Toracotomia de Reanimacion, Dra Mayla Perdomo, Colombia
1. Toracotomía de Reanimación
Mayla Andrea Perdomo Amar
Magíster en Administración – MBA, Universidad EAFIT
Especialista en Medicina de Urgencias, Universidad CES
Docente Universidad de Antioquia
2. Maras, Moray.Perú
“Yo creo en el inconmensurable poder del amor;
el amor verdadero puede soportar cualquier
circunstancia y atravesar cualquier distancia”
3. EPIDEMIOLOGIA
• Del trauma, 25-50% Trauma torácico
penetrante y cerrado.
• Trauma tórax ocasiona 50% mortalidad en
trauma civil.
• Mayoría pacientes requiere manejo no qx, con
o sin sonda a tórax.
• 10 -15% requieren toracotomía de
reanimación
Hunt PA. Injury, Int J Care Injured. 2006;37:1-19
4.
5. Qué es?
• Procedimiento realizado a un paciente en extremis,
inmediatamente en el lugar de la lesión o en la sala de
emergencias o en la sala de cirugía, como parte integral
de la resucitación inicial.
• Creada 1960, Beall y colegas: pacientes moribundos con
trauma tórax penetrante.
• Términos:
- Toracotomía en el departamento de emergencias
- Toracotomía emergente
- Toracotomía temprana - Toracotomía de reanimación
Edens JW. Jam Coll Surg. 2009;209:188-197
Hunt PA. Injury, Int J. Care Injured. 2006;37: 1-19
6. Objetivos
• Evacuación de taponamiento cardíaco
• Aliviar neumotórax a tensión
• Control directo sangrado intratorácico
• Control de un embolismo aéreo masivo o de una
fístula broncopleural masiva: clampear hilio
pulmonar
• Realizar masaje cardíaco directo
• Clampear la aorta descendente: controlar la
hemorragia subdiafragmática y redistribuir el flujo
sanguíneo: cerebral y coronario.
Edens JW. Jam Coll Surg. 2009;209:188-197
Hunt PA. Injury, Int J. Care Injured. 2006;37: 1-19
J. Wayne M. Surg Clin N Am. 2007;87:95-118
7. Tomar la decisión: 3 Factores a
considerar
• Mecanismo de lesión
• Localización de la lesión
• Signos de vida (estado fisiológico)
Rhee PM. J Am Coll Surg. 2000;190:288-298
Hunt PA. Injury, Int J. Care Injured. 2006;37: 1-19
J. Wayne M. Surg Clin N Am. 2007;87:95-118
8. MECANISMO DE LESIÓN
• No es un factor aislado de no utilidad, excepto en
trauma de cráneo devastador.
• Sobrevida general: 7.4% - 7.8%
- Trauma cerrado: 1.4% – 1.6%
- Trauma penetrante: 8.8% - 11.2%:
HACP 16.8%
HAF 4.3%
3 Factores a considerar
Moore EE. The Journal of Trauma. 2011;70(2):334-339
Powell DW. J Am Coll Surg. 2004;2:211-215
Rhee PM. J Am Coll Surg. 2000;190:288-298
9. LOCALIZACIÓN DE LA LESIÓN
• Sobrevida:
• Mayor en lesiones torácicas, 10.7%
• Lesiones abdominales 4.5%
• Lesiones múltiples 0.7%
• Si la lesión es cardíaca, es mas Alta: 19.4% - 31.1%
• HACP ventrículo 30% sobrevida
• HAF ventrículo 16% sobrevida
3 Factores a considerar
Moore EE. The Journal of Trauma. 2011;70(2):334-339
Powell DW. J Am Coll Surg. 2004;2:211-215
Rhee PM. J Am Coll Surg. 2000;190:288-298
Mayor sobrevida:
Lesiones cardíacas por
Arma cortopunzante,
con taponamiento
10. Estado fisiológico
Hunt PA. Int. J. Care Injured 2006;37:1-19
Lorenz HP. J Trauma. 1992;32:780-8
Estado fisiológico es el mayor predictor de sobrevida
Menor o nula
sobrevida
Mejor sobrevida
11. ESTADO FISIOLÓGICO, SIGNOS DE VIDA
• Sobrevida:
• Signos presentes a la llegada hospital, 11.5%
• Signos ausentes a la llegada hospital, 2.6%
• Signos presentes durante transporte, 8.9%
