HEADACHE
Ms Tarika Sharma
Assistant Professor
MMCON, MMU, Ambala
Headache
Headache or cephalgia is one of the most common of
all human complaints.
Symptom rather than disease entity.
May indicate organic disease, a stress response,
vasodilatation (migraine), skeletal muscle tension (
tension headache), or a combination of factors.
Primary headache
Primary headache is one for which no
organic cause can be identified.
These types of headache include: migraine,
tension- type, and cluster headaches.
SECONDARY HEADACHE
Symptom associated with an organic cause,
such as brain tumor or an aneurysm.
The International Classification of Headache
Disorders (Part I)
The International Classification of Headache
Disorders (Part II)
The International Classification of Headache
Disorders (Part III)
Primary Headaches
Secondary Headaches
Painful cranial neuropathies, other facial
pains and other headaches
CAUSES OF HEADACHE
Brain abscess
Brain tumor
Encephalitis
Hypertension
Meningitis
Intracerebral haemorrhage
Sinusitits
Subarachnoid hemorrhage
Postconcussional syndrome
Migraine
Migraine is derived from the word ‘hemicrania’ or ‘half-
a-head’
Migraine is a symptom complex characterized by
periodic and recurrent attacks of severe headache
lasting from 4 -72 hours in adults.
Primarily a vascular disturbance that occurs more
commonly in women and has a high familial tendency.
Incidence highest in adults 20- 35 yrs of age.
Typical time of onset: puberty
Migraine
Episodic, lasting 4-72 h, associated with nausea and/or
vomiting, photophobia (increased sensitivity to light) and
phonophobia (increased sensitivity to sound )and interferes
with day-to-day functioning.
Headache has a throbbing or pulsatile quality and is often
unilateral (2/3rds of patients) although may become
generalised
MIGRAINE (Factors associated with an
attack)
Menstrual pattern
Stress (often as crisis is resolving)
Fasting or missing a meal
Certain foods containing tyramine, monosodium
glutamate, nitrites or milk products.eg chocolate,
alcohol, cheese
Extreme changes in weather
Use of OCPs
Migraine ( types)
Without aura (headache, nausea, vomiting,
photophobia, phonophobia)
With aura ( visual: flashing lights; speech
difficulty; sensory disturbances with paresis
of arms or legs; motor changes with
weakness + additional symptoms)
Pathophysiology ( migraine)
Abnormal metabolism of serotonin.
Rise in plasma serotonin, which dilates the cerebral vessels.
Dysfunction of the brain stem pathways that normally modulate
sensory input .
Cerebral signs and symptoms of migraine, Pulsating , throbbing
pain.
(Related to the cranial blood vessels, the innervation of the
vessels, and the reflex connections in the brain stem.)
CLINICAL MANIFESTATIONS (MIGRAINE
WITH AURA)
Migraine with aura …4 phases.
1. PRODROME (Occurs hours to days before a migraine headache;
depression, food cravings, feeling cold, anorexia, increased urination,
diarrhea or constipation )
2. AURA PHASE ( Lasts less than 1 hour; focal neurologic symptoms.
Visual disturbances ( light flashes and bright spots) are common and
may be hemianopic .
3. HEADACHE PHASE ( Throbbing headache ---unilateral in 60%,
intensifies over several hours; often associated with photophobia, nausea
and vomiting; 4-72 hours)
4. RECOVERY PHASE (TERMINATION AND POSTDROME)
Pain gradually subsides; muscle contraction in neck and scalp is
common, exhaustion , mood changes. May sleep for long hrs
Migraine (Management)
AVOIDING TRIGGERS
Dietary : caffeine, chocolate, cheese, nuts,
yoghurt
Stress, smoking, inadequate sleep , hunger
MEDICAL MANAGEMENT ( MIGRAINE)
ABORTIVE ( SYMPTOMATIC) APPROACH
PREVENTIVE APPROACH
1. TRIPTANS , serotonin receptor agonists cause
vasoconstriction, reduce inflammation, reduce pain
transmission.
SUMATRIPTAN, NARATRIPTAN, RIZATRIPTAN, ALMOTRIPTAN.
2. ERGOTAMINES..acts on smooth muscle…prolonged
constriction of cranial blood vessels.
3. Newer AEDs – gabapentin and topiramate under research
Preventive Drug therapy ( Migraine)
Beta-blocking agents, propranolol
and metoprolol.
Amitriptyline hydrochloride, Valproate, and
several serotonin antagonist pizotifen,
methysergide.
Calcium antagonists (verapamil HCl)
Tension Headache
Tight bands around head
Very common and hard to relieve
Affects 3% of population
More prevalent in women than men ( 1.5: 1)
Commonest age at onset: second decade
Causes are sources of state of tension.
