4.0 guidelines for prescribing glasses in children


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4.0 guidelines for prescribing glasses in children

  1. 1. Gauri S. Shrestha, M.Optom, FIACLE Sujata Rizal, B.Optom Final Year Prativa Devkota, Final year
  2. 2. Uncorrected refractive error leads to: Amblyopia Delayed visual maturation Deviation of the eyes
  3. 3. 1. Is the refractive error within the normal range for the child’s age? 2. Will this particular child’s refractive error emmetropise? 3. Will this level of refractive error disrupt normal visual development or functional vision? 4. Will prescribing spectacles improve visual function or functional vision? 5. Will prescribing glasses interfere with the normal process of emmetropisation?
  4. 4.  Knowledge about the normal refractive error of infants  Emmetropization process  The benefits and risks of prescribing spectacles  Any threat to normal visual development by the prescription
  5. 5.  Most infants are hyperopic by +2.00 D with SD of 2.0  In Initial 3 months, controversy over refractive status to be static or decreasing.  Average is +2.16 D
  6. 6.  There is rapid decrease in refractive status from 3 months to 12 months.  The error decrease to 1. 36 D at 9 months  What happens after ……12 months?  This is followed by a period of slower change until two years for hyperopes and four to five years for myopes
  7. 7.  Few children myopic at birth  most of those who are either myopic or  hyperopic will emmetropise.  The rate of emmetropisation is generally proportional to the initial error. ◦ those who start off close to emmetropia or with a low amount of hyperopia show little change ◦ those who have higher ametropia generally show greater and faster changes
  8. 8.  Higher prevalence of astigmatism at birth but decrease in degree and magnitude ◦ 69% of children have astigmatism greater than 1.00 D at birth  90% Swedish children with astigmatism of +1 or more was reported to decrease. Astigmatism of 1 or more (%) Age 8-30% 1.2 yrs 4-24% 3-4 yrs
  9. 9.  Higher astigmatism decrease more rapidly in initial 3-4 years  Significant WTR, ATR and oblique astigmatism ( least common) are all more common  all types of astigmatism decrease, with infants losing approximately two-thirds of their astigmatism between nine and 21 months (most common: 1.5- 2 yrs)
  10. 10.  Anisometropia (≥1D) is more common in infants than adults.  spherical anisometropia remains more common in children compared with adults up to four years of life  Anisometropia is transient in relatively lower level of refractive error, for example, 2.50 D or less and may not lead to amblyopia
  11. 11.  Probability of emmetropisation decreases as hyperopia increases.  Probability is < than 50 % for three-month-olds, who had a cycloplegic spherical equivalent refraction greater than 5.00 D.
  12. 12.  Hyperopic children with ATR astigmatism at six months maintained their hyperopia (approximately 2.00 D on average)  Hyperopic children with WTR astigmatism lost their hyperopia (ending up at approximately 0.75 D hyperopic at age six years)
  13. 13.  Main tool to predict emmetropisation is to monitor the refraction.  Those who emmetropise, lose approximately one- half of their spherical equivalent refractive error in the first year and approximately one-third between nine and 21 months.  With regards to astigmatism, approximately two- thirds of the astigmatism is lost between nine and 21 months.
  14. 14.  No way to predict with certainty whether a particular child’s anisometropia is transient or will remain into adulthood.  Following things should be taken into account:  monitor a child over a period of four to six months  consider VA—if amblyopia is already present it requires treatment  higher levels of anisometropia (for example, >3.00 D or more and particularly 5.00 D or more) are less likely to be transient.  children with low vision are less likely to fully emmetropise .  Therefore, these children can be prescribed, with the main consideration being to optimize visual function.
  15. 15.  Association between lack of emmetropisation and the presence of strabismus.  However, its not clear whether strabismus interferes with emmetropisation or whether those who do not emmetropise, and thus maintain their higher hyperopia, are more likely to develop strabismus
  16. 16.  Uncorrected high refractive error (hyperopia, astigmatism and anisometropia) during the first few years of life is a risk factor for amblyopia  There is an increased chance of monocular or binocular amblyopia in one-year-olds with 3.50 D or more in one meridian,
  17. 17.  Partial correction of hyperopia greater than 3.50 D at nine to 11 months resulted in improved VA at four years of age and may reduce the incidence of eso  In children with 5.00 D or more, ◦ 25 to 43% have acuity of 6/12 or worse ◦ 87 per cent have acuity worse than 6/6. ◦ Poor accommodation and stereopsis
  18. 18.  In the study by Atkinson and colleagues, there was no difference in the reduction of hyperopia comparing those who were fitted with a partial prescription and the controls.
  19. 19.  Many authors recommend monitoring the refraction (hyperopia, myopia or astigmatism) in infants and toddlers before prescribing.  Frequently unchanging or increasing refractions are associated with amblyopia.
  20. 20.  Only demonstrable amblyopia indicate immediate prescribing  The other main factor, which will influence one’s likelihood of prescribing for hyperopia, is the presence of heterophoria.  Correction of hyperopia to optimize alignment
  21. 21.  Prescribing if the refraction is outside the 95% limits for a particular age.  Main aim is to bring the uncorrected portion just within the normal range, for example, to the 95% limit.  This would leave a large stimulus for emmetropisation and therefore potentially encourage a greater amount of emmetropisation
  22. 22.  It seems that the child’s accommodation cannot overcome the very large uncorrected hyperopia but a correction that is small enough to bring them just within the normal range allows them to accommodate for the remaining hyperopia, resulting in esotropia.  Thus, when a larger prescription is given, it is imperative to see the child approximately four to six weeks after the prescribing appointment
  23. 23.  >3.50 D in one or both meridian  Atkinson’s protocol based on the plus cylinder format at this was: ◦ sphere : prescribe 1.0 D less than the least meridian ◦ Cylinder: prescribe half of the astigmatism if >2.50 D
  24. 24.  < -5.0 D during the first year, reduce by 2.0 D. Under correct because emmetropisation occurs in myopia
  25. 25.  In study of Mohindra and Held (1981), refracted 400 children ≤5 yrs using near retinoscopy.  For children 0 to 4 weeks they found bell shaped distribution of refractive error ranging from -14 D to + 12D gradually narrowing to range of -3 to +4 D at age of 129 to 256 weeks.  This demonstrate the process of emmetropization is evident.
