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Estrategias Diagnósticas en 
Reumatología 
Dr. Daniel Xibillé Friedmann, MSc
Bases del razonamiento clínico 
• Conocimiento 
• Habilidad (velocidad y agilidad) 
• Establecimiento de mapas mentales 
– Experto 
– Novato 
• Pensamiento no analítico 
– Patrones
Paciente 
• “La rodilla me dolió mucho anoche. El dolor 
me despertó. Cuando me fuí a dormir estaba 
bien. Ahora está inflamada. Es el peor dolor 
que he tenido. He tenido problemas similares 
en la misma rodilla, una vez hace 9 meses, la 
otra hace dos años. No me ha molestado 
excepto en esas dos ocasiones.”
Novato 
• Masculino de 54 años con dolor en la rodilla. 
Inició anoche. No refiere trauma previo. A la 
exploración sus signos vitales son normales. La 
rodilla se encuentra inflamada, eritematosa y 
dolorosa a la palpación. Le duele mucho al 
explorar el rango de movimiento. Ha 
presentado este problema en dos ocasiones 
previas.
Experto 
• Masculino de 54 años con artritis aguda de la 
rodilla derecha que lo despertó. Niega 
antecedentes y se refiere asintomático al 
acostarse. Tiene el antecedente de dos 
episodios similares de dolor hace 9 meses y 
dos años con periodos libres de dolor entre 
ellos. Hoy se encuentra afebril. Su rodilla está 
inflamada, dolorosa a la palpación y 
eritematosa.
Diferencias 
• No hay estructura definida en la presentación 
novata 
• El experto generó una representación del 
problema e impresión diagnóstica 
• Guía subsecuente 
• Transformación a términos clínicos 
• Una sola frase “Monoartritis aguda recurrente 
en un hombre adulto”
Abordaje en dos pasos 
• Mapa mental no analítico 
• Basado en evidencia y experiencia 
• Dos mapas: 
– Mapas creados mediante el conocimiento de una 
enfermedad 
– Mapa generado al revisar a un paciente
Tipos de diagnóstico 
Diagnóstico 
Sindromático 
Diagnóstico 
Nosológico 
Diagnóstico 
Etiológico
3 
I- MAIN SYNDROMES 
The aim of our first step is to establish the main syndrome which is most representative of our patients’ 
clinical picture (pattern recognition). The main syndromes proposed below represent our diagnostic strategy, 
matured and revised over the years. Other experts may identify other operating patterns deemed useful for 
their clinical practice. 
Figure 1 - Main rheumatological syndromes5 
4 Reumatologia Prática. JAP da Silva. Diagnosteo, Publishers. Coimbra. Portugal. 2004. 
5 Published under permission. Diagnósteo, Publishers. Coimbra, Portugal.
Síndromes regionales 
• Dolor en una área específica 
– Periarticular 
– Articular 
– Neurogénico 
– Referido
6 
Table n°1 - Distinctive features of regional syndromes 
Periarticular 
Pain 
Articular pain Neurogenic 
pain 
Referred Pain 
Enquiry Selective painful 
movements 
All joint 
movements are 
painful 
Disaestesic. 
Aggravated by 
compression of 
nerve or 
mobilization of 
the spine 
Unrelated to 
movement. 
“Visceral” timing 
Pain on 
motion 
Active> passive. 
Selected motions 
Active ~ passive 
Several 
directions 
Normal. If root 
pain: Pain on 
motion of the 
affected spine 
segment 
Normal 
Range of 
Motion 
Active motion can 
be limited by 
pain. Passive 
motion: full 
Can be limited in 
active and 
passive motion 
Normal Normal 
Resisted 
mobilization 
Pain on specific 
manoeuvres 
No effect No effect No effect 
Local 
Palpation 
Pain upon 
affected structure 
Possible: 
Crepitus, 
swelling, effusion, 
heat. 
Pain along joint 
margin 
Normal Normal 
Neurological 
examination 
Normal Normal May be 
abnormal 
Normal
Eular On-line Course on Rheumatic Diseases – module n°1 
Prof. José da Silva, Prof. Karen Lisbeth Faarvang, Dr. Catia Duarte 
Figure 2 - Most common sites and origins of referred pain 
Table n°1 - Distinctive features of regional syndromes 
Periarticular 
Pain 
Articular pain Neurogenic 
pain 
Referred Pain 
Enquiry Selective painful 
movements 
All joint 
movements are 
painful 
Disaestesic. 