• Ausentes en el campo, 1.2%
• NO hay claro factor predictor de muerte
3 Factores a considerar
Moore EE. The Journal of Trauma. 2011;70(2):334-339
Rhee PM. J Am Coll Surg. 2000;190:288-298
12. Hunt PA. Int. J. Care Injured 2006;37:1-19
PACIENTE EN EXTREMIS CON TRAUMA DE TÓRAX
PREDICTORES DE
POBRE
PRONÓSTICO
• Trauma cerrado
• RCP prehospitalario
> 5 min
• TEC o múltiples
traumas asociados
INDICACIONES
PARA
TORACOTOMIA DE
EMERGENCIA
Trauma penetrante +
SV en la escena
Signos de Vida
INDICACIONES
PARA
TORACOTOMIA DE
EMERGENCIA
Hipotensión severa +
1 de las siguientes:
- Hemorragia
intratorácica
- Taponamiento
- Embolismo aéreo
- Hemorragia
extratorácica severa
SIGNOS DE VIDA
Actividad eléctrica en EKG,
con o sin pulso
Reflejos corneano, pupilar
o nauseoso
GCS > 3
NO SI
13. INDICACIONES
• Paro cardiorespiratorio por trauma penetrante
torácico aislado con evidencia de signos de vida
antes de la llegada a urgencias.
• Hipotensión persistente por trauma debido a
hemorragia intratorácica sin respuesta a reanimación
con líquidos.
• Hipotensión severa persistente con evidencia de
embolismo sistémico aéreo o taponamiento
cardíaco.
Rhee PM. J Am Coll Surg. 2000;190:288-298
Hunt PA. Injury, Int J. Care Injured. 2006;37: 1-19
J. Wayne M. Surg Clin N Am. 2007;87:95-118
14. INDICACIONES
• Masaje prehospitalario por menos de 5 min sin
intubación
• Intubación extrahospitalaria exitosa
RELATIVAS
• Presencia de signos de vida a la llegada a
emergencias independiente del mecanismo
• Trauma penetrante abdominal con algún signo de
vida en el campo
• Trauma cerrado con paro presenciado en urgencias
Rhee PM. J Am Coll Surg. 2000;190:288-298
Hunt PA. Injury, Int J. Care Injured. 2006;37: 1-19
J. Wayne M. Surg Clin N Am. 2007;87:95-118
15. • Retrospectivo, 50 pctes toracotomía por heridas
abdominales, enero 2000- diciembre 2006
• Signos de vida: 84% en la escena y 78% en urgencias
• 8 pacientes (16%) sobrevivieron neurológicamente
intactos = 98% HPAF abdominales (75% tx vascular
mayor, 25% heridas de hígado)
Seamon MJ. J Trauma 2008;64:1-8
La toracotomía de emergencia prelaparotomía brinda
beneficios en la supervivencia de pacientes in extremis con
heridas penetrantes abdominales asociadas a hemorragia
16. CONTRAINDICACIONES
• Ausencia de signos de vida o paciente en asistolia al
ingreso, asociado a trauma tórax cerrado.
• Ausencia de signos de vida en la escena y al ingreso,
con RCP mayor a 5minutos.
• Trauma multisistémico asociado a TEC severo o
trauma torácico.
• RCP más de 15 min en trauma penetrante o cerrado
mayor de 5min
• Trauma cerrado o penetrante sin signos vitales en el
sitio del evento
Rhee PM. J Am Coll Surg. 2000;190:288-298
Hunt PA. Injury, Int J. Care Injured. 2006;37: 1-19
J. Wayne M. Surg Clin N Am. 2007;87:95-118
17. • Estudio multicéntrico prospectivo.
• Sobrevida 56 pacientes al alta.
• Mayor lesiones torácicas, 77%, abdomen 9%,
extremidades 7%, cuello 4%, cráneo 4%
Moore EE. The Journal of Trauma. 2011;70(2):334-339
Conclusión: Toracotomía de reanimación en ED, no es útil en:
-RCP prehospitalaria mayor a 10 minutos, en trauma cerrado sin respuesta
-RCP prehospitalaria mayor a 15 minutos, en trauma penetrante sin respuesta
-Asistolia en ausencia de taponamiento cardíaco
19. Conclusiones:
-Sobrevida en trauma cerrado es muy baja.
