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Evaluation & management of
Supra-ventricular Tachycardia
in Myocardial Infarction
1
No Disclosure
2
How much time would you take to react?
3
How much time would you take to react?
4
How much time would you take to react?
5
Well known
Early/ late
Bad consequences
Ventricular Arrhythmia
in Myocardial Infarction
6
Supra-ventricular Arrhythmia
in Myocardial Infarction
How common?
When does it happen?
How bad it is?
7
How common?
Cristal et al, Annals of Internal Medicine, 1975
N =
154/ 350
44%
Serrano et al, Clin. Cardiol. 1995
25%
N = 131
8
AF + hyper-acute AWMI
AF: 10-20%
9
Which subset of MI?
SVT in AWMI carries high mortality
Cristal et al, Annals of Internal Medicine, 1975
N = 350
10
PSVT in IWMI
Chest pain After adenosine
Subramanian et al, IPEJ 2012
11
However! AF in IWMI
Sugiura et al, The American Journal Of Cardiology , 1991
More in RVMI
12
Holter’s POV
13
Incidences
ST: AWMI AF: IWMI/ PWMI/ RVMI
Rajhans R et al. Int J Adv Med. 2017
STEMI
50 patients
24 hours
Atrial infarction
Right atrial infarction + atrial arrhythmia + inferior myocardial infarction
14
How to diagnose atrial infarction?
Liu et al, Circulation 1961 Sivertssen et al, Am J Cardiol 1973
15
Complications of atrial infarction
Shakir et al, Can J Cardiol 2007
16
When does it happen?
N = 350
Mostly within 24 hrs
Cristal et al, Annals of Internal Medicine, 1975
17
How long is actually bad?
N= 35/ 340
AFL lasting >48 hrs is 100% killer
Blumlein et al, The Western Journal Of Medicine , 1981
18
AFL + AWMI
19
The later the deadlier!
AWMI/ LAD/ MVD/ LVF/ IABP
Serrano et al, Clin. Cardiol. 1995
20
After PCI! Reperfusion arrhythmia
AVN blockers in APC
Carey et al, American Journal Of Critical Care 2015
21
Reperfusion arrhythmia
after Primary PCI in STEMI
Gorenek et al, EuroIntervention 2014
22
How bad it is?
2/3 patients with SVT have pulmonary edema or shock
Cristal et al, Annals of Internal Medicine, 1975
23
Which is first?
SVT
Pulm edema
Shock
24
Why does it happen?
Ischemia to atrium,
conduction system
Primary
IWMI
Secondary
AWMI
25
Aftermath of SVT in MI
Atrial boosting lost/ high atrial pressure
Increased O2 demand/ supply
shear stress/ plaque rupture
Low BP promoting stasis & coagulation
26
Coronary blood supply to
the cardiac conduction system
Ramanathan et al, Arq. Bras. Cardiol 2009
27
LBBB + AWMI
28
SGARBOSA is not enough!
29
Sgarbosa et al, N Engl J Med 1996 Smith et al, Annals of Emergency Medicine 2012
Sp 95-98%, Sn 15-20% Sp 95-98%, Sn 60-80%
RBBB + AWMI
30
BBB in MI
31
IWMIAWMI
Shah et al, J Clin Diagn Res. 2014N = 100
ALCAPA
32
Mimickers # 1
33
Mimickers # 2
WPW
34
Mimickers # 3
Hyperkalaemia
Mimicker # 4
Coronary embolism/ thrombosuction 35
Terminator 3
36
Beta
blockers
Within
24 hours
Amiodarone
If persistent
DC
shock
if
hemodynamic
instability
DCC = direct-current cardioversion,
D+Q = combination digitalis and quini- dine, V = verapamil Serrano et al, Clin. Cardiol. 1995
AF/ AFL in MI: DAPT VS TAPT
Steffel et al, EHJ 2018
37
PIONEER
REDUAL
ENTRUST
AUGUSTUS
Choose Your Weapon Wisely!
NORSTENT: Bønaa et al, N Engl J Med. 2016
BMS is as good as DES
LEADERS FREE: Morice et al, Int J Cardiol. 2017
LAA occluders
BIOFREEDOM
RFA 38
What we have learned?
39
25-44%
AF
worst
The later
the worse
More in
IWMI
Worse in
AWMI
Poor
hemo
dynamics
Masquerading
morphology
SVT in MI: Ten Commandments
• Ignorance is NOT bliss!
• Not so uncommon
• Mimickers: WPW/ coronary embolism/ ALCAPA/ electrolytes
• Which MI: AWMI/ IWMI?
• When: <12 hrs/ >12 hours?
• What: AF is worst.
• How bad: pulm edema/ shock?
• How long: normalised/ persistent?
• Antiarrhythmics: beta blockers/ Amiodarone/ DC shock
• Management: ischemic risk/ bleeding risk
40
Thank you

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