Aggravated by 
compression of 
nerve or 
mobilization of 
the spine 
Unrelated to 
movement. 
“Visceral” timing
Síndrome de dolor generalizado 
• Difuso 
• Fibromialgia 
– Dolor generalizado, migratorio asociado a ejercicio 
– Cefalea, ansiedad, trastornos del sueño 
– Diferentes patrones 
• Diagnóstico diferencial adecuado 
– AR 
– LES 
Sjögren 
– Polimialgia reumática 
– EA 
– Polimiositis 
– Hipotiroidismo 
– Hipoparatiroidismo
Lumbalgia y cervicalgia 
• Anatomicamente complejas 
• Estudios de imágen 
• Mec´nico vs. inflamatorio 
• Signos de alarma 
• Condiciones musculares, neurológicas, etc.
Eular On-line Course on Rheumatic Diseases – module n°1 
Prof. José da Silva, Prof. Karen Lisbeth Faarvang, Dr. Catia Duarte 
Table n°2 - Common causes of low back pain, suggestive manifestations and alarm signals. 
“Red Flags” 
Back pain with inflammatory rhythm 
Localized pain 
Nocturnal pain 
Visceral or constitutional symptoms 
Onset before age 30 or after 50 
Pain at movement in all directions 
History of neoplasm 
Risk or evidence of osteoporosis 
Neurological manifestations 
Sacroiliitis 
Spondylodiscitis 
Metastases 
Osteoporotic fracture 
Neurogenic pain 
Referred pain 
Interspinous ligamentitis 
“No Red flags” Acute mechanical low back pain 
Chronic mechanical low back pain 
Fibromyalgia 
Reasoning regarding neck pain follows a similar rationale. 
In most cases the pain has a mechanical rhythm. It is triggered by movement and relieved by rest. In adults, 
most of these conditions are caused by spondyloarthrosis. In many other cases, particularly in young people, 
there is no apparent cause for the pain, and it is thought to be the result of mild articular instability and 
irritation of the nerves and muscle bundles leading to painful reflex muscle contractions. Both situations 
should be treated conservatively, aiming to relieve the pain and restore function, without any specific 
aetiological intervention. In a few cases, the pain may be neurogenic, inflammatory, infectious, neoplastic or 
psychogenic in origin. The clues for such special conditions are similar to those described above. The 
possibility of referred pain, from the heart, lung apex and shoulder must be kept in mind. Acute 
lymphadenopathy, thyroiditis and meningitis represent important non-rheumatic causes of neck pain.
Síndrome articular 
• Dolor localizado, crepitación, aumento de 
volúmen, calor. 
• Mecánico vs. inflamatorio 
• Otros síntomas 
• Exacerbaciones/remisiones 
• Actitudes forzadas 
• Fluctuación
Diferencias entre inflamación y daño 
articular 
Inflamación Daño 
RAM Prolongada Corta 
Rigidez a la inmovilidad Prolongada Corta 
Calor + - 
Dolor a la posición forzada Si No 
Inflamación de tejido blando + - 
Sinovitis o “derrame” +++ +/- 
Crepitación - +++ 
Deformidad - +/- 
Inestabilidad - +/-
Síndrome de “Osteoporosis” 
• Factores de riesgo 
– Postmenopausia 
– Menopausia precoz 
– IMC bajo 
– Sedentarismo 
– Ingesta pobre de lacteos 
– Antecedentes familiares o personales de fractura 
– Malabsorción, hipogonadismo, hipertiroidismo, 
hiperparatiroidismo, uso de alcohol, esteroides, etc.
