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Foot and Ankle Sports
Selene G. Parekh, MD, MBA
Associate Professor of Surgery
Partner, North Carolina Orthopaedic Clinic
Department of Orthopaedic Surgery
Adjunct Faculty Fuqua Business School
Duke University
Durham, NC
919.471.9622
http://seleneparekhmd.com
Twitter: @seleneparekhmd
Ankle Problems in the
Athlete
Athletic Injuries
• Participation in organized/recreational sports
continues to increase
• Injuries can be debilitating
• Recognition, prompt treatment and rehabilitation
• Prevent long term disability
Athletic Injuries
• 59% of adults participate in
exercise
• 11% jog
• 25% run ≥ 2 miles/day
• 800 footstrikes/mile
• Impact 3-8 x body wt
• Running injuries vary between
37%-57%
Athletic Injuries
• Risk Factors
• Intrinsic
• Individual’s physical and
personality traits
• Extrinsic
• Training techniques
• Weekly running mileage
greater than 40 miles
• Playing surfaces
• Equipment
• Break-away bases
Athletic Injuries
• Etiologic Factors
• Biomechanical abnml
• Flexibility
• Strength
• Footwear & orthoses
• Playing surfaces
Athletic Injuries: Etiology
• Biomechanical abnormalities
• Pelvic obliquity only predictor of injury
• <10% running injuries are due to biomechanical
problems
• 70-80% runners treated with orthoses improve
• Need to wean use
Athletic Injuries: Etiology
• Flexibility
• Prevention
• Tight Achilles
• Decreased DF
HV, turf toe, midfoot
strain, ankle sprain,
Achilles tendinitis,
calf strain
• Decr. MTP ROM
• HR dancers predispose
to injury
• Hypermobility
• Posterior ankle pain
• Pathologic laxity
• Recurrent ankle sprain
Athletic Injuries: Etiology
• Strength
• Weakness  injury
• >10% difference in extremity strength puts player
at risk
• Soccer & volleyball players risk for injury and
recurrence due to weakness
• Restore normal strength ratio though isotonic and
isokinetic strengthening
Athletic Injuries: Etiology
• Footwear and orthoses
• Improper fit
• Tight/loose, neurapraxia
• Load  cushioning
• Reduced calcaneal stress fx in military recruits
• Loss of sensory feedback
• Torque
• Cleating  ankle injuries
• Too much, too little traction
Athletic Injuries: Etiology
• Playing Surfaces
• Concrete, synthetic track
• Grass vs. artificial turf
• Role not completely defined
Chronic Leg Pain
• Concurrent with foot/ankle complaints
• Differential
• Chronic compartment syndrome
• Medial tibial stress syndrome
• Stress fractures
• Muscle strain/tendinitis
• Nerve entrapment
• Vascular disease
• Fascial herniation
• Radiculopathy
Chronic Leg Pain
• Incidence
• 150 patients
• 33% chronic compartment syndrome
• 25% stress fx
• 14% muscle strain, 13% medial tibial stress syndrome
• 10% nerve entrapment, 4% venous disease
• Shin splints
• Periostitis
• 10-15% all running injuries
Exercise-Induced Compartment Syndrome
Exercise-Induced Compartment Syndrome
• Incidence
• Unclear
• 70% runners, 20% ball and puck
• Clinical Features
• Pain predictable after certain distance, duration,
speed
• Aching, cramping, stabbing, fullness
• Neurologic symptoms
• Anterior, deep posterior compartments
Exercise-Induced Compartment Syndrome
• Diagnosis
• Clinical
• Rest: Minimal tenderness
• Exercise to provoke sx
• Muscle herniation 20-60% pts
• DPN/SPN
• Radiologic
• Stress fx
• Occult tumor
• Bone scan
• MRI
Exercise-Induced Compartment Syndrome
• Criteria for Diagnosis
• Pressure measurements
• Resting, post-exercise measurements
• Secondary
• Pressure >15mmHg pre-exercise
• Pressure >30mmHg 1 min post-exercise
• Pressure >15mmHg 5-10 min post-exercise
Exercise-Induced Compartment Syndrome
• Conservative treatment
• Dependent on athletes goals
• Activity modification
• Rest  gradual increase in exercise
• Ice, NSAID’s
• Insoles, orthoses
• Surgical treatment
• Fasciotomies
Exercise-Induced Compartment Syndrome
• Results
• Good with proper patient and techniques
• 80-95% excellent results
• 20% decrease in strength
