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Approach to Musculoskeletal
Injuries
Dr. Ashraf Hussein, ER consultant
OVERVIEW
• Significant percentage of all emergency
room care rendered.
• In HUP ED, over 6 month period, survey
showed chief complaint of musculoskeletal
problem comprised approx. 11% of all
patients (1 in 9).
Musculoskeletal Injuries
• ED physician needs a coherent, systematic
approach to orthopaedic complaints.
• Severity ranges from trivial sprains to life-
or limb-threatening trauma.
• Often acute trauma is the cause of the
presentation.
• Pain and decreased ROM are the main
symptoms.
What We Commonly See
• Musculoskeletal disorders commonly seen
in the ED include:
– Soft Tissue Injuries (strains and sprains)**
– Fractures (open, closed, long bone, pelvic, occult)
– Dislocations
– Infections (soft-tissue, bite wounds)**
– Effusions **
– Deep venous thrombosis
Less Common But…
• Musculoskeletal disorders less commonly
seen in the ED, but unmistakably important:
– Spinal Injuries
– Crush Injuries
– Compartment syndrome
FRACTURES
Fractures
• A partial or complete break in a bone.
– Bone is the only tissue in the human body other
than liver that heals by regeneration instead of
by scarring.
– For regeneration to occur the bone must be
immobilized to allow uninterrupted formation
of new bone.
Fractures
• New Bone Formation:
• A hematoma forms between realigned Fx fragments.
• Hematopoietic cells in the hematoma secrete growth
factors (GF’s).
• GF’s stimulate formation of granulation tissue at the
Fx ends, slowly resorbing the hematoma.
• A primary callus forms, progressing from a soft
callus to hard callus.
• Final phase of healing: during remodeling the bone
reassumes its original architecture.
Fractures
• Nomenclature of fractures is essential to
successful Fx management in the ED
• Adequate description:
– Open or closed?
– Which bone involved?
– Location within the bone?
– Direction of the main fracture line?
– Number of fragments?
– Alignment and displacement of the fragments?
– Complications?
Closed Fracture
Open Elbow Fracture
Open Fractures
• ORTHOPEDIC EMERGENCY
• Immediate control of hemorrhage.
• Splinting +/- reduction.
• Copious irrigation.
• Early administration of sufficient analgesia,
appropriate antibiotics, and tetanus
prophylaxis.
• Emergent consultation w/ orthopaedics for all
Type II and Type III open Fx’s (and some Type
I’s).
Fractures-Open Classification
I <1cm long, minimal contamination, low energy force
ABX: 1st/2nd Ceph for 3d
II >1cm long, moderate contamination and force
ABX: 1st/2nd Ceph plus aminoglyc for 3d
III High energy, comminuted fx, extensive tissue
damage, enough tissue to cover wound, extensive
contamination, arterial injury
ABX: 1st/2nd Ceph plus aminoglyc for 5d
PELVIC FRACTURES
Pelvic Fractures
• Least common fracture (3%)
• Most are result of auto-collisions
• Commonly associated with other injuries
• Pelvis contains many important structures:
– Iliac vessels, urogenital organs, nerve plexi…..
• Patients can sustain large volume blood loss
Clinical Evaluation
• Any patient assessment begins with the ABC’s
(Airway, Breathing, Circulation)
• Complete neurologic and vascular exam
• Have high suspicion of intra-abdominal
injuries
• Physical exam:
– Ecchymosis or contusion around hips, perineum
– Pelvic instability with stressing
– Suspect if signs of urologic/gyn findings: blood at
urethral meatus, high-riding prostate
– High force mechanisms also associated (mvc,
femur fx)
Pelvic Fracture-Management
• #1--Control of bleeding vessels
• In hemodynamically unstable pts, consider
angiography
• #2--Surgical management of the broken
bone can proceed LATER after life-
threatening conditions are controlled.
LONG BONE FRACTURES
Long Bone Fractures
• Fractures of the femur, humerus, tibia/fibula
• Blunt and penetrating trauma
• Requires high energy to break bone,
therefore look for other injuries.
• Bone has a generous blood supply.
• Does patient have associated bleeding
disorder?
Long Bone Fractures
• Fx’s cause localized bleeding and this can
be substantial resulting in hypovolemic
shock.