Síndrome óseo 
• Dolor profundo, difuso, continuo, sin relación 
con movimiento 
• Nocturno 
• Tumores, inflamación periosteo, enfermedad 
ósea 
• Metástasis (columna, cinturas) 
• EF tiende a ser normal
Síndrome sistémico 
• Puede acompañar a otros síndromes 
• Enfermedades de tejido conectivo 
• Vasculítis 
• Síntomas y signos constitucionales
Table n°3 - Main systemic manifestations associated with rheumatic diseases 
Associated diseases (in descending order of frequency) 
Constitutional manifestations 
Fever 
Weight loss 
Severe fatigue 
Systemic lupus erythematosus 
Systemic sclerosis 
Rheumatoid arthritis 
Mixed connective tissue disease 
Vasculitis 
Skin manifestations 
Photosensitivity 
Skin rash 
Scleroderma 
Purpura 
Livedo reticularis 
Ulcers 
Alopecia 
Telangiectasia 
Heliotrope 
Gottron’s papules 
Systemic lupus erythematosus 
Systemic sclerosis 
Dermatomyositis 
Mixed connective tissue disease 
Overlap syndromes 
Vasculitis 
Mucosal manifestations 
Oral and genital aphthae 
Dry eyes and mouth 
Red eye 
Balanitis 
Sjögren’s syndrome 
Systemic lupus erythematosus 
Rheumatoid arthritis 
Reactive arthritis 
Ankylosing spondylitis 
Behçet’s disease and other vasculitis 
Serositis 
Connective tissue diseases 
Rheumatoid arthritis 
Raynaud’s phenomenon Idiopathic Raynaud’s phenomenon 
Systemic sclerosis 
Systemic lupus erythematosus 
Arterial or venous thrombosis Vasculitis 
Antiphospholipid syndrome 
Recurrent abortion Antiphospholipid syndrome 
Dysphagia Systemic sclerosis 
Dyspnea Connective tissue diseases 
Lower limb edema, 
Connective tissue diseases 
hypertension 
Lymphadenopathy Connective tissue diseases 
Muscular weakness Myositis, 
Overlap syndromes 
Convulsions 
Psychosis 
Peripheral neuropathy 
Systemic lupus erythematosus 
Vasculitis
Síndromes Pediatricos
Patrones articulares
pattern of distribution, 2) how the condition began and how it developed over time; 3) the presence or 
absence of inflammatory low back pain; 4) accompanying extra-articular manifestations. 
Once you have this information, you may classify the arthritis according to a number of parameters, which will 
help you find and support a final diagnosis: 
14 
Number of joints affected 
Monoarthritis: one single joint involved 
Oligoarthritis: 2 to 4 joints involved 
Polyarthritis: 5 or more joints involved 
Acute versus Chronic 
Acute: onset in hours or days 
Chronic: onset over weeks or months 
Additive versus Migratory 
Additive: the affected joints are added progressively 
Migratory. The inflammatory process flits from one joint to another 
Persistent versus Recurrent 
Persistent: once it has set, the arthritis persists over the time 
Recurrent: episodes or crisis of arthritis separated by symptom-free intervals 
predominantly Proximal versus predominantly Distal 
Proximal: arthritis mainly involves large joints, i.e, proximal to the wrist or ankle, 
and the spine 
Distal: the arthritis mainly involves the small joints of the hands and feet, with or 
without the wrist and ankle 
Symmetrical versus Assymmetrical 
Symmetrical: affects approximately the same joint groups of each side of the 
body 
Asymmetrical: there is no relationship between the joints involved on either side 
of the body 
With or without inflammatory low back pain 
With or without systemic manifestations
Eular On-line Course on Rheumatic Diseases – module n°1 
Prof. José da Silva, Prof. Karen Lisbeth Faarvang, Dr. Catia Duarte 
Table n°5 - Most common causes of secondary osteoarthritis. 
Fracture involving the articular surface Preexisting arthritis 
Meniscectomy Axial deviations 
Articular instability Aseptic necrosis 
Intra-articular loose bodies Chondrocalcinosis 
Osteochondritis dissecans 
Particularly demanding occupations 
Patterns of inflammatory joint disease 
The Inflammatory articular syndrome is suggested by pain with “inflammatory rhythm” and confirmed by the 
demonstration of joint inflammation (diffuse elastic swelling around the joint with or without redness and heat). 
When evaluating a patient with arthritis, it is important to determine: 1) which joints are affected and their 
pattern of distribution, 2) how the condition began and how it developed over time; 3) the presence or 
absence of inflammatory low back pain; 4) accompanying extra-articular manifestations. 