Symptoms:
Band like squeezing , tight or pressure sensation
Distributed diffusely or concentrated in temples and occipital region
May be associated with depression, anxiety or stress
TENSION HEADACHE
PATHOPHYSIOLOGY ( TENSION HEADACHE)
Emotional or physical stress
contraction of muscles in neck and scalp
TENSION HEADACHE
TENSION TYPE HEADACHE
Steady, constant feeling of pressure ; usually
begins in forehead, temple or back of neck.
Band like ……..a weight on top of my head
Treatment ( tension headache)
Analgesics Not Very Helpful
Relaxation Therapy
Amitryptyline : 10-50 mg/dl ( Anti
Depressant) irrespective of depression
Cluster headache
Usual tendency to occur repetitively over a few
weeks
More common in men
Pain is severe, located in orbit, local redness
swelling
Dilation of orbital and nearby extracranial
arteries
CLINICAL MANIFESTATIONS
CLUSTER HEADACHE
Unilateral and come in clusters of 1-8 daily;
excruciating pain localized to eye and orbit and
radiating to facial and temporal regions.
Watering of eyes and nasal congestion
15 min – 3 hrs
crescendo- decrescendo pattern
CLUSTER HEADACHE
Management
1. 100% Oxygen by face mask for 15 minutes
2. Ergotamine tartarate, sumatriptan,
corticosteroids.
Characteristics
Tension type headache
Chronic and less severe
Most common type
• Cluster headaches
Severe form of vascular headache.
5 Times more frequently in men than in women.
Temporal arteritis
A condition in which the temporal arteries,
which supply blood to the head and brain,
become inflamed or damaged.
It is also known as cranial arteritis or
giant cell arteritis.
Temporal arteritis…
This condition leads to a throbbing
headache that’s usually in the temples
CRANIAL ARTERITIS
Cause of headache in older population ( >
70 yrs)
Inflammation of the cranial arteries is
characterized by a severe headache
localized in the region of the temporal
arteries.
Inflammation may be generalized or focal.
Causes
May be linked to the body’s autoimmune
response.
Excessive doses of antibiotics and certain
severe infections have been linked to
temporal arteritis.
Pathophysiology ( cranial arteritis)
Immune vasculitis
immune complexes deposited in blood
vessels walls
Vascular injury and inflammation
Cranial arteritis
CLINICAL MANIFESTATIONS
CRANIAL ARTERITIS
General manifestations – fatigue, wt loss, malaise and
fever.
Inflammation over the involved artery
sometimes tender, swollen or nodular temporal artery
is visible
Pain on chewing
Headache localized (temporal headache)
visual problems due to ischemia of involved structures
MEDICAL MANAGEMENT
CRANIAL ARTERTIS
1. Corticosteroids
2. Analgesics
ASSESSMENT AND DIAGNOSTIC
EVALUATION
Detailed history ( med surg illness, medication history, family
history , stress, occupational history-toxic substances, complete
description of headache)
Physical assessment of head and neck
Complete neurologic examination
CT, cerebral angiography, MRI : to detect underlying cause
Electromyography ; sustained contraction of neck , scalp or
facial muscles.
Laboratory tests: CBC, ESR, glucose, creatinine, Thyroid
hormone levels
NURSING MANAGEMENT
GOALS :
To enhance pain relief
To treat acute event of headache
To prevent recurrent episodes
NURSING MANAGEMENT
RELIEVING PAIN
1. Individualized treatment depending upon type. During attack ---
Provide comfort; quiet dark environment; head of the bed elevated to
30 degrees; Symptomatic treatment (antiemetics)
2. Analgesics
3. Antidepressants
4. Muscle relaxants
5. Local heat or massage
6. Diversion therapies
Teaching Patients Self-Care
Education about the type of headache, its mechanism
(if known), and appropriate changes in lifestyle to avoid
triggers.
Regular sleep, meals, exercise, and avoidance of dietary
triggers may be helpful in avoiding headaches.
The patient with tension headaches needs teaching and
reassurance that the headache is not due to a brain tumor.
This is a common unspoken fear.
Teaching Patients Self-Care…
Stress reduction techniques, such as biofeedback,
exercise programs, and meditation, are examples of
nonpharmacologic therapies that may prove helpful.
Patients and their families need to be reminded of
the importance of following the prescribed
treatment regimen for headache and keeping follow-
up appointments.
Thank you!

Headache

  • 1.
    HEADACHE Ms Tarika Sharma AssistantProfessor MMCON, MMU, Ambala
  • 2.
