head
 Stabilizer of the back of
the shoulder
Rotator Cuff
 Teres Minor
 Rotates the shoulder out
from the side
Rotator Cuff
 Subscapularis
 Stabilizes the front of the
shoulder
 Rotates the arm inward
Rotator Cuff Balance
 Proper function depends
upon balance between all
muscle and ligament forces
around the shoulder
Tendon Degeneration
 Age-related changes
 Decreased vascularity at
the tendon attachment
to the bone
 Leads to weak tendon
that tears easily
Rotator Cuff Tears
 Tears begin where the
stresses are the
greatest
 Tendon fibers fail a few
at a time or all at once
 Arm may be at rest
 Torn fibers retract when
torn
Humeral Head
Partial Supraspinatus
Tear
Consequences of rupture
 Increasing loads applied to the intact fibers
 Muscle fibers become detached from the bone resulting in weakness
Full Thickness Tears
 Loads are concentrated
at the margins of the
tear
 Further tearing occurs
with smaller loads
 Partial tears become
complete
 Smaller tears become
large
 Large tears eventually
become unfixable
Early Cuff Failure
 Compression of the
humeral head is less
effective
 Deltoid pulls head upward
 Upward pull of the deltoid
results in cuff abrasion &
further cuff damage
Progressive Tearing
 Spacer effect of the cuff is lost
 Humeral head displaces superiorly
 Biceps tendon eventually ruptures
Chronic Cuff Failure
 Humeral head forms a
joint with the arch above
 Secondary joint disease
occurs called cuff tear
arthropathy
Chronic Cuff Tears
 Muscle atrophy
 Fatty infiltration of
muscle belly
 Tendon retraction
 Bone osteoporosis
 Loss of muscle and
tendon excursion
 Irreversible
 Progressively worse
Fatty
infiltration
with muscle
wasting
Healthy
muscle, no fat
stripes
Prevalence of Rotator Cuff Tears
 Cadaver studies 7-40%
 MRI & Ultrasound studies
34% of asymptomatic individuals
54% of asymptomatic individuals over 60y
 Ultrasound study
13% of asymptomatic individuals: 50-59y
51% of asymptomatic individuals: over 80y
Prevalence of Rotator Cuff Tears
 40%: no history of strenuous physical labor
 50%: no history of trauma
 Frequently bilateral
 Many heavy laborers never get cuff tears
Healing Potential
 None without surgery
Cuff tears never heal spontaneously
Without a blood supply, there is never any chance
a cuff healing spontaneously
 51% of asymptomatic RCT become symptomatic
 Small tears increase in size slowly
 Large tears progress more quickly
Non Rotator Cuff Shoulder Pain
 Pain to the back of shoulder
upper back or neck
 Pain to top of shoulder
 Think arthritis of the neck
 Pain beyond the elbow
 Think pinched nerve in the neck
Radiographs
 Acromial shape
 Position of humeral head
 AC arthritis
 Calcific tendinitis
 Glenohumeral arthritis
 Destructive lesions
Partial Tendon Tears: Acromioplasty, Excise and 1O Repair
 For tears >50% tendon
thickness
 Post-op treat same as a
full thickness RC repair
Rotator Cuff Repair
 Arthroscopic
 Open repair
Irreparable Rotator Cuff Tears
 Superior capsular reconstruction
For massive irreparable cuff tears
No arthritis
Younger patients
Older patients who do not want a
shoulder replacement
Restores superior capsule restraining
superior translation
Allows deltoid and pectoralis to
function normally
Superior Capsular Reconstruction
 Indications
 Irreparable Supraspinatus and Infraspinatus tears
 Failed non-operative management
 Intolerable shoulder pain
 Subjectively unacceptable dysfunction
 Contraindications
 Moderate to severe arthropathy
 Significant bone defects
 Absent deltoid, latissimus dorsi or pectoralis function
 Shoulder stiffness
Superior Capsular Reconstruction
https://www.youtube.com/watch?v=R2UO1eiIJRI
Biceps Tendon
Biceps Tendon
 Tearing
 SLAP
 Subluxation
Shredded
biceps
tendon
Normal
biceps
tendon
Anatomic Shoulder Replacement
 Osteoarthritis
 Intact rotator cuff
 Sufficient remaining bone
 Intact neuromuscular function
Arhtritic shoulder
Anatomic
shoulder
replacement
Reverse Shoulder Replacements
Reverse shoulder replacements are helpful
when treating arthritis associated with
irreparable rotator cuff tears in patients
unable to lift the arm due to tendon tears.
