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Crisis de hiperglicemia en pacientes adultos con Diabetes Mellitus MARIA ALEJANDRA ENRIQUEZ R I MEDICINA INTERNA FUSM
Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate. Crisis de hiperglicemia en DM
CRITERIOS DIAGNOSTICOS PARA CAD Y HHS Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate. Crisis de Hiperglicemia en DMEpidemiologia
Patogenesis Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
Patogenesis                  Kitabchi, Abbas E; Epidemiology and pathogenesis of diabetic ketoacidosis and                   Hyperosmolar hiperglycemic state,  Agosto 2009
Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
Factoresprecipitantes Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
Factores precipitantes Data from: Kitabchi, AE, Umpierrez, GE, Murphy, MB, et al. Management of hyperglycemic crises in patients with diabetes mellitus (Technical Review). Diabetes Care 2001; 24:131
Factores precipitantes Data from: Kitabchi, AE, Umpierrez, GE, Murphy, MB, et al. Management of hyperglycemic crises in patients with diabetes mellitus (Technical Review). Diabetes Care 2001; 24:131
CAD desarrollo rápido, 24-horas, dolor abdominal y respiración Kussmaul  HHS insidiosa, con poliuria, polidipsia y pérdida de peso, varios días antes de su ingreso. Sintomas neurológicos progresivos incluyendo letargo, signos focales, y obnubilación, progresar a coma en etapas posteriores Presentación Clínica Kitabchi Abbas, Clinical Features and Diagnosis of Diabetics Ketoacidosis and Hyperosmolar  Hyperglicemic State in adults Enero 2010, Uptodate
Osmolaridad elevada causa alt. Neurológicas.  >320 mmol/kg    Osmolaridad efectiva= 2Na + glucemia/18 urea no induce cambios intracelulares en volumen o el gradiente osmótico de la membrana celular Diuresis osmótica, perdida de agua Na Y K. Osmolaridad normal, y sx neurológicos obliga a buscar otras causas Presentación clínica Kitabchi Abbas, treatment of diagnosis  of Diabetic Ketoacidosis and Hyperosmolar Hyperglicemic state in adults Enero 2010, Uptodate
Dolor abdominal, En CAD 40%, en relación con  severidad de acidosis Se relaciona con HCO3 5-15 Lento vaciamiento gástrico Desequilibrio H – Electrolítico Si persiste post a resolución de CAD buscar otras causas. Presentación clínica Kitabchi Abbas, Clinical Features And diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic state in adults, Jan 2010, Uptodate.
Hallazgos de Laboratorio Diabetes Care, American Diabetes Association, July 2009
Hallazgos de laboratorio Kitabchi Abbas, Clinical features and diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
Hallazgos de laboratorio Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
La gravedad de acidosis metabólica depende  La tasa de producción de cetoacidos. La duración de la producción cetoácido Tasa de excreción urinaria. ( depende funcion renal) El anión gap: Estimación de la cantidad de aniones no medidos en suero     anion gap = Na sérico - (Cl + HCO3) Hallazgos de Laboratorio Kitabchi Abbas, Clinical Features and diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults,  Jan  2009, Uptodate.
Hallazgos de Laboratorio Kitabchi Abbas, Features and diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Jan 2009, Uptodate.
Hallazgos de Laboratorio Kitabchi Abbas, Clinical Features and diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Jan 2009, Uptodate.
Hallazgos de laboratorio ,[object Object]
Para diagnostico de pancreatitis basarse en clínica
Puede elevarse amilasa por producción en glan. salivaresKitabchi Abbas, Clinical Features and diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Jan 2009, Uptodate.
Hallazgos de Laboratorio Kitabchi Abbas, Clinical Features and  diadnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Jan 2009, Uptodate.
Tratamiento Evaluación inicial examen físico  deben centrarse en: Vía aérea, respiración y circulación (ABC)   Estado mental Posibles factores precipitantes  Estado de hidratación y volemia La evaluación inicial de laboratorio: Glucosa sérica Electrolitos (con cálculo del anión gap), Azoados Hemograma completo con diferencial Uroanálisis  y cetonas en la orina con tira reactiva Osmolaridad de plasma Cetonas en suero  Gases  arteriales   Electrocardiograma Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
Tratamiento  Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.