• Complications
• Swelling
• Hematoma
• Infection
• Herniation
• Nerve entrapment
• Recurrence
Stress Fractures: Tibia & Fibula
• Abnormal repetitive loads causing imbalance of
resorption over formation
• Running > basketball > soccer > skating >
aerobics > ballet
• Athletes with decreased mineral content
• Amenorrheic females
Stress Fractures: Tibia & Fibula
• Clinical features
• Recent change in routine
• Insidious, localized tenderness, swelling
• Diagnosis
• X-ray initially negative
• (2-3 weeks) radiodense haze, periosteal rxn
• Transverse cortical lucency
• Bone scan
Stress Fractures: Tibia & Fibula
• Conservative Treatment
• Rest, cessation of activity
• NWB PWB to WBAT
• As symptoms allow
• Swimming, pool running, cycling
• Resume competition full motion, strength, minimal
tenderness
• Surgical Treatment
• Excision, bone grafting
• PEMF bone stimulation
• Intramedullary fixation
Gastrocnemius-Soleus Strain
• Common injury
• Racquet sports, basketball, running, skiing
• Plantaris tear
• Clinical Features
• Sudden knee extension while crouched w/ ankle DF
• Sudden sharp pain
• Difficulty with toe-off
Gastrocnemius-Soleus Strain
• Treatment
• Casting/immobilization
• RICE
• WBAT
• Active dorsiflexion
• Gentle passive stretching more functional
recovery
Nerve Entrapment Syndromes
• Superficial Peroneal Nerve
• Neuritic pain, burning, tingling, radiation
• Tenderness over area of compression
• R/o compartment syndrome, double-crush
• Tx
• Injections, modalities, thermogesic creams,
neuroactive medications, shoe modifications
• Surgical nerve release, fasciotomy
Popliteal Artery Entrapment Syndrome
• Rare, mimics pain chronic compartment
syndrome
• Recognition important as may lead to
amputation
Popliteal Artery Entrapment Syndrome
Popliteal Artery Entrapment Syndrome
• Clinical
• Cramping pain, intermittent claudication
• 67% bilaterality
• Pulses diminished or absent
• Knee hyperextended, ankle dorsiflexed
• Knee warm, foot cool
• Ischemia, thrombosis; palpable mass
Popliteal Artery Entrapment Syndrome
• Diagnostics
• Provocative testing (treadmill) absent pulses
• Doppler, U/S  flow, aneurysm
• Biplanar arteriography after exercise
• Deviation, occlusion, dilatation
• Treatment
• Release
• Bypass grafting
Venous Disease
• Effort thrombosis
• Pain, edema, distended veins, discoloration
• Venography
• Tx: anticoagulation  heparin, coumadin
• Thrombophlebitis
• Immobilization after injury
• Air travel
• Dehydration
• Oral contraceptives
• Alcohol/drug use
Delayed-Onset Muscle Soreness
• Unaccustomed to activity
• Eccentric muscle contraction  microinjury,
subcellular level
• Inability to generate maximal force
• Pain, fatigue
• Loss of performance 24-48hrs
• Resolve 5-7 days
• Tx
• Symptomatic treatment
Medial Ankle Sprains
• Rare, associated with lateral, bony injuries
• 2 portions:
• Superficial deltoid
• Deep deltoid
DeepSuperficial
Medial Ankle Sprains
• Biomechanics of deltoid
• Prohibits abduction
• Secondary restraint against anterior translation ankle
• Deep deltoid
• Greatest restraint lateral translation (after fibula)
• Highest load to failure
• Valgus tilt  superficial & deep rupture
Medial Ankle Sprains
• Clinical Evaluation
• Lateral injury, fracture, syndesmotic injury
• Abduction, eversion
• Other injuries
• Maisonneuve, FDL, PTT, FHL, tibial & saph n
• Radiographs
• Assoc fx, AP stress  valgus tilt, normal with
incomplete injury
• MRI
Medial Ankle Sprains
• Conservative Treatment
• Dependent upon associated injuries
• Walking cast, boot 6-8wks
• Functional treatment
• Surgical treatment
• Inability to reduce medial clear space
• Direct repair
• Suture, suture anchors
• Stirrup brace 6 months after surgery
Medial Ankle Sprains
Sinus Tarsi Syndrome
• Related to subtalar instability
• Nebulous condition w/ pain in lateral ankle &
sinus tarsi
• Pathologic anatomy not clearly defined
• Scarring or degenerative changes to soft tissues
