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Anatomical considerations
of the shoulder girdle
IMPINGMENT SYNDROME
& ROTATOR CUFF TEAR
Mohamed Hamdi Elgawadi ,MD,FRCS (T&O)
 Definition:
 Abnormal Contact
Compression, Crushing
Encroachment, Entrapment
Of the Rotator Cuff
 Impingement" syndromes of the
shoulder includes 4 items ( Rossi,
1998):
 1-Subacromial impingement
syndrome
 2-Subcoracoid impingement syndrome
 3-Posterior superior glenoid rim
impingement syndrome
 4- Suprascapular nerve impingement
syndrome
Subcoracoid impingment
Decreased coracohumeral distance (less than 6 mm)
Normal 8.7 mm
Coracoplasty
Internal impingment
(GIRD)
caused by impingement of posterior under-
surface of supraspinatus tendon on
the posterosuperior glenoid rim
BENNETT LESION
INTERNAL VS EXTERNAL
Internal impingement refers to pathology on undersurface of rotator cuff
in contrast to
subacromial or "external" impingement which occurs on bursal side of rotator cuff
Anatomical considerations
of the shoulder girdle
Anatomical cosideration of
the shoulder girdle
Created wlth
TYPES OF ACROMION
Outlet is bounded by
CAarch
Strong arch resist upward thrust
(William and Worwick ,1980)
AC joint
Plane synovial diarthrodial
Stability by AC &CC ligaments
Vestigial (continuation of
pectoralis minor) (Batman ,1972)
Thickening of the
clavipectoral fascia (Iannotti et
al. ,1991)
Triangular to trapizoid
FUNCTIONS:
Humeral head guide
Prevent upward migration
of humeral head.
 The largest
 Roof is related to deltoid,
acromion and to CA
ligament
 Base is related to RC and
greater tuberosity of the
humerus
 SS
 IS
 Sb.S
• TMn
The critical zone
1 cm medial to SS attachment
 similar zone in IS & Sb.S
Area of anastomosis
Limits potentiality for healing
 Lindholm has described
an area of avascularity in
the supraspinatus tendon
proximal to its insertion
into the greater tuberosity.
 Dynamic reason
 Histological transition
that takes place from
tendon to calcified
fibrocartilage to bone
Pathological considerations
of subacromial
impingement syndrome
 Stage I lesion:
 Reversible oedema and haemorrhage in the tendon with bursal
irritation , below 25 years
 Stage II :
 Tendonitis and fibrosis with bursal thickening, superficial cuff
fibers dissociation , residual damage ,25-40 years
 Stage III :
 bony osteophytes appear, tendon wear and ruptures, partial
thickness &full thickness tear ,above 40 years
 Stage IV:
 Cuff tear arthropathy
 Jobe&Jobe in1983
 Stage I : tendonitis, of SS and LHB, with oedema
 Stage II : fibers dissociation
 Stage III : rotator cuff tears of less than 1cm
 Stage IV : the tear is more than 1 cm.
 Paulos et.al in 1996 stated that there are two distinct types
of impingement syndrome: primary and secondary.
1ry form is the classic form of Neer
Over 40 years
Treated by acromioplasty
2ry (normal subacromial anatomy)
Instability, trauma or overuse
Any age
Treated by managimg the cause
 1-Anatomical etiology. .
 2-Truamatic. Etiology
 3-Vascular. Etiology
 4-Degenerative etiology
 Impingement =
 4 factors interplay
Anatomical considerations
of the shoulder girdle
Diagnosis of subacromial
impingment syndrome
CLINICAL ISSUE
 Symptoms:
 Pain
 Limitation of motions
 Weakness
 Signs:
 Arc of pain
 Crepitus
 Weakness
 Impingement sign
 Impingement test
 Cuff tear:
 Dropping sign
 Shrugging sign
 Heamoarthrosis
 Cuff tear arthropathy:
 Advanced painful limitation of movement
1. Forward elevation
(maximum arm-trunk angle)
2. Abduction (note classic
painful arc)
4. External r
(arm comfo
otation
rtably at side)
3. External rotation
(arm at 90 degree abduction
 The “lift-off” test can
help to assess
subscapularis
integrity
 Resisted ER
 Infraspinatus
 Teres Minor
 Neer sign for
impingement
 Hawkins Test
 Other causes of painful shoulder
 AC arthritis
 RC tear
 Frozen shoulder
 Calcific tendonitis
 LHB tendonitis
 GH instability ,arthritis
 AHSB (acute haemorrhagic subacromialbursitis)
 Snapping scapula
 Cervical radiculopathy C5-C6
Anatomical considerations
of the shoulder girdle
Radiological assessment of
subacromial impingment
syndrome
positive in late stage II and
 (A) Plain -X-ray:
stage III
 1-True AP view
 2-The 30˚ caudal tilt view
 3-supraspintus out let view
 4-Axillary view
2-The 30˚ caudal tilt view
4-Axillary view
 8 to 12
millimeters
 Squaring and or decalcification of
the greater tuberosity
 Anterior acromial spur and or
erosions
 Prominent under surface of
acromioclavicular joint
 Loss of acromiohumeral distance
(normal 7-13 mm.)
