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ANEURYSM
DR. BIPUL
THAKUR
1
INTRODUCTION
• The term ANEURYSM is derived from the Greek word ANEURYSMA
meaning “ a widening”.
• Normal arterial diameter is dependent on age , gender, body size
and other factors.
• An Aneurysm is defined as a localized dilation at least 50 % larger
than an adjacent normal portion of the same artery.
• ECTASIA- Arterial dilation less than 50% above normal
• ARTERIOMEGALY– Diffuse arterial enlargement involving several
arterial segments with an increase in diameter greater than 50%
2
3
CLASSIFICATION
True/False Aneurysm
 True aneurysm : contains all layers of arterial wall.
 False aneurysm :dilation covered by thick fibrinous capsule.
4
PSEUDOANEURYSM
 Injury to wall of vessel allows blood to escape from vessel into
adjacent tissue.
 Extravasated blood coagulates and becomes a mass along side the
vessel.
 This mass of blood (hematoma) gives the impression that there is
an aneurysm
5
6
BASED ON LOCATION
LOCATION FREQUENCY
• ABDOMINAL AORTA 65%
• THORACIC AORTA 19%
• AA + ILIAC 13%
• THORACOABDOMINAL 2%
• ISOLATED ILIAC 1%
7
LOCATION-PERIPHERAL
LOCATION FREQUENCY
 POPLITEAL 70%
 FEMORAL together make upto 90%
 CAROTID 4%
 SUBCLAVIAN 2%
 CEREBRAL 2%
 SPLENIC 1%
 MESENTRIC 0.5%
 RENAL 0.5%
8
BASED ON MORPHOLOGY
 FUSIFORM:
SYMMETRICAL CIRCUMFERENTIAL ENLARGEMENT INVOLVING
ALL LAYERS OF THE ARTERY WALL.
 SACCULAR:
ANEURYSMAL DEGENERATION AFFECTING ONLY PART OF THE
ARTERIAL CIRCUMFERENCE.
9
10
ABDOMINAL AORTIC ANEURYSM
 INCIDENCE: 2% TO 5% OF ALL MEN OVER 60 YRS
 COMMON IN MEN ABOVE 60YRS
PEAK OF 5% AT 80 YRS IN MEN
PEAK OF 4.5% AT 90 YRS IN WOMEN
 GREATER - FIRST DEGREE RELATIVES
TALLER INDIVIDUAL
11
Risk Factors
 Atherosclerosis (90%)
-Smoking
-Hyperlipidemia
- Diabetes
 Gender
- Males > Females
 Age
 Familial History
- 20% of patients with AA have 1st Degree relative
 Connective tissue disease
- Marfan Syndrome- defect in Fibrillin structure
- Ehlers-Danlos - defect in pro-collagen type 3
12
13
14
AAA Sequelae
 Natural history :
- gradual and/or sporadic expansion
- accumulation of mural thrombus
 Complications
- rupture
- thromboembolic events
- compression of adjacent structures
15
Progression of AAA
 Pathological changes cause the aorta wall to
- become thinner
- bulge
- rupture
16
AAA ANNUAL RUPTURE RISK 17
American Association for Vascular Surgery and Society for
Vascular Surgery
 Risk of rupture of an abdominal aortic aneurysm
(AAA) over time according to the first measurement
of aneurysm diameter in 1792 men and 465 women.
The risk of rupture increased markedly in aneurysms
larger than 5.5 cm in diameter
18
Powell, JT, Greenhalgh, RM, N Engl J Med 2003; 348:1895
CLINICAL PRESENTATION
AAA are asymptomatic before rupture in 75%
 Abdominal palpation may show a pulsatile abdominal mass.
 Vague abdominal and back discomfort.
 Large aneurysms – GI symptoms– early satiety and vomiting.
