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(for undergraduate 3rd Integrated Medicine)
Compiled by Ahmed Redwan, 2021 © COPYRIGHTED
Assist. Lecturer Int. Med. Depart.
El-Minia University, EGYPT.
Topics Discussed:
 Conscious level and mentality.
 Appearance
 Built
 Complexion
 Decubitus
 Vital signs: (pulse, BP, Temperature, R.R.)
 U.L & L.L.
 Head & Neck
 Other system review; Chest, Abdomen ,
Neurological, …
 Summary flowchart for CVS EXAMINATION.
Conscious level and mentality.
 Intellectual functions with cloudy mentality may be affected
with generalized atherosclerosis that also may lead to
Transient ischemic attack (T.I.A).
 Embolic manifestations : M.S ,A.F. leads to Cerebral Stroke.
 Hypertensive Encephalopathy (e.g. Posterior reversible
encephalopathy Syndrome (PRES) and cerebral Hge->Fits).
 Pulmonary edema also affects the patient consciousness
especially after development of Respiratory Failure (hypoxia
+/- hypercapnia)
 Disease Association CVS + NS:
 Fridriech’s ataxia with Cardiomyopathy
 Parkinson’s disease with primary orthostatic hypotension + syncope.
Appearance
Toxemia with:
 I.E.
 Rh.Fever
 Chest infection
 Pericarditis
 Myocard. Infarction & pulm. Embolism.
Cardiac Cachexia
Cachexia
 Cachexia refers to severe muscle and
fat loss, anorexia and marked weight
loss due to an underlying chronic
disease condition leading to lower life
expectancy.
 The title cachexia, from the Greek
kakos (i.e., bad) and hexis (i.e.,
condition or appearance), or 'bad
condition‘.
Cachexia D.D.
Cachexia is a serious and underestimated
consequence in many conditions:
 Cancer
 Organ failure
 Chronic conditions like chronic heart failure,
chronic kidney disease, chronic obstructive
pulmonary disease
 Infectious diseases like tuberculosis, acquired
immunodeficiency syndrome(AIDS)
 Inflammatory disorders like rheumatoid
arthritis
Built
1.Height:
Marfan's syndrome: (Tall+arachnodactyly+pes-
cavas-high arched palate+ ectopia lentis) dissecting
aortic aneurysm, aortic incompetence and MVP.
2.Dwarf undergrowth: Congenital heart disease or
in long standing heart disease since childhood.
2.Weight (BMI):
Underweight: Prolonged heart disease whatever
congenial or rheumatic.
Overweight: Atherosclerosis, HTN, MI.
3. Well developed upper part and under
developed lower part in coarctation of the aorta.
Marfan’s wide span as compared to
height:
Thumb test in Marfan syndrome
Aortic Coarctation
Decubitus
 Orthopnea: Left sided heart failure.
 Squatting: Congenital cyanotic heart
disease.
 Prayer (knee-chest): Pericardial
effusion, mediastinal syndrome.
CARDIOVASCULAR
Squatting position
Complexion:
Pallor:
 Anaemia (congest. Gastropathy) , Low
cardiac output, Shock.
 Facial pallor with acute MI, pallor with
anemic HF.
 Pallor also commonly associated with:
Endocarditis, Rheumatic fever.
Jaundice:
Causes of jaundice in a cardiac patient:
1. Hemolytic J.: in pulmonary infarction, artificial
valves.
2. Hepato-cellular J.: in marked hepatic
congestion (cardiac cirrhosis)due to severe Rt.
sided HF - pericardial effusion -TS & TR.
3. Obstructive J.: bile & inspissation of bile in
canalicular lumen.
4. Associated viral hepatitis: the Commonest !!.
Cyanosis
1. Central: Congenital cyanotic heart
disease (F4)- Advanced heart failure.
2. Peripheral: L.C.O.P., C.H.F., Peripheral
vascular disorders.
3. False (chemical) cyanosis:
Methemoglobinemia in nitrate ttt.
4. N.B.: "differential cyanosis": Means
cyanosis more apparent in lower part of
the body in reversed P.D.A.