-Secuelas neurológicas en los sobrevivientes son severas
•Algunos autores recomiendan que la toracotomía debe estar
contraindicada en pacientes con trauma cerrado sin signos de
vida en la escena o al ingreso a urgencias.
•Hacen falta estudios validar esta conclusión.
Khorzandi M. Interactive CardioVascular and Thoracic Surgery 16 (2013) 509–516
20. Cursos para:
Enseñar indicaciones, contraindicaciones
Anatomía, apertura del tórax, masaje cardíaco directo,
clamp de la aorta para controlar hemorragia,
corregir taponamiento cardíaco
Konig T. Emergency Medicine 2013, 21(Suppl 1):A2
21.
22. Es útil realizar Masaje cardíaco
externo en trauma?
• La recomendación de masaje cardíaco externo en
trauma con shock profundo, ha sido realizada sin
documentación de los efectos o beneficios
hemodinámicos.
• Sirve para “comprar tiempo” mientras se corrigen
los problemas causantes.
• No aumenta la presión arterial diastólica en
pacientes con presión ventricular izq de fin de
diástole disminuída, por lo cual no perfunde.
Semin Thorac Cardiovasc Surg 2008;20:13-18
Luna GK. The Journal of Trauma. 1989;29(10):1430-1433
23. Presión arterial diastólica en trauma con
RCP, masaje externo
Luna GK. The Journal of Trauma. 1989;29(10):1430-1433
Bajo o nulo incremento, el aumento es al tratar la causa
24. Efectos del masaje cardíaco
externo
• Presión perfusión cerebral y coronaria: 10% de la
basal.
• Mantenimiento mínimo de la perfusión de órganos
vitales, por lo cual se produce una relación
tiempo/pronóstico:
- Baja sobrevida luego de 15 minutos
- Nula sobrevida posterior a 30 minutos
Luna GK. The Journal of Trauma. 1989;29(10):1430-1433
25. Masaje cardíaco directo
• Ciclo = presión de perfusión
sistémica cercana a la
normal
• 40-100 compresiones/min,
dependiendo de la
frecuencia:
- Volumen: 2500-3300
- Flujo miocárdico: 150-250
- Flujo cerebral: 600-
800ml/min
Masaje cardíaco externo
• Compresiones = menor
presión de perfusión
sistémica
• Aporte del masaje
independiente de la
frecuencia:
- Volumen: 1200
- Flujo miocárdico: 70
- Flujo cerebral: 450 ml/min
Luna GK. The Journal of Trauma. 1989;29(10):1430-1433
26. Efectos del masaje cardíaco
externo
• Taponamiento cardíaco: hay un incremento en presión
intrapericárdica, esta es aumentada aún más por masaje
externo y ventilación positiva, lleva a:
- Mayor compromiso del llenado ventricular.
- Disminución de la presión diastólica
- Reducción perfusión coronaria
• Hipovolemia: corazón vacío.
• Lesiones comunes: ruptura cardíaca, aórtica,
neumotórax, lesiones vasculares exanguinantes.
Luna GK. The Journal of Trauma. 1989;29(10):1430-1433
27. Masaje cardíaco externo en
trauma, qué podemos concluir?
• Masaje cardíaco externo de rutina en pacientes con
trauma DEBE ser reevaluado.
• NO debe retardar la corrección de la causa del
shock en trauma: inadecuado volúmen intravascular
y ventricular.
• Los efectos hemodinámicos y los beneficios del
masaje cardíaco externo en pacientes médicos no
pueden extrapolarse al paciente con trauma.
Vanden Hoek TL. Circulation. 2010; 122;S829-S861
Semin Thorac Cardiovasc Surg 2008;20:13-18
Luna GK. The Journal of Trauma. 1989;29(10):1430-1433
28. Masaje cardíaco externo en
trauma, qué podemos concluir?
• Masaje cardíaco externo es útil en trauma por:
electrocución, lesiones por rayo, ahogamiento, en las
cuales existe alteración de la conducción cardíaca,
FV, paro respiratorio.
• Retardo en el transporte aumenta la mortalidad y
morbilidad.