 Inflammatory aneurysms- pain /fever /ureteral obstruction
19
CLINICAL PRESENTATION
 AAA RUPTURE
 Syncope
 Back abdominal pain
 Shock
 Sudden death (rupture)
-Ripping or tearing pain
- Atypical pain (groin, flank., hip, bladder )
-Nausea ,vomiting ,tenesmus
-Cullen sign : periumbilical ecchymosis
-Grey-Turner sign: flank ecchymosis
- Hematemesis, melena, hematochezia (aortic-enteric fistula)
20
DIAGNOSIS
PHYSICAL EXAMINATION
-Detection of expansile pulsation
- unreliable about 50%-- false negative
- large hypogastric aneurysm palpated on rectal examination
 INVESTIGATIONS
- X- RAY
- USG
- CT/MRI / MR ANGIOGRAPHY
- ARTERIOGRAPHY
21
X-RAY
 Lumbar Spine Radiograph:
Egg shell Pattern of Calcification
22
USG
 most useful and least expensive mode of diagnosis.
 Measuring transverse aneurysmal diameter.
 Screening /Surveillance/ follow up after endovascular repair.
- Best used to assess progression of AAA size
- Average expansion of 0.4cm/year
 Can’t detect Rupture.
23
CT Imaging
 Precise test – provides good images of aorta , aortic lumen branch
vessels and adjacent retroperitoneal structures.
 Shows size and extent of aneurysm and relation to renal and iliac
arteries.
 SPIRAL CT – 3 dimensional image.
24
25
MRI/ MR ANGIOGRAPHY
 Useful for planning and follow up of endovascular repair.
 Less sensitive than CT scan in identifying accessory renal
arteries.
26
27
Chaikof EL, Dalman RL, Eskandari MK, et al. The Society for Vascular Surgery practice guidelines on the care of patients with an abdominal aortic aneurysm. J Vasc
Surg 2018; 67:2.
28
Treatment options
 OPEN SURGERY (OSR)
 Endovascular Aneurysm Repair (EVAR)
29
Indications
 The AAA diameter is greater than 5.5 cm in diameter in men. In
women, it might be considered from a smaller diameter (5.0 cm)
 The patient with AAA is symptomatic
 The AAA is rapidly expanding (more than 1 cm/year) irrespective of
the absolute diameter
30
31
Complications –AAA Surgery
• Early :
- Myocardial ischemia
- Renal failure
-Postoperative Pneumonia
-Paralytic Ileus
-Colonic ischemia
-Distal Embolisation
-Paraplegia
• Late :
-Anastomotic Pseudoaneurysms
-Aorto-enteric Fistula
-Graft Occlusion
-Graft Infection
32
ENDOVASCULAR REPAIR
 TRANSLUMINAL PLACEMENT OF A GRAFT WITHIN THE
ANEURYSM THAT COMPLETELY EXCLUDES THE SAC
FROM GENERAL CIRCULATION
 INTRODUCTED BY PARODI IN 1990
 SUCCESS RATE 98 %
 CONVERTION RATE 12 %
 OVERALL HOSPITAL MORTALITY < 2.5 %
33
Benefits
 Theoretical
- Reduced complications and mortality
- Decreased hospitalization
-Decreased cost
 Realized
- Same number of complications but different types
- Less systemic complications, same mortality
- Shorter respiratory support
- Decreased ICU and hospital stay
-Decreased blood loss
34
DISADVANTAGES
 CUSTOMISED FOR EACH PATIENT
 FOLLOW UP IS CRUCIAL
 LONG TERM IMPLICATIONS AWAITED
 ENDOLEAK 14 – 20 %
 CONTRAST INDUCED NEPHROPATHY
 HIGH COST
35
36
37
Complications of EAAA repair
 Injuries to arteries of access – Iliac/ Supra Renal
 Embolization - micro – Renal Failure
- distal – Ischemia
 Procedure related: Groin hematoma, wound infection, Post-
implantation syndrome
 Device related : Migration, detachment, rupture, stenosis, kinking,
endoleak
38
Endoleaks
 Leak around proximal or distal attachment sites
 Persistent flow in aneurysm sac
 Risks :
- Expansion
-Rupture
39
40
Chaikof EL, Blankensteijn JD, Harris PL, et al. Reporting standards for endovascular aortic aneurysm repair. J
Vasc Surg 2002; 35:1048
RUPTURED AAA
 anteriorly into the peritoneal cavity (20%) or postero-laterally into
the retroperitoneal space (80%) causing Retroperitoneal hematoma
 Less than 50% of patients with rupture survive to reach hospital.