Cyanosis
VITAL SIGNS
Temperature
Fever in CVS:
 IE
 Rh.Fever
 Chest infection
 Pericarditis
 Myocard. Infarction & pulm. Embolism
 DVT and thrombophlepitis
 Associated fever
Pulse:
Feel the right radial artery to
determine, pulse rate , rhythm, volume,
character equality (volume) in both upper
limbs, radiofemoral delay (Aortic
coarctation) and state of arterial wall.
One should also examine all
peripheral pulses in the carotides
brachial,femorals, post. tibial and dorsalis
pedis as routine specially in cardiac cases
with history of embolization.
PULSE RHYTM
Normally sinus regular.
Path. Irregularities:
A- Regular irregular: Extrasystole
B- Irregular ( total) irregularity: A.F. ,V.F
Tachycardia
During sleep
Drugs:
B blockers
Ca #
Digitalis.
R.R.
Tachypnea:
Pulm edema
H.F, MI, pulm. embolism
Associated chest infection.
Complication: Resp. Failure.
Distressed sweaty or clammy
('diaphoretic') Acute MI
Upper Limb
• 1-Dorsal aspect of the hands:
Tobacco "tar" staining
Peripheral cyanosis.
Temperature.
Clubbing(cyanotic HD or toxic I.E.)
I.E. THROMBOEMBOLC MANIFESTATIONS:
 Splinter haemorrhages (linear, reddish brown marks along the
axis of the finger and to nails, thought to be due to circulating
immune complexes).
 Jenway’s lesions
 Oslar’s nodules
• Sc nodules
• Arthritis due to Rheumatic fever.
• Abnormal movement (chorea)
• Xanthomatas around tendons
Upper Limb
• 1-Dorsal aspect of the hands:
Tobacco "tar" staining
Peripheral cyanosis.
Temperature.
Clubbing(cyanotic HD or toxic I.E.)
I.E. THROMBOEMBOLC MANIFESTATIONS:
 Splinter haemorrhages (linear, reddish brown marks,
multiple >2, along the axis of the finger and to nails,
thought to be due to circulating immune complexes).
 Jenway’s lesions
 Osler’s nodules
• Sc nodules (Rh. Fever).
• Arthritis due to Rheumatic fever.
• Abnormal movement (chorea) H.F.
• Xanthomatas around tendons
Upper Limb
2-Palmar aspect of the hands:
• Janeway lesions - painless red spots/macules, which blanch on
pressure, on the thenar/hypothenar eminences of the Palms, and
soles of the feet.
• Osler's nodes – small painful raised erythematous lesions
(nodules) which are rare but found most often on the pads of the
fingers (finger pulps) And toes representing digital microinfarction.
3-Palmar and extensor surfaces of the hands:
xanthomata (yellow skin or tendon nodules from lipid deposits).
4-Entire skin surface: petechiae.
Lower Limb
• Bilat. Bitting LL edema wih H.F
• Unilat edema: redness, hotness, pain,
tense, tender swollen calf muscles in
DVT --- risk for PE.
• Clubbing
• Peripheral pulsations.
• Differential cyanosis
D.V.T.
Head & neck
Toxic look: endocarditis, rheumatic fever, severe
pulmonary infection.
• Malar flush: M.S (tight).
• Yellow-bluish complexion: Cyanoicterus features: In
tricuspid valve disease (cardiac cirrhosis).
• Mongolic features: Fallot's tetralogy and V.S.D.
• Turner: Coarctation of aorta.
• Ehlers-Danlos syndrome: joint hypermobility, elastic
skin, easy bruising: mitral valve prolapse, aortic (and
other arterial) aneurysms.
• Low set ear, big lips, depressive nose: may appear in
congenital aorticstenosis.
• Butter fly: S.L.E.
Cont.
 Thyrotoxicosis: (heat intolerance, weight loss,
tremor) : tachycardia.
 Hypothyroidism• (cold intolerance, fatigue,
weight gain, depression): Bradycardia.
 Acromegaly: (enlargement of hands and feet,
prominent eye brow): cardiomyopathy.
Ehler-Danlos Syn.
Turner
Down’s Synd.
 Down's syndrome: (microgenia,
macroglossia, epicanthic folds.single
transverse palmar crease): complete
atrioventricular septal defects (e.g.
