Tilt table is a padded table that can be elevated from horizontal position to vertical position. It is used in a therapeutic setting for physiological accommodation to upright position, facilitate early weight bearing, cardiovascular conditioning etc.
The Tilt Table Test is used to evaluate causes of syncope (fainting) by reproducing symptoms while closely monitoring the patient. During the test, the patient lies on a table that is tilted to 60-80 degrees for up to 45 minutes. Electrodes monitor heart rate and a blood pressure cuff measures pressure. If no symptoms occur, medication may be administered to increase heart rate and tilt continued. Fainting during the test confirms vasovagal or neurocardiogenic syncope caused by abnormal nervous system reflexes lowering blood pressure and heart rate. The test aims to safely reproduce symptoms to identify causes of fainting.
Utility value of tilt table testing in evaluationUday Prashant
I had presented in CARE Highlights session and book is being published on this topic by LAMBERT publications, Germany
http://www.google.co.in/url?sa=t&rct=j&q=&esrc=s&frm=1&source=web&cd=1&cad=rja&ved=0CCoQFjAA&url=http%3A%2F%2Fwww.amazon.in%2FEvaluation-Unexplained-Syncope-Young-Adults%2Fdp%2F3843373175&ei=lzVtUvbtCIfSrQemkYDwCg&usg=AFQjCNEK_NmIVC5j5LcLSr2hKbYFwMmRuw&sig2=okLwwgOdFiPgw4GPk7mugQ&bvm=bv.55123115,d.bmk
O documento descreve um estudo sobre o teste de inclinação (tilt test) em 348 pacientes. Os principais resultados foram:
1) O tilt test foi positivo em 53,2% dos casos, semelhante aos achados da literatura.
2) A resposta mais comum foi a síncope vasovagal (32% dos pacientes).
3) Foi necessário usar medicação sensibilizadora (Isordil) em quase 90% dos pacientes para induzir a síncope.
This document provides an overview of syncope (transient loss of consciousness). It defines syncope and discusses mechanisms and common causes. Cardiac causes include arrhythmias and structural heart issues. Common non-cardiac causes are neurocardiogenic syncope and orthostatic hypotension. The document outlines how to take a history and perform an exam for a syncope patient. It recommends ECG, monitoring, and risk stratification. The summary emphasizes differentiating cardiac from non-cardiac syncope, as cardiac causes have higher mortality and recommends not missing life-threatening conditions.
This document provides an outline on syncope (transient loss of consciousness). It defines syncope, describes the mechanisms, classifications, clinical features, diagnosis and treatment. Key points include:
- Syncope is caused by transient global hypoperfusion of the brain due to low cardiac output or systemic vascular resistance.
- Causes are classified as cardiac (e.g. arrhythmias, structural issues), non-cardiac (e.g. neurological, metabolic), or unknown.
- Common causes include vasovagal, orthostatic hypotension, arrhythmias, and carotid sinus hypersensitivity.
- Evaluation involves history, physical exam, ECG and sometimes tilt table testing or cardiac
O documento descreve a síncope vasovagal, definindo-a como uma perda súbita da consciência associada à perda do tônus muscular, com recuperação espontânea. Explica que ocorre devido a uma estimulação excessiva do sistema nervoso vagal, causando bradicardia e vasodilatação. Detalha possíveis tratamentos como beta-bloqueadores, mineralocorticóides e antidepressivos para controlar os sintomas.
Tilt table is a padded table that can be elevated from horizontal position to vertical position. It is used in a therapeutic setting for physiological accommodation to upright position, facilitate early weight bearing, cardiovascular conditioning etc.
The Tilt Table Test is used to evaluate causes of syncope (fainting) by reproducing symptoms while closely monitoring the patient. During the test, the patient lies on a table that is tilted to 60-80 degrees for up to 45 minutes. Electrodes monitor heart rate and a blood pressure cuff measures pressure. If no symptoms occur, medication may be administered to increase heart rate and tilt continued. Fainting during the test confirms vasovagal or neurocardiogenic syncope caused by abnormal nervous system reflexes lowering blood pressure and heart rate. The test aims to safely reproduce symptoms to identify causes of fainting.
