This document discusses evaluating TMEM33 as a prognostic biomarker for cervical cancer and its correlation with immune infiltration. The author used four cervical cancer cell lines and one normal epithelial cell line, transfecting them with siRNAs. Western blot analysis and quantitative RT-PCR were used to detect changes in TMEM33 expression levels and proliferation rates with decreased TMEM33. The results suggest TMEM33 may be involved in cervical cancer progression and metastasis, and that the tumor microenvironment can stimulate tumor development by causing immune suppression and escape. The author concludes that evaluating diseases with multiple techniques can help determine appropriate treatments and develop more understanding of cancer behavior, and that TMEM33 shows potential as an early biomarker for cervical cancer.