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Crisis de hiperglicemia en pacientes adultos con diabetes

  • 1. Crisis de hiperglicemia en pacientes adultos con Diabetes Mellitus MARIA ALEJANDRA ENRIQUEZ R I MEDICINA INTERNA FUSM
  • 2. Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate. Crisis de hiperglicemia en DM
  • 3. CRITERIOS DIAGNOSTICOS PARA CAD Y HHS Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 4. Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate. Crisis de Hiperglicemia en DMEpidemiologia
  • 5. Patogenesis Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 6. Patogenesis Kitabchi, Abbas E; Epidemiology and pathogenesis of diabetic ketoacidosis and Hyperosmolar hiperglycemic state, Agosto 2009
  • 7. Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 8. Factoresprecipitantes Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 9. Factores precipitantes Data from: Kitabchi, AE, Umpierrez, GE, Murphy, MB, et al. Management of hyperglycemic crises in patients with diabetes mellitus (Technical Review). Diabetes Care 2001; 24:131
  • 10. Factores precipitantes Data from: Kitabchi, AE, Umpierrez, GE, Murphy, MB, et al. Management of hyperglycemic crises in patients with diabetes mellitus (Technical Review). Diabetes Care 2001; 24:131
  • 11. CAD desarrollo rápido, 24-horas, dolor abdominal y respiración Kussmaul HHS insidiosa, con poliuria, polidipsia y pérdida de peso, varios días antes de su ingreso. Sintomas neurológicos progresivos incluyendo letargo, signos focales, y obnubilación, progresar a coma en etapas posteriores Presentación Clínica Kitabchi Abbas, Clinical Features and Diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults Enero 2010, Uptodate
  • 12. Osmolaridad elevada causa alt. Neurológicas. >320 mmol/kg Osmolaridad efectiva= 2Na + glucemia/18 urea no induce cambios intracelulares en volumen o el gradiente osmótico de la membrana celular Diuresis osmótica, perdida de agua Na Y K. Osmolaridad normal, y sx neurológicos obliga a buscar otras causas Presentación clínica Kitabchi Abbas, treatment of diagnosis of Diabetic Ketoacidosis and Hyperosmolar Hyperglicemic state in adults Enero 2010, Uptodate
  • 13. Dolor abdominal, En CAD 40%, en relación con severidad de acidosis Se relaciona con HCO3 5-15 Lento vaciamiento gástrico Desequilibrio H – Electrolítico Si persiste post a resolución de CAD buscar otras causas. Presentación clínica Kitabchi Abbas, Clinical Features And diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic state in adults, Jan 2010, Uptodate.
  • 14. Hallazgos de Laboratorio Diabetes Care, American Diabetes Association, July 2009
  • 15. Hallazgos de laboratorio Kitabchi Abbas, Clinical features and diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 16. Hallazgos de laboratorio Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 17. La gravedad de acidosis metabólica depende La tasa de producción de cetoacidos. La duración de la producción cetoácido Tasa de excreción urinaria. ( depende funcion renal) El anión gap: Estimación de la cantidad de aniones no medidos en suero     anion gap = Na sérico - (Cl + HCO3) Hallazgos de Laboratorio Kitabchi Abbas, Clinical Features and diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Jan 2009, Uptodate.
  • 18. Hallazgos de Laboratorio Kitabchi Abbas, Features and diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Jan 2009, Uptodate.
  • 19. Hallazgos de Laboratorio Kitabchi Abbas, Clinical Features and diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Jan 2009, Uptodate.
  • 20.
  • 21. Para diagnostico de pancreatitis basarse en clínica
  • 22. Puede elevarse amilasa por producción en glan. salivaresKitabchi Abbas, Clinical Features and diagnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Jan 2009, Uptodate.
  • 23. Hallazgos de Laboratorio Kitabchi Abbas, Clinical Features and diadnosis of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Jan 2009, Uptodate.
  • 24. Tratamiento Evaluación inicial examen físico deben centrarse en: Vía aérea, respiración y circulación (ABC) Estado mental Posibles factores precipitantes Estado de hidratación y volemia La evaluación inicial de laboratorio: Glucosa sérica Electrolitos (con cálculo del anión gap), Azoados Hemograma completo con diferencial Uroanálisis y cetonas en la orina con tira reactiva Osmolaridad de plasma Cetonas en suero Gases arteriales Electrocardiograma Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 25. Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 26. Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 27. Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 28. Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 29. Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 30. Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 31. Tratamiento Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct 2009, Uptodate.
  • 32. Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Jan. 2009, Uptodate. Kitabchi Abbas, treatment of Diabetics Ketoacidosis and Hyperosmolar Hyperglicemic State in adults, Oct. 2009, Uptodate. Diabetes Care, American Diabetes Association, July 2009 Bibliografia