VSD), F4, repeated chest infection (the
commonest cause of death rather than
H.F. !!!!).
What a beautiful creature !!!:
Marfan Syndrome
Eye
 Peticheal haemorrhage inhypertension.
 Roth spots: Flame shaped retinal Hge
with cotton wool center (clear center)
 Palate: High arched palate: congenital
heart diseases,Tonsills: Tonsillitis
rheumatic fever.
Roth’s spots
Roth’s spots
Neck
Neck veins: venous waves and venous
pressure (Congestion) by examination of the
internal jugular vein (the window of the right
atrium).
Normally there is no congestion , but only normal
pulsation.
Thrill: Palpate the neck for thrill.
Causes:
A.S.Thyrotoxicosis, Aneurysm of carotid artery.
Corneal arcus in young patient.
(Familial Hypercholestrol.)
Also young !!
Abdomen
C-ABDOMINAL EXAMINATION:
1-Liver:-Enlarged & tender in: RVF,
pericardial disease-Enlarged, tender with
expansile pulsation: TR. TS-Enlarged firm,
with sharp border: cardiac cirrhosis
2-Spleen: enlarged in RVF, pericardial
disease3-Ascites: after edema in RVF, or
ascites precox in TR. TS. Pericardial
diseas.
BACK EXAMINATION:
1) Murmurs: - Bruit:of MR left scapolar regionOf coarctation
of aorta: interscapular region -Bnist of renal artery
stenosis in renal angle.
2) Crepitations:-Bilateral basal crepitation: LVF-Bubbling
coarse crepitations pulmonary edema
3) Ewart's sign: collapse of left lower lobe in pericardial
effusion
4) Vertebrae - Scapula:-Spina bifida: associated congenital
anomaly in congenital HD-Low back pain, stiffness &
limited spine mobility m ankylosing spondylitis
associated with AR -Anastomosis around scapula in
coarctation of aorta
5) Skin: Cafe au lait patches in neurofibromatosas
associated with pheochromocytoma & IHSS.
6) Sacral edema: pitting edema at sacral area in prolonged
bed ridden patients with Heart failure.
Thank You
‫هللا‬ ‫بحمد‬ ‫تم‬

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General examination cardiac patient

  • 1. (for undergraduate 3rd Integrated Medicine) Compiled by Ahmed Redwan, 2021 © COPYRIGHTED Assist. Lecturer Int. Med. Depart. El-Minia University, EGYPT.
  • 2. Topics Discussed:  Conscious level and mentality.  Appearance  Built  Complexion  Decubitus  Vital signs: (pulse, BP, Temperature, R.R.)  U.L & L.L.  Head & Neck  Other system review; Chest, Abdomen , Neurological, …  Summary flowchart for CVS EXAMINATION.
  • 3. Conscious level and mentality.  Intellectual functions with cloudy mentality may be affected with generalized atherosclerosis that also may lead to Transient ischemic attack (T.I.A).  Embolic manifestations : M.S ,A.F. leads to Cerebral Stroke.  Hypertensive Encephalopathy (e.g. Posterior reversible encephalopathy Syndrome (PRES) and cerebral Hge->Fits).  Pulmonary edema also affects the patient consciousness especially after development of Respiratory Failure (hypoxia +/- hypercapnia)  Disease Association CVS + NS:  Fridriech’s ataxia with Cardiomyopathy  Parkinson’s disease with primary orthostatic hypotension + syncope.
  • 4. Appearance Toxemia with:  I.E.  Rh.Fever  Chest infection  Pericarditis  Myocard. Infarction & pulm. Embolism. Cardiac Cachexia
  • 5.
  • 6. Cachexia  Cachexia refers to severe muscle and fat loss, anorexia and marked weight loss due to an underlying chronic disease condition leading to lower life expectancy.  The title cachexia, from the Greek kakos (i.e., bad) and hexis (i.e., condition or appearance), or 'bad condition‘.