C/F:
 “Classic triad:”
-Severe abdominal pain -Hypotension -Pulsatile mass
 Less common symptoms:
-Groin/flank pain, hematuria, groin hernia all secondary to
increased intra-abdominal pressure
- Congestive Heart Failure with JVD and abdominal bruit if patient
has ruptured into the Vena Cava
41
Rupture outcomes
 Operative mortality: 50%
 Combined Mortality(Community + Hospital) : 80-90 %
 More than one third of rupture cases die outside the hospital
42
AAA - RUPTURE
• Treatment
A) EMERGENCY SURGERY : First successful repair of a ruptured AAA
was by Cooley and DeBakey in 1954
• More than 98% of ruptures occur below the renal arteries.
• Exploration -- through MIDLINE incision
IF aorta approached at infra renal level– it should be clamped
PROXIMAL CONTROL– At the level of diaphragm
• OTHER METHODS : Compression at diaphragm
Placement of aortic balloon catheters
Foley catheters via puncture of the aneurysm
43
THORACO ABDOMINAL AORTIC
 Aneurysms that involve the thoracic and abdominal aorta or those
aneurysms including the visceral aortic segments
 Etiology: - degenerative– 80%
- sequelae of chronic dissection– 20%
 C/f-- sudden development of severe pain– back /epigastric/flank
pain
 others– hoarseness/cough hemoptysis /dysphagia lusoria
44
Crawford classification
 Type I – involves Descending thoracic aorta and abdominal aorta
proximal to renal arteries.
 Type II – Most of DTA + AA distal to renal arteries
 Type III – Involves aorta distal to renal arteries.
 Type IV – Involves all or most of the abdominal aorta including the
Para visceral segment.
45
SYPHILITIC ANEURYSM
 Seen in tertiary stage of syphilis with obliterative endarteritis of
vasa vasorum and aortitis
 Roughening of intima : “Tree barking”
 Involves the thoracic aorta
 Complications include rupture, aortic insufficiency, and narrowing
of coronary ostia
46
PERIPHERAL ANEURYSM
 FEMORAL ARTERY ANEURYSM
 POPLITEAL ARTERY ANEURYSM
 TIBIAL ARTERY ANEURYSM
47
FEMORAL ANEURYSM
 Common peripheral aneurysm
 Incidence- 7.39 / 1OOOOO Population
 TRUE aneurysm- Non specific etiology
 FALSE aneurysm - anastomotic/traumatic catheter-
induced/infected
 TYPE 1 – Limited to Common Femoral Artery
 TYPE 2 – Involving orifice of Profunda Femoris artery
48
Femoral aneurysm
 Asymptomatic with pulsatile mass
 Local pain – pressure on adjacent nerve
 Limb edema/venous distention- venous compression
 Lower extremity ischemia with intermittent claudication /rest
pain/gangrene
 COMPLICATIONS -THROMBOSIS
- RUPTURE
- EMBOLIZATION
49
OPERATIVE MANAGEMENT
 TYPE 1 ANEURYSM:
- INTERPOSITION GRAFT OF DACRON
- e PTFE
 TYPE 2 ANEURYSM:
-INTERPOSITION GRAFT TO SFA/ PFA with re-implantation
of the other artery.
- SYNDACTYLIZATION- Suturing of Superficial and Profunda
femoris arteries together to form a common lumen
50
POPLITEAL ANEURYSM
 commonest ( 70% )
 65% bilateral with 25% associated with AAA
 Etiology related to chronic flexion/extension
 Associated aneurysms – other leg, femoral, aortic
51
 Clinical features-
-Swelling in popliteal region which is smooth, soft ,
pulsatile , well localized, warm compressible , often with thrill and
bruit.