Semin Thorac Cardiovasc Surg 2008;20:13-18
Louis RM. Resuscitation.1987;15:9-11
Mattox KL. The Journal of Trauma.1982;22(11):934-936
29. Toracotomía de reanimación
CONCLUSIONES
• Evaluar 3 factores: mecanismo, localización lesión, estado
fisiológico
• Lesiones cardíacas tienen mejor pronóstico, así sean
penetrantes por HACP, HAF, cerradas.
• Mayor sobrevida: asociada a taponamiento cardíaco
• Estudios: estado neurológico normal 92.4%
• Beneficio es mayor 2.4 veces que el costo.
• Ausencia signos de vida en el campo es mal pronóstico, pero
no predictor claro y 100% de muerte.
Edens JW. Jam Coll Surg. 2009;209:188-197
Hunt PA. Injury, Int J. Care Injured. 2006;37: 1-19
There are numerous different terms for emergency
thoracotomy. These depend on the circumstances in
which the procedure is performed, the status of the
patient and the location of the procedure itself.
Examples include emergency department thoracotomy,
emergent thoracotomy, early thoracotomy,
resuscitative thoracotomy, etc. This can make interpretation
of the available data and comparison of
studies difficult, due to the use of the terms interchangeably.
In general, emergency thoracotomy can
be undertaken at any stage of the resuscitative
process, and so can be defined in terms of the
urgency of the procedure in relation to the patients’
physiological status.
Thereby, emergency thoracotomy may be defined
as that occurring either immediately at the site of
injury, in the emergency department, or in the
operating room, as an integral part of the initial
resuscitation. An urgent thoracotomy is one that
takes place under more controlled circumstances
and in the context of appropriate physiological
stability and hitherto successful resuscitation.
Lastly, an elective (or formal) thoracotomy one that
is performed during the course of elective surgical
access to the thorax, such as for coronary artery
bypass surgery.
The goals of EDT
are to release pericardial tamponade or tension pneumothorax,
to directly control and repair intrathoracic hemorrhage,
to allow open cardiac massage, and to cross clamp
the thoracic aorta, restoring and maintaining perfusion to
the heart and brain and preventing additional blood loss
from distal sites of hemorrhage.
Systemic
air embolism will occur if air enters pulmonary
veins as a result of low pulmonary venous
pressure, increased airways pressure, or both. The
consequences may be catastrophic if pulmonary
venous gas embolises to the coronary vessels, heart
chambers or cerebral arteries. The incidence of
systemic air embolism has been estimated to be
4—14%, with two thirds resulting from penetrating
injury and one third from blunt trauma.80,8
Despite being a subtle clinical entity, systemic air
embolus should be considered in any major thoracic
injury resulting from either penetrating or blunt
trauma. It is often diagnosed only when sudden
circulatory collapse occurs immediately after
tracheal intubation and the initiation of positive
pressure ventilation. This collapse is typically unresponsive
to conventional resuscitation. The unexplained
development of a neurological deficit, or
seizures, in the absence of a head injury implies
cerebral air embolism unless proven otherwise.81
The current recommendation for treatment of
systemic air embolus associated, with unilateral
lung injury, is immediate thoracotomy, in the emergency
department if necessary, in order to clamp
the hilum of the injured lung so as to arrest the
passage of air into the systemic circulation.88 Bubbles
may be noted in the vasculature on thoracotomy.
