2. Venous Anatomy of Lower Limbs
Superficial venous system
Deep venous system
Perforator veins
3. Venous valves
The venous valves are abundant in the distal lower
extremity and number of valves decreases
proximally, with no valves in superior and inferior
vena cava
Delicate structures
Prevent reverse flow in the veins
Ensure that the blood is pumped from the superficial
to the deep system and back towards the heart when
the patient is walking
4. Perforator veins
Connect superficial to deep veins at various
levels.
Travel from superficial fascia through an
opening in the deep fascia before entering the
deep veins.
The direction of blood flow - from superficial to
deep veins.
Guarded by valves so that the flow is
unidirectional, i.e. Towards deep veins.
Reversal of flow occurs due to incompetence of
perforators which will lead to varicose veins
6. Varicose Veins
Permanently dilated , elongated veins with
Tortuous veins causing pathological ( Reverse flow)
circulation.
Risk factors
Female sex
Prolonged standing
Raised intra abdominal pressure
Increased progesterone
High heels
7. The etiology of varicose veins can be subdivided into
three categories: primary, secondary, and congenital (see
the image below).
8. Classification Of Varicose Veins
Anatomical
Long Saphenous
System
Short
Saphenous
System
Perforator
Incompetence
Size Of Varices
Thread
Veins
Reticular
Veins
1- 4mm
Varicosities
>4mm
CEAP Classification
Clinical
Etiological
Anatomical
Pathophysiological
9. Thread veins, also called spider
veins, are tiny prominent veins just
below the skin surface.
The best treatment for thread veins is
microsclerotherapy
15. Clinical Signs
• Saphenofemoral incompetence
Brodie-trendelenberg’s test I
• Perforator incompetence
Brodie-trendelenberg’s test II
• DVT
Perthe’s test / modified
perthe’s
• Perforator incompetence
Tourniquet’s test
• Valvular incompetence
Schwartz test
• Perforator site localisation
Fegan test
• Blow outs = perforators
Pratt’s test
17. Investigation In Varicose Veins
Localise the anatomical location of the disease
Nature of the lesion
Rule out DVT
18. Contd…
Venous Doppler
DUPLEX scan
Doppler combined with B mode Ultrasound
Functional and anatomical information
DVT well made out.
Uniphasic signal – normal
Biphasic signal – reversal flow
19. Contd…
Venography
Ascending venography
• Dorsal venous arch – canulated
• Tourniquet at malleoli
• Dye injected
• X-rays taken
• DVT/perforator status
Descending venography
• Ascending venogram not possible
• Contrast through femoral vein
• Valvular incompetence
20.
21. Conservative management
Elastic crepe bandage – stockings
30-40mm Hg
Elevation of limbs
Above the level of heart
Graded compression stockings
22. Contd..
Unna boot
Nonelastic compression
Zinc oxide, calamine, and glycerine
Dressing changed once in a week
Infection should not be there
Compression methods
Reduce ambulatory venous pressure
Trans capillary leakage
Improve cutaneous micro circulation
23. Medications
Calcium dobesilate
Improves lymph flow, reduce edema
Diosmin
Protects venous valves / anti inflammatory
Not proven much beneficial
24. Sclerotherapy
Complete sclerosis of the venous wall
Indications
Uncomplicated perforator incompetence
Smaller varices
Recurrent varices
Isolated varices
Aged/patients not fit for surgery.
25. Contd…
Sclerosants used are
Sodium tetradecyl sulphate
Sodium morrhuate
Ethanolamine oleate
Polidocanol
Mechanism of action
Aseptic inflammation
Perivenous fibrosis
Endothelial damage
Obliteration by intimal approximation
26. Technique
23 gauge needle
in to vein and
emptied
0.5 -1 ml
of
sclerosant
Immediate
compressio
n bandage
Proper
endothelial
apposition
May have
to be
repeated
after 2-4
weeks later
31. Contd…
Perforator incompetence
Subfascial ligation of perforators-- Linton’s method (The Linton
procedure involves making a long incision across the calf including the diseased tissue,
forming a skin/soft tissue/fascial flap, and ligating the perforator veins under direct
visualization.)
Percutaneous endovenous RFA of incompetent perforator veins
Stab avulsion method
subfascial endoscopic perforator surgery(SEPS)
The cause of primary varicose veins is valvular insufficiency of the superficial veins, most commonly at the SFValve, resulting in venous hypertension.
Secondary varicose veins are mainly caused by DVT that leads to chronic deep venous obstruction or valvular insufficiency. Long-term clinical sequelae from this have been called the postthrombotic syndrome. This category also includes catheter-associated DVT. Pregnancy-induced and progesterone-induced venous wall and valve weakness worsened by expanded circulating blood volume and enlarged uterus can compress the inferior vena cava and hinder venous return from the lower extremities. Trauma is another possible cause of secondary varicose veins.
The congenital category includes any venous malformations. Examples include Klippel-Trenaunay variants and avalvulia.