Once you have this information, you may classify the arthritis according to a number of parameters, which will 
help you find and support a final diagnosis: 
Number of joints affected 
Monoarthritis: one single joint involved 
Oligoarthritis: 2 to 4 joints involved 
Polyarthritis: 5 or more joints involved 
Acute versus Chronic
Patrones 
• Monoartritis aguda 
– Gota 
– Artrítis séptica 
• Monoartritis crónica 
– Artritis séptica 
– OA/sx regional 
– Hidrartrosis 
– Osteonecrosis 
– Artropatía de Charcot 
– Tumores (sinovitis villonodular pigmentada)
Patrones 
• Oligo/poliartritis crónica asimétrica 
– OA 
– AR 
– Apso, ARe 
– Gota tofacea crónica 
• Oligoartritis proximal 
– EA seronegativas 
• Artritis en la cintura escapular y pélvica 
– PMR
Patrones 
• Oligo/poliartritis aguda + fiebre 
– Infecciones virales 
– Still 
– LES, Behcet, PM, AR 
• Dolor inflamatorio en columna vertebral 
• EA seronegativas 
• Poliartritis con manifestaciones sistémicas 
– LES
Atención óptima 
• Enfoque en problemas relevantes 
• Evaluar la validez de la información 
• Escuchar al paciente 
• Explorar adecuadamente 
• Resumir los datos 
• Uso sensato de laboratorio y gabinete

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M1 estrategia diagnóstica

  • 1. Estrategias Diagnósticas en Reumatología Dr. Daniel Xibillé Friedmann, MSc
  • 2. Bases del razonamiento clínico • Conocimiento • Habilidad (velocidad y agilidad) • Establecimiento de mapas mentales – Experto – Novato • Pensamiento no analítico – Patrones
  • 3. Paciente • “La rodilla me dolió mucho anoche. El dolor me despertó. Cuando me fuí a dormir estaba bien. Ahora está inflamada. Es el peor dolor que he tenido. He tenido problemas similares en la misma rodilla, una vez hace 9 meses, la otra hace dos años. No me ha molestado excepto en esas dos ocasiones.”
  • 4. Novato • Masculino de 54 años con dolor en la rodilla. Inició anoche. No refiere trauma previo. A la exploración sus signos vitales son normales. La rodilla se encuentra inflamada, eritematosa y dolorosa a la palpación. Le duele mucho al explorar el rango de movimiento. Ha presentado este problema en dos ocasiones previas.
  • 5. Experto • Masculino de 54 años con artritis aguda de la rodilla derecha que lo despertó. Niega antecedentes y se refiere asintomático al acostarse. Tiene el antecedente de dos episodios similares de dolor hace 9 meses y dos años con periodos libres de dolor entre ellos. Hoy se encuentra afebril. Su rodilla está inflamada, dolorosa a la palpación y eritematosa.
  • 6. Diferencias • No hay estructura definida en la presentación novata • El experto generó una representación del problema e impresión diagnóstica • Guía subsecuente • Transformación a términos clínicos • Una sola frase “Monoartritis aguda recurrente en un hombre adulto”
  • 7. Abordaje en dos pasos • Mapa mental no analítico • Basado en evidencia y experiencia • Dos mapas: – Mapas creados mediante el conocimiento de una enfermedad – Mapa generado al revisar a un paciente
  • 8. Tipos de diagnóstico Diagnóstico Sindromático Diagnóstico Nosológico Diagnóstico Etiológico
  • 9. 3 I- MAIN SYNDROMES The aim of our first step is to establish the main syndrome which is most representative of our patients’ clinical picture (pattern recognition). The main syndromes proposed below represent our diagnostic strategy, matured and revised over the years. Other experts may identify other operating patterns deemed useful for their clinical practice. Figure 1 - Main rheumatological syndromes5 4 Reumatologia Prática. JAP da Silva. Diagnosteo, Publishers. Coimbra. Portugal. 2004. 5 Published under permission. Diagnósteo, Publishers. Coimbra, Portugal.
  • 10. Síndromes regionales • Dolor en una área específica – Periarticular – Articular – Neurogénico – Referido
  • 11. 6 Table n°1 - Distinctive features of regional syndromes Periarticular Pain Articular pain Neurogenic pain Referred Pain Enquiry Selective painful movements All joint movements are painful Disaestesic. Aggravated by compression of nerve or mobilization of the spine Unrelated to movement. “Visceral” timing Pain on motion Active> passive. Selected motions Active ~ passive Several directions Normal. If root pain: Pain on motion of the affected spine segment Normal Range of Motion Active motion can be limited by pain. Passive motion: full Can be limited in active and passive motion Normal Normal Resisted mobilization Pain on specific manoeuvres No effect No effect No effect Local Palpation Pain upon affected structure Possible: Crepitus, swelling, effusion, heat. Pain along joint margin Normal Normal Neurological examination Normal Normal May be abnormal Normal
  • 12. Eular On-line Course on Rheumatic Diseases – module n°1 Prof. José da Silva, Prof. Karen Lisbeth Faarvang, Dr. Catia Duarte Figure 2 - Most common sites and origins of referred pain Table n°1 - Distinctive features of regional syndromes Periarticular Pain Articular pain Neurogenic pain Referred Pain Enquiry Selective painful movements All joint movements are painful Disaestesic. Aggravated by compression of nerve or mobilization of the spine Unrelated to movement. “Visceral” timing
  • 13. Síndrome de dolor generalizado • Difuso • Fibromialgia – Dolor generalizado, migratorio asociado a ejercicio – Cefalea, ansiedad, trastornos del sueño – Diferentes patrones • Diagnóstico diferencial adecuado – AR – LES Sjögren – Polimialgia reumática – EA – Polimiositis – Hipotiroidismo – Hipoparatiroidismo
  • 14. Lumbalgia y cervicalgia • Anatomicamente complejas • Estudios de imágen • Mec´nico vs. inflamatorio • Signos de alarma • Condiciones musculares, neurológicas, etc.
