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ACUTE ABDOMEN Shynal Dutt - s180271
Mashal – s200604
Q1 - ACUTE ABDOMEN DEFINITION
• Defined as a sudden onset of severe abdominal pain
• Developing in less than 1 week’s duration
• Requiring admission to hospital for urgent attention and treatment, which has
not been previously investigated or treated’
Q2 - Be able to correlate abdominal
anatomy to the presentation of an
acute abdomen.
ABDOMINAL
BOUNDARIES
• Superior – diaphragm
• Inferior- pelvic inlet
• Anterior- anterior abdominal wall
• Posterior – lumbar vertebrae, upper part of bony
pelvis, psoas and quadratus lumborum muscles
Abdominal quadrants
ABDOMINAL
PAIN
TYPES OF ABDOMINAL PAIN
1. Visceral Pain- dull and deep-
seated pain usually localized
vaguely to the area occupied by the
viscus during development, and is
referred to the overlying skin of the
abdominal wall
1
2. Parietal Pain (Somatic)- sharp or
knife-like in nature, and is usually
well localized to the affected area.
2
3. Referred Pain – pain/ discomfort
at a site distant from the affected
organ, reflects same
embroyological nerve root.
3
Q5 - Be able to correlate the
pathophysiology of appendicitis to
its clinical presentation
• 2-20 cm in length
• <6mm in diameter
• <2mm wall thickness
• Supplied by the appendicular artery ->
ileocecal artery – SMA
• Innervation
• Sympathetic – Nerve fibres from
T10
• Parasympathetic – Vagus nerve
Aetiology
Appendicitis – Acute onset of an Inflamed appendix
Primary aetiology that has been proposed is appendiceal (luminal) obstruction
• Children/ young adults – Hyperplasia of lymphoid follicles or initiated by infection
• Adults – Fibrosis/ stricture, fecalith (hard faecal mass), obstructing neoplasm (benign or
malignant)
• Other causes – Parasites or foreign body
Pathophysiology
1. Luminal obstruction
2. Overgrowth of bacteria – Occurs within diseases appendix – Invade wall and propagate
further
3. Inflammation and swelling (Lumen becomes mucus filled and distended)
4. Increased luminal and intramural pressure
5. Ischemia due to thrombosis, occlusion of small vessels in appendiceal wall, and stasis of
lymphatic flow
6. Perforation
7. Localised abscess formation or diffuse peritonitis
Clinical features
• Highly variable pain
• Sequence of events  Preceding period of anorexia followed by central abdominal
pain then vomiting, and it concludes with pain in the RLQ (Right anterior Iliac Fossa)
• Initially - Gnawing pain in periumbilical region -> Obstruction and inflammation of the
appendix. Mediated through visceral pain [C] fibres as a mid-gut pain
• Post transmural - Serosa of appendix and parietal peritoneum become involved ->
Localised sharp pain which is mediated through somatic pain [A-delta] fibres in the
RIF
Clinical features continued
• Tachycardia
• Fever 37.5-38.5 degrees – Generally occurs later
• Furred tongue
• Pt will remain still as best as possible
• Coughing exacerbates pain
• Foetor oris +/- flushing
• Shallow breaths
Atypical presentations – Indigestion, flatulence, bowel irregularity, diarrhea, and generalised malaise
Hx and Physical exam
Non-specific – Progressive fever, anorexia, N/V, diarrhoea/ constipation
Abdominal pain – Migratory, dull colicky periumbilical pain or sharp RIF pain
Physical exam – Inspection, palpation, percussion, auscultation + special tests
Special tests –
McBurney’s sign
• Guarding and rebound tenderness
present at McBurney’s point – 1/3 the
distance from the anterior superior iliac
spine to the umbilicus on the right side
Rovsing sign
• Palpation of the Pt LIF will bring about
pain in the Pt RIF – Indirect tenderness –
Indicative of right-sided local peritoneal
irritation
Psoas sign
• Pt in left lateral position – Extension of
the hip will bring about pain in RIF –
Associated with retrocecal appendix
• Psoas borders peritoneal cavity. Stretching/
contracting the muscle causes friction against
the inflamed tissue
Obturator sign
• Associated with pelvic appendix – Pts
knee and hip are flexed and then internal
rotation of the right hip takes place. RLQ
pain elicited
Investigations
Laboratory
• Mild leukocytosis – Left shit – Indicates immature WBCs
present
• B-hCG – Rule out ectopic pregnancy
• Higher leukocyte counts seen with perforation
• Urinalysis – Rule out renal/ urological causes
Imaging
• US – 90-94%. Also assists in ruling out gynae causes
• CT scan – 94-100% accuracy. Appendix will appear thick
walled, enlarged (>6mm) with inflammatory changes.
