https://hartfordsportsorthopedics.com/
In this presentation by Dr. Mazzara, he discusses work-related injuries to the shoulder and knee. This presentation highlights:
Why workers' compensation matters
Justice v. science
Age-related cartilage changes in the knee
Meniscus injuries
Knee arthroscopy
Total knee replacement
Shoulder anatomy
Rotator cuff injuries
Rotator cuff repair
Biceps tendon injuries
Shoulder replacement
Reverse shoulder replacement
To learn more, please visit: https://hartfordsportsorthopedics.com/shoulder-overview-south-windsor-rocky-hill-glastonbury-ct/ and https://hartfordsportsorthopedics.com/knee-anatomy-acl-injury-south-windsor-rocky-hill-glastonbury-ct/.
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Shoulder & Knee Conditions | Work-Related Injuries | South Windsor, Rocky Hill Glastonbury CT
1. Shoulder and knee
conditions in work-related
Injuries
______JAMES T MAZZARA, MD
ORTHOPEDIC ASSOCIATES OF HARTFORD, PC
MANCHESTER / ROCKY HILL
HARTFORD HOSPITAL GLASTONBURY SURGERY CENTER
CONNECTICUT JOINT REPLACEMENT INSTITUTE @ ST FRANCIS
HOSPITAL
ECHN
2. Contact Information
James T Mazzara, MD
Orthopedic Associates of Hartford, PC
29 Haynes Street
Manchester, CT 06040
_________________
150 Enterprise Drive
Rocky Hill, CT 06067
860-649-2267
www.HartfordSportsOrthopedics.com
3. Why Workers’ Comp Matters
The workers’ compensation system is a
medical driven legal compromise described as
a “Grand Bargain” between labor and
employees
4. Causation
The cause (injury or history of exposure)
is medically probable and occurred
while at work or with a specific event
The effect is medically probable.
Condition is medically diagnosable &
consistent with the symptoms, history,
examination and clinical studies.
6. Causation
Medical findings are compatible with
the effects of an injury
Sufficient exposure is present to have
caused the condition
The weight of evidence supports the
disease as having an occupational
rather than non-occupational cause.
7. The Method
1. Identify evidence of disease
2. Review and assess available epidemiologic
evidence for a causal relationship
3. Obtain and assess evidence of exposure
4. Consider other relevant factors
5. Judge the validity of the facts (Plausibility)
6. Form conclusions about the work-relatedness
of the condition
8. 1. Identify the condition
History
Exam
Clinical studies
Make the correct diagnosis
9. 2. Review & assess epidemiologic
evidence for causal relationship
AMA Guides to Disease and Injury Causation
Second edition
Analyses the studies based on study design, quality
and value
Strength of evidence
From Very strong evidence to No Risk evidence
10. 3. Risk of Exposure
Evidence of exposure
What tasks. Quantified. Standardized
Personal measurement
Exposure of other workers
Defined job category or trade
Defined by employer
11. 4. Other relevant factors
How does the individual perform the job
How long on the job, previous job
Has the job changed
Other jobs
Modifications
Previous restrictions or impairments
Pre existing conditions
Hobbies
12. 5. Judge the facts
Temporality
Strength of association
Dose response relationship
Consistency
Coherence to current science
Plausibility
Reversibility
13. Form a Conclusion
Convert data from the whole to data for the individual
May not be intuitively obvious
Work relatedness involves the concept of medical and
legal causation
These may be mutually exclusive
Medical causation is scientific
Legal causation from a desire for social justice
14. Justice v. Science
The courts are not based in science
Therefore, laws are not scientifically derived
Accepted wisdom if justice and fair play that
underlie the concept of proximate cause are
subject to many interpretations in WC law.
Each state may determine for itself what
constitutes proximate cause
Judges and legislature can and will substitute
convenience for science
The judges have the final say
15. Connecticut Law
Threshold Standard
Reasonable degree of medical probability
(certainty)
Proving causal relationship is on the claimant
Can only be met by competent medical evidence
At least one doctor must state RDOMC
20. Age-related Cartilage Changes
Older cartilage is less
cellular
Cartilage cells do not
reproduce after
growth plates close
Chondrocytes only in
lower layers
Chondrocytes
maintain extracellular
matrix
Bad news for aging
cartilage
Water content
decreases
23. Meniscectomy
Medial meniscectomy
50-70% reduction in femoral
condyle contact w/ meniscus
100% increase in contact
stress
Lateral meniscectomy
Total meniscectomy: 40-50%
decrease in contact area
Increased contact stresses in
lateral compartment 200-
300%
Contributes to articular
cartilage damage and
degeneration
24. Meniscectomy and Laxity
In ACLR, additional MM resection increased whereas MM repair
preserved knee laxity in comparison with the ACLR knee with intact
menisci.
Neither LM resection or LM repair showed a significant effect on
knee laxity.
Surgeons should make every effort to repair the meniscus whenever
possible to avoid the residual postoperative laxity present in the
meniscus-deficient knee.
Medial Meniscus Resection Increases and Medial Meniscus Repair Preserves Anterior Knee Laxity:
A Cohort Study of 4497 Patients With Primary Anterior Cruciate Ligament Reconstruction
Riccardo Cristiani, MD*, Erik Rönnblad, MD, Björn Engström, MD, PhD, ...
First Published October 24, 2017
26. Radiographic Evaluation
Standing AP
Standing 45o flexion PA
Lateral
Patellofemoral
Each x-ray position tells us something different about
the location of problems and the alignment of the
knee.
