This document discusses a cancer theory involving mutational and wild-normal receptors. Mutational receptors are overexpressed via mutations and upregulation, continuously sending signals from outside that amplify the mutational receptors. However, suppressive normal DNA should not allow transcription or expression of wild-normal receptors, leading to their downregulation. Cancer cell identity depends on a combination of genes, transcription factors, and proteins from both mutational and wild-normal receptors.

1. “EFRUZHU
CANCER (CARCİNOGENESİS) THEORY”
MUTATİONAL RECEPTORS AND WİLD=NORMAL RECEPTORS.
DETAİLED COMMENTS 6. = VOİCED 6.
MUTATİONAL RECEPTORS OVER EXPRESS VİA MUTATİONS GROUP
İDENTİTY AND UP REGULATİON POSİTİON BECAUSE SENDİNG
SİGNALS CONTİNUOUS FROM OUTSİDE MAKE EFFECT THAT
PRODUCİNG –AMPLİFY MUTATİONAL RECEPTOR.BUT SUPPRESSİVE
NORMAL DNA SHOUL NOT TRANSCRİPTİON EXPRESSİON SO WİLD –
NORMAL RECEPTORS DOWN REGULATİON.CHİMERİC-HYBRİD:
GENES, TRANSCİPTİON FACTORS AND PROTEİNS DEPENDENCE
CANCER CELL BİNARY =DUAL İDENTİTY.