- thrombosis and emboli can cause distal gangrene
- rupture : torrential hemorrhage
 Treatment- ANEURYSMORRHAPHY- Repair with arterial graft using
PTFE, dacron
- ENDOLUMINAL STENTING
52
 National Institute for Health and Care Excellence (NICE) in the UK
published guidelines in 2020 recommending open surgical repair
unless contraindicated, reserving EVAR for high-risk patients or
those with a hostile abdomen.
 This compares to the European Society for Vascular Surgery 2019
guidelines, which recommend EVAR as the first-line treatment
option with open surgical repair to be considered for patients with
long life expectancy.
53
THANK YOU
54

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ANEURYSMS , TYPES AND THERE MANAGEMENT.pptx

  • 2. INTRODUCTION • The term ANEURYSM is derived from the Greek word ANEURYSMA meaning “ a widening”. • Normal arterial diameter is dependent on age , gender, body size and other factors. • An Aneurysm is defined as a localized dilation at least 50 % larger than an adjacent normal portion of the same artery. • ECTASIA- Arterial dilation less than 50% above normal • ARTERIOMEGALY– Diffuse arterial enlargement involving several arterial segments with an increase in diameter greater than 50% 2
  • 3. 3
  • 4. CLASSIFICATION True/False Aneurysm  True aneurysm : contains all layers of arterial wall.  False aneurysm :dilation covered by thick fibrinous capsule. 4
  • 5. PSEUDOANEURYSM  Injury to wall of vessel allows blood to escape from vessel into adjacent tissue.  Extravasated blood coagulates and becomes a mass along side the vessel.  This mass of blood (hematoma) gives the impression that there is an aneurysm 5
  • 6. 6
  • 7. BASED ON LOCATION LOCATION FREQUENCY • ABDOMINAL AORTA 65% • THORACIC AORTA 19% • AA + ILIAC 13% • THORACOABDOMINAL 2% • ISOLATED ILIAC 1% 7
  • 8. LOCATION-PERIPHERAL LOCATION FREQUENCY  POPLITEAL 70%  FEMORAL together make upto 90%  CAROTID 4%  SUBCLAVIAN 2%  CEREBRAL 2%  SPLENIC 1%  MESENTRIC 0.5%  RENAL 0.5% 8
  • 9. BASED ON MORPHOLOGY  FUSIFORM: SYMMETRICAL CIRCUMFERENTIAL ENLARGEMENT INVOLVING ALL LAYERS OF THE ARTERY WALL.  SACCULAR: ANEURYSMAL DEGENERATION AFFECTING ONLY PART OF THE ARTERIAL CIRCUMFERENCE. 9
  • 10. 10
  • 11. ABDOMINAL AORTIC ANEURYSM  INCIDENCE: 2% TO 5% OF ALL MEN OVER 60 YRS  COMMON IN MEN ABOVE 60YRS PEAK OF 5% AT 80 YRS IN MEN PEAK OF 4.5% AT 90 YRS IN WOMEN  GREATER - FIRST DEGREE RELATIVES TALLER INDIVIDUAL 11
  • 12. Risk Factors  Atherosclerosis (90%) -Smoking -Hyperlipidemia - Diabetes  Gender - Males > Females  Age  Familial History - 20% of patients with AA have 1st Degree relative  Connective tissue disease - Marfan Syndrome- defect in Fibrillin structure - Ehlers-Danlos - defect in pro-collagen type 3 12
  • 13. 13
  • 14. 14
  • 15. AAA Sequelae  Natural history : - gradual and/or sporadic expansion - accumulation of mural thrombus  Complications - rupture - thromboembolic events - compression of adjacent structures 15
  • 16. Progression of AAA  Pathological changes cause the aorta wall to - become thinner - bulge - rupture 16
  • 17. AAA ANNUAL RUPTURE RISK 17 American Association for Vascular Surgery and Society for Vascular Surgery
  • 18.  Risk of rupture of an abdominal aortic aneurysm (AAA) over time according to the first measurement of aneurysm diameter in 1792 men and 465 women. The risk of rupture increased markedly in aneurysms larger than 5.5 cm in diameter 18 Powell, JT, Greenhalgh, RM, N Engl J Med 2003; 348:1895