– Humerus: 200-500cc
– Unilateral tibia/fibula: 400-800cc
– Femur Fx: 1000-1500cc
Long Bone Fracture
Management
• ABC’s
• Neurovascular exam (vascular +/- nerve injury)
• Splint involved extremity
– Reduction decreases pain, bleeding
• Orthopedic consultation for definitive
management
• Complications:
– Fat-emboli syndrome
– Blood loss
Fracture Complications
• Vascular Injuries
– Most commonly occur in open Fx’s, Fx-dislocations,
or widely displaced Fx’s and at sites where the
vessels lie in close proximity to the bone or @ sites
where the vessels are held in a relatively fixed
position.
• Classic signs: The 5 P’s: Pain, Pallor,
Pulselessness (or diminished pulse),
Paresthesia, and Paralysis.
– Location of Fx and MOI dictate need to assess for
potential vascular injury in asymptomatic patient.
Fracture Complications
• Nerve Injuries
– Occur more frequently than vascular injuries in
assoc. w/ Fx.
– Can occur 2/2 blunt trauma, along path of
penetrating trauma, or be caused by the Fx
fragments themselves.
– Nerves are @ increased risk of injury when
they are superficial to the skin, lie close to the
bone, or span a joint, making them susceptible
to stretch injury.
Fracture Complications
• Fat Emboli Syndrome (FES)
– Most common form of non-thrombotic
embolism.
– Single or multiple long bone fractures in young
or pelvic/hip fractures in elderly predispose to
FES.
– 20% of patients w/ pelvic or long bone
fractures have detectable fat droplets in their
blood.
• Fat Emboli Syndrome (FES)
– Vast majority remain asymptomatic
– Has characteristic clinical course:
1. Fracture sustained.
2. Other than fracture-associated pain, patient is
asymptomatic for 12-36 hours.
3. Sudden onset of life-threatening syndrome characterized
by rapid cardiopulmonary and neurologic deterioration,
agitation, hallucinations, delirium, coma, hypoxia,
dyspnea, tachypnea, and tachycardia leading to DIC and
ARDS.
Deep Venous Thrombosis
Deep Venous Thrombosis
• Clot forming in one of the deep veins of an
extremity: Legs > Arms.
• If clot propagates above the popliteal fossa,
substantial risk of piece of clot breaking free,
embolizing to the pulmonary circulation.
• Risk of respiratory distress, hypoxia, pleuritic
chest pain, circulatory compromise, death.
• Doppler ultrasound; CXR; V/Q Scan; spiral CT
• Treatment: anticoagulation
CRUSH INJURY
Crush Injuries
• First descriptions from military records,
bombings in England and Europe during world
wars
• Now more commonly seen in natural disasters,
building collapse, acts of terrorism, after
poisioning, after drug overdoses
• Injury results from prolonged continuous
pressure on a body part, typically an extremity
Crush Injuries
• Under direct pressure, cellular ischemia incurred
causing loss cellular integrity
• Cells leak K+ and myoglobin
• Influx of ions into the cells causing irreversible
cell death
• Can have large fluid volume shifts
• Electrolyte abnormalities:
– Hyperkalemia, hyperphosphatemia, myoglobinemia,
hypocalcemia, metabolic acidosis
Crush Injuries
• Myoglobin concentrates in
the renal tubules
obstructive nephropathy
 acute renal failure
• When ARF occurs,
mortality 20-40%
• Arrythmias
• Concern for sepsis with
devitalized tissue
Crush Injuries-Treatment
• Early consideration /recognition
• Fluid resuscitation
– ARF approaches 100% if hydration
delayed >12 hours
• Alkalinize the urine –add sodium
bicarbonate to IVF
– prevents myoglobin precipitation and
enhances excretion
COMPARTMENT
SYNDROME
Compartment Syndrome
• Occurs when pressure w/i soft tissues in a
fixed body compartment increases to level
that exceeds venous pressure,
compromising venous blood flow, and
limiting capillary perfusion.
• Leads to muscle ischemia and necrosis.
• TRUE ORTHOPEDIC EMERGENCY
Compartment Syndrome
• Contributing Factors
• External:
– Conditions that reduced size of muscle
compartment (casts/splints); occlusive
dressing; eschar of burns
• Internal:
– Conditions that increase compartment volume:
bleeding, swelling, fluid extravasation into
tissue
Hand and Foot
Compartments
CS-Recognition
• Suspect with long bone fx, crush injuries
• Presents as pain out of proportion to
physical findings, +/- hypoesthesia,
pulselessness (late).