Utility value of tilt table testing in evaluationUday Prashant
I had presented in CARE Highlights session and book is being published on this topic by LAMBERT publications, Germany
http://www.google.co.in/url?sa=t&rct=j&q=&esrc=s&frm=1&source=web&cd=1&cad=rja&ved=0CCoQFjAA&url=http%3A%2F%2Fwww.amazon.in%2FEvaluation-Unexplained-Syncope-Young-Adults%2Fdp%2F3843373175&ei=lzVtUvbtCIfSrQemkYDwCg&usg=AFQjCNEK_NmIVC5j5LcLSr2hKbYFwMmRuw&sig2=okLwwgOdFiPgw4GPk7mugQ&bvm=bv.55123115,d.bmk
O documento descreve um estudo sobre o teste de inclinação (tilt test) em 348 pacientes. Os principais resultados foram:
1) O tilt test foi positivo em 53,2% dos casos, semelhante aos achados da literatura.
2) A resposta mais comum foi a síncope vasovagal (32% dos pacientes).
3) Foi necessário usar medicação sensibilizadora (Isordil) em quase 90% dos pacientes para induzir a síncope.
This document provides an overview of syncope (transient loss of consciousness). It defines syncope and discusses mechanisms and common causes. Cardiac causes include arrhythmias and structural heart issues. Common non-cardiac causes are neurocardiogenic syncope and orthostatic hypotension. The document outlines how to take a history and perform an exam for a syncope patient. It recommends ECG, monitoring, and risk stratification. The summary emphasizes differentiating cardiac from non-cardiac syncope, as cardiac causes have higher mortality and recommends not missing life-threatening conditions.
This document provides an outline on syncope (transient loss of consciousness). It defines syncope, describes the mechanisms, classifications, clinical features, diagnosis and treatment. Key points include:
- Syncope is caused by transient global hypoperfusion of the brain due to low cardiac output or systemic vascular resistance.
- Causes are classified as cardiac (e.g. arrhythmias, structural issues), non-cardiac (e.g. neurological, metabolic), or unknown.
- Common causes include vasovagal, orthostatic hypotension, arrhythmias, and carotid sinus hypersensitivity.
- Evaluation involves history, physical exam, ECG and sometimes tilt table testing or cardiac
O documento descreve a síncope vasovagal, definindo-a como uma perda súbita da consciência associada à perda do tônus muscular, com recuperação espontânea. Explica que ocorre devido a uma estimulação excessiva do sistema nervoso vagal, causando bradicardia e vasodilatação. Detalha possíveis tratamentos como beta-bloqueadores, mineralocorticóides e antidepressivos para controlar os sintomas.
El documento describe el síncope neurocardiogénico, una pérdida breve de conciencia causada por una disminución transitoria del flujo sanguíneo al cerebro. Define el síncope y explica que es uno de los síncopes reflejos más comunes, caracterizado por la pérdida súbita de la conciencia con incapacidad de mantenerse de pie y recuperación espontánea. También enumera posibles síntomas durante un episodio sincopal como palidez, sudoración, piel fría, pupilas dilatadas, mare
El documento describe las diferentes causas y tipos de síncope. El síncope se define como una pérdida transitoria de conocimiento debido a una hipoperfusión cerebral transitoria y puede deberse a causas cardíacas, vasovagales, ortostáticas u otras. La evaluación de un paciente con síncope incluye la historia clínica, examen físico y diversos exámenes para determinar la causa subyacente.
Syncope is defined as a transient loss of consciousness due to decreased cerebral blood flow. It is commonly caused by neurally-mediated reflexes, orthostatic hypotension, or cardiac arrhythmias. Evaluation involves determining the cause through history, physical exam, ECG and tests like tilt table testing, echocardiogram, and cardiac monitoring. Treatment depends on the underlying cause and may include lifestyle changes, medications, pacemakers, or defibrillators.