  • 19. CLINICAL PRESENTATION AAA are asymptomatic before rupture in 75%  Abdominal palpation may show a pulsatile abdominal mass.  Vague abdominal and back discomfort.  Large aneurysms – GI symptoms– early satiety and vomiting.  Inflammatory aneurysms- pain /fever /ureteral obstruction 19
  • 20. CLINICAL PRESENTATION  AAA RUPTURE  Syncope  Back abdominal pain  Shock  Sudden death (rupture) -Ripping or tearing pain - Atypical pain (groin, flank., hip, bladder ) -Nausea ,vomiting ,tenesmus -Cullen sign : periumbilical ecchymosis -Grey-Turner sign: flank ecchymosis - Hematemesis, melena, hematochezia (aortic-enteric fistula) 20
  • 21. DIAGNOSIS PHYSICAL EXAMINATION -Detection of expansile pulsation - unreliable about 50%-- false negative - large hypogastric aneurysm palpated on rectal examination  INVESTIGATIONS - X- RAY - USG - CT/MRI / MR ANGIOGRAPHY - ARTERIOGRAPHY 21
  • 22. X-RAY  Lumbar Spine Radiograph: Egg shell Pattern of Calcification 22
  • 23. USG  most useful and least expensive mode of diagnosis.  Measuring transverse aneurysmal diameter.  Screening /Surveillance/ follow up after endovascular repair. - Best used to assess progression of AAA size - Average expansion of 0.4cm/year  Can’t detect Rupture. 23
  • 24. CT Imaging  Precise test – provides good images of aorta , aortic lumen branch vessels and adjacent retroperitoneal structures.  Shows size and extent of aneurysm and relation to renal and iliac arteries.  SPIRAL CT – 3 dimensional image. 24
  • 25. 25
  • 26. MRI/ MR ANGIOGRAPHY  Useful for planning and follow up of endovascular repair.  Less sensitive than CT scan in identifying accessory renal arteries. 26
  • 27. 27 Chaikof EL, Dalman RL, Eskandari MK, et al. The Society for Vascular Surgery practice guidelines on the care of patients with an abdominal aortic aneurysm. J Vasc Surg 2018; 67:2.
  • 28. 28
  • 29. Treatment options  OPEN SURGERY (OSR)  Endovascular Aneurysm Repair (EVAR) 29
  • 30. Indications  The AAA diameter is greater than 5.5 cm in diameter in men. In women, it might be considered from a smaller diameter (5.0 cm)  The patient with AAA is symptomatic  The AAA is rapidly expanding (more than 1 cm/year) irrespective of the absolute diameter 30
  • 31. 31
  • 32. Complications –AAA Surgery • Early : - Myocardial ischemia - Renal failure -Postoperative Pneumonia -Paralytic Ileus -Colonic ischemia -Distal Embolisation -Paraplegia • Late : -Anastomotic Pseudoaneurysms -Aorto-enteric Fistula -Graft Occlusion -Graft Infection 32
  • 33. ENDOVASCULAR REPAIR  TRANSLUMINAL PLACEMENT OF A GRAFT WITHIN THE ANEURYSM THAT COMPLETELY EXCLUDES THE SAC FROM GENERAL CIRCULATION  INTRODUCTED BY PARODI IN 1990  SUCCESS RATE 98 %  CONVERTION RATE 12 %  OVERALL HOSPITAL MORTALITY < 2.5 % 33
  • 34. Benefits  Theoretical - Reduced complications and mortality - Decreased hospitalization -Decreased cost  Realized - Same number of complications but different types - Less systemic complications, same mortality - Shorter respiratory support - Decreased ICU and hospital stay -Decreased blood loss 34
  • 35. DISADVANTAGES  CUSTOMISED FOR EACH PATIENT  FOLLOW UP IS CRUCIAL  LONG TERM IMPLICATIONS AWAITED  ENDOLEAK 14 – 20 %  CONTRAST INDUCED NEPHROPATHY  HIGH COST 35
  • 36. 36
  • 37. 37
  • 38. Complications of EAAA repair  Injuries to arteries of access – Iliac/ Supra Renal  Embolization - micro – Renal Failure - distal – Ischemia  Procedure related: Groin hematoma, wound infection, Post- implantation syndrome  Device related : Migration, detachment, rupture, stenosis, kinking, endoleak 38