Measure intra-compartmental pressure
when considering compartment
syndrome
Pressures >40mmHg considered dangerous
Compartment Syndrome
Compartment syndrome
should be suspected in
long bone Fx’s and Fx’s
associated w/ significant
vascular injuries or
pronounced swelling.
Intra-compartment pressures
must be measured once the
issue of compartment
syndrome is raised.
SPINAL INJURY
Spinal Injuries
• Devastating injuries
• >80% occur in young males
• Motor vehicle accidents, falls from height,
gunshot wound
• Worrisome presentations:
– pain over spine in setting of trauma
– loss of motor function
– incontinence
– priapism
Spinal Injuries
• Additional risk factors for spinal PAIN:
– Metastatic cancer
– Osteoporosis, rheumatic dz, steroid use
(compression fracture)
– IV drug use (epidural abscess)
– Spinal hardware
Spinal Injury
• Assessment
– ABCs
– Immobilize neck and back
– GCS, motor/sensory/sphincter tone exam
• Imaging
– Plain c-spine films (lateral only detects >85%
of cervical spine injuries)
– CT/MRI for injuries with neuro deficits and
identifiable spine fractures.
• Identifiable spine
fractures require
Orthopedic OR
Neurosurgical
consultation
Subluxation and Dislocation
Subluxation and Dislocation
• Acute or chronic ligamentous laxity/tearing
can result in subluxation or dislocation of a
joint.
• Classic example: glenohumeral joint:
– Subluxation: 1 bone becomes partially
disarticulated from the other; articular surfaces
remain partially intact.
– Dislocation: bones completely disarticulated;
no parts of articular surfaces are in contact.
Dislocations
• Nomenclature is straightforward:
– Most occur @ a joint formed by 2 bones and
the dislocation is named after the affected joint.
– Direction of dislocation refers to the position of
the distal bone in relation to the proximal.
• Clinically:
– Pain, deformity, decreased ROM.
– Certain dislocations are associated w/
specific complications, which must be ruled
out in the routine evaluation of the
injury…e.g., the axillary nerve (12%) and
the musculocutaneous nerve (2%) are @
risk in anterior dislocations of the
glenohumeral joint.
– Smooth, timely reduction is mandatory.
Bilateral Hip Dislocations
More dislocations
QUESTIONS ?

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9- MusculoskeletalOrthopedic Emergencies.ppt

  • 1. Approach to Musculoskeletal Injuries Dr. Ashraf Hussein, ER consultant
  • 2. OVERVIEW • Significant percentage of all emergency room care rendered. • In HUP ED, over 6 month period, survey showed chief complaint of musculoskeletal problem comprised approx. 11% of all patients (1 in 9).
  • 3. Musculoskeletal Injuries • ED physician needs a coherent, systematic approach to orthopaedic complaints. • Severity ranges from trivial sprains to life- or limb-threatening trauma. • Often acute trauma is the cause of the presentation. • Pain and decreased ROM are the main symptoms.
  • 4. What We Commonly See • Musculoskeletal disorders commonly seen in the ED include: – Soft Tissue Injuries (strains and sprains)** – Fractures (open, closed, long bone, pelvic, occult) – Dislocations – Infections (soft-tissue, bite wounds)** – Effusions ** – Deep venous thrombosis
  • 5. Less Common But… • Musculoskeletal disorders less commonly seen in the ED, but unmistakably important: – Spinal Injuries – Crush Injuries – Compartment syndrome
  • 7. Fractures • A partial or complete break in a bone. – Bone is the only tissue in the human body other than liver that heals by regeneration instead of by scarring. – For regeneration to occur the bone must be immobilized to allow uninterrupted formation of new bone.
  • 8. Fractures • New Bone Formation: • A hematoma forms between realigned Fx fragments. • Hematopoietic cells in the hematoma secrete growth factors (GF’s). • GF’s stimulate formation of granulation tissue at the Fx ends, slowly resorbing the hematoma. • A primary callus forms, progressing from a soft callus to hard callus. • Final phase of healing: during remodeling the bone reassumes its original architecture.
  • 9. Fractures • Nomenclature of fractures is essential to successful Fx management in the ED • Adequate description: – Open or closed? – Which bone involved? – Location within the bone? – Direction of the main fracture line? – Number of fragments? – Alignment and displacement of the fragments? – Complications?