within sinus tarsi
• Loss of nerve function  loss of proprioception
Sinus Tarsi Syndrome
• Diagnosis
• Clinical Evaluation
• Pain lateral ankle, sinus tarsi
• Prior inversion injury
• Instability rare
• Radiologic Evaluation
• X-rays- normal
• Subtalar arthrography
• Absence of microrecesses, interdigitation
• MRI
• Fibrosis, fluid collections
Sinus Tarsi Syndrome
• Conservative treatment
• Injection
• Surgical treatment
• Exploratory, sx may persist
• Resect fibrofatty tissue & extensions from IER
• Subtalar arthroscopy
• Results
• 96 cases; 69% excellent, 25% good, 6% failures
Os Trigonum/Posterior
Impingement
• First Described
Rosenmuller
• First Described as
Fx Shepherd 1822
• Asymptomatic
• Separate
Ossification Center
• Age 8-11
• Incidence 1.7 - 7%
Os Trigonum/Posterior
Impingement
• No side predominence
• Vary in size
• Normal anatomic variation
• Mild extension
• Thumb shaped projection - 1 cm (Stieda’s
Process)
Os Trigonum/Posterior
Impingement
• Symptoms
• Pain with PF
• Young Athlete/Dancer
• Spectrum
• Acute – Fx
• Gradual - FHL Tendinitis
Os Trigonum/Posterior
Impingement
• Conservative
• Immobilization
• NSAID
• Surgical – Excision
Prone Ankle Scope
Evaluation
• 54% of talar dome
• Os trigonum
• Haglund’s
• Achilles insertion site
Foot Problems in the
Athlete
Goals & Objectives
• Specific problems
• Midfoot
• Forefoot
Os Peroneum
• Ossification
• Complete
• Less than complete
• Multipartite
• Location
• Lateral border calc & plantar lateral cuboid
• Useful marker for ruptured PL
Painful Os Peroneum Syndrome
(POS) & PL Disruption
POPS/Peroneus Long.
Tendonitis/Tear
• Conservative treatment
• Cast immobilization (20% success)
• Physical therapy
• Surgical treatment
• Os peroneum excision/tendon repair
• Peroneal tenodesis
Forefoot Sprains
•First MTP joint/ “turf toe”
•Etiology
•Hard artificial surface, flexible shoes
•True incidence not defined, but many
reports indicate a fairly common entity
•Functional disability
•Push-off impaired
•Compromised forward drive and
running
•More missed games & practices than
other injuries
Forefoot Sprains
• Mechanisms of injury
• Hyperextension of the MTP joint
• Foot dorsiflexed, forefoot fixed, heel raised
• Joint capsule tears, compression injury to the dorsal articular
surface with extremes of dorsiflexion
Forefoot Sprains
• Etiologic factors
• Shoe-surface interface
• Flexible shoes
• Hardness of surface
• Loss of resiliency with time
• Enhanced friction
• Fixes foot allows extremes of motion
• Excessive DF
• In those with limited DF
Forefoot Sprains
• Radiographs
• R/o fx, avulsions, impaction
• Other methods usually not necessary
Forefoot Sprains
• Conservative treatment
• RICE, NSAID’s
• Taping, boot
• Increase stiffness in shoe
• Custom molded insert/Morton’s extension
• Rehab once decrease in sx allows
Forefoot Sprains
• Surgical treatment
• Rarely necessary
• Instability
• Loose bodies, osteochondral injury
• Ligamentous disruption
• Sesamoid retraction
• Repair plantar plate, FHB, long flexor transfer
Metatarsal Stress Fractures
• Etiology
• Injury to opposite limp 
increased stress to normal
limb
• Muscle fatigue  increase in
loading
• Hard surfaces, poorly
cushioned shoes, heavy heel
strike
• Rigid cavus feet  decrease
shock absorption of the
midfoot  increase stress on
MT’s
• Hypermobile first ray or long
2nd MT
Stress Fractures
• High index of suspicion
• Focal tenderness
• Min/no swelling
• X-rays nl < 3-4 wks
• Bone scan, MRI, & CT scans
Treatment
• Activity modifications
• Non-impact sports
• Avoid casting if possible
Treatment
• Shoe modifications
• Rigid cavus foot
• Flexible, cushioned arch support
• Hyperpronation/hypermobile 1st ray
• Soft medial arch support
• Long 2nd MT
• MT pad
RE
ECT
the ankle
the foot

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Lecture 47 parekh sports f&a

  • 1. Foot and Ankle Sports Selene G. Parekh, MD, MBA Associate Professor of Surgery Partner, North Carolina Orthopaedic Clinic Department of Orthopaedic Surgery Adjunct Faculty Fuqua Business School Duke University Durham, NC 919.471.9622 http://seleneparekhmd.com Twitter: @seleneparekhmd
  • 2. Ankle Problems in the Athlete
  • 3. Athletic Injuries • Participation in organized/recreational sports continues to increase • Injuries can be debilitating • Recognition, prompt treatment and rehabilitation • Prevent long term disability
  • 4. Athletic Injuries • 59% of adults participate in exercise • 11% jog • 25% run ≥ 2 miles/day • 800 footstrikes/mile • Impact 3-8 x body wt • Running injuries vary between 37%-57%
  • 5. Athletic Injuries • Risk Factors • Intrinsic • Individual’s physical and personality traits • Extrinsic • Training techniques • Weekly running mileage greater than 40 miles • Playing surfaces • Equipment • Break-away bases
  • 6. Athletic Injuries • Etiologic Factors • Biomechanical abnml • Flexibility • Strength • Footwear & orthoses • Playing surfaces
  • 7. Athletic Injuries: Etiology • Biomechanical abnormalities • Pelvic obliquity only predictor of injury • <10% running injuries are due to biomechanical problems • 70-80% runners treated with orthoses improve • Need to wean use
  • 8. Athletic Injuries: Etiology • Flexibility • Prevention • Tight Achilles • Decreased DF HV, turf toe, midfoot strain, ankle sprain, Achilles tendinitis, calf strain • Decr. MTP ROM • HR dancers predispose to injury • Hypermobility • Posterior ankle pain • Pathologic laxity • Recurrent ankle sprain
  • 9. Athletic Injuries: Etiology • Strength • Weakness  injury • >10% difference in extremity strength puts player at risk • Soccer & volleyball players risk for injury and recurrence due to weakness • Restore normal strength ratio though isotonic and isokinetic strengthening
  • 10. Athletic Injuries: Etiology • Footwear and orthoses • Improper fit • Tight/loose, neurapraxia • Load  cushioning • Reduced calcaneal stress fx in military recruits • Loss of sensory feedback • Torque • Cleating  ankle injuries • Too much, too little traction
  • 11. Athletic Injuries: Etiology • Playing Surfaces • Concrete, synthetic track • Grass vs. artificial turf • Role not completely defined
  • 12. Chronic Leg Pain • Concurrent with foot/ankle complaints • Differential • Chronic compartment syndrome • Medial tibial stress syndrome • Stress fractures • Muscle strain/tendinitis • Nerve entrapment • Vascular disease • Fascial herniation • Radiculopathy
  • 13. Chronic Leg Pain • Incidence • 150 patients • 33% chronic compartment syndrome • 25% stress fx • 14% muscle strain, 13% medial tibial stress syndrome • 10% nerve entrapment, 4% venous disease • Shin splints • Periostitis • 10-15% all running injuries
  • 15. Exercise-Induced Compartment Syndrome • Incidence • Unclear • 70% runners, 20% ball and puck • Clinical Features • Pain predictable after certain distance, duration, speed • Aching, cramping, stabbing, fullness • Neurologic symptoms • Anterior, deep posterior compartments
  • 16. Exercise-Induced Compartment Syndrome • Diagnosis • Clinical • Rest: Minimal tenderness • Exercise to provoke sx • Muscle herniation 20-60% pts • DPN/SPN • Radiologic • Stress fx • Occult tumor • Bone scan • MRI
  • 17. Exercise-Induced Compartment Syndrome • Criteria for Diagnosis • Pressure measurements • Resting, post-exercise measurements • Secondary • Pressure >15mmHg pre-exercise • Pressure >30mmHg 1 min post-exercise • Pressure >15mmHg 5-10 min post-exercise
  • 18. Exercise-Induced Compartment Syndrome • Conservative treatment • Dependent on athletes goals • Activity modification • Rest  gradual increase in exercise • Ice, NSAID’s • Insoles, orthoses • Surgical treatment • Fasciotomies
  • 19. Exercise-Induced Compartment Syndrome • Results • Good with proper patient and techniques • 80-95% excellent results • 20% decrease in strength • Complications • Swelling • Hematoma • Infection • Herniation • Nerve entrapment • Recurrence