Reverse Shoulder Replacements
Reverse shoulder replacement
Anatomic shoulder replacement
Reverse Shoulder Replacement
 Indications
 Pseudoparalysis
 Antero-superior escape
 Acute 3-4 part fractures in
the elderly
 Nonunion or malunion of
tuberosity
 Failed arthroplasty
 Rheumatoid arthritis
Thank You
Be careful out there.

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Shoulder & Knee Conditions | Work-Related Injuries | South Windsor, Rocky Hill Glastonbury CT

  • 1. Shoulder and knee conditions in work-related Injuries ______JAMES T MAZZARA, MD ORTHOPEDIC ASSOCIATES OF HARTFORD, PC MANCHESTER / ROCKY HILL HARTFORD HOSPITAL GLASTONBURY SURGERY CENTER CONNECTICUT JOINT REPLACEMENT INSTITUTE @ ST FRANCIS HOSPITAL ECHN
  • 2. Contact Information James T Mazzara, MD Orthopedic Associates of Hartford, PC 29 Haynes Street Manchester, CT 06040 _________________ 150 Enterprise Drive Rocky Hill, CT 06067 860-649-2267 www.HartfordSportsOrthopedics.com
  • 3. Why Workers’ Comp Matters  The workers’ compensation system is a medical driven legal compromise described as a “Grand Bargain” between labor and employees
  • 4. Causation  The cause (injury or history of exposure) is medically probable and occurred while at work or with a specific event  The effect is medically probable. Condition is medically diagnosable & consistent with the symptoms, history, examination and clinical studies.
  • 5. Causation  Cause & effect are probably related
  • 6. Causation  Medical findings are compatible with the effects of an injury  Sufficient exposure is present to have caused the condition  The weight of evidence supports the disease as having an occupational rather than non-occupational cause.
  • 7. The Method  1. Identify evidence of disease  2. Review and assess available epidemiologic evidence for a causal relationship  3. Obtain and assess evidence of exposure  4. Consider other relevant factors  5. Judge the validity of the facts (Plausibility)  6. Form conclusions about the work-relatedness of the condition
  • 8. 1. Identify the condition  History  Exam  Clinical studies  Make the correct diagnosis
  • 9. 2. Review & assess epidemiologic evidence for causal relationship  AMA Guides to Disease and Injury Causation  Second edition  Analyses the studies based on study design, quality and value  Strength of evidence  From Very strong evidence to No Risk evidence
  • 10. 3. Risk of Exposure  Evidence of exposure What tasks. Quantified. Standardized Personal measurement Exposure of other workers Defined job category or trade Defined by employer
  • 11. 4. Other relevant factors  How does the individual perform the job  How long on the job, previous job  Has the job changed  Other jobs  Modifications  Previous restrictions or impairments  Pre existing conditions  Hobbies
  • 12. 5. Judge the facts  Temporality  Strength of association  Dose response relationship  Consistency  Coherence to current science  Plausibility  Reversibility
  • 13. Form a Conclusion  Convert data from the whole to data for the individual  May not be intuitively obvious  Work relatedness involves the concept of medical and legal causation  These may be mutually exclusive  Medical causation is scientific  Legal causation from a desire for social justice
  • 14. Justice v. Science  The courts are not based in science  Therefore, laws are not scientifically derived  Accepted wisdom if justice and fair play that underlie the concept of proximate cause are subject to many interpretations in WC law.  Each state may determine for itself what constitutes proximate cause  Judges and legislature can and will substitute convenience for science  The judges have the final say
  • 15. Connecticut Law  Threshold Standard Reasonable degree of medical probability (certainty)  Proving causal relationship is on the claimant  Can only be met by competent medical evidence  At least one doctor must state RDOMC
  • 16. Aggravation  Permanent worsening of a prior, or underlying, condition by an event or exposure
  • 17. Exacerbation  Temporary worsening of a prior, or underlying, condition by an event or exposure that will or has returned to baseline
  • 20. Age-related Cartilage Changes  Older cartilage is less cellular  Cartilage cells do not reproduce after growth plates close  Chondrocytes only in lower layers  Chondrocytes maintain extracellular matrix  Bad news for aging cartilage  Water content decreases
