4. Example :
M.A.M , a 40 yrs old male patient , from el maragha ,Sohag
.He works as an engineer , married for 12 years now and
has 2 off spring , the youngest is 3 years old and the oldest
is 5 years old . He is a heavy smoker (20 CPD for 14 years).
He is Rt handed.
N.B:
Smoking index = CPD X total duration in years
Mild if <100
Moderate if 101-300
9. HISTORY OF
PRESENT ILLNESS
1-Motor
2-Sensory
3-Cranial nerves
4-Speech
5-Sphincteric
6-Cognition
7-Symptoms of
increased ICP.
8-Others :Sleep
disturbances - -
Autonomic Dysfunction.
This Photo by Unknown Author is licensed under CC BY
14. MOTOR SYSTEM
MOTOR WEAKNESS:
1- OCD>>>Onset :sudden دقائق
–
ساعات
Acute أسبوعين
subacute أسبوعين
–
شهرين
Chronic >شهرين
Course : regressive – Progressive - Stationary
Duration
15. MOTOR SYSTEM
MOTOR WEAKNESS:
2-Distribution :
Rt vs lt
Symmertcal or Asymmetrical
UL vs LL
Distal vs proximal
Flexors vs extensors
3-Tone
4-Fasciculations (twitches)
5-Wasting
6-Ambulance : without support - with minimal support – with
maximum
support - wheel chair bound – bed ridden
16. EXAMPLE :
The condition started 10 days ago when the patient developed
weakness of acute onset , regressive course of Rt UL & LL . Distal
more than proximal . The patient felt his limb flai. There was no
wasting or muscle twitches . The ptient is ambulant with maximal
support . there were no manifestations affecting the other side
17. MOTOR SYSTEM :
INVOLUNTARY MOVEMENTS:
Fits :
Attacks of involuntary movement, could occur during sleep
1. Description
a) Prodroma
b) Aura
c) Ictus
d) Post Ictal state
2. Duration
3. Frequency
18. MOTOR SYSTEM
INVOLUNTARY MOVEMENTS:
Tremors
1-OCD
2-Distribution
3-Description
a) Static → Parkonism
b) Kinetic → Ataxia
c) Postural
Fine → Anxiety, Thyrotoxicosis, senile, familial (essential)
Course (Flapping) → Organ Failure
4- Effect of stress: ↑
5- Effect of movement: ↓
6- Effect of sleep: x
7-interfering with activities of daily living.
19.
20. MOTOR SYSTEM
COORDINATION
Motor Incoordination : Tremors & Gait
Swaying from side to side during walking
Side Difference: no/more to the Rt/Lt side
Q: ممكن تمشي لما ؟ توازن بعدم تحس ممكن هل ؟ ايه مشيتك اخبار
تحدف
؟ ناحية علي
Effect on dim light:
a) No change: Cerebellar
b) Increase: Mixed cerebellar & deep sensory
c) Only in dim light: Deep sensory
3. Association
a) Staccato speech, nystagmus: Cerebellar
b) Basin sign, Stamping gait: Deep Sensory
21. EXAMPLE :
The patient complains of Abnormal involuntary
movements affecting Rt UL and LL that occur on action
and increase on approaching the target . He also
experiences unsteadiness of gait with deviation to the
Rt on walking which is of the same onset and course
as weakness
22. HISTORY OF
PRESENT ILLNESS
1-Motor
2-Sensory
3-Cranial nerves
4-Speech
5-Sphincteric
6-Cognition
7-Symptoms of
increased ICP.
8-Others :Sleep
disturbances - -
Autonomic Dysfunction.
This Photo by Unknown Author is licensed under CC BY
25. Define pain and parasthesia in terms
of the following :
-Type and character
-Location and distribution
-Radiation
-Severity
-Persistent or in attacks
-PPT factors: stress – touch- sleep
deprivation- neck movements
-Relieving factors eg : sleep , stress
management
41. HISTORY OF
PRESENT ILLNESS
1-Motor
2-Sensory
3-Cranial nerves
4-Speech
5-Sphincteric
6-Cognition
7-Symptoms of
increased ICP.
8-Others :Sleep
disturbances - -
Autonomic Dysfunction.
This Photo by Unknown Author is licensed under CC BY
42. SPEECH & LANGUAGE
-change in speech >>>become slurred ،, Volume of speech, tone
Q: ؟ الكالم في لته فيه كان او عليك تقل لسانك هل
-Fluency
-word finding difficulty
-understand spoken speech
-difficulty in naming objects.
