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Dr. Mohammad Masoom Parwez
Academic Resident
AIIMS Bhopal
 Barium swallow -- esophagus
 Barium meal--- stomach and duodenum
 Barium meal follow through--- small and large gut
 Smooth narrowing, no irregularity of mucosal lining
 Due to loss of ganglion cells in myenteric plexus
 Retention of food and fluids– dilatation of esophagus– recurrent aspiration
pneumonitis--- increased incidence of CA
 Pseudoachalasia– due to intraluminal growth/ extraluminal compression
 Presentation: young adults/ dysphagia to liquids– solids & liquids/ retrosternal
discomfort/ regurgitation/ aspiration pneumonia/ weight loss and anemia
 Inv: endoscopy, manometry
 Medical mx: CCB oral nifedipine, botulinum toxin inj
 Surgical Mx: endoscopic dilatation, Hellers cardiomyotomy with partial fundoplication
 Irregular/ incoordinated contractions – high intraesophageal pressure (400-500 mm
Hg)
 Marked hypertrophy of circular muscles
 Presentation: Dysphagia, chest pain
 Cork screw esophagus
 Treatment: esophageal dilatation, long esophageal myotomy
 Presentation: dysphagia to solids– both solids and liquids/ regurgitation/ anorexia &
weight loss/ hoarseness/ neck mass/ hematemesis/ melena
 Inv: endoscopy – biopsy, EUS, CT
 Staging– AJCC 8th edition
 Spread: direct/ lymphatic/ distant
 Predisposing factors: diet, smoking, alcohol, barretts esophagus, achalasia, chr
esophagitis
 Types: squamous/ adenoCA
 Sx: subtotal/ total esophagectomy with esophagogastric anastomosis + CT/RT
 Palliation: stenting, laser/electrocautery to excise tumor, brachytherapy
 Barium enema
 DCBE
Saw tooth
appearance
Rose thorn ulcers
String sign of
kantor
Lead
pipe
colon
Apple core
ERCP
T- tube cholangiogram
Breast and lymph node
 Based on cells of origin:
 Ductal – epth cells lining the ducts
 Lobular-- cells lining lobules/acini
 Pathological types:
 Carcinoma in situ– upto basement memb
 Infiltrating– basement memb breached; invade connective tissue
 Scirrhous– small unencapsulated tumor with diminished vascularity; cuts with gritty sensation
 Medullary– young women; tumor bulky but softer
 Colloidal– bulky, soft tumor; microscopically cells abundant in mucin; signet cells
 Inflammatory– highly aggressive, poor prognosis, breast swollen, warm, painful with rich
vascularity
 Pagets disease of nipple– eczema over NAC– eventual destruction, large ovoid pagets cells
with abundant cytoplasm
 Skin changes:
Peau d’ Orange: orange peel appearance–
subdermal lymphatics– skin tense, thickened and
edematous
Tethering: coopers ligament, skin dimples on
pushing the lump
Skin nodules: direct invasion of the skin
Cancer en cuirasse: advanced stage; skin
become fixed and indurated, satellite nodules,
involves whole of breast
 Risk factors:
 Age
 Sex
 SES
 Diet
 Addiction
 Early menarche, late menopause, late first pregnancy
 HRT/OCP
 Prior radiation
 Family history
 Genetic
 Prior breast CA
 Modes of spread:
 Direct: adjacent quadrant; chest wall; pectoral muscles; serratus anterior; overlying
skin
 Lymphatic: permeation and embolization; axillary and internal mammary nodes; supra
and infraclavicular nodes, opposite breast and axilla; peritoneum and ovaries
 Hematogenous: bones (lumbar vertebra, femur, thoracic vertebra, ribs, skull); liver;
lungs; brain; adrenal; ovaries
X-rays & Specimen.pptx
X-rays & Specimen.pptx
X-rays & Specimen.pptx
X-rays & Specimen.pptx
X-rays & Specimen.pptx
X-rays & Specimen.pptx
X-rays & Specimen.pptx
X-rays & Specimen.pptx

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X-rays & Specimen.pptx

  • 1. Dr. Mohammad Masoom Parwez Academic Resident AIIMS Bhopal
  • 2.  Barium swallow -- esophagus  Barium meal--- stomach and duodenum  Barium meal follow through--- small and large gut
  • 3.
  • 4.
  • 5.
  • 6.
  • 7.
  • 8.
  • 9.
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  • 11.  Smooth narrowing, no irregularity of mucosal lining  Due to loss of ganglion cells in myenteric plexus  Retention of food and fluids– dilatation of esophagus– recurrent aspiration pneumonitis--- increased incidence of CA  Pseudoachalasia– due to intraluminal growth/ extraluminal compression  Presentation: young adults/ dysphagia to liquids– solids & liquids/ retrosternal discomfort/ regurgitation/ aspiration pneumonia/ weight loss and anemia  Inv: endoscopy, manometry  Medical mx: CCB oral nifedipine, botulinum toxin inj  Surgical Mx: endoscopic dilatation, Hellers cardiomyotomy with partial fundoplication
  • 12.
  • 13.  Irregular/ incoordinated contractions – high intraesophageal pressure (400-500 mm Hg)  Marked hypertrophy of circular muscles  Presentation: Dysphagia, chest pain  Cork screw esophagus  Treatment: esophageal dilatation, long esophageal myotomy
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  • 18.  Presentation: dysphagia to solids– both solids and liquids/ regurgitation/ anorexia & weight loss/ hoarseness/ neck mass/ hematemesis/ melena  Inv: endoscopy – biopsy, EUS, CT  Staging– AJCC 8th edition  Spread: direct/ lymphatic/ distant  Predisposing factors: diet, smoking, alcohol, barretts esophagus, achalasia, chr esophagitis  Types: squamous/ adenoCA  Sx: subtotal/ total esophagectomy with esophagogastric anastomosis + CT/RT  Palliation: stenting, laser/electrocautery to excise tumor, brachytherapy
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  • 62.  Based on cells of origin:  Ductal – epth cells lining the ducts  Lobular-- cells lining lobules/acini  Pathological types:  Carcinoma in situ– upto basement memb  Infiltrating– basement memb breached; invade connective tissue  Scirrhous– small unencapsulated tumor with diminished vascularity; cuts with gritty sensation  Medullary– young women; tumor bulky but softer  Colloidal– bulky, soft tumor; microscopically cells abundant in mucin; signet cells  Inflammatory– highly aggressive, poor prognosis, breast swollen, warm, painful with rich vascularity  Pagets disease of nipple– eczema over NAC– eventual destruction, large ovoid pagets cells with abundant cytoplasm
  • 63.  Skin changes: Peau d’ Orange: orange peel appearance– subdermal lymphatics– skin tense, thickened and edematous Tethering: coopers ligament, skin dimples on pushing the lump Skin nodules: direct invasion of the skin Cancer en cuirasse: advanced stage; skin become fixed and indurated, satellite nodules, involves whole of breast
  • 64.  Risk factors:  Age  Sex  SES  Diet  Addiction  Early menarche, late menopause, late first pregnancy  HRT/OCP  Prior radiation  Family history  Genetic  Prior breast CA
  • 65.  Modes of spread:  Direct: adjacent quadrant; chest wall; pectoral muscles; serratus anterior; overlying skin  Lymphatic: permeation and embolization; axillary and internal mammary nodes; supra and infraclavicular nodes, opposite breast and axilla; peritoneum and ovaries  Hematogenous: bones (lumbar vertebra, femur, thoracic vertebra, ribs, skull); liver; lungs; brain; adrenal; ovaries