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On April 9th 2013 took place at Vall d’Hebron Institut de Recerca (VHIR) the seminar ‘Multi-stage, multimodal approaches for regenerative stroke therapies’, conducted by Pr. Aurel Popa-Wagner, PhD, Professor of Experimental Neurology at the Department of Neurology and Head of the Research Department.
Every six minutes a stroke occurs in Spain, the first cause of death in women and the second for men. In the world, it is estimated that there are 4.5 million deaths a year from stroke. Almost one in four men and nearly one in five women aged 45 years can expect to have a stroke if they live to their 85th year. It is estimated that by 2023 there will be an absolute increase in the number of patients experiencing a first ever stroke of about 30% compared with 1983.
It is known that intracellular vessel occlusion has a strong age dependency. As long as aging is an important risk factor for stroke, aged animals are being used in the laboratory because of its relevance in clinical rehabilitation and cellular studies. In addition, Pr. Aurel Popa-Wagner and his research group have shown that potential mechanisms for self-repair also operate in the post-ischemic aged brain. Young and aged animals affected by stroke differ in their post-stroke response, being more pronounced at 14 days post-stroke, where downregulated genes decrease in young rats and increases in aged rats, and the opposite occurs with the upregulated genes.
Referring to therapy, Pr. Aurel Popa-Wagner and his research group concluded that including both physical methods and methods of cellular therapy is more effective for improving recovery of function in aged rats after stroke than therapies aimed at only a single target system. This is, for example, combining neurogenesis, functional in the subventricular zone of the adult brain, with G-CSF (stem cell mobilisator granulocyte-colony stimulating factor), significantly effective in treatment of aged rats after stroke due to that it reduces the mortality rate in rats. In the first post-stroke hours, induced hypothermia with hydrogen sulfide diminish inflammation and improve neurorehabilitation in aged rats by simultaneously targeting multiple points of intervention. This technique could have a higher probability of success in treating the illness, as the Pr. Popa-Wagner affirms. The next step in post-stroke therapy, according to the speaker, might be the combination of nanotherapy with cell therapy.Multi-stage, multimodal Approaches for regenerative stroke therapies
Multi-stage, multimodal Approaches for regenerative stroke therapiesVall d'Hebron Institute of Research (VHIR)
La infección activa (IA) por el CMV es frecuente en el paciente crítico sin inmunosupresión canónica (0-55% de los pacientes seropositivos para el CMV). Esta es comúnmente el resultado de la reactivación viral en el tracto respiratorio inferior, sin que pueda descartarse la reinfección como origen, en algunos casos. La IA por el CMV se ha asociado consistentemente con una mayor mortalidad, especialmente relacionada con el desarrollo de ALI/ARDS, una estancia en UCI más prolongada, una mayor duración del período de ventilación mecánica y un riesgo mayor de “superinfección” bacteriana y fúngica. No existen, sin embargo, vínculos incontestables de causalidad. Solo un ensayo clínico controlado puede precisar el papel del CMV como agente patogénico en este grupo de pacientes. En este seminario se tratarán los siguientes temas: (i) posible patogenia de la infección por el CMV en el paciente crítico; (ii) ensayos clínicos de tratamiento antiviral en marcha; (iii) factores biológicos (genómicos e inmunológicos) de riesgo para el desarrollo de IA en estos pacientes.Cytomegalovirus (CMV) in Intensive Care Unit (ICU) patients (David Navarro, PhD)
Cytomegalovirus (CMV) in Intensive Care Unit (ICU) patients (David Navarro, PhD)Vall d'Hebron Institute of Research (VHIR)
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On April 9th 2013 took place at Vall d’Hebron Institut de Recerca (VHIR) the seminar ‘Multi-stage, multimodal approaches for regenerative stroke therapies’, conducted by Pr. Aurel Popa-Wagner, PhD, Professor of Experimental Neurology at the Department of Neurology and Head of the Research Department.
Every six minutes a stroke occurs in Spain, the first cause of death in women and the second for men. In the world, it is estimated that there are 4.5 million deaths a year from stroke. Almost one in four men and nearly one in five women aged 45 years can expect to have a stroke if they live to their 85th year. It is estimated that by 2023 there will be an absolute increase in the number of patients experiencing a first ever stroke of about 30% compared with 1983.
It is known that intracellular vessel occlusion has a strong age dependency. As long as aging is an important risk factor for stroke, aged animals are being used in the laboratory because of its relevance in clinical rehabilitation and cellular studies. In addition, Pr. Aurel Popa-Wagner and his research group have shown that potential mechanisms for self-repair also operate in the post-ischemic aged brain. Young and aged animals affected by stroke differ in their post-stroke response, being more pronounced at 14 days post-stroke, where downregulated genes decrease in young rats and increases in aged rats, and the opposite occurs with the upregulated genes.
Referring to therapy, Pr. Aurel Popa-Wagner and his research group concluded that including both physical methods and methods of cellular therapy is more effective for improving recovery of function in aged rats after stroke than therapies aimed at only a single target system. This is, for example, combining neurogenesis, functional in the subventricular zone of the adult brain, with G-CSF (stem cell mobilisator granulocyte-colony stimulating factor), significantly effective in treatment of aged rats after stroke due to that it reduces the mortality rate in rats. In the first post-stroke hours, induced hypothermia with hydrogen sulfