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PRINCIPLES OF TREATMENT
OF ABSCESS AND
PHLEGMONS OF
MAXILLOFACIAL REGION
Kritika
Singh
Parth
Parmar
INFECTION
• Definition: Infection is the pathological state
resulting from the invasion of the body by
pathogenic microorganisms
.•The reaction of the tissues to the presence of
these microorganisms and the toxins generated
by them is INFLAMMATION
TYPES OF INFECTION
• Bacterial Infection: not only one particular
specie, but due to a mixture of species which
make up the oral flora
.• Fungal Infection: Actinomycosis
• Viral Infection: Not recognized because they
are complicated early by secondary bacterial
infection
.Parasitic infection: Very rare( ex. Lyshmoniasis)
ETIOLOGY OF INFECTION
The majority of oral, facial, and neck infections
are odontogenic in origin.
Although they may be caused by infections
from
• Antrum (maxillary sinus)
• Major salivary glands
• Specific Infections of jaws
(osteoradionecrosis, osteomyelitis)
ODONTOGENIC CAUSES OF
INFECTIONS
1. Decay (caries) reaching the dental pulp= pulpitis,
this in turn spreads to supporting bone resulting in
periapical abscess which in turn may spread
subperiosteally
2. Periapical abscess may occur in seemingly intact
but devitalized teeth (trauma, cracks or decay under
fillings)
3. Periodontal diseases
4. Erupting teeth (especially partially impacted lower
third molars) can result in inflammation and infection
PREDISPOSING FACTORS FOR
ACUTE ORAL INFECTIONS
1 Endocrine disturbances
II. Nutritional deficiency (decreases resistance)
III. Chemical compounds used in dentistry (arsenic)
IV. Blood disorders (leukemia, anemia)
V. General diseases (syphilis, TB)
VI. Immunological diseases (AIDS)
VII. Trauma - Fractures of jaw,Pressure type of
anesthesia, Improper use of surgical burs without
cooling
MICROBIOLOGY
Odontogenic infections are multimicrobial:
• Gram (+) cocci, aerobic and anaerobic:
• Streptococci and their anaerobic counterpart,
peptostreptococci
• Staphylococci, and their anaerobic counterpart, peptococci
• Gram (+) rods:
• Lactobacillus, diphtheroids, Actinomyces
• Gram (-) rods:
RATE OF SPREAD OF INFECTION
Rate depends on:
• Virulence of the invading microbes
• Dosage or number of these microbes
• Host resistanceSeverity of
infection=(Virulence x Dose)/Resistance
CLINICALLY ODONTOGENIC
INFECTIONS CAN BE
DISTINGUISHED IN THREE
PERIODS:
• Period of periapical of dento-alveolar abscess (in
which the initial lesion develops)
• Period of extension to the adjacent bone and facial
spaces
• Period of serious complications (embolism,
septicemia, pyemia)
ANATOMICAL FACTORS
INFLUENCING THE DIRECTION OF
SPREAD WITHIN TISSUE
1. The site of the source of infection (maxilla or
mandible and even the particular segment of the jaw)
.2. The point at which pus escapes from the bone and
discharges into the soft tissues (linguopalatally or
labiobuccally).
3. The natural barriers to the spread of pus in the
tissues, such as by layers of fascia or muscle or the
jaw bones themselves.
FACIAL SPACES
Fascial spaces are fascia-lined areas that can be
eroded or distended by purulent exudate
.• These areas are potential spaces that do not
exist in healthy people but become filled during
infections.
PRINCIPLES OF TREATMENT OF
OROFACIAL INFECTIONS
1. Remove the cause.
2.Establish drainage.
3.Institute antibiotic therapy.
4.Supportive care, including proper rest and nutrition.
BASIC PRINCIPLES OF LOCAL
TREATMENT
Surgical treatment of abscess and phlegmon is based
on the pus drainage, and therefore in all cases the
following steps should be followed.
1. Incision
2. Opening the abscess
3.Taking of smear to determine the bacteria
4.Drainage of abscess
ABSCESS OF OROFACIAL SPACES
CANINE FOSSA(INFRA-ORBITAL)
Borders:
• Superior: inferior margin of the orbit
• Inferior: alveolar process of maxilla
Mesial: margin of periform aperture
• Lateral: zygomatic-maxillary suture
CANINE FOSSA INFECTION
Odontogenic origin:
maxillary canines and premolars
Nonodontogenic:
• Skin infections
• TraumaHematoma
Infection can be superficial (due to dermatologic
infections) or can be deep (between muscles and
bone-odontogenic infection), and the infections
quickly spreads from superficial to deep and vice
CLINICAL PRESENTATION (CANINE
FOSSA)
Edema, localized in the infraorbital region, which
spreads towards the lower eyelid and side of the nose
as far as the corner of the mouth
.• There is also obliteration of the nasolabial fold, and
somewhat of the mucolabial fold.
• The edema at the infraorbital region is painful and
there is fluctuation during palpation, and later on the
skin becomes taut and shiny due to suppuration, while
its color is reddish(fig)
TREATMENT OF CANINE FOSSA
ABSCESS
1. The incision for drainage is performed intraorally at
the mucobuccal fold (parallel to the alveolar bone), in
the canine region.