 GH instability
 RC tear arthropathy
 Dynamic and static
 Subjective
 Non invasive
 Cheap
Arthrography
Full thickness tear
CT scanning
 Magnetic resonance imaging is the most useful diagnostic
modality for shoulder disorders ( Rossi, 1998).
*SENETIVE AND SPECIFIC
*MORE OBJECTIVE (OPERATOR INDEPENDENT)
*NON INVASIVE
*HIGHER SOFT TISSIUE CONTRAST
*MULIPLANER(SAGITTAL,CORONAL&AXIAL)
 More accurate than X ray in evaluating acromion
 Can predict intra substance and partial thickness tear
 Assessment of bursa, LHB, labrum and CA ligament
MRI grade of RC evaluation
G 0 ……… normal
G I ………tendonitis increased signal
G II ……… increased signal and disturbed morphology
G III ……..subacromial subdeltoid fat plane disruption ”partialtear”
G IV………fluid in the bursa “complete tear”
Anatomical considerations
of the shoulder girdle
Treatment of subacromial
impingment syndrome
prophylactic curative
Conservative
-rest
-NSAID
-corticosteroid injection
-physical therapy
Surgical
-CA ligament release
-Ant.Acromioplasty
-lat. Clavicle excision
-repair of RC tear
-biceps tenodesis
-epsilon of calcified
deposit
-combination
 Above 40 years
 No rotator cuff tear
 Persistent disability
 Pain can be eliminated by impingement test.
 Under 40 years
 Stage II impingement lesions
 or any young active patient with his work requiring
an over head movement of the arm
 if conservative ttt fail.
 Good prognostic signs:
 (1)A well motivated patient over 40 years old.
 (2)Absence of posterior capsularstiffness.
 (3)Presence of subacromial crepitus.
 (4)Pain relieved by the subacromial injection of lidocaine.
 (5)A condition that is unrelated to the patient's occupation.
 Poor prognostic signs:
 (1)Age less than 40 years old.
 (2)Stiffness.
 (3)Absence of subacromial crepitus.
 (4)Lack of relieve by subacromial injection.
 (5)Attribution of problem to occupation.
 (6)Concomitant evidence of glenohumeral instability.
 (7)Neurogenic cuff muscle weakness.
 Arthroscope.
 Mini open repair.
 Open repair.
 Indication:
-Patient are unable to tolerate either the
surgery or post operative rehabilitation of
an open procedure.
 Procedure :-
1Glenohumeral inspection.
2 Bursectomy.
3Determination of cuff reliability.
4- Identification of tear geometry.
5- Resection of coracoacromial ligament.
6Acromioplasty.
7Greater tuberosity preparation.
8- Anchor placement.
9 Suture placement.
10 Knot tying.
 Post operative management:
- 6 weeks passive ROM.
- Then active ROM.
- Strengthening excersise at 3 month.
- Rehabilitation continue for 6 - 12 month.
 The technique combines Arthroscopic
subacromial decompression with open
tendon repair through a small deltoid split.
 Advantages:-
- Lower preoperative morbidity.
- Short hospital stay.
- Cosmetic incision.
- Short rehabilitation period.
- Preservation of deltoid origin during repair.
 Patient selection:-
Mini open repair is usually reserved for
patient with small to medium sized tears
<3cm without significant retraction.