  • 39. Endoleaks  Leak around proximal or distal attachment sites  Persistent flow in aneurysm sac  Risks : - Expansion -Rupture 39
  • 40. 40 Chaikof EL, Blankensteijn JD, Harris PL, et al. Reporting standards for endovascular aortic aneurysm repair. J Vasc Surg 2002; 35:1048
  • 41. RUPTURED AAA  anteriorly into the peritoneal cavity (20%) or postero-laterally into the retroperitoneal space (80%) causing Retroperitoneal hematoma  Less than 50% of patients with rupture survive to reach hospital. C/F:  “Classic triad:” -Severe abdominal pain -Hypotension -Pulsatile mass  Less common symptoms: -Groin/flank pain, hematuria, groin hernia all secondary to increased intra-abdominal pressure - Congestive Heart Failure with JVD and abdominal bruit if patient has ruptured into the Vena Cava 41
  • 42. Rupture outcomes  Operative mortality: 50%  Combined Mortality(Community + Hospital) : 80-90 %  More than one third of rupture cases die outside the hospital 42
  • 43. AAA - RUPTURE • Treatment A) EMERGENCY SURGERY : First successful repair of a ruptured AAA was by Cooley and DeBakey in 1954 • More than 98% of ruptures occur below the renal arteries. • Exploration -- through MIDLINE incision IF aorta approached at infra renal level– it should be clamped PROXIMAL CONTROL– At the level of diaphragm • OTHER METHODS : Compression at diaphragm Placement of aortic balloon catheters Foley catheters via puncture of the aneurysm 43
  • 44. THORACO ABDOMINAL AORTIC  Aneurysms that involve the thoracic and abdominal aorta or those aneurysms including the visceral aortic segments  Etiology: - degenerative– 80% - sequelae of chronic dissection– 20%  C/f-- sudden development of severe pain– back /epigastric/flank pain  others– hoarseness/cough hemoptysis /dysphagia lusoria 44
  • 45. Crawford classification  Type I – involves Descending thoracic aorta and abdominal aorta proximal to renal arteries.  Type II – Most of DTA + AA distal to renal arteries  Type III – Involves aorta distal to renal arteries.  Type IV – Involves all or most of the abdominal aorta including the Para visceral segment. 45
  • 46. SYPHILITIC ANEURYSM  Seen in tertiary stage of syphilis with obliterative endarteritis of vasa vasorum and aortitis  Roughening of intima : “Tree barking”  Involves the thoracic aorta  Complications include rupture, aortic insufficiency, and narrowing of coronary ostia 46
  • 47. PERIPHERAL ANEURYSM  FEMORAL ARTERY ANEURYSM  POPLITEAL ARTERY ANEURYSM  TIBIAL ARTERY ANEURYSM 47
  • 48. FEMORAL ANEURYSM  Common peripheral aneurysm  Incidence- 7.39 / 1OOOOO Population  TRUE aneurysm- Non specific etiology  FALSE aneurysm - anastomotic/traumatic catheter- induced/infected  TYPE 1 – Limited to Common Femoral Artery  TYPE 2 – Involving orifice of Profunda Femoris artery 48
  • 49. Femoral aneurysm  Asymptomatic with pulsatile mass  Local pain – pressure on adjacent nerve  Limb edema/venous distention- venous compression  Lower extremity ischemia with intermittent claudication /rest pain/gangrene  COMPLICATIONS -THROMBOSIS - RUPTURE - EMBOLIZATION 49
  • 50. OPERATIVE MANAGEMENT  TYPE 1 ANEURYSM: - INTERPOSITION GRAFT OF DACRON - e PTFE  TYPE 2 ANEURYSM: -INTERPOSITION GRAFT TO SFA/ PFA with re-implantation of the other artery. - SYNDACTYLIZATION- Suturing of Superficial and Profunda femoris arteries together to form a common lumen 50
  • 51. POPLITEAL ANEURYSM  commonest ( 70% )  65% bilateral with 25% associated with AAA  Etiology related to chronic flexion/extension  Associated aneurysms – other leg, femoral, aortic 51
