This document discusses anatomical considerations related to impingement syndrome and rotator cuff tears. It describes four types of impingement syndromes, including subacromial impingement, which is the most common. Stages of subacromial impingement are outlined from stage I involving tendonitis to stage IV with cuff tear arthropathy. Diagnosis is mainly clinical through evaluation of range of motion and impingement tests. Imaging such as MRI can further assess tendon integrity and tear size. Treatment options include conservative measures, arthroscopic subacromial decompression, or open repair of torn rotator cuff tendons.
6. Internal impingment
(GIRD)
caused by impingement of posterior under-
surface of supraspinatus tendon on
the posterosuperior glenoid rim
BENNETT LESION
7. INTERNAL VS EXTERNAL
Internal impingement refers to pathology on undersurface of rotator cuff
in contrast to
subacromial or "external" impingement which occurs on bursal side of rotator cuff
10. Outlet is bounded by
CAarch
Strong arch resist upward thrust
(William and Worwick ,1980)
AC joint
Plane synovial diarthrodial
Stability by AC &CC ligaments
11. Vestigial (continuation of
pectoralis minor) (Batman ,1972)
Thickening of the
clavipectoral fascia (Iannotti et
al. ,1991)
Triangular to trapizoid
FUNCTIONS:
Humeral head guide
Prevent upward migration
of humeral head.
12. The largest
Roof is related to deltoid,
acromion and to CA
ligament
Base is related to RC and
greater tuberosity of the
humerus
13. SS
IS
Sb.S
• TMn
The critical zone
1 cm medial to SS attachment
similar zone in IS & Sb.S
Area of anastomosis
Limits potentiality for healing
14.
15. Lindholm has described
an area of avascularity in
the supraspinatus tendon
proximal to its insertion
into the greater tuberosity.
Dynamic reason
Histological transition
that takes place from
tendon to calcified
fibrocartilage to bone
17. Stage I lesion:
Reversible oedema and haemorrhage in the tendon with bursal
irritation , below 25 years
Stage II :
Tendonitis and fibrosis with bursal thickening, superficial cuff
fibers dissociation , residual damage ,25-40 years
Stage III :
bony osteophytes appear, tendon wear and ruptures, partial
thickness &full thickness tear ,above 40 years
Stage IV:
Cuff tear arthropathy
18. Jobe&Jobe in1983
Stage I : tendonitis, of SS and LHB, with oedema
Stage II : fibers dissociation
Stage III : rotator cuff tears of less than 1cm
Stage IV : the tear is more than 1 cm.
Paulos et.al in 1996 stated that there are two distinct types
of impingement syndrome: primary and secondary.
1ry form is the classic form of Neer
Over 40 years
Treated by acromioplasty
2ry (normal subacromial anatomy)
Instability, trauma or overuse
Any age
Treated by managimg the cause
31. positive in late stage II and
(A) Plain -X-ray:
stage III
1-True AP view
2-The 30˚ caudal tilt view
3-supraspintus out let view
4-Axillary view
37. Squaring and or decalcification of
the greater tuberosity
Anterior acromial spur and or
erosions
Prominent under surface of
acromioclavicular joint
Loss of acromiohumeral distance
(normal 7-13 mm.)
GH instability
RC tear arthropathy
38. Dynamic and static
Subjective
Non invasive
Cheap
Arthrography
Full thickness tear
CT scanning
39. Magnetic resonance imaging is the most useful diagnostic
modality for shoulder disorders ( Rossi, 1998).
*SENETIVE AND SPECIFIC
*MORE OBJECTIVE (OPERATOR INDEPENDENT)
*NON INVASIVE
*HIGHER SOFT TISSIUE CONTRAST
*MULIPLANER(SAGITTAL,CORONAL&AXIAL)
40. More accurate than X ray in evaluating acromion
Can predict intra substance and partial thickness tear
Assessment of bursa, LHB, labrum and CA ligament
MRI grade of RC evaluation
G 0 ……… normal
G I ………tendonitis increased signal
G II ……… increased signal and disturbed morphology
G III ……..subacromial subdeltoid fat plane disruption ”partialtear”
G IV………fluid in the bursa “complete tear”
44. Above 40 years
No rotator cuff tear
Persistent disability
Pain can be eliminated by impingement test.
Under 40 years
Stage II impingement lesions
or any young active patient with his work requiring
an over head movement of the arm
if conservative ttt fail.
45. Good prognostic signs:
(1)A well motivated patient over 40 years old.
(2)Absence of posterior capsularstiffness.
(3)Presence of subacromial crepitus.
(4)Pain relieved by the subacromial injection of lidocaine.
(5)A condition that is unrelated to the patient's occupation.
Poor prognostic signs:
(1)Age less than 40 years old.
(2)Stiffness.
(3)Absence of subacromial crepitus.
(4)Lack of relieve by subacromial injection.
(5)Attribution of problem to occupation.
(6)Concomitant evidence of glenohumeral instability.
(7)Neurogenic cuff muscle weakness.
60. Impingement syndrome, presents one of the most
common cause of a painful shoulder
Diagnosis of impingement syndrome is mainly a
CLINICAL issue
Arthroscopic subacromial decompression carry more
advantages over open acromioplasty