27. X-Rays: Standing Films- AP
Standing x-rays are performed
with the knee in different
positions. Each position reveals
something about the location
and degree of arthritis or other
conditions that may lead to
knee pain.
This is a standing x-ray with
knees straight (extended).
28. X-Rays: Standing Films- PA w/ Flexion
This is an x-ray with the
knee bent (flexed) 45
degrees.
Properly positioned knee x-
rays can reveal a degree of
arthritis that may be missed
on non-weight bearing x-ray
views.
29. X-Rays
This weight bearing tunnel view can
reveal the degree of arthritis not seen
on other x-rays
This patellar view reveals the position of
the patella in mid flexion.
40. Microfracture: Predictors of Outcome
Poorer results in certain
patients
Increased age
Malalignment
Higher BMI (obesity)
Rim height (condition of
adjacent cartilage)
Proper technique and
rehabilitation
42. MACI: Autogenous Chondrocyte Implantation via
Collagen Membrane
A sample of the
patient’s own cartilage
is sent to the lab,
regenerated and
implanted into a
collagen membrane
for reimplantation into
the patient’s knee.
43. Arthroscopy &
Microfracture in the
Degenerative Knee
Different than in non-
degenerative knees
Surrounding cartilage is
thinned
Dense, sclerotic bone
Joint scarring and
contractures
Synovitis
Osteophytes (Bone Spurs)
44. Total Knee Replacement
>50% of patients over 65 have at least one joint with OA
Over 250,000 TKA performed annually
Excellent survival and function in >90% at 10-20 years
45. Knee Osteoarthritis – What does it look
like?
Knee with OsteoarthritisHealthy Knee
Tibia
(shin bone)
Cartilage
Femur
(thigh bone)
46. Knee Replacement Surgery
Considered when
nonsurgical
interventions aren’t
alleviating pain
The only long-term
solution to knee pain
58. Rotator Cuff Balance
Proper function depends
upon balance between all
muscle and ligament forces
around the shoulder
59. Tendon Degeneration
Age-related changes
Decreased vascularity at
the tendon attachment
to the bone
Leads to weak tendon
that tears easily
60. Rotator Cuff Tears
Tears begin where the
stresses are the
greatest
Tendon fibers fail a few
at a time or all at once
Arm may be at rest
Torn fibers retract when
torn
Humeral Head
Partial Supraspinatus
Tear
61. Consequences of rupture
Increasing loads applied to the intact fibers
Muscle fibers become detached from the bone resulting in weakness
62. Full Thickness Tears
Loads are concentrated
at the margins of the
tear
Further tearing occurs
with smaller loads
Partial tears become
complete
Smaller tears become
large
Large tears eventually
become unfixable
63. Early Cuff Failure
Compression of the
humeral head is less
effective
Deltoid pulls head upward
Upward pull of the deltoid
results in cuff abrasion &
further cuff damage
64. Progressive Tearing
Spacer effect of the cuff is lost
Humeral head displaces superiorly
Biceps tendon eventually ruptures
65. Chronic Cuff Failure
Humeral head forms a
joint with the arch above
Secondary joint disease
occurs called cuff tear
arthropathy
66. Chronic Cuff Tears
Muscle atrophy
Fatty infiltration of
muscle belly
Tendon retraction
Bone osteoporosis
Loss of muscle and
tendon excursion
Irreversible
Progressively worse
Fatty
infiltration
with muscle
wasting
Healthy
muscle, no fat
stripes
67. Prevalence of Rotator Cuff Tears
Cadaver studies 7-40%
MRI & Ultrasound studies
34% of asymptomatic individuals
54% of asymptomatic individuals over 60y
Ultrasound study
13% of asymptomatic individuals: 50-59y
51% of asymptomatic individuals: over 80y
68. Prevalence of Rotator Cuff Tears
40%: no history of strenuous physical labor
50%: no history of trauma
Frequently bilateral
Many heavy laborers never get cuff tears
69. Healing Potential
None without surgery
Cuff tears never heal spontaneously
Without a blood supply, there is never any chance
a cuff healing spontaneously
51% of asymptomatic RCT become symptomatic
Small tears increase in size slowly
Large tears progress more quickly
70. Non Rotator Cuff Shoulder Pain
Pain to the back of shoulder
upper back or neck
Pain to top of shoulder
Think arthritis of the neck
Pain beyond the elbow
Think pinched nerve in the neck
71. Radiographs
Acromial shape
Position of humeral head
AC arthritis
Calcific tendinitis
Glenohumeral arthritis
Destructive lesions
72. Partial Tendon Tears: Acromioplasty, Excise and 1O Repair
For tears >50% tendon
thickness
Post-op treat same as a
full thickness RC repair
74. Irreparable Rotator Cuff Tears
Superior capsular reconstruction
For massive irreparable cuff tears
No arthritis
Younger patients
Older patients who do not want a
shoulder replacement
Restores superior capsule restraining
superior translation
Allows deltoid and pectoralis to
function normally
75. Superior Capsular Reconstruction
Indications
Irreparable Supraspinatus and Infraspinatus tears
Failed non-operative management
Intolerable shoulder pain
Subjectively unacceptable dysfunction
Contraindications
Moderate to severe arthropathy
Significant bone defects
Absent deltoid, latissimus dorsi or pectoralis function
Shoulder stiffness
80. Reverse Shoulder Replacements
Reverse shoulder replacements are helpful
when treating arthritis associated with
irreparable rotator cuff tears in patients
unable to lift the arm due to tendon tears.