ROTATOR CUFF ARTHROPATHY
Reverse TSA
TSA
GLENOID LOOSENING BY ( ROCKING
HORSE PHENOMENON)
 Impingement syndrome, presents one of the most
common cause of a painful shoulder
 Diagnosis of impingement syndrome is mainly a
CLINICAL issue
 Arthroscopic subacromial decompression carry more
advantages over open acromioplasty
THANK YOU
VISIT

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IMPINGMENT SYNDROME & ROTATOR CUFF TEAR.pptx

  • 1. Anatomical considerations of the shoulder girdle IMPINGMENT SYNDROME & ROTATOR CUFF TEAR Mohamed Hamdi Elgawadi ,MD,FRCS (T&O)
  • 2.  Definition:  Abnormal Contact Compression, Crushing Encroachment, Entrapment Of the Rotator Cuff
  • 3.  Impingement" syndromes of the shoulder includes 4 items ( Rossi, 1998):  1-Subacromial impingement syndrome  2-Subcoracoid impingement syndrome  3-Posterior superior glenoid rim impingement syndrome  4- Suprascapular nerve impingement syndrome
  • 4. Subcoracoid impingment Decreased coracohumeral distance (less than 6 mm) Normal 8.7 mm
  • 6. Internal impingment (GIRD) caused by impingement of posterior under- surface of supraspinatus tendon on the posterosuperior glenoid rim BENNETT LESION
  • 7. INTERNAL VS EXTERNAL Internal impingement refers to pathology on undersurface of rotator cuff in contrast to subacromial or "external" impingement which occurs on bursal side of rotator cuff
  • 8. Anatomical considerations of the shoulder girdle Anatomical cosideration of the shoulder girdle
  • 10. Outlet is bounded by CAarch Strong arch resist upward thrust (William and Worwick ,1980) AC joint Plane synovial diarthrodial Stability by AC &CC ligaments
  • 11. Vestigial (continuation of pectoralis minor) (Batman ,1972) Thickening of the clavipectoral fascia (Iannotti et al. ,1991) Triangular to trapizoid FUNCTIONS: Humeral head guide Prevent upward migration of humeral head.
  • 12.  The largest  Roof is related to deltoid, acromion and to CA ligament  Base is related to RC and greater tuberosity of the humerus
  • 13.  SS  IS  Sb.S • TMn The critical zone 1 cm medial to SS attachment  similar zone in IS & Sb.S Area of anastomosis Limits potentiality for healing
  • 14.
  • 15.  Lindholm has described an area of avascularity in the supraspinatus tendon proximal to its insertion into the greater tuberosity.  Dynamic reason  Histological transition that takes place from tendon to calcified fibrocartilage to bone
  • 17.  Stage I lesion:  Reversible oedema and haemorrhage in the tendon with bursal irritation , below 25 years  Stage II :  Tendonitis and fibrosis with bursal thickening, superficial cuff fibers dissociation , residual damage ,25-40 years  Stage III :  bony osteophytes appear, tendon wear and ruptures, partial thickness &full thickness tear ,above 40 years  Stage IV:  Cuff tear arthropathy
  • 18.  Jobe&Jobe in1983  Stage I : tendonitis, of SS and LHB, with oedema  Stage II : fibers dissociation  Stage III : rotator cuff tears of less than 1cm  Stage IV : the tear is more than 1 cm.  Paulos et.al in 1996 stated that there are two distinct types of impingement syndrome: primary and secondary. 1ry form is the classic form of Neer Over 40 years Treated by acromioplasty 2ry (normal subacromial anatomy) Instability, trauma or overuse Any age Treated by managimg the cause
  • 19.  1-Anatomical etiology. .  2-Truamatic. Etiology  3-Vascular. Etiology  4-Degenerative etiology  Impingement =  4 factors interplay
  • 20. Anatomical considerations of the shoulder girdle Diagnosis of subacromial impingment syndrome
  • 21. CLINICAL ISSUE  Symptoms:  Pain  Limitation of motions  Weakness  Signs:  Arc of pain  Crepitus  Weakness  Impingement sign  Impingement test
  • 22.  Cuff tear:  Dropping sign  Shrugging sign  Heamoarthrosis  Cuff tear arthropathy:  Advanced painful limitation of movement
  • 23. 1. Forward elevation (maximum arm-trunk angle) 2. Abduction (note classic painful arc) 4. External r (arm comfo otation rtably at side) 3. External rotation (arm at 90 degree abduction
  • 24.  The “lift-off” test can help to assess subscapularis integrity
  • 25.