  • 20. Stress Fractures: Tibia & Fibula • Abnormal repetitive loads causing imbalance of resorption over formation • Running > basketball > soccer > skating > aerobics > ballet • Athletes with decreased mineral content • Amenorrheic females
  • 21. Stress Fractures: Tibia & Fibula • Clinical features • Recent change in routine • Insidious, localized tenderness, swelling • Diagnosis • X-ray initially negative • (2-3 weeks) radiodense haze, periosteal rxn • Transverse cortical lucency • Bone scan
  • 22. Stress Fractures: Tibia & Fibula • Conservative Treatment • Rest, cessation of activity • NWB PWB to WBAT • As symptoms allow • Swimming, pool running, cycling • Resume competition full motion, strength, minimal tenderness • Surgical Treatment • Excision, bone grafting • PEMF bone stimulation • Intramedullary fixation
  • 23. Gastrocnemius-Soleus Strain • Common injury • Racquet sports, basketball, running, skiing • Plantaris tear • Clinical Features • Sudden knee extension while crouched w/ ankle DF • Sudden sharp pain • Difficulty with toe-off
  • 24. Gastrocnemius-Soleus Strain • Treatment • Casting/immobilization • RICE • WBAT • Active dorsiflexion • Gentle passive stretching more functional recovery
  • 25. Nerve Entrapment Syndromes • Superficial Peroneal Nerve • Neuritic pain, burning, tingling, radiation • Tenderness over area of compression • R/o compartment syndrome, double-crush • Tx • Injections, modalities, thermogesic creams, neuroactive medications, shoe modifications • Surgical nerve release, fasciotomy
  • 26. Popliteal Artery Entrapment Syndrome • Rare, mimics pain chronic compartment syndrome • Recognition important as may lead to amputation
  • 28. Popliteal Artery Entrapment Syndrome • Clinical • Cramping pain, intermittent claudication • 67% bilaterality • Pulses diminished or absent • Knee hyperextended, ankle dorsiflexed • Knee warm, foot cool • Ischemia, thrombosis; palpable mass
  • 29. Popliteal Artery Entrapment Syndrome • Diagnostics • Provocative testing (treadmill) absent pulses • Doppler, U/S  flow, aneurysm • Biplanar arteriography after exercise • Deviation, occlusion, dilatation • Treatment • Release • Bypass grafting
  • 30. Venous Disease • Effort thrombosis • Pain, edema, distended veins, discoloration • Venography • Tx: anticoagulation  heparin, coumadin • Thrombophlebitis • Immobilization after injury • Air travel • Dehydration • Oral contraceptives • Alcohol/drug use
  • 31. Delayed-Onset Muscle Soreness • Unaccustomed to activity • Eccentric muscle contraction  microinjury, subcellular level • Inability to generate maximal force • Pain, fatigue • Loss of performance 24-48hrs • Resolve 5-7 days • Tx • Symptomatic treatment
  • 32. Medial Ankle Sprains • Rare, associated with lateral, bony injuries • 2 portions: • Superficial deltoid • Deep deltoid DeepSuperficial
  • 33. Medial Ankle Sprains • Biomechanics of deltoid • Prohibits abduction • Secondary restraint against anterior translation ankle • Deep deltoid • Greatest restraint lateral translation (after fibula) • Highest load to failure • Valgus tilt  superficial & deep rupture
  • 34. Medial Ankle Sprains • Clinical Evaluation • Lateral injury, fracture, syndesmotic injury • Abduction, eversion • Other injuries • Maisonneuve, FDL, PTT, FHL, tibial & saph n
  • 35. • Radiographs • Assoc fx, AP stress  valgus tilt, normal with incomplete injury • MRI Medial Ankle Sprains
  • 36. • Conservative Treatment • Dependent upon associated injuries • Walking cast, boot 6-8wks • Functional treatment • Surgical treatment • Inability to reduce medial clear space • Direct repair • Suture, suture anchors • Stirrup brace 6 months after surgery Medial Ankle Sprains
  • 37. Sinus Tarsi Syndrome • Related to subtalar instability • Nebulous condition w/ pain in lateral ankle & sinus tarsi • Pathologic anatomy not clearly defined • Scarring or degenerative changes to soft tissues within sinus tarsi • Loss of nerve function  loss of proprioception
