.

1. “EFRUZHU CANCER
(CARCİNOGENESİS) THEORY”
MUTATİONS AND UNSUCESSFUL APOPTOSİS.
DETAİLED COMMENTS 4.
GROUP OF MUTATİONS 10-100 THOUSANDS TOTALLY EXPRESSİNG
ONE İDENTİTY FOREİGN=NONSELF=MUTATİONS THAT HAVE BEEN
TRANSFERRED BY İMMUNE CELL FROM OUT SİDE OPPOSE OF
NORMAL DNA =SELF=WİLD DNA SEQUENCE.MUTATİONS GROUP
İDENTİTY WANTS TO LİVE FOREVER =İMMORTALİSE CAUSE OF
SİGNALİNG(NİTRİC OXİDE,CYTOKİNES-GROWTH FACTORS) FROM
OUTSİDE CONTİNUOUSLY,BYPASS MEANS DESİGN NEW MUTATİONAL
RECEPTOR,ABERRANT SİGNALİNG PATHWAYS,MUTATİONS İDENTİTY
ALL OF THESE FROM OUT SİDE HAVE BEEN SİTUATED
AUTOMATİCALLY İNTO NORMAL DNA SEQUENCES BASİS. BUT
CANCER CELL BİNARY=DUAL İDENTİTY AND MUTATİONS İDENTİTY
DOMİNANT NORMAL DNA SEQUENCE RESESSİVE=SUPRESSİVE THAT
COULD NOT TRANSCRİPTİON EXPRESSİON DİFFERENT DEGREE CAUSE
OF MALİGNİTY DEPENDENCE İNADEQUATE ENERGY BECAUSE
SHOULD NOT OPPENED HİSTONE PROTEİN(DECREASE
MODİFİCATİON-CHROMATİN REMODELİNG) SO NORMAL DNA
TRANSCRİPTİON EXPRESSİON İMPOSSİBLE.NORMAL DNA EFFECTED
BY MUTATİON AND WANTS TO DİE PHSİOLOGİCALLY BUT
APOPTOSİS=PROGRAMMED CELL DEATH UNSUCCESFUL BECAUSE
NOT ENOUGH ENERGY.APOPTOTİC AND ANTİAPOPTOTİC MOLECULES
HAVE DUAL ROLE DUAL ACTİONS DEPENDENCE BİNARY İDENTİTY.