2. A hemostat is then inserted, which is placed at the
depth of the purulent accumulation until it comes into
contact with bone, while the index finger of the
nondominant hand palpates the infraorbital margin.
3. Finally, a rubber drain is placed, which is stabilized
with a suture on the mucosa.
BUCCAL SPACE
Borders:
• Superior: inferior margin of zygomatic arch
Inferior: Inferior margin of mandible
Anterior: Corner of the mouth
Posterior: anterior margin of Masseter muscle
Internal: Buccinator muscle
Layers:
• SkinSuperficial fascia
Buccinator muscle
BUCCAL SPACE INFECTION
Odontogenic: (most frequently)maxillary and
mandibular posterior teeth.
Nonodontogenic: adenophlegmon of facial lymph
nodes.
*Secondary spread of infection:
• Superiorly: pterygopalatine space
• Inferiorly: pterygomandibular space
CLINICAL PRESENTATION OF
BUCCAL ABSCESS
• Swelling of the cheek, which extends from the
zygomatic arch as far as the inferior border of the
mandible, and from the anterior border of the ramus
to the corner of themouth
.• The skin appears taut and red, with or without
fluctuation of the abscess.
• There can be intraoral bulging.
TREATMENT OF BUCCAL SPACE
ABSCESS
Access to the buccal space is usually intraoral for three main
reasons:
1. Because the abscess fluctuates intraorally in the majority of
cases.
2 . To avoid injuring the facial nerve.
3. For esthetic reasons.
The intraoral incision is made at the posterior region of the
mouth, in an anteroposterior direction and very carefully in
order to avoid injury of the parotid duct. A hemostat is then
used to explore the space thoroughly.
An extraoral incision is made when intraoral access would not
SUBMENTAL SPACE
Borders:
• Lateral: anterior bellies of the right and left Digastric
muscles
• Anterior: internal border of corpus of mandible
• Posterior: Hyoid bone
• Roof: Mylohoid muscle
• Lower margin: Skin and Platysma muscle
SUBMENTAL SPACE INFECTION
Odontogenic: from frontal teeth of the mandible
Nonodontogenic: adenophlegmon or trauma
*Secondary spread: from submandibular or sublingual
spaces
CLINICAL PRESENTATION OF
SUBMENTAL ABSCESS
Edema in submental region which can spread to
submandibular space.
• Palpation is painful and fluctuation is present
• Hyperemia of skin
• Functional disturbances in protrusion of the jaw
• Dysphagia or discomfort while swallowing can be
present.
THANK YOU…

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Principles of treatment of abscess.pptx

  • 1. PRINCIPLES OF TREATMENT OF ABSCESS AND PHLEGMONS OF MAXILLOFACIAL REGION Kritika Singh Parth Parmar
  • 2. INFECTION • Definition: Infection is the pathological state resulting from the invasion of the body by pathogenic microorganisms .•The reaction of the tissues to the presence of these microorganisms and the toxins generated by them is INFLAMMATION
  • 3. TYPES OF INFECTION • Bacterial Infection: not only one particular specie, but due to a mixture of species which make up the oral flora .• Fungal Infection: Actinomycosis • Viral Infection: Not recognized because they are complicated early by secondary bacterial infection .Parasitic infection: Very rare( ex. Lyshmoniasis)
  • 4. ETIOLOGY OF INFECTION The majority of oral, facial, and neck infections are odontogenic in origin. Although they may be caused by infections from • Antrum (maxillary sinus) • Major salivary glands • Specific Infections of jaws (osteoradionecrosis, osteomyelitis)
  • 5. ODONTOGENIC CAUSES OF INFECTIONS 1. Decay (caries) reaching the dental pulp= pulpitis, this in turn spreads to supporting bone resulting in periapical abscess which in turn may spread subperiosteally 2. Periapical abscess may occur in seemingly intact but devitalized teeth (trauma, cracks or decay under fillings) 3. Periodontal diseases 4. Erupting teeth (especially partially impacted lower third molars) can result in inflammation and infection
  • 6. PREDISPOSING FACTORS FOR ACUTE ORAL INFECTIONS 1 Endocrine disturbances II. Nutritional deficiency (decreases resistance) III. Chemical compounds used in dentistry (arsenic) IV. Blood disorders (leukemia, anemia) V. General diseases (syphilis, TB) VI. Immunological diseases (AIDS) VII. Trauma - Fractures of jaw,Pressure type of anesthesia, Improper use of surgical burs without cooling
  • 7. MICROBIOLOGY Odontogenic infections are multimicrobial: • Gram (+) cocci, aerobic and anaerobic: • Streptococci and their anaerobic counterpart, peptostreptococci • Staphylococci, and their anaerobic counterpart, peptococci • Gram (+) rods: • Lactobacillus, diphtheroids, Actinomyces • Gram (-) rods:
  • 8. RATE OF SPREAD OF INFECTION Rate depends on: • Virulence of the invading microbes • Dosage or number of these microbes • Host resistanceSeverity of infection=(Virulence x Dose)/Resistance
  • 9. CLINICALLY ODONTOGENIC INFECTIONS CAN BE DISTINGUISHED IN THREE PERIODS: • Period of periapical of dento-alveolar abscess (in which the initial lesion develops) • Period of extension to the adjacent bone and facial spaces • Period of serious complications (embolism, septicemia, pyemia)
  • 10. ANATOMICAL FACTORS INFLUENCING THE DIRECTION OF SPREAD WITHIN TISSUE 1. The site of the source of infection (maxilla or mandible and even the particular segment of the jaw) .2. The point at which pus escapes from the bone and discharges into the soft tissues (linguopalatally or labiobuccally). 3. The natural barriers to the spread of pus in the tissues, such as by layers of fascia or muscle or the jaw bones themselves.