  • 26.  Resisted ER  Infraspinatus  Teres Minor
  • 27.  Neer sign for impingement
  • 29.  Other causes of painful shoulder  AC arthritis  RC tear  Frozen shoulder  Calcific tendonitis  LHB tendonitis  GH instability ,arthritis  AHSB (acute haemorrhagic subacromialbursitis)  Snapping scapula  Cervical radiculopathy C5-C6
  • 30. Anatomical considerations of the shoulder girdle Radiological assessment of subacromial impingment syndrome
  • 31. positive in late stage II and  (A) Plain -X-ray: stage III  1-True AP view  2-The 30˚ caudal tilt view  3-supraspintus out let view  4-Axillary view
  • 32. 2-The 30˚ caudal tilt view
  • 34.  8 to 12 millimeters
  • 35.
  • 36.
  • 37.  Squaring and or decalcification of the greater tuberosity  Anterior acromial spur and or erosions  Prominent under surface of acromioclavicular joint  Loss of acromiohumeral distance (normal 7-13 mm.)  GH instability  RC tear arthropathy
  • 38.  Dynamic and static  Subjective  Non invasive  Cheap Arthrography Full thickness tear CT scanning
  • 39.  Magnetic resonance imaging is the most useful diagnostic modality for shoulder disorders ( Rossi, 1998). *SENETIVE AND SPECIFIC *MORE OBJECTIVE (OPERATOR INDEPENDENT) *NON INVASIVE *HIGHER SOFT TISSIUE CONTRAST *MULIPLANER(SAGITTAL,CORONAL&AXIAL)
  • 40.  More accurate than X ray in evaluating acromion  Can predict intra substance and partial thickness tear  Assessment of bursa, LHB, labrum and CA ligament MRI grade of RC evaluation G 0 ……… normal G I ………tendonitis increased signal G II ……… increased signal and disturbed morphology G III ……..subacromial subdeltoid fat plane disruption ”partialtear” G IV………fluid in the bursa “complete tear”
  • 41.
  • 42. Anatomical considerations of the shoulder girdle Treatment of subacromial impingment syndrome
  • 43. prophylactic curative Conservative -rest -NSAID -corticosteroid injection -physical therapy Surgical -CA ligament release -Ant.Acromioplasty -lat. Clavicle excision -repair of RC tear -biceps tenodesis -epsilon of calcified deposit -combination
  • 44.  Above 40 years  No rotator cuff tear  Persistent disability  Pain can be eliminated by impingement test.  Under 40 years  Stage II impingement lesions  or any young active patient with his work requiring an over head movement of the arm  if conservative ttt fail.
  • 45.  Good prognostic signs:  (1)A well motivated patient over 40 years old.  (2)Absence of posterior capsularstiffness.  (3)Presence of subacromial crepitus.  (4)Pain relieved by the subacromial injection of lidocaine.  (5)A condition that is unrelated to the patient's occupation.  Poor prognostic signs:  (1)Age less than 40 years old.  (2)Stiffness.  (3)Absence of subacromial crepitus.  (4)Lack of relieve by subacromial injection.  (5)Attribution of problem to occupation.  (6)Concomitant evidence of glenohumeral instability.  (7)Neurogenic cuff muscle weakness.
  • 46.
  • 47.  Arthroscope.  Mini open repair.  Open repair.
  • 48.  Indication: -Patient are unable to tolerate either the surgery or post operative rehabilitation of an open procedure.
  • 49.  Procedure :- 1Glenohumeral inspection. 2 Bursectomy. 3Determination of cuff reliability. 4- Identification of tear geometry. 5- Resection of coracoacromial ligament.
  • 50. 6Acromioplasty. 7Greater tuberosity preparation. 8- Anchor placement. 9 Suture placement. 10 Knot tying.
  • 51.  Post operative management: - 6 weeks passive ROM. - Then active ROM. - Strengthening excersise at 3 month. - Rehabilitation continue for 6 - 12 month.
  • 52.
  • 53.  The technique combines Arthroscopic subacromial decompression with open tendon repair through a small deltoid split.
  • 54.  Advantages:- - Lower preoperative morbidity. - Short hospital stay. - Cosmetic incision. - Short rehabilitation period. - Preservation of deltoid origin during repair.
  • 55.  Patient selection:- Mini open repair is usually reserved for patient with small to medium sized tears <3cm without significant retraction.
  • 56.
  • 58. Reverse TSA TSA GLENOID LOOSENING BY ( ROCKING HORSE PHENOMENON)
  • 59.
  • 60.  Impingement syndrome, presents one of the most common cause of a painful shoulder  Diagnosis of impingement syndrome is mainly a CLINICAL issue  Arthroscopic subacromial decompression carry more advantages over open acromioplasty