  • 7. Cachexia D.D. Cachexia is a serious and underestimated consequence in many conditions:  Cancer  Organ failure  Chronic conditions like chronic heart failure, chronic kidney disease, chronic obstructive pulmonary disease  Infectious diseases like tuberculosis, acquired immunodeficiency syndrome(AIDS)  Inflammatory disorders like rheumatoid arthritis
  • 8. Built 1.Height: Marfan's syndrome: (Tall+arachnodactyly+pes- cavas-high arched palate+ ectopia lentis) dissecting aortic aneurysm, aortic incompetence and MVP. 2.Dwarf undergrowth: Congenital heart disease or in long standing heart disease since childhood. 2.Weight (BMI): Underweight: Prolonged heart disease whatever congenial or rheumatic. Overweight: Atherosclerosis, HTN, MI. 3. Well developed upper part and under developed lower part in coarctation of the aorta.
  • 9.
  • 10. Marfan’s wide span as compared to height:
  • 11. Thumb test in Marfan syndrome
  • 13.
  • 14. Decubitus  Orthopnea: Left sided heart failure.  Squatting: Congenital cyanotic heart disease.  Prayer (knee-chest): Pericardial effusion, mediastinal syndrome.
  • 15.
  • 17.
  • 19. Complexion: Pallor:  Anaemia (congest. Gastropathy) , Low cardiac output, Shock.  Facial pallor with acute MI, pallor with anemic HF.  Pallor also commonly associated with: Endocarditis, Rheumatic fever.
  • 20.
  • 21.
  • 22. Jaundice: Causes of jaundice in a cardiac patient: 1. Hemolytic J.: in pulmonary infarction, artificial valves. 2. Hepato-cellular J.: in marked hepatic congestion (cardiac cirrhosis)due to severe Rt. sided HF - pericardial effusion -TS & TR. 3. Obstructive J.: bile & inspissation of bile in canalicular lumen. 4. Associated viral hepatitis: the Commonest !!.
  • 23.
  • 24. Cyanosis 1. Central: Congenital cyanotic heart disease (F4)- Advanced heart failure. 2. Peripheral: L.C.O.P., C.H.F., Peripheral vascular disorders. 3. False (chemical) cyanosis: Methemoglobinemia in nitrate ttt. 4. N.B.: "differential cyanosis": Means cyanosis more apparent in lower part of the body in reversed P.D.A.
  • 26.
  • 27.
  • 29. Temperature Fever in CVS:  IE  Rh.Fever  Chest infection  Pericarditis  Myocard. Infarction & pulm. Embolism  DVT and thrombophlepitis  Associated fever
  • 30. Pulse: Feel the right radial artery to determine, pulse rate , rhythm, volume, character equality (volume) in both upper limbs, radiofemoral delay (Aortic coarctation) and state of arterial wall. One should also examine all peripheral pulses in the carotides brachial,femorals, post. tibial and dorsalis pedis as routine specially in cardiac cases with history of embolization.
  • 31.
  • 32. PULSE RHYTM Normally sinus regular. Path. Irregularities: A- Regular irregular: Extrasystole B- Irregular ( total) irregularity: A.F. ,V.F
  • 33.
  • 34.
  • 35.
  • 38.
  • 39.
  • 40. R.R. Tachypnea: Pulm edema H.F, MI, pulm. embolism Associated chest infection. Complication: Resp. Failure. Distressed sweaty or clammy ('diaphoretic') Acute MI
  • 41. Upper Limb • 1-Dorsal aspect of the hands: Tobacco "tar" staining Peripheral cyanosis. Temperature. Clubbing(cyanotic HD or toxic I.E.) I.E. THROMBOEMBOLC MANIFESTATIONS:  Splinter haemorrhages (linear, reddish brown marks along the axis of the finger and to nails, thought to be due to circulating immune complexes).  Jenway’s lesions  Oslar’s nodules • Sc nodules • Arthritis due to Rheumatic fever. • Abnormal movement (chorea) • Xanthomatas around tendons
  • 42. Upper Limb • 1-Dorsal aspect of the hands: Tobacco "tar" staining Peripheral cyanosis. Temperature. Clubbing(cyanotic HD or toxic I.E.) I.E. THROMBOEMBOLC MANIFESTATIONS:  Splinter haemorrhages (linear, reddish brown marks, multiple >2, along the axis of the finger and to nails, thought to be due to circulating immune complexes).  Jenway’s lesions  Osler’s nodules • Sc nodules (Rh. Fever). • Arthritis due to Rheumatic fever. • Abnormal movement (chorea) H.F. • Xanthomatas around tendons
  • 43. Upper Limb 2-Palmar aspect of the hands: • Janeway lesions - painless red spots/macules, which blanch on pressure, on the thenar/hypothenar eminences of the Palms, and soles of the feet. • Osler's nodes – small painful raised erythematous lesions (nodules) which are rare but found most often on the pads of the fingers (finger pulps) And toes representing digital microinfarction. 3-Palmar and extensor surfaces of the hands: xanthomata (yellow skin or tendon nodules from lipid deposits). 4-Entire skin surface: petechiae.