  • 52.  Clinical features- -Swelling in popliteal region which is smooth, soft , pulsatile , well localized, warm compressible , often with thrill and bruit. - thrombosis and emboli can cause distal gangrene - rupture : torrential hemorrhage  Treatment- ANEURYSMORRHAPHY- Repair with arterial graft using PTFE, dacron - ENDOLUMINAL STENTING 52
  • 53.  National Institute for Health and Care Excellence (NICE) in the UK published guidelines in 2020 recommending open surgical repair unless contraindicated, reserving EVAR for high-risk patients or those with a hostile abdomen.  This compares to the European Society for Vascular Surgery 2019 guidelines, which recommend EVAR as the first-line treatment option with open surgical repair to be considered for patients with long life expectancy. 53

Editor's Notes

  1. * Typically identified by abdominal ultrasound obtained to evaluate palpable abdominal mass, for screening purposes, or follow-up evaluation of another identified aneurysm (eg, thoracic aortic aneurysm, popliteal artery aneurysm). AAA has also been identified incidentally on plain abdominal radiography, spine imaging studies, and others.
  2. * Permissive hypotension targets systolic BP 80 to 100 mmHg. ¶ Intravenous contrast is not absolutely required to diagnose rupture but is highly desired if endovascular repair is an option. Δ Ultrasound can be performed at the bedside or in the operating room. In a patient with an appropriate history and clinical findings, ultrasound may not be necessary. ◊ In the absence of overt rupture, no radiologic signs have been proven to predict AAA rupture for certain, but signs associated with impending AAA rupture have included: crescent sign, layering hematoma, aortic blebs, aortic draping over a vertebral body, irregular aortic wall, breaks in calcification of the aortic wall, and localized areas of higher attenuation within mural thrombus. In combination with an AAA of >5 cm, these signs may indicate a rapidly changing aneurysm and urgency for repair. § The aneurysm is managed as with other asymptomatic AAA. ¥ For patients with symptomatic (ruptured, nonruptured) AAA, an endovascular first approach is generally preferred if anatomically suitable and institutional resources, endovascular devices, and expertise are available. Otherwise, open surgical repair can be performed. ‡ Initial wire access and aortic occlusion balloon placement can precede repair as a temporizing measure to limit bleeding. † Obtain CT angiography, if contrast study not previously performed.
  3. Post-implantation syndrome (PIS) was initially registered as a fever and leucocytosis syndrome following the implantation of a stent graft in the aorta. The etiology of PIS is due to the attribution of endovascular reconstruction to the systemic inflammatory reaction.
  4. The types of endoleaks after an endovascular repair of an abdominal aortic aneurysm are as follows:Type I is due to an incompetent seal at the proximal (Ia) or distal (Ib) attachment site. Type II results from flow into and out of the aneurysm sac from one or more patent branch vessels (lumbar or inferior mesenteric artery). Type III results from dissociation of modular components. Type IV is due to leaks through the porous graft material. Endoleak of undefined origin (type V) is continued aneurysm sac expansion without a demonstrable endoleak on any imaging modality. It is also referred to as endotension
  5. Dysphagia lusoria : aberrant right subclavian artery  (ARSA) that compresses the esophagus.