Este documento describe estimuladores del tono vagal como la fenilefrina y la metoxamina. Estas sustancias actúan a través de receptores adrenérgicos para causar vasoconstricción y aumentar la presión arterial, además de estimular el sistema vagal causando bradicardia. Se usan para tratar hipotensión y algunas arritmias. Su acción depende del receptor adrenérgico estimulado y si es de acción directa, indirecta o mixta.
Este documento presenta las guías de práctica clínica de la Sociedad Europea de Cardiología para el manejo del síndrome coronario agudo sin elevación del segmento ST. Proporciona recomendaciones sobre el diagnóstico, la evaluación del riesgo, el tratamiento farmacológico y de revascularización, así como consideraciones especiales para poblaciones como ancianos y diabéticos. Fue desarrollado por un grupo de trabajo de expertos y revisado por comités de la ESC.
El documento describe el síncope neurocardiogénico, una pérdida breve de conciencia causada por una disminución transitoria del flujo sanguíneo al cerebro. Define el síncope y explica que es uno de los síncopes reflejos más comunes, caracterizado por la pérdida súbita de la conciencia con incapacidad de mantenerse de pie y recuperación espontánea. También enumera posibles síntomas durante un episodio sincopal como palidez, sudoración, piel fría, pupilas dilatadas, mare
El documento describe las diferentes causas y tipos de síncope. El síncope se define como una pérdida transitoria de conocimiento debido a una hipoperfusión cerebral transitoria y puede deberse a causas cardíacas, vasovagales, ortostáticas u otras. La evaluación de un paciente con síncope incluye la historia clínica, examen físico y diversos exámenes para determinar la causa subyacente.
Syncope is defined as a transient loss of consciousness due to decreased cerebral blood flow. It is commonly caused by neurally-mediated reflexes, orthostatic hypotension, or cardiac arrhythmias. Evaluation involves determining the cause through history, physical exam, ECG and tests like tilt table testing, echocardiogram, and cardiac monitoring. Treatment depends on the underlying cause and may include lifestyle changes, medications, pacemakers, or defibrillators.
Este documento describe estimuladores del tono vagal como la fenilefrina y la metoxamina. Estas sustancias actúan a través de receptores adrenérgicos para causar vasoconstricción y aumentar la presión arterial, además de estimular el sistema vagal causando bradicardia. Se usan para tratar hipotensión y algunas arritmias. Su acción depende del receptor adrenérgico estimulado y si es de acción directa, indirecta o mixta.
Este documento presenta las guías de práctica clínica de la Sociedad Europea de Cardiología para el manejo del síndrome coronario agudo sin elevación del segmento ST. Proporciona recomendaciones sobre el diagnóstico, la evaluación del riesgo, el tratamiento farmacológico y de revascularización, así como consideraciones especiales para poblaciones como ancianos y diabéticos. Fue desarrollado por un grupo de trabajo de expertos y revisado por comités de la ESC.
Guía de práctica clínica de la ESC para el manejo del síndrome coronario agud...
Editor's Notes
Osteoporosis is a condition characterized by a decrease in the density of bone, decreasing its strength and resulting in fragile bones. This disorder of the skeleton weakens the bone and results in frequent fractures (breaks) in the bones. Normal bone is composed of protein, collagen, and calcium, all of which give bone its strength. Bones that are affected by osteoporosis can break (fracture) with relatively minor injury that normally would not cause a bone to fracture.//This device is what we called the Magnetoteraphy device. This picture shown is a model known as The PMT Qs devices. This PMT Qs devices apply ELF (Extremely Low Frequency) magnetic fields in parallel to the longitudinal axis of the body using solenoids, or perpendicular to the area to be treated using the Flexa applicators. The flexible applicators are particularly suited for dependant treatment and for healing fractures of the long bones. The smaller solenoids are ideal for carrying out magnetotherapy treatments that are specific for the limbs, while the larger solenoids are perfect for use on more extended areas, up to total body treatment, which is particularly effective for diffused arthrosis and osteoporosis. However, not all commercialized magnetoteraphy that are available in the market are suitable to treat osteoporosis.//