Optimal investigation
• X-ray – Free air – Perforation or not
Alvarado score
10 point score system - Used for predicting
acute appendicitis
Mnemonic - MANTRELS
Score
• 5-6 = Compatible diagnosis with
appendicitis
• 7-8 = Probably acute appendicitis
• 9-10 = Very probable acute appenditis
diagnosis
If suspected, admission/ surgery required
Management
of
appendicitis
• Hydrate and correct electrolyte abnormalities (if
excessive vomiting)
• Appendectomy – Gold standard
• Some cases can be treated with antibiotics -
Metronidazole + cefuroxime
Laparoscopic surgery
• Wound infection is less likely
• Reduced pain and hospital stay
• Sooner return to normal activity
• Reduced costs outside hospital
• Intra-abdominal abscess is twice as likely
Open surgery
• Short duration of surgery and lower operation costs
Complications
• Perforation
• Appendix mass – Inflamed appendix becomes covered with omentum
• Pelvic abscess
• Sepsis
Q6 – Be able to describe the cardinal
symptoms of bowel obstruction
• Obstruction which inhibits movement of bowel contents
Epidemiology
• High likelihood in males (4:1)
• Children – Intussusception – Sliding of a part of the intestine into an adjacent part
• Adults – Hernia and adhesion
• Elderly – Adhesions and colorectal tumours
Aetiology
Location Causes
Intraluminal Gallstone ileus, ingested foreign body, faecal impaction
Mural Cancer, inflammatory strictures, intussusception,
lymphoma, Meckel’s diverticulum, diverticular strictures
Extramural Hernias, adhesions, peritoneal metastasis, volvulus
Cardinal symptoms of bowel
obstruction
Diagnosis of dynamic intestinal obstruction based on 4 principles (cardinal
features)
• Pain – Colicky or cramping
• Distention
• Vomiting – Early in proximal, late in distal
• Absolute constipation – Early in distal, late in proximal
Obstruction classification & Features
Small bowel obstruction
• High small bowel obstruction – Vomiting -> pain -> distension
• Low small bowel obstruction – Pain -> distension -> vomiting
Large bowel obstruction – Distension -> pain -> vomiting

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Acute-abdomen-and-Bowel-obstruction.pptx

  • 1. ACUTE ABDOMEN Shynal Dutt - s180271 Mashal – s200604
  • 2. Q1 - ACUTE ABDOMEN DEFINITION • Defined as a sudden onset of severe abdominal pain • Developing in less than 1 week’s duration • Requiring admission to hospital for urgent attention and treatment, which has not been previously investigated or treated’
  • 3. Q2 - Be able to correlate abdominal anatomy to the presentation of an acute abdomen.
  • 4. ABDOMINAL BOUNDARIES • Superior – diaphragm • Inferior- pelvic inlet • Anterior- anterior abdominal wall • Posterior – lumbar vertebrae, upper part of bony pelvis, psoas and quadratus lumborum muscles
  • 7. TYPES OF ABDOMINAL PAIN 1. Visceral Pain- dull and deep- seated pain usually localized vaguely to the area occupied by the viscus during development, and is referred to the overlying skin of the abdominal wall 1 2. Parietal Pain (Somatic)- sharp or knife-like in nature, and is usually well localized to the affected area. 2 3. Referred Pain – pain/ discomfort at a site distant from the affected organ, reflects same embroyological nerve root. 3
  • 8. Q5 - Be able to correlate the pathophysiology of appendicitis to its clinical presentation • 2-20 cm in length • <6mm in diameter • <2mm wall thickness • Supplied by the appendicular artery -> ileocecal artery – SMA • Innervation • Sympathetic – Nerve fibres from T10 • Parasympathetic – Vagus nerve
  • 9. Aetiology Appendicitis – Acute onset of an Inflamed appendix Primary aetiology that has been proposed is appendiceal (luminal) obstruction • Children/ young adults – Hyperplasia of lymphoid follicles or initiated by infection • Adults – Fibrosis/ stricture, fecalith (hard faecal mass), obstructing neoplasm (benign or malignant) • Other causes – Parasites or foreign body
  • 10. Pathophysiology 1. Luminal obstruction 2. Overgrowth of bacteria – Occurs within diseases appendix – Invade wall and propagate further 3. Inflammation and swelling (Lumen becomes mucus filled and distended) 4. Increased luminal and intramural pressure 5. Ischemia due to thrombosis, occlusion of small vessels in appendiceal wall, and stasis of lymphatic flow 6. Perforation 7. Localised abscess formation or diffuse peritonitis
  • 11. Clinical features • Highly variable pain • Sequence of events  Preceding period of anorexia followed by central abdominal pain then vomiting, and it concludes with pain in the RLQ (Right anterior Iliac Fossa) • Initially - Gnawing pain in periumbilical region -> Obstruction and inflammation of the appendix. Mediated through visceral pain [C] fibres as a mid-gut pain • Post transmural - Serosa of appendix and parietal peritoneum become involved -> Localised sharp pain which is mediated through somatic pain [A-delta] fibres in the RIF