  • 22. Meniscus  Cushion, shock absorber, stabilizer  Compression results in hoop (circumferential) stress  Laterally 70% load transmitted  Medially 50% load transmitted
  • 23. Meniscectomy  Medial meniscectomy  50-70% reduction in femoral condyle contact w/ meniscus  100% increase in contact stress  Lateral meniscectomy  Total meniscectomy: 40-50% decrease in contact area  Increased contact stresses in lateral compartment 200- 300%  Contributes to articular cartilage damage and degeneration
  • 24. Meniscectomy and Laxity  In ACLR, additional MM resection increased whereas MM repair preserved knee laxity in comparison with the ACLR knee with intact menisci.  Neither LM resection or LM repair showed a significant effect on knee laxity.  Surgeons should make every effort to repair the meniscus whenever possible to avoid the residual postoperative laxity present in the meniscus-deficient knee.  Medial Meniscus Resection Increases and Medial Meniscus Repair Preserves Anterior Knee Laxity: A Cohort Study of 4497 Patients With Primary Anterior Cruciate Ligament Reconstruction  Riccardo Cristiani, MD*, Erik Rönnblad, MD, Björn Engström, MD, PhD, ...  First Published October 24, 2017
  • 25. Orthopedic Evaluation  Activity level  Expectations & Goals  Symptoms  Examination  Alignment  Hip exam  Knee Exam  ROM  Meniscal signs  Patellar mobility
  • 26. Radiographic Evaluation  Standing AP  Standing 45o flexion PA  Lateral  Patellofemoral  Each x-ray position tells us something different about the location of problems and the alignment of the knee.
  • 27. X-Rays: Standing Films- AP Standing x-rays are performed with the knee in different positions. Each position reveals something about the location and degree of arthritis or other conditions that may lead to knee pain. This is a standing x-ray with knees straight (extended).
  • 28. X-Rays: Standing Films- PA w/ Flexion This is an x-ray with the knee bent (flexed) 45 degrees. Properly positioned knee x- rays can reveal a degree of arthritis that may be missed on non-weight bearing x-ray views.
  • 29. X-Rays This weight bearing tunnel view can reveal the degree of arthritis not seen on other x-rays This patellar view reveals the position of the patella in mid flexion.
  • 35. Knee Arthroscopy: Microfracture  Full thickness joint cartilage defects  Unstable full thickness lesions  Osteoarthritis with proper knee alignment  Not for partial thickness defects
  • 37. Knee Arthroscopy: Microfracture Cartilage injury Loose cartilage removed After microfracture
  • 40. Microfracture: Predictors of Outcome  Poorer results in certain patients Increased age Malalignment Higher BMI (obesity) Rim height (condition of adjacent cartilage) Proper technique and rehabilitation
  • 41. Autogenous Cartilage Implantation  Focal cartilage defects  Intact meniscus  Meniscus transplant  Normal alignment
  • 42. MACI: Autogenous Chondrocyte Implantation via Collagen Membrane  A sample of the patient’s own cartilage is sent to the lab, regenerated and implanted into a collagen membrane for reimplantation into the patient’s knee.
  • 43. Arthroscopy & Microfracture in the Degenerative Knee  Different than in non- degenerative knees Surrounding cartilage is thinned Dense, sclerotic bone Joint scarring and contractures Synovitis Osteophytes (Bone Spurs)
  • 44. Total Knee Replacement  >50% of patients over 65 have at least one joint with OA  Over 250,000 TKA performed annually  Excellent survival and function in >90% at 10-20 years
  • 45. Knee Osteoarthritis – What does it look like? Knee with OsteoarthritisHealthy Knee Tibia (shin bone) Cartilage Femur (thigh bone)
  • 46. Knee Replacement Surgery  Considered when nonsurgical interventions aren’t alleviating pain  The only long-term solution to knee pain
  • 51. The Shoulder Complex Deltoid Muscle Pectoralis Clavicle (Collar Bone)
  • 54. Rotator Cuff  Supraspinatus  Active in any elevation of the arm  Stabilizes the shoulder joint
  • 55. Rotator Cuff  Infraspinatus  Depressor of the humeral head  Stabilizer of the back of the shoulder
  • 56. Rotator Cuff  Teres Minor  Rotates the shoulder out from the side
  • 57. Rotator Cuff  Subscapularis  Stabilizes the front of the shoulder  Rotates the arm inward