-difficulty in reading
-difficulty in writing
N.B: donot forget OCD
43. IMPORTANT SPEECH PROBLEMS:
Dysarthria: Difficulty in articulation of speech but the patient can
understand spoken words.
Motor Aphasia: inability to speak but the patient can
understand spoken words.
Sensory Aphasia: inability to understand spoken words.
Mixed Aphasia (Global) : inability to speak with inability to
understand spoken words.
44. HISTORY OF
PRESENT ILLNESS
1-Motor
2-Sensory
3-Cranial nerves
4-Speech
5-Sphincteric
6-Cognition
7-Symptoms of
increased ICP.
8-Others :Sleep
disturbances - -
Autonomic Dysfunction.
This Photo by Unknown Author is licensed under CC BY
47. HISTORY OF
PRESENT ILLNESS
1-Motor
2-Sensory
3-Cranial nerves
4-Speech
5-Sphincteric
6-
Cognition
7-Symptoms of
increased ICP.
8-Others :Sleep
disturbances - -
Autonomic Dysfunction.
This Photo by Unknown Author is licensed under CC BY
48. COGNITION
-Take history from the relative in addition to the patient
-conscious level.
-Attention
-orientation
-Memory
-Calculation
-Apraxia: inability to do previously learnt actions.
-Behaviour
-Agnosia : inability to recognize faces , objects..etc
-Mood disturbances
49. HISTORY OF
PRESENT ILLNESS
1-Motor
2-Sensory
3-Cranial nerves
4-Speech
5-Sphincteric
6-Cognition
7-
Symptoms
of increased
ICP.
8-Others :Sleep
disturbances - -
Autonomic Dysfunction.
This Photo by Unknown Author is licensed under CC BY
50. SYMPTOMS OF ICP:
Headache:
– Character: Bursting,
– Location: Allover
– Timing: more severe in early morning
– What aggravate: ↑ by leaning forward & coughing – What relief it: ↓
partially by vomiting & analgesics
Vomiting:
– Character: Projectile (propulsive not preceded by nausea), – Timing:
common in the morning
– Relation to meals : no
Blurring of vision: (due to papilloedema): the details of image is not
apparent even from a near distance.
Convulsions: Focal or generalized
Alteration of consciousness: drowsy, stupor or coma.
51. HISTORY OF
PRESENT ILLNESS
1-Motor
2-Sensory
3-Cranial nerves
4-Speech
5-Sphincteric
6-Cognition
7-Symptoms of increased ICP.
8-Others
:Sleep
disturbances
- -Autonomic
Dysfunction.
This Photo by Unknown Author is licensed under CC BY
54. Past History
-Similar attacks .
-Previous illnesses .
-Trauma or accident
-operations.
-blood transfusion
-Previous medication.
-In children, a brief history
of motor & mental
58. CASE
FORMULATI
ON
Short summary of:
• Relevant points in personal
history : Age , sex , special
habits.
.Relevant family and past
history.
.OCD
.Patient’s symptoms
formulated in scientific terms .
60. DIAGNOSIS: ( PROVISIONAL )
1- Clinical diagnosis
• According to motor deficite e.g.
– Rt Sided hemiplgia, Rt UMNL VII & XII.
– Spinal Paraplegia, Painless Retention of Urine.
– Mixed Cerebellar & Deep sensory ataxia.
61. CONT. DIAGNOSIS:
2- Anatomical diagnosis
• Whether it is focal, systemic or disseminated or multifocal.
– Focal:
All symptoms could be explained by one area in the nervous system (Not respecting neurological
systems).
– Systemic:
Bilateral & symmetric distribution of the whole system or systems (Selective affection of 1 or more
neurological systems that could be explained by well known systemic disease). EX :
PD
– Disseminated:
Bilateral & asymmetric (Dissemination in time & space). EX; MS
62. CONT: DIAGNOSIS
• Where is the Lesion? Eg:
– Lt Internal capsule.
– Bilateral Cerebellar tracts, Posterior column.
– Rt Optic Nerve, Lt Cerebelar Tract, Rt Δ Tract.
63. CONT: DIAGNOSIS:
What is the lesion ? VITAMIN C & D
Vascular>>>>sudden onset , regressive or stationary course.
Infectious/Inflammatory>>> rapid onset and regressive course
Traumatic>>> sudden onset ,regressive or stationary course.
Autoimmune
Metabolic/Toxic
Idiopathhic
Neoplastic>>>gradual onset and progressive course
Congenital>> onset since birth and stationary course
Degenerative / Hereditary >>> gradual onset and progressive