2. OVERVIEW
• Significant percentage of all emergency
room care rendered.
• In HUP ED, over 6 month period, survey
showed chief complaint of musculoskeletal
problem comprised approx. 11% of all
patients (1 in 9).
3. Musculoskeletal Injuries
• ED physician needs a coherent, systematic
approach to orthopaedic complaints.
• Severity ranges from trivial sprains to life-
or limb-threatening trauma.
• Often acute trauma is the cause of the
presentation.
• Pain and decreased ROM are the main
symptoms.
4. What We Commonly See
• Musculoskeletal disorders commonly seen
in the ED include:
– Soft Tissue Injuries (strains and sprains)**
– Fractures (open, closed, long bone, pelvic, occult)
– Dislocations
– Infections (soft-tissue, bite wounds)**
– Effusions **
– Deep venous thrombosis
5. Less Common But…
• Musculoskeletal disorders less commonly
seen in the ED, but unmistakably important:
– Spinal Injuries
– Crush Injuries
– Compartment syndrome
7. Fractures
• A partial or complete break in a bone.
– Bone is the only tissue in the human body other
than liver that heals by regeneration instead of
by scarring.
– For regeneration to occur the bone must be
immobilized to allow uninterrupted formation
of new bone.
8. Fractures
• New Bone Formation:
• A hematoma forms between realigned Fx fragments.
• Hematopoietic cells in the hematoma secrete growth
factors (GF’s).
• GF’s stimulate formation of granulation tissue at the
Fx ends, slowly resorbing the hematoma.
• A primary callus forms, progressing from a soft
callus to hard callus.
• Final phase of healing: during remodeling the bone
reassumes its original architecture.
9. Fractures
• Nomenclature of fractures is essential to
successful Fx management in the ED
• Adequate description:
– Open or closed?
– Which bone involved?
– Location within the bone?
– Direction of the main fracture line?
– Number of fragments?
– Alignment and displacement of the fragments?
– Complications?
12. Open Fractures
• ORTHOPEDIC EMERGENCY
• Immediate control of hemorrhage.
• Splinting +/- reduction.
• Copious irrigation.
• Early administration of sufficient analgesia,
appropriate antibiotics, and tetanus
prophylaxis.
• Emergent consultation w/ orthopaedics for all
Type II and Type III open Fx’s (and some Type
I’s).
13. Fractures-Open Classification
I <1cm long, minimal contamination, low energy force
ABX: 1st/2nd Ceph for 3d
II >1cm long, moderate contamination and force
ABX: 1st/2nd Ceph plus aminoglyc for 3d
III High energy, comminuted fx, extensive tissue
damage, enough tissue to cover wound, extensive
contamination, arterial injury
ABX: 1st/2nd Ceph plus aminoglyc for 5d
15. Pelvic Fractures
• Least common fracture (3%)
• Most are result of auto-collisions
• Commonly associated with other injuries
• Pelvis contains many important structures:
– Iliac vessels, urogenital organs, nerve plexi…..
• Patients can sustain large volume blood loss
16. Clinical Evaluation
• Any patient assessment begins with the ABC’s
(Airway, Breathing, Circulation)
• Complete neurologic and vascular exam
• Have high suspicion of intra-abdominal
injuries
• Physical exam:
– Ecchymosis or contusion around hips, perineum
– Pelvic instability with stressing
– Suspect if signs of urologic/gyn findings: blood at
urethral meatus, high-riding prostate
– High force mechanisms also associated (mvc,
femur fx)
17.
18.
19. Pelvic Fracture-Management
• #1--Control of bleeding vessels
• In hemodynamically unstable pts, consider
angiography
• #2--Surgical management of the broken
bone can proceed LATER after life-
threatening conditions are controlled.
21. Long Bone Fractures
• Fractures of the femur, humerus, tibia/fibula
• Blunt and penetrating trauma
• Requires high energy to break bone,
therefore look for other injuries.
• Bone has a generous blood supply.
• Does patient have associated bleeding
disorder?
22. Long Bone Fractures
• Fx’s cause localized bleeding and this can
be substantial resulting in hypovolemic
shock.
– Humerus: 200-500cc
– Unilateral tibia/fibula: 400-800cc
– Femur Fx: 1000-1500cc
25. Fracture Complications
• Vascular Injuries
– Most commonly occur in open Fx’s, Fx-dislocations,
or widely displaced Fx’s and at sites where the
vessels lie in close proximity to the bone or @ sites
where the vessels are held in a relatively fixed
position.
• Classic signs: The 5 P’s: Pain, Pallor,
Pulselessness (or diminished pulse),
Paresthesia, and Paralysis.
– Location of Fx and MOI dictate need to assess for
potential vascular injury in asymptomatic patient.
26. Fracture Complications
• Nerve Injuries
– Occur more frequently than vascular injuries in
assoc. w/ Fx.
– Can occur 2/2 blunt trauma, along path of
penetrating trauma, or be caused by the Fx
fragments themselves.
– Nerves are @ increased risk of injury when
they are superficial to the skin, lie close to the
bone, or span a joint, making them susceptible
to stretch injury.