  • 15. Eular On-line Course on Rheumatic Diseases – module n°1 Prof. José da Silva, Prof. Karen Lisbeth Faarvang, Dr. Catia Duarte Table n°2 - Common causes of low back pain, suggestive manifestations and alarm signals. “Red Flags” Back pain with inflammatory rhythm Localized pain Nocturnal pain Visceral or constitutional symptoms Onset before age 30 or after 50 Pain at movement in all directions History of neoplasm Risk or evidence of osteoporosis Neurological manifestations Sacroiliitis Spondylodiscitis Metastases Osteoporotic fracture Neurogenic pain Referred pain Interspinous ligamentitis “No Red flags” Acute mechanical low back pain Chronic mechanical low back pain Fibromyalgia Reasoning regarding neck pain follows a similar rationale. In most cases the pain has a mechanical rhythm. It is triggered by movement and relieved by rest. In adults, most of these conditions are caused by spondyloarthrosis. In many other cases, particularly in young people, there is no apparent cause for the pain, and it is thought to be the result of mild articular instability and irritation of the nerves and muscle bundles leading to painful reflex muscle contractions. Both situations should be treated conservatively, aiming to relieve the pain and restore function, without any specific aetiological intervention. In a few cases, the pain may be neurogenic, inflammatory, infectious, neoplastic or psychogenic in origin. The clues for such special conditions are similar to those described above. The possibility of referred pain, from the heart, lung apex and shoulder must be kept in mind. Acute lymphadenopathy, thyroiditis and meningitis represent important non-rheumatic causes of neck pain.
  • 16. Síndrome articular • Dolor localizado, crepitación, aumento de volúmen, calor. • Mecánico vs. inflamatorio • Otros síntomas • Exacerbaciones/remisiones • Actitudes forzadas • Fluctuación
  • 17. Diferencias entre inflamación y daño articular Inflamación Daño RAM Prolongada Corta Rigidez a la inmovilidad Prolongada Corta Calor + - Dolor a la posición forzada Si No Inflamación de tejido blando + - Sinovitis o “derrame” +++ +/- Crepitación - +++ Deformidad - +/- Inestabilidad - +/-
  • 18. Síndrome de “Osteoporosis” • Factores de riesgo – Postmenopausia – Menopausia precoz – IMC bajo – Sedentarismo – Ingesta pobre de lacteos – Antecedentes familiares o personales de fractura – Malabsorción, hipogonadismo, hipertiroidismo, hiperparatiroidismo, uso de alcohol, esteroides, etc.