    Headache Headache or cephalgiais one of the most common of all human complaints. Symptom rather than disease entity. May indicate organic disease, a stress response, vasodilatation (migraine), skeletal muscle tension ( tension headache), or a combination of factors.
  • 3.
    Primary headache Primary headacheis one for which no organic cause can be identified. These types of headache include: migraine, tension- type, and cluster headaches.
  • 4.
    SECONDARY HEADACHE Symptom associatedwith an organic cause, such as brain tumor or an aneurysm.
  • 5.
    The International Classificationof Headache Disorders (Part I)
  • 6.
    The International Classificationof Headache Disorders (Part II)
  • 7.
    The International Classificationof Headache Disorders (Part III)
  • 8.
  • 9.
  • 10.
    Painful cranial neuropathies,other facial pains and other headaches
  • 11.
    CAUSES OF HEADACHE Brainabscess Brain tumor Encephalitis Hypertension Meningitis Intracerebral haemorrhage Sinusitits Subarachnoid hemorrhage Postconcussional syndrome
  • 13.
    Migraine Migraine is derivedfrom the word ‘hemicrania’ or ‘half- a-head’ Migraine is a symptom complex characterized by periodic and recurrent attacks of severe headache lasting from 4 -72 hours in adults. Primarily a vascular disturbance that occurs more commonly in women and has a high familial tendency. Incidence highest in adults 20- 35 yrs of age. Typical time of onset: puberty
  • 14.
    Migraine Episodic, lasting 4-72h, associated with nausea and/or vomiting, photophobia (increased sensitivity to light) and phonophobia (increased sensitivity to sound )and interferes with day-to-day functioning. Headache has a throbbing or pulsatile quality and is often unilateral (2/3rds of patients) although may become generalised
  • 15.
    MIGRAINE (Factors associatedwith an attack) Menstrual pattern Stress (often as crisis is resolving) Fasting or missing a meal Certain foods containing tyramine, monosodium glutamate, nitrites or milk products.eg chocolate, alcohol, cheese Extreme changes in weather Use of OCPs
  • 16.
    Migraine ( types) Withoutaura (headache, nausea, vomiting, photophobia, phonophobia) With aura ( visual: flashing lights; speech difficulty; sensory disturbances with paresis of arms or legs; motor changes with weakness + additional symptoms)
  • 17.
    Pathophysiology ( migraine) Abnormalmetabolism of serotonin. Rise in plasma serotonin, which dilates the cerebral vessels. Dysfunction of the brain stem pathways that normally modulate sensory input . Cerebral signs and symptoms of migraine, Pulsating , throbbing pain. (Related to the cranial blood vessels, the innervation of the vessels, and the reflex connections in the brain stem.)
  • 18.
    CLINICAL MANIFESTATIONS (MIGRAINE WITHAURA) Migraine with aura …4 phases. 1. PRODROME (Occurs hours to days before a migraine headache; depression, food cravings, feeling cold, anorexia, increased urination, diarrhea or constipation ) 2. AURA PHASE ( Lasts less than 1 hour; focal neurologic symptoms. Visual disturbances ( light flashes and bright spots) are common and may be hemianopic . 3. HEADACHE PHASE ( Throbbing headache ---unilateral in 60%, intensifies over several hours; often associated with photophobia, nausea and vomiting; 4-72 hours) 4. RECOVERY PHASE (TERMINATION AND POSTDROME) Pain gradually subsides; muscle contraction in neck and scalp is common, exhaustion , mood changes. May sleep for long hrs
  • 19.
    Migraine (Management) AVOIDING TRIGGERS Dietary: caffeine, chocolate, cheese, nuts, yoghurt Stress, smoking, inadequate sleep , hunger
  • 20.
    MEDICAL MANAGEMENT (MIGRAINE) ABORTIVE ( SYMPTOMATIC) APPROACH PREVENTIVE APPROACH 1. TRIPTANS , serotonin receptor agonists cause vasoconstriction, reduce inflammation, reduce pain transmission. SUMATRIPTAN, NARATRIPTAN, RIZATRIPTAN, ALMOTRIPTAN. 2. ERGOTAMINES..acts on smooth muscle…prolonged constriction of cranial blood vessels. 3. Newer AEDs – gabapentin and topiramate under research
  • 21.
    Preventive Drug therapy( Migraine) Beta-blocking agents, propranolol and metoprolol. Amitriptyline hydrochloride, Valproate, and several serotonin antagonist pizotifen, methysergide. Calcium antagonists (verapamil HCl)
  • 22.
