The document discusses a cancer theory related to histone proteins and chromatin remodeling. It suggests that normal DNA histones do not undergo sufficient modification and remodeling to allow for adequate gene transcription and expression due to low energy levels. This inhibits aerobic glycolysis and mitochondrial function, resulting in very low ATP production through anaerobic glycolysis alone. As a result, signaling, proliferation, and mutations are more likely to occur that increase histone modification and allow for gene expression.

1. “EFRUZHU
CANCER (CARCİNOGENESİS) THEORY”
HİSTONE PROTEİNS AND CHROMATİNE REMODELİNG.
DETAİLED COMMENTS 10. = VOİCED 10.
DNA SEQUENCE GENES HAVE POSSİBLE TRANSCRİPTİON EXPRESSİON
APPROXİMATELY 10 NANO METER SHOULD BE OPPENED.BUT
NORMAL DNA HİSTONES MODİFİCATİON DECREASES AND NOT
SUİTABLE TRANSCRİPTİON EXPRESSİON CAUSE OF İNADEQUATE
ENERGY DEGREE/LEVEL AND CHROMATİNE REMODELİNG
UNSUCCESSFUL.MİTHOCHONDRİON AND AEROBİC GLYCOLYSİS
İNHİBİTON OR VERY LOW LEVEL ENERGY GENERATİON,ANAEROBİC
GLYCOLYSİS DOMİNATİVE ATP PRODUCTİON CAPASİTY VERY LOW SO
STİMULATİNG,SİGNALİNG AND PROLİFERATİVE CONDİTİON WORKS
SİDE OF MUTATİONS GROUP İDENTİTY THAT SHOULD HİSTONE
MODİFİCATİON İNCREASİNG(SHOULD OPPENED) AND
TRANSCRİPTİON EXPRESSİON WOULD BECOME POSSİBLE.