  • 12. Open Fractures • ORTHOPEDIC EMERGENCY • Immediate control of hemorrhage. • Splinting +/- reduction. • Copious irrigation. • Early administration of sufficient analgesia, appropriate antibiotics, and tetanus prophylaxis. • Emergent consultation w/ orthopaedics for all Type II and Type III open Fx’s (and some Type I’s).
  • 13. Fractures-Open Classification I <1cm long, minimal contamination, low energy force ABX: 1st/2nd Ceph for 3d II >1cm long, moderate contamination and force ABX: 1st/2nd Ceph plus aminoglyc for 3d III High energy, comminuted fx, extensive tissue damage, enough tissue to cover wound, extensive contamination, arterial injury ABX: 1st/2nd Ceph plus aminoglyc for 5d
  • 15. Pelvic Fractures • Least common fracture (3%) • Most are result of auto-collisions • Commonly associated with other injuries • Pelvis contains many important structures: – Iliac vessels, urogenital organs, nerve plexi….. • Patients can sustain large volume blood loss
  • 16. Clinical Evaluation • Any patient assessment begins with the ABC’s (Airway, Breathing, Circulation) • Complete neurologic and vascular exam • Have high suspicion of intra-abdominal injuries • Physical exam: – Ecchymosis or contusion around hips, perineum – Pelvic instability with stressing – Suspect if signs of urologic/gyn findings: blood at urethral meatus, high-riding prostate – High force mechanisms also associated (mvc, femur fx)
  • 17.
  • 18.
  • 19. Pelvic Fracture-Management • #1--Control of bleeding vessels • In hemodynamically unstable pts, consider angiography • #2--Surgical management of the broken bone can proceed LATER after life- threatening conditions are controlled.
  • 21. Long Bone Fractures • Fractures of the femur, humerus, tibia/fibula • Blunt and penetrating trauma • Requires high energy to break bone, therefore look for other injuries. • Bone has a generous blood supply. • Does patient have associated bleeding disorder?
  • 22. Long Bone Fractures • Fx’s cause localized bleeding and this can be substantial resulting in hypovolemic shock. – Humerus: 200-500cc – Unilateral tibia/fibula: 400-800cc – Femur Fx: 1000-1500cc
  • 24. Management • ABC’s • Neurovascular exam (vascular +/- nerve injury) • Splint involved extremity – Reduction decreases pain, bleeding • Orthopedic consultation for definitive management • Complications: – Fat-emboli syndrome – Blood loss
  • 25. Fracture Complications • Vascular Injuries – Most commonly occur in open Fx’s, Fx-dislocations, or widely displaced Fx’s and at sites where the vessels lie in close proximity to the bone or @ sites where the vessels are held in a relatively fixed position. • Classic signs: The 5 P’s: Pain, Pallor, Pulselessness (or diminished pulse), Paresthesia, and Paralysis. – Location of Fx and MOI dictate need to assess for potential vascular injury in asymptomatic patient.
  • 26. Fracture Complications • Nerve Injuries – Occur more frequently than vascular injuries in assoc. w/ Fx. – Can occur 2/2 blunt trauma, along path of penetrating trauma, or be caused by the Fx fragments themselves. – Nerves are @ increased risk of injury when they are superficial to the skin, lie close to the bone, or span a joint, making them susceptible to stretch injury.
  • 27. Fracture Complications • Fat Emboli Syndrome (FES) – Most common form of non-thrombotic embolism. – Single or multiple long bone fractures in young or pelvic/hip fractures in elderly predispose to FES. – 20% of patients w/ pelvic or long bone fractures have detectable fat droplets in their blood.
  • 28. • Fat Emboli Syndrome (FES) – Vast majority remain asymptomatic – Has characteristic clinical course: 1. Fracture sustained. 2. Other than fracture-associated pain, patient is asymptomatic for 12-36 hours. 3. Sudden onset of life-threatening syndrome characterized by rapid cardiopulmonary and neurologic deterioration, agitation, hallucinations, delirium, coma, hypoxia, dyspnea, tachypnea, and tachycardia leading to DIC and ARDS.