  • 11. FACIAL SPACES Fascial spaces are fascia-lined areas that can be eroded or distended by purulent exudate .• These areas are potential spaces that do not exist in healthy people but become filled during infections.
  • 12. PRINCIPLES OF TREATMENT OF OROFACIAL INFECTIONS 1. Remove the cause. 2.Establish drainage. 3.Institute antibiotic therapy. 4.Supportive care, including proper rest and nutrition.
  • 13. BASIC PRINCIPLES OF LOCAL TREATMENT Surgical treatment of abscess and phlegmon is based on the pus drainage, and therefore in all cases the following steps should be followed. 1. Incision 2. Opening the abscess 3.Taking of smear to determine the bacteria 4.Drainage of abscess
  • 15. CANINE FOSSA(INFRA-ORBITAL) Borders: • Superior: inferior margin of the orbit • Inferior: alveolar process of maxilla Mesial: margin of periform aperture • Lateral: zygomatic-maxillary suture
  • 16. CANINE FOSSA INFECTION Odontogenic origin: maxillary canines and premolars Nonodontogenic: • Skin infections • TraumaHematoma Infection can be superficial (due to dermatologic infections) or can be deep (between muscles and bone-odontogenic infection), and the infections quickly spreads from superficial to deep and vice
  • 17. CLINICAL PRESENTATION (CANINE FOSSA) Edema, localized in the infraorbital region, which spreads towards the lower eyelid and side of the nose as far as the corner of the mouth .• There is also obliteration of the nasolabial fold, and somewhat of the mucolabial fold. • The edema at the infraorbital region is painful and there is fluctuation during palpation, and later on the skin becomes taut and shiny due to suppuration, while its color is reddish(fig)
  • 18. TREATMENT OF CANINE FOSSA ABSCESS 1. The incision for drainage is performed intraorally at the mucobuccal fold (parallel to the alveolar bone), in the canine region. 2. A hemostat is then inserted, which is placed at the depth of the purulent accumulation until it comes into contact with bone, while the index finger of the nondominant hand palpates the infraorbital margin. 3. Finally, a rubber drain is placed, which is stabilized with a suture on the mucosa.
  • 19. BUCCAL SPACE Borders: • Superior: inferior margin of zygomatic arch Inferior: Inferior margin of mandible Anterior: Corner of the mouth Posterior: anterior margin of Masseter muscle Internal: Buccinator muscle Layers: • SkinSuperficial fascia Buccinator muscle
  • 20. BUCCAL SPACE INFECTION Odontogenic: (most frequently)maxillary and mandibular posterior teeth. Nonodontogenic: adenophlegmon of facial lymph nodes. *Secondary spread of infection: • Superiorly: pterygopalatine space • Inferiorly: pterygomandibular space
  • 21. CLINICAL PRESENTATION OF BUCCAL ABSCESS • Swelling of the cheek, which extends from the zygomatic arch as far as the inferior border of the mandible, and from the anterior border of the ramus to the corner of themouth .• The skin appears taut and red, with or without fluctuation of the abscess. • There can be intraoral bulging.
  • 22. TREATMENT OF BUCCAL SPACE ABSCESS Access to the buccal space is usually intraoral for three main reasons: 1. Because the abscess fluctuates intraorally in the majority of cases. 2 . To avoid injuring the facial nerve. 3. For esthetic reasons. The intraoral incision is made at the posterior region of the mouth, in an anteroposterior direction and very carefully in order to avoid injury of the parotid duct. A hemostat is then used to explore the space thoroughly. An extraoral incision is made when intraoral access would not
  • 23. SUBMENTAL SPACE Borders: • Lateral: anterior bellies of the right and left Digastric muscles • Anterior: internal border of corpus of mandible • Posterior: Hyoid bone • Roof: Mylohoid muscle • Lower margin: Skin and Platysma muscle
  • 24. SUBMENTAL SPACE INFECTION Odontogenic: from frontal teeth of the mandible Nonodontogenic: adenophlegmon or trauma *Secondary spread: from submandibular or sublingual spaces
  • 25. CLINICAL PRESENTATION OF SUBMENTAL ABSCESS Edema in submental region which can spread to submandibular space. • Palpation is painful and fluctuation is present • Hyperemia of skin • Functional disturbances in protrusion of the jaw • Dysphagia or discomfort while swallowing can be present.