27. Fracture Complications
• Fat Emboli Syndrome (FES)
– Most common form of non-thrombotic
embolism.
– Single or multiple long bone fractures in young
or pelvic/hip fractures in elderly predispose to
FES.
– 20% of patients w/ pelvic or long bone
fractures have detectable fat droplets in their
blood.
28. • Fat Emboli Syndrome (FES)
– Vast majority remain asymptomatic
– Has characteristic clinical course:
1. Fracture sustained.
2. Other than fracture-associated pain, patient is
asymptomatic for 12-36 hours.
3. Sudden onset of life-threatening syndrome characterized
by rapid cardiopulmonary and neurologic deterioration,
agitation, hallucinations, delirium, coma, hypoxia,
dyspnea, tachypnea, and tachycardia leading to DIC and
ARDS.
30. Deep Venous Thrombosis
• Clot forming in one of the deep veins of an
extremity: Legs > Arms.
• If clot propagates above the popliteal fossa,
substantial risk of piece of clot breaking free,
embolizing to the pulmonary circulation.
• Risk of respiratory distress, hypoxia, pleuritic
chest pain, circulatory compromise, death.
• Doppler ultrasound; CXR; V/Q Scan; spiral CT
• Treatment: anticoagulation
34. Crush Injuries
• First descriptions from military records,
bombings in England and Europe during world
wars
• Now more commonly seen in natural disasters,
building collapse, acts of terrorism, after
poisioning, after drug overdoses
• Injury results from prolonged continuous
pressure on a body part, typically an extremity
35. Crush Injuries
• Under direct pressure, cellular ischemia incurred
causing loss cellular integrity
• Cells leak K+ and myoglobin
• Influx of ions into the cells causing irreversible
cell death
• Can have large fluid volume shifts
• Electrolyte abnormalities:
– Hyperkalemia, hyperphosphatemia, myoglobinemia,
hypocalcemia, metabolic acidosis
36. Crush Injuries
• Myoglobin concentrates in
the renal tubules
obstructive nephropathy
acute renal failure
• When ARF occurs,
mortality 20-40%
• Arrythmias
• Concern for sepsis with
devitalized tissue
37. Crush Injuries-Treatment
• Early consideration /recognition
• Fluid resuscitation
– ARF approaches 100% if hydration
delayed >12 hours
• Alkalinize the urine –add sodium
bicarbonate to IVF
– prevents myoglobin precipitation and
enhances excretion
39. Compartment Syndrome
• Occurs when pressure w/i soft tissues in a
fixed body compartment increases to level
that exceeds venous pressure,
compromising venous blood flow, and
limiting capillary perfusion.
• Leads to muscle ischemia and necrosis.
• TRUE ORTHOPEDIC EMERGENCY
40. Compartment Syndrome
• Contributing Factors
• External:
– Conditions that reduced size of muscle
compartment (casts/splints); occlusive
dressing; eschar of burns
• Internal:
– Conditions that increase compartment volume:
bleeding, swelling, fluid extravasation into
tissue
43. CS-Recognition
• Suspect with long bone fx, crush injuries
• Presents as pain out of proportion to
physical findings, +/- hypoesthesia,
pulselessness (late).
45. Compartment Syndrome
Compartment syndrome
should be suspected in
long bone Fx’s and Fx’s
associated w/ significant
vascular injuries or
pronounced swelling.
Intra-compartment pressures
must be measured once the
issue of compartment
syndrome is raised.
47. Spinal Injuries
• Devastating injuries
• >80% occur in young males
• Motor vehicle accidents, falls from height,
gunshot wound
• Worrisome presentations:
– pain over spine in setting of trauma
– loss of motor function
– incontinence
– priapism
48. Spinal Injuries
• Additional risk factors for spinal PAIN:
– Metastatic cancer
– Osteoporosis, rheumatic dz, steroid use
(compression fracture)
– IV drug use (epidural abscess)
– Spinal hardware
49. Spinal Injury
• Assessment
– ABCs
– Immobilize neck and back
– GCS, motor/sensory/sphincter tone exam
• Imaging
– Plain c-spine films (lateral only detects >85%
of cervical spine injuries)
– CT/MRI for injuries with neuro deficits and
identifiable spine fractures.
53. Subluxation and Dislocation
• Acute or chronic ligamentous laxity/tearing
can result in subluxation or dislocation of a
joint.
• Classic example: glenohumeral joint:
– Subluxation: 1 bone becomes partially
disarticulated from the other; articular surfaces
remain partially intact.
– Dislocation: bones completely disarticulated;
no parts of articular surfaces are in contact.
54. Dislocations
• Nomenclature is straightforward:
– Most occur @ a joint formed by 2 bones and
the dislocation is named after the affected joint.
– Direction of dislocation refers to the position of
the distal bone in relation to the proximal.
55. • Clinically:
– Pain, deformity, decreased ROM.
– Certain dislocations are associated w/
specific complications, which must be ruled
out in the routine evaluation of the
injury…e.g., the axillary nerve (12%) and
the musculocutaneous nerve (2%) are @
risk in anterior dislocations of the
glenohumeral joint.
– Smooth, timely reduction is mandatory.