  • 19. Síndrome óseo • Dolor profundo, difuso, continuo, sin relación con movimiento • Nocturno • Tumores, inflamación periosteo, enfermedad ósea • Metástasis (columna, cinturas) • EF tiende a ser normal
  • 20. Síndrome sistémico • Puede acompañar a otros síndromes • Enfermedades de tejido conectivo • Vasculítis • Síntomas y signos constitucionales
  • 21. Table n°3 - Main systemic manifestations associated with rheumatic diseases Associated diseases (in descending order of frequency) Constitutional manifestations Fever Weight loss Severe fatigue Systemic lupus erythematosus Systemic sclerosis Rheumatoid arthritis Mixed connective tissue disease Vasculitis Skin manifestations Photosensitivity Skin rash Scleroderma Purpura Livedo reticularis Ulcers Alopecia Telangiectasia Heliotrope Gottron’s papules Systemic lupus erythematosus Systemic sclerosis Dermatomyositis Mixed connective tissue disease Overlap syndromes Vasculitis Mucosal manifestations Oral and genital aphthae Dry eyes and mouth Red eye Balanitis Sjögren’s syndrome Systemic lupus erythematosus Rheumatoid arthritis Reactive arthritis Ankylosing spondylitis Behçet’s disease and other vasculitis Serositis Connective tissue diseases Rheumatoid arthritis Raynaud’s phenomenon Idiopathic Raynaud’s phenomenon Systemic sclerosis Systemic lupus erythematosus Arterial or venous thrombosis Vasculitis Antiphospholipid syndrome Recurrent abortion Antiphospholipid syndrome Dysphagia Systemic sclerosis Dyspnea Connective tissue diseases Lower limb edema, Connective tissue diseases hypertension Lymphadenopathy Connective tissue diseases Muscular weakness Myositis, Overlap syndromes Convulsions Psychosis Peripheral neuropathy Systemic lupus erythematosus Vasculitis
  • 22.
  • 25. pattern of distribution, 2) how the condition began and how it developed over time; 3) the presence or absence of inflammatory low back pain; 4) accompanying extra-articular manifestations. Once you have this information, you may classify the arthritis according to a number of parameters, which will help you find and support a final diagnosis: 14 Number of joints affected Monoarthritis: one single joint involved Oligoarthritis: 2 to 4 joints involved Polyarthritis: 5 or more joints involved Acute versus Chronic Acute: onset in hours or days Chronic: onset over weeks or months Additive versus Migratory Additive: the affected joints are added progressively Migratory. The inflammatory process flits from one joint to another Persistent versus Recurrent Persistent: once it has set, the arthritis persists over the time Recurrent: episodes or crisis of arthritis separated by symptom-free intervals predominantly Proximal versus predominantly Distal Proximal: arthritis mainly involves large joints, i.e, proximal to the wrist or ankle, and the spine Distal: the arthritis mainly involves the small joints of the hands and feet, with or without the wrist and ankle Symmetrical versus Assymmetrical Symmetrical: affects approximately the same joint groups of each side of the body Asymmetrical: there is no relationship between the joints involved on either side of the body With or without inflammatory low back pain With or without systemic manifestations
  • 26. Eular On-line Course on Rheumatic Diseases – module n°1 Prof. José da Silva, Prof. Karen Lisbeth Faarvang, Dr. Catia Duarte Table n°5 - Most common causes of secondary osteoarthritis. Fracture involving the articular surface Preexisting arthritis Meniscectomy Axial deviations Articular instability Aseptic necrosis Intra-articular loose bodies Chondrocalcinosis Osteochondritis dissecans Particularly demanding occupations Patterns of inflammatory joint disease The Inflammatory articular syndrome is suggested by pain with “inflammatory rhythm” and confirmed by the demonstration of joint inflammation (diffuse elastic swelling around the joint with or without redness and heat). When evaluating a patient with arthritis, it is important to determine: 1) which joints are affected and their pattern of distribution, 2) how the condition began and how it developed over time; 3) the presence or absence of inflammatory low back pain; 4) accompanying extra-articular manifestations. Once you have this information, you may classify the arthritis according to a number of parameters, which will help you find and support a final diagnosis: Number of joints affected Monoarthritis: one single joint involved Oligoarthritis: 2 to 4 joints involved Polyarthritis: 5 or more joints involved Acute versus Chronic
  • 27. Patrones • Monoartritis aguda – Gota – Artrítis séptica • Monoartritis crónica – Artritis séptica – OA/sx regional – Hidrartrosis – Osteonecrosis – Artropatía de Charcot – Tumores (sinovitis villonodular pigmentada)
  • 28. Patrones • Oligo/poliartritis crónica asimétrica – OA – AR – Apso, ARe – Gota tofacea crónica • Oligoartritis proximal – EA seronegativas • Artritis en la cintura escapular y pélvica – PMR
  • 29. Patrones • Oligo/poliartritis aguda + fiebre – Infecciones virales – Still – LES, Behcet, PM, AR • Dolor inflamatorio en columna vertebral • EA seronegativas • Poliartritis con manifestaciones sistémicas – LES
  • 30. Atención óptima • Enfoque en problemas relevantes • Evaluar la validez de la información • Escuchar al paciente • Explorar adecuadamente • Resumir los datos • Uso sensato de laboratorio y gabinete