  • 44.
  • 45.
  • 46.
  • 47. Lower Limb • Bilat. Bitting LL edema wih H.F • Unilat edema: redness, hotness, pain, tense, tender swollen calf muscles in DVT --- risk for PE. • Clubbing • Peripheral pulsations. • Differential cyanosis
  • 48.
  • 50. Head & neck Toxic look: endocarditis, rheumatic fever, severe pulmonary infection. • Malar flush: M.S (tight). • Yellow-bluish complexion: Cyanoicterus features: In tricuspid valve disease (cardiac cirrhosis). • Mongolic features: Fallot's tetralogy and V.S.D. • Turner: Coarctation of aorta. • Ehlers-Danlos syndrome: joint hypermobility, elastic skin, easy bruising: mitral valve prolapse, aortic (and other arterial) aneurysms. • Low set ear, big lips, depressive nose: may appear in congenital aorticstenosis. • Butter fly: S.L.E.
  • 51. Cont.  Thyrotoxicosis: (heat intolerance, weight loss, tremor) : tachycardia.  Hypothyroidism• (cold intolerance, fatigue, weight gain, depression): Bradycardia.  Acromegaly: (enlargement of hands and feet, prominent eye brow): cardiomyopathy.
  • 54. Down’s Synd.  Down's syndrome: (microgenia, macroglossia, epicanthic folds.single transverse palmar crease): complete atrioventricular septal defects (e.g. VSD), F4, repeated chest infection (the commonest cause of death rather than H.F. !!!!).
  • 55. What a beautiful creature !!!:
  • 57. Eye  Peticheal haemorrhage inhypertension.  Roth spots: Flame shaped retinal Hge with cotton wool center (clear center)  Palate: High arched palate: congenital heart diseases,Tonsills: Tonsillitis rheumatic fever.
  • 60. Neck Neck veins: venous waves and venous pressure (Congestion) by examination of the internal jugular vein (the window of the right atrium). Normally there is no congestion , but only normal pulsation. Thrill: Palpate the neck for thrill. Causes: A.S.Thyrotoxicosis, Aneurysm of carotid artery.
  • 61.
  • 62.
  • 63. Corneal arcus in young patient. (Familial Hypercholestrol.)
  • 65. Abdomen C-ABDOMINAL EXAMINATION: 1-Liver:-Enlarged & tender in: RVF, pericardial disease-Enlarged, tender with expansile pulsation: TR. TS-Enlarged firm, with sharp border: cardiac cirrhosis 2-Spleen: enlarged in RVF, pericardial disease3-Ascites: after edema in RVF, or ascites precox in TR. TS. Pericardial diseas.
  • 66. BACK EXAMINATION: 1) Murmurs: - Bruit:of MR left scapolar regionOf coarctation of aorta: interscapular region -Bnist of renal artery stenosis in renal angle. 2) Crepitations:-Bilateral basal crepitation: LVF-Bubbling coarse crepitations pulmonary edema 3) Ewart's sign: collapse of left lower lobe in pericardial effusion 4) Vertebrae - Scapula:-Spina bifida: associated congenital anomaly in congenital HD-Low back pain, stiffness & limited spine mobility m ankylosing spondylitis associated with AR -Anastomosis around scapula in coarctation of aorta 5) Skin: Cafe au lait patches in neurofibromatosas associated with pheochromocytoma & IHSS. 6) Sacral edema: pitting edema at sacral area in prolonged bed ridden patients with Heart failure.
  • 67.