Selective ventilation of the uninjured lung may
be a life-saving alternative procedure in unilateral
Injuries
MASAJE CARDIACO DIRECTO
Ciclo = presión de perfusión sistémica cercana a la normal
40-100 compresiones/min, dependiendo de la frecuencia:
Volumen: 2500-3300
Flujo miocárdico: 150-250
Flujo cerebral: 600-800ml/min
MASAJE CARDIACO EXTERNO
Compresiones = menor presión de perfusión sistémica, sólo en diástole
Aporte del masaje independiente de la frecuencia:
Volumen: 1200
Flujo miocárdico: 70
Flujo cerebral: 450 ml/min
analysis
of 251 cases of cardiac injury following emergency
thoracotomy, Henderson et al. showed that
survival rates diminished significantly with higher
physiological indices: the overall survival rate in this
study was 18.7%. The findings suggested that emergency
thoracotomy is most helpful for patients with
limited penetrating cardiac injuries who have significant
physiological impairment secondary to pericardial
tamponade.40
Significant differences
Lewis and Knottenbelt, in a series of 45 cases,
suggested that the primary indication for resuscitative
emergency thoracotomy was an observable
pericardial tamponade, and that results were shown
to be good for this single indication. However, they
noted that emergency thoracotomy was justifiable
for all patients with cardiac arrest, or persisting
severe hypotension, following penetrating thoracic
trauma, as not all cases of tamponade are clinically
obvious on admission. They also concluded that the
case for emergency thoracotomy in blunt trauma
remained debatable.52 In a detailed literature
review, the American College of Surgeons Committee
on Trauma recommended that emergency thoracotomy
be performed rarely in blunt thoracic
trauma with cardiopulmonary arrest, due to low
survival rates and poor neurological outcomes.1
Grove et al. repeated these conclusions, reporting
no survivors at all from a total of 19 cases, over a 2-
year period, of blunt multisystem trauma, including
thoracic injury requiring emergency thoracotomy
INDICACIONES
Cardiorespiratory arrest following isolated
penetrating thoracic trauma, with evidence
of signs of life before arrival in
emergency department
Post traumatic persistent
hypotension due to
intrathoracic haemorrhage,
unresponsive to fluid resuscitation
Persistent severe hypotension,
with evidence of
systemic air embolism or
pericardial tamponade
La toracotomía en este escenario trae doble beneficio: dar masaje cardíaco directo y clampar la aorta
Los pacientes no presentaron complicaciones de la oclusión de la aorta: sobrecarga del VI, lesiones esofágicas, avulsión de arterias intercostales o paraplejía. Aunque no se midió flujo coronario o cerebral los resultados medidos en términos de estado neurológico son excelentes
LIMITACIONES DEL ESTUDIO
Experiencia de un solo centro con gran historia en el manejo de trauma penetrante
Estudio retrospectivo de lesiones poco comunes en pocos pacientes (aunque este es uno de los reportes con mas pacientes en shock exanguinante abdominal)
La toracotomía de emergencia en trauma penetrante de abdomen no es inútil en la resucitación de pacientes exangües como se mencionaba en varias publicaciones anteriores
Centros de trauma americanos
best evidence topic in cardiothoracic surgery was written according to a structured protocol. The question addressed was whether
there is any role for resuscitative emergency department thoracotomy in severe blunt trauma. Emergency thoracotomy is an accepted
intervention for patients with penetrating cardiothoracic trauma. However, its role in blunt trauma has been challenged and has been a
subject of considerable debate. Altogether, 186 relevant papers were identified, of which 14 represented the best evidence to answer
the question. The author, journal, date, country of publication and relevant outcomes are tabulated. The 14 studies comprised
2 systematic reviews and 12 retrospective studies. The systematic review performed by the Trauma Committee of the American College
of Surgeons included 42 studies and a cumulative total of 2193 blunt trauma patients who underwent an emergency department
thoracotomy, reporting a survival rate of 1.6%. According to this review, 15% of the survivors suffered from neurological sequelae, but
survivors from both penetrating and blunt trauma were included. A systematic review comprising 24 studies reported a survival rate of
1.4% among 1047 blunt trauma patients. Of the retrospective studies, 11 report poor survival rates, ranging from 0 to 6%. Only one
study reports a higher survival rate (12.2%). Five of the studies reported on the neurological outcome of survivors. The majority of the
studies suffered from limitations due to the small number of included cases. The reported survival after an emergency department
thoracotomy for blunt trauma is very low in the vast majority of available studies. Furthermore, the neurological sequelae in the few
survivors are frequent and severe. Interestingly, some author groups recommend that emergency department thoracotomy should be
contraindicated in cases of blunt trauma with no signs of life at the scene of trauma or on arrival at the emergency department. Larger,
well-designed series will be required to reach a consensus on valid prognostic factors and specific subgroups of blunt trauma patients
with substantial chances of survival.
The reported survival after an emergency department
thoracotomy for blunt trauma is very low in the vast majority of available studies. Furthermore, the neurological sequelae in the few
survivors are frequent and severe. Interestingly, some author groups recommend that emergency department thoracotomy should be
contraindicated in cases of blunt trauma with no signs of life at the scene of trauma or on arrival at the emergency department. Larger,
well-designed series will be required to reach a consensus on valid prognostic factors and specific subgroups of blunt trauma patients
with substantial chances of survival.