  • 12. Clinical features continued • Tachycardia • Fever 37.5-38.5 degrees – Generally occurs later • Furred tongue • Pt will remain still as best as possible • Coughing exacerbates pain • Foetor oris +/- flushing • Shallow breaths Atypical presentations – Indigestion, flatulence, bowel irregularity, diarrhea, and generalised malaise
  • 13. Hx and Physical exam Non-specific – Progressive fever, anorexia, N/V, diarrhoea/ constipation Abdominal pain – Migratory, dull colicky periumbilical pain or sharp RIF pain Physical exam – Inspection, palpation, percussion, auscultation + special tests
  • 14. Special tests – McBurney’s sign • Guarding and rebound tenderness present at McBurney’s point – 1/3 the distance from the anterior superior iliac spine to the umbilicus on the right side
  • 15. Rovsing sign • Palpation of the Pt LIF will bring about pain in the Pt RIF – Indirect tenderness – Indicative of right-sided local peritoneal irritation
  • 16. Psoas sign • Pt in left lateral position – Extension of the hip will bring about pain in RIF – Associated with retrocecal appendix • Psoas borders peritoneal cavity. Stretching/ contracting the muscle causes friction against the inflamed tissue
  • 17. Obturator sign • Associated with pelvic appendix – Pts knee and hip are flexed and then internal rotation of the right hip takes place. RLQ pain elicited
  • 18. Investigations Laboratory • Mild leukocytosis – Left shit – Indicates immature WBCs present • B-hCG – Rule out ectopic pregnancy • Higher leukocyte counts seen with perforation • Urinalysis – Rule out renal/ urological causes Imaging • US – 90-94%. Also assists in ruling out gynae causes • CT scan – 94-100% accuracy. Appendix will appear thick walled, enlarged (>6mm) with inflammatory changes. Optimal investigation • X-ray – Free air – Perforation or not
  • 19. Alvarado score 10 point score system - Used for predicting acute appendicitis Mnemonic - MANTRELS Score • 5-6 = Compatible diagnosis with appendicitis • 7-8 = Probably acute appendicitis • 9-10 = Very probable acute appenditis diagnosis If suspected, admission/ surgery required
  • 20. Management of appendicitis • Hydrate and correct electrolyte abnormalities (if excessive vomiting) • Appendectomy – Gold standard • Some cases can be treated with antibiotics - Metronidazole + cefuroxime Laparoscopic surgery • Wound infection is less likely • Reduced pain and hospital stay • Sooner return to normal activity • Reduced costs outside hospital • Intra-abdominal abscess is twice as likely Open surgery • Short duration of surgery and lower operation costs
  • 21. Complications • Perforation • Appendix mass – Inflamed appendix becomes covered with omentum • Pelvic abscess • Sepsis
  • 22. Q6 – Be able to describe the cardinal symptoms of bowel obstruction • Obstruction which inhibits movement of bowel contents Epidemiology • High likelihood in males (4:1) • Children – Intussusception – Sliding of a part of the intestine into an adjacent part • Adults – Hernia and adhesion • Elderly – Adhesions and colorectal tumours
  • 23. Aetiology Location Causes Intraluminal Gallstone ileus, ingested foreign body, faecal impaction Mural Cancer, inflammatory strictures, intussusception, lymphoma, Meckel’s diverticulum, diverticular strictures Extramural Hernias, adhesions, peritoneal metastasis, volvulus
  • 24. Cardinal symptoms of bowel obstruction Diagnosis of dynamic intestinal obstruction based on 4 principles (cardinal features) • Pain – Colicky or cramping • Distention • Vomiting – Early in proximal, late in distal • Absolute constipation – Early in distal, late in proximal
  • 25. Obstruction classification & Features Small bowel obstruction • High small bowel obstruction – Vomiting -> pain -> distension • Low small bowel obstruction – Pain -> distension -> vomiting Large bowel obstruction – Distension -> pain -> vomiting

Editor's Notes

  1. - Migration of pain to the right iliac fossa - Anorexia [urinalysis to look for acetone as an indication of anorexia; add urine dipstick for ketonuria >2+] - Nausea/Vomiting - Tenderness in the right iliac fossa - Rebound pain [Can be replaced with other indirect signs such as the Rovsing sign; Dunphy sign; Markle test; or percussion tenderness] - Elevated temperature (fever) [> 37.3 C] - Leukocytosis - Shift of leukocytes to the left
  2. High small bowel obstruction – Vomiting occurs early. It is profuse in nature and causes rapid dehydration. Pain present with minimal distension and little evidence of dilated small bowel loops on radiography Low small bowel obstruction – Pain predominant with central distension. Vomiting is delayed. Multiple dilated small bowel loops seen on radiography Large bowel obstruction – Distension is early and pronounced. Pain less severe. Vomiting and dehydration are later features. Proximal colon is distended.