  • 38. Sinus Tarsi Syndrome • Diagnosis • Clinical Evaluation • Pain lateral ankle, sinus tarsi • Prior inversion injury • Instability rare • Radiologic Evaluation • X-rays- normal • Subtalar arthrography • Absence of microrecesses, interdigitation • MRI • Fibrosis, fluid collections
  • 39. Sinus Tarsi Syndrome • Conservative treatment • Injection • Surgical treatment • Exploratory, sx may persist • Resect fibrofatty tissue & extensions from IER • Subtalar arthroscopy • Results • 96 cases; 69% excellent, 25% good, 6% failures
  • 40. Os Trigonum/Posterior Impingement • First Described Rosenmuller • First Described as Fx Shepherd 1822 • Asymptomatic • Separate Ossification Center • Age 8-11 • Incidence 1.7 - 7%
  • 41. Os Trigonum/Posterior Impingement • No side predominence • Vary in size • Normal anatomic variation • Mild extension • Thumb shaped projection - 1 cm (Stieda’s Process)
  • 42. Os Trigonum/Posterior Impingement • Symptoms • Pain with PF • Young Athlete/Dancer • Spectrum • Acute – Fx • Gradual - FHL Tendinitis
  • 43. Os Trigonum/Posterior Impingement • Conservative • Immobilization • NSAID • Surgical – Excision
  • 45. Evaluation • 54% of talar dome • Os trigonum • Haglund’s • Achilles insertion site
  • 46. Foot Problems in the Athlete
  • 47. Goals & Objectives • Specific problems • Midfoot • Forefoot
  • 48. Os Peroneum • Ossification • Complete • Less than complete • Multipartite • Location • Lateral border calc & plantar lateral cuboid • Useful marker for ruptured PL
  • 49. Painful Os Peroneum Syndrome (POS) & PL Disruption
  • 50. POPS/Peroneus Long. Tendonitis/Tear • Conservative treatment • Cast immobilization (20% success) • Physical therapy • Surgical treatment • Os peroneum excision/tendon repair • Peroneal tenodesis
  • 51. Forefoot Sprains •First MTP joint/ “turf toe” •Etiology •Hard artificial surface, flexible shoes •True incidence not defined, but many reports indicate a fairly common entity •Functional disability •Push-off impaired •Compromised forward drive and running •More missed games & practices than other injuries
  • 52. Forefoot Sprains • Mechanisms of injury • Hyperextension of the MTP joint • Foot dorsiflexed, forefoot fixed, heel raised • Joint capsule tears, compression injury to the dorsal articular surface with extremes of dorsiflexion
  • 53. Forefoot Sprains • Etiologic factors • Shoe-surface interface • Flexible shoes • Hardness of surface • Loss of resiliency with time • Enhanced friction • Fixes foot allows extremes of motion • Excessive DF • In those with limited DF
  • 54. Forefoot Sprains • Radiographs • R/o fx, avulsions, impaction • Other methods usually not necessary
  • 55. Forefoot Sprains • Conservative treatment • RICE, NSAID’s • Taping, boot • Increase stiffness in shoe • Custom molded insert/Morton’s extension • Rehab once decrease in sx allows
  • 56. Forefoot Sprains • Surgical treatment • Rarely necessary • Instability • Loose bodies, osteochondral injury • Ligamentous disruption • Sesamoid retraction • Repair plantar plate, FHB, long flexor transfer
  • 57. Metatarsal Stress Fractures • Etiology • Injury to opposite limp  increased stress to normal limb • Muscle fatigue  increase in loading • Hard surfaces, poorly cushioned shoes, heavy heel strike • Rigid cavus feet  decrease shock absorption of the midfoot  increase stress on MT’s • Hypermobile first ray or long 2nd MT
  • 58. Stress Fractures • High index of suspicion • Focal tenderness • Min/no swelling • X-rays nl < 3-4 wks • Bone scan, MRI, & CT scans
  • 59. Treatment • Activity modifications • Non-impact sports • Avoid casting if possible
  • 60. Treatment • Shoe modifications • Rigid cavus foot • Flexible, cushioned arch support • Hyperpronation/hypermobile 1st ray • Soft medial arch support • Long 2nd MT • MT pad