    Tension Headache Tight bandsaround head Very common and hard to relieve Affects 3% of population More prevalent in women than men ( 1.5: 1) Commonest age at onset: second decade Causes are sources of state of tension. Symptoms: Band like squeezing , tight or pressure sensation Distributed diffusely or concentrated in temples and occipital region May be associated with depression, anxiety or stress
  • 23.
  • 24.
    PATHOPHYSIOLOGY ( TENSIONHEADACHE) Emotional or physical stress contraction of muscles in neck and scalp TENSION HEADACHE
  • 25.
    TENSION TYPE HEADACHE Steady,constant feeling of pressure ; usually begins in forehead, temple or back of neck. Band like ……..a weight on top of my head
  • 26.
    Treatment ( tensionheadache) Analgesics Not Very Helpful Relaxation Therapy Amitryptyline : 10-50 mg/dl ( Anti Depressant) irrespective of depression
  • 27.
    Cluster headache Usual tendencyto occur repetitively over a few weeks More common in men Pain is severe, located in orbit, local redness swelling Dilation of orbital and nearby extracranial arteries
  • 28.
    CLINICAL MANIFESTATIONS CLUSTER HEADACHE Unilateraland come in clusters of 1-8 daily; excruciating pain localized to eye and orbit and radiating to facial and temporal regions. Watering of eyes and nasal congestion 15 min – 3 hrs crescendo- decrescendo pattern
  • 29.
    CLUSTER HEADACHE Management 1. 100%Oxygen by face mask for 15 minutes 2. Ergotamine tartarate, sumatriptan, corticosteroids.
  • 30.
    Characteristics Tension type headache Chronicand less severe Most common type • Cluster headaches Severe form of vascular headache. 5 Times more frequently in men than in women.
  • 31.
    Temporal arteritis A conditionin which the temporal arteries, which supply blood to the head and brain, become inflamed or damaged. It is also known as cranial arteritis or giant cell arteritis.
  • 32.
    Temporal arteritis… This conditionleads to a throbbing headache that’s usually in the temples
  • 33.
    CRANIAL ARTERITIS Cause ofheadache in older population ( > 70 yrs) Inflammation of the cranial arteries is characterized by a severe headache localized in the region of the temporal arteries. Inflammation may be generalized or focal.
  • 34.
    Causes May be linkedto the body’s autoimmune response. Excessive doses of antibiotics and certain severe infections have been linked to temporal arteritis.
  • 35.
    Pathophysiology ( cranialarteritis) Immune vasculitis immune complexes deposited in blood vessels walls Vascular injury and inflammation Cranial arteritis
  • 36.
    CLINICAL MANIFESTATIONS CRANIAL ARTERITIS Generalmanifestations – fatigue, wt loss, malaise and fever. Inflammation over the involved artery sometimes tender, swollen or nodular temporal artery is visible Pain on chewing Headache localized (temporal headache) visual problems due to ischemia of involved structures
  • 37.
    MEDICAL MANAGEMENT CRANIAL ARTERTIS 1.Corticosteroids 2. Analgesics
  • 38.
    ASSESSMENT AND DIAGNOSTIC EVALUATION Detailedhistory ( med surg illness, medication history, family history , stress, occupational history-toxic substances, complete description of headache) Physical assessment of head and neck Complete neurologic examination CT, cerebral angiography, MRI : to detect underlying cause Electromyography ; sustained contraction of neck , scalp or facial muscles. Laboratory tests: CBC, ESR, glucose, creatinine, Thyroid hormone levels
  • 39.
    NURSING MANAGEMENT GOALS : Toenhance pain relief To treat acute event of headache To prevent recurrent episodes
  • 40.
    NURSING MANAGEMENT RELIEVING PAIN 1.Individualized treatment depending upon type. During attack --- Provide comfort; quiet dark environment; head of the bed elevated to 30 degrees; Symptomatic treatment (antiemetics) 2. Analgesics 3. Antidepressants 4. Muscle relaxants 5. Local heat or massage 6. Diversion therapies
  • 41.
    Teaching Patients Self-Care Educationabout the type of headache, its mechanism (if known), and appropriate changes in lifestyle to avoid triggers. Regular sleep, meals, exercise, and avoidance of dietary triggers may be helpful in avoiding headaches. The patient with tension headaches needs teaching and reassurance that the headache is not due to a brain tumor. This is a common unspoken fear.
  • 42.
    Teaching Patients Self-Care… Stressreduction techniques, such as biofeedback, exercise programs, and meditation, are examples of nonpharmacologic therapies that may prove helpful. Patients and their families need to be reminded of the importance of following the prescribed treatment regimen for headache and keeping follow- up appointments.
  • 43.

Editor's Notes

  • #18 serotonin, a vasoactive neurotransmitter found in platelets and cells of the brainThe