  26. 26.  In a study of 1,432 unselected children, Sorsby, Benjamin, and sheridan (1961) found the mean ocular refraction under cycloplegia, decreased from +2.33D at age 3yrs to 0.93 D at age of 14yrs in boys and from 2.96D at age 3yrs to 0.64D at age 15yrs for girls.
  27. 27.  Prevalence of myopia is found to be steadily increasing during school years.  In study Blum, Peters, and Bettman found that prevalence of myopia increased from about 2 % at age 6 to 15 % at age 15.
  28. 28.  the refraction with higher hyperopia and with emmetropia remains unchanged  the refraction with moderate hyperopia still shows a drift towards emmetropia up to nine or 10 years of age  In the school years, myopia should be corrected for function with full correction
  29. 29. When to consider prescribing What to prescribe? Outside the 95% range of refraction at any age according to any currently available data. This guideline could be applied to other refractive errors also. Prescribe so as to leave the uncorrected hyperopia somewhat above the mean for the age 3 to 6 months if outside the 95% range (Partial correction) In addition to the level of hyperopia determined by cycloplegic refraction, factors that would indicate correction are VA poorer than 6/100 plus non- cycloplegic (Mohindra) refraction that is
  30. 30. When to consider prescribing What to prescribe? Comments, rationale and references > 3.50 in one or more meridian at age of 1 year upwards Give partial prescription Atkinson’s protocol : prescribe 1D less than least hyperopic meridian Based on randomised clinical trials of Atkinson and colleagues and natural history study of Ingram and colleagues >2.50 at 4 years upward Still give partial correction of hyperopia Under correct by 1 to 1.50 D* Based on studies of visual function and functional vision and Mayer and co- worker’s > 1. 50 D in the school years without symptom Full or near full correction may be given at this age, as emmetropisation has Studies on visual function show that hyperopia ranging from > 1.00 to > 2.00 D may impact visual function .
  31. 31. When to consider prescribing What to prescribe Comments, rationale and references >2.50 D at 15 months of age upwards Decrease cylinder by 1.00 D or give 50% 15 month of period is the most critical period > 2. 00 D at 2 years or upward Give partial cylinder up to 3 to 4 years after which give full cylinder Based on finding of better VA in children whose astigmatism was corrected at this age and at 2 years approximately 5 to 10% have astigmatism > 2.00D > 1.50 D at 4 years upwards Give full cylinder and in case of previously uncorrected high astigmatism, reduced prescription may be given to allow child to adapt Based on studies of Cowen and Bobier 95th percentile for astigmatism was 1.25 in children in mean age of 4 years > 1.00 D oblique astigmatism from Correct ¾ to the age of 2 and then correct the full Oblique astigmatism is risk factor for amblyopia. Mayer and
  32. 32. When to consider prescribing What to prescribe Comments, rationale and references > 3.00D at 1 year upward Prescribe full anisometropia if amblyopia is already present, if there is no amblyopia reduced anisometropic correction could be considered > 3.00 D of anisometropia is less likely to be transient according to report of Abrahamsson > 1.00 D but < 3.00 D after 1 year of age Monitor for 4 to 6 months and if persist prescribe myopia and hyperopia according to age Based on report of transient myopia > 1.00 D of spherical hyperopic anisometropia, ≥2.00D of spherical myopic anisometropia, > 1.50 D of cylindrical anisometropia after Prescribe myopia and hyperopia according to age This level of amblyopia is found to be amblyogenic at this age
  33. 33. When to consider prescribing What to prescribe Comments, rationale, and refrences < -2 .00D myopia from one year Reduce by 0.50 or 1.00 D until school age MEPED study showed that < -1.2 to -1.7 is the lower end of 95% range in African American 4 years to early school years In correction of low amount of myopia improves vision , correct it. Give full correction for high amount of myopia Congdon and colleagues found the correction of <0.75 D improved VA. School age myopia Prescribe full correction. In cases of myopia with near esophoria , larger lag of accomodation or shorter habitual reading distance, PAL may be considered Guideline for bifocal correction based on the correction of Myopia Evaluation Trial Study for 6 to 11 years old
  34. 34.  Overcorrect by 2 to 3.00D because child’s world is near.  After 2 to 3 years, distance correction with bifocal is better option
  35. 35.  Prescription of spectacles thus must be done keeping factor like emmetropization and amblyogenic factor in mind.
  36. 36.  Susan J Leat . To prescribe or not to prescribe? Guidelines for spectacle prescribing in infants and children. Clin Exp Optom 2011; 94: 6: 514–527.  Jane Farbrother. Spectacle prescribing in childhood: a survey of hospital optometrist. Br. J. Ophthalmol.  SEAN P. DONAHUE. Prescribing Spectacles in Children: A Pediatric Ophthalmologist’s Approach. Optometry and Vision Science, Vol. 84, No. 2, February 2007  Ashwin Sainani. Special considerations for prescription of glasses in children. Journal of Clinical Ophthalmology and Research - Sep-Dec 2013 - Volume 1 - Issue 3