  • 30. Deep Venous Thrombosis • Clot forming in one of the deep veins of an extremity: Legs > Arms. • If clot propagates above the popliteal fossa, substantial risk of piece of clot breaking free, embolizing to the pulmonary circulation. • Risk of respiratory distress, hypoxia, pleuritic chest pain, circulatory compromise, death. • Doppler ultrasound; CXR; V/Q Scan; spiral CT • Treatment: anticoagulation
  • 31.
  • 32.
  • 34. Crush Injuries • First descriptions from military records, bombings in England and Europe during world wars • Now more commonly seen in natural disasters, building collapse, acts of terrorism, after poisioning, after drug overdoses • Injury results from prolonged continuous pressure on a body part, typically an extremity
  • 35. Crush Injuries • Under direct pressure, cellular ischemia incurred causing loss cellular integrity • Cells leak K+ and myoglobin • Influx of ions into the cells causing irreversible cell death • Can have large fluid volume shifts • Electrolyte abnormalities: – Hyperkalemia, hyperphosphatemia, myoglobinemia, hypocalcemia, metabolic acidosis
  • 36. Crush Injuries • Myoglobin concentrates in the renal tubules obstructive nephropathy  acute renal failure • When ARF occurs, mortality 20-40% • Arrythmias • Concern for sepsis with devitalized tissue
  • 37. Crush Injuries-Treatment • Early consideration /recognition • Fluid resuscitation – ARF approaches 100% if hydration delayed >12 hours • Alkalinize the urine –add sodium bicarbonate to IVF – prevents myoglobin precipitation and enhances excretion
  • 39. Compartment Syndrome • Occurs when pressure w/i soft tissues in a fixed body compartment increases to level that exceeds venous pressure, compromising venous blood flow, and limiting capillary perfusion. • Leads to muscle ischemia and necrosis. • TRUE ORTHOPEDIC EMERGENCY
  • 40. Compartment Syndrome • Contributing Factors • External: – Conditions that reduced size of muscle compartment (casts/splints); occlusive dressing; eschar of burns • Internal: – Conditions that increase compartment volume: bleeding, swelling, fluid extravasation into tissue
  • 41.
  • 43. CS-Recognition • Suspect with long bone fx, crush injuries • Presents as pain out of proportion to physical findings, +/- hypoesthesia, pulselessness (late).
  • 44. Measure intra-compartmental pressure when considering compartment syndrome Pressures >40mmHg considered dangerous
  • 45. Compartment Syndrome Compartment syndrome should be suspected in long bone Fx’s and Fx’s associated w/ significant vascular injuries or pronounced swelling. Intra-compartment pressures must be measured once the issue of compartment syndrome is raised.
  • 47. Spinal Injuries • Devastating injuries • >80% occur in young males • Motor vehicle accidents, falls from height, gunshot wound • Worrisome presentations: – pain over spine in setting of trauma – loss of motor function – incontinence – priapism
  • 48. Spinal Injuries • Additional risk factors for spinal PAIN: – Metastatic cancer – Osteoporosis, rheumatic dz, steroid use (compression fracture) – IV drug use (epidural abscess) – Spinal hardware
  • 49. Spinal Injury • Assessment – ABCs – Immobilize neck and back – GCS, motor/sensory/sphincter tone exam • Imaging – Plain c-spine films (lateral only detects >85% of cervical spine injuries) – CT/MRI for injuries with neuro deficits and identifiable spine fractures.
  • 50. • Identifiable spine fractures require Orthopedic OR Neurosurgical consultation
  • 51.
  • 53. Subluxation and Dislocation • Acute or chronic ligamentous laxity/tearing can result in subluxation or dislocation of a joint. • Classic example: glenohumeral joint: – Subluxation: 1 bone becomes partially disarticulated from the other; articular surfaces remain partially intact. – Dislocation: bones completely disarticulated; no parts of articular surfaces are in contact.
  • 54. Dislocations • Nomenclature is straightforward: – Most occur @ a joint formed by 2 bones and the dislocation is named after the affected joint. – Direction of dislocation refers to the position of the distal bone in relation to the proximal.
  • 55. • Clinically: – Pain, deformity, decreased ROM. – Certain dislocations are associated w/ specific complications, which must be ruled out in the routine evaluation of the injury…e.g., the axillary nerve (12%) and the musculocutaneous nerve (2%) are @ risk in anterior dislocations of the glenohumeral joint. – Smooth, timely reduction is mandatory.
  • 56.
  • 59.