  • 58. Rotator Cuff Balance  Proper function depends upon balance between all muscle and ligament forces around the shoulder
  • 59. Tendon Degeneration  Age-related changes  Decreased vascularity at the tendon attachment to the bone  Leads to weak tendon that tears easily
  • 60. Rotator Cuff Tears  Tears begin where the stresses are the greatest  Tendon fibers fail a few at a time or all at once  Arm may be at rest  Torn fibers retract when torn Humeral Head Partial Supraspinatus Tear
  • 61. Consequences of rupture  Increasing loads applied to the intact fibers  Muscle fibers become detached from the bone resulting in weakness
  • 62. Full Thickness Tears  Loads are concentrated at the margins of the tear  Further tearing occurs with smaller loads  Partial tears become complete  Smaller tears become large  Large tears eventually become unfixable
  • 63. Early Cuff Failure  Compression of the humeral head is less effective  Deltoid pulls head upward  Upward pull of the deltoid results in cuff abrasion & further cuff damage
  • 64. Progressive Tearing  Spacer effect of the cuff is lost  Humeral head displaces superiorly  Biceps tendon eventually ruptures
  • 65. Chronic Cuff Failure  Humeral head forms a joint with the arch above  Secondary joint disease occurs called cuff tear arthropathy
  • 66. Chronic Cuff Tears  Muscle atrophy  Fatty infiltration of muscle belly  Tendon retraction  Bone osteoporosis  Loss of muscle and tendon excursion  Irreversible  Progressively worse Fatty infiltration with muscle wasting Healthy muscle, no fat stripes
  • 67. Prevalence of Rotator Cuff Tears  Cadaver studies 7-40%  MRI & Ultrasound studies 34% of asymptomatic individuals 54% of asymptomatic individuals over 60y  Ultrasound study 13% of asymptomatic individuals: 50-59y 51% of asymptomatic individuals: over 80y
  • 68. Prevalence of Rotator Cuff Tears  40%: no history of strenuous physical labor  50%: no history of trauma  Frequently bilateral  Many heavy laborers never get cuff tears
  • 69. Healing Potential  None without surgery Cuff tears never heal spontaneously Without a blood supply, there is never any chance a cuff healing spontaneously  51% of asymptomatic RCT become symptomatic  Small tears increase in size slowly  Large tears progress more quickly
  • 70. Non Rotator Cuff Shoulder Pain  Pain to the back of shoulder upper back or neck  Pain to top of shoulder  Think arthritis of the neck  Pain beyond the elbow  Think pinched nerve in the neck
  • 71. Radiographs  Acromial shape  Position of humeral head  AC arthritis  Calcific tendinitis  Glenohumeral arthritis  Destructive lesions
  • 72. Partial Tendon Tears: Acromioplasty, Excise and 1O Repair  For tears >50% tendon thickness  Post-op treat same as a full thickness RC repair
  • 73. Rotator Cuff Repair  Arthroscopic  Open repair
  • 74. Irreparable Rotator Cuff Tears  Superior capsular reconstruction For massive irreparable cuff tears No arthritis Younger patients Older patients who do not want a shoulder replacement Restores superior capsule restraining superior translation Allows deltoid and pectoralis to function normally
  • 75. Superior Capsular Reconstruction  Indications  Irreparable Supraspinatus and Infraspinatus tears  Failed non-operative management  Intolerable shoulder pain  Subjectively unacceptable dysfunction  Contraindications  Moderate to severe arthropathy  Significant bone defects  Absent deltoid, latissimus dorsi or pectoralis function  Shoulder stiffness
  • 78. Biceps Tendon  Tearing  SLAP  Subluxation Shredded biceps tendon Normal biceps tendon
  • 79. Anatomic Shoulder Replacement  Osteoarthritis  Intact rotator cuff  Sufficient remaining bone  Intact neuromuscular function Arhtritic shoulder Anatomic shoulder replacement
  • 80. Reverse Shoulder Replacements Reverse shoulder replacements are helpful when treating arthritis associated with irreparable rotator cuff tears in patients unable to lift the arm due to tendon tears.
  • 81. Reverse Shoulder Replacements Reverse shoulder replacement Anatomic shoulder replacement
  • 82. Reverse Shoulder Replacement  Indications  Pseudoparalysis  Antero-superior escape  Acute 3-4 part fractures in the elderly  Nonunion or malunion of tuberosity  Failed arthroplasty  Rheumatoid arthritis
  • 83. Thank You Be careful out there.