These insights and courses are aimed at empowering
clinicians to identify and apply a beneficial intervention
to those that need it, when they need it. Ludwig Rehn’s
pertinent conclusion following the first successful RT
over a century ago remains: “many lives can be saved
that were previously counted as lost [2].”
insight into the
anatomy, advice about optimal team preparation and a
simulated demonstration of a pre hospital case ‘moulage’
on a manikin. Students were shown and allowed to
practice myocardial suture repair, discuss cases and gain
insight into the post operative care of patients after successful
thoracotomy.
Es inefectivo en paro secundario a taponamiento cardíaco, neumotórax a tensión y hemorragia
To determine the hemodynamic effects of external massage in profound
shock, hypotension was induced in baboons. Pressures obtained with external
massage were compared to spontaneous intra-arterial pressures before
compression. Although external massage increased systolic pressures in both
tamponade and hypovolemia, diastolic pressures were consistently decreased.
We conclude that CCCPR does not augment arterial pressure in the clinical
situations associated with decreased LVEDV and is unlikely to provide organ
perfusion for trauma victims
massage
during cardiac tamponade.
Chemically Induced Cardiac Arrest
CPR CPR
Initiated Terminated
Post-Arrest
~
Systolic Diastolic
108(90-125) 12(8-15)
DBP ----------f,,,,,,,,,,
O,-L..-------:;t,...-----!~-r----=='----r-=.-;
Overdose
20
4
80
Pre Arrest
Spontaneous
Systolic Diastolic
115(110-120) 60(50-70)
Augmentation with CPR (torr)
~ ~ ~
+98 +6 +40
100
120
SBP
Hemodynamics
FIG. 3. Arterial pressure changes resulting from closed chest massage
during cardiac arrest.
Arterial
Pressure 60
(torr)
tion or dysrhythmia is associated with normal cardiac
and intravascular volumes and, particularly, normal or
elevated left ventricular volume. In contrast, the inadequate
cardiac output that results from trauma or surgically
correctable shock is due to markedly reduced left
ventricular volume secondary to inadequate intravascular
volume (hypovolemic shock) or markedly restricted
ventricular filling (tamponade). The dramatic reduction
in blood available to exit the left ventricle during external
cardiac massage would logically indicate that cardiac
output and vital organ perfusion would also be significantly
less than in a normovolemic patient with unobstructed
ventricular filling. Although our experimental
model did not directly measure cardiac output or vital
organ perfusion, there was a significant difference in the
intra-arterial pressures generated in the models of
trauma shock as compared to that of cardiac arrest. Not
only was the average augmentation in the systolic, diastolic,
and mean arterial pressures greater in the cardiac
model, the absolute increment in pressure was markedly
less when external massage was initiated in the presence
of reduced left ventricular volumes.
The dramatic rise in intrapericardial pressure that
resulted from external massage in the setting of cardiac
tamponade raises a particular concern about the impact
of external massage in this clinical setting. Our findings
are consistent with those of other investigators who have
Es inefectivo en paro secundario a taponamiento cardíaco, neumotórax a tensión y hemorragia
To determine the hemodynamic effects of external massage in profound
shock, hypotension was induced in baboons. Pressures obtained with external
massage were compared to spontaneous intra-arterial pressures before
compression. Although external massage increased systolic pressures in both
tamponade and hypovolemia, diastolic pressures were consistently decreased.
We conclude that CCCPR does not augment arterial pressure in the clinical
situations associated with decreased LVEDV and is unlikely to provide organ
perfusion for trauma victims
Es inefectivo en paro secundario a taponamiento cardíaco, neumotórax a tensión y hemorragia
To determine the hemodynamic effects of external massage in profound
shock, hypotension was induced in baboons. Pressures obtained with external
massage were compared to spontaneous intra-arterial pressures before
compression. Although external massage increased systolic pressures in both
tamponade and hypovolemia, diastolic pressures were consistently decreased.
We conclude that CCCPR does not augment arterial pressure in the clinical
situations associated with decreased LVEDV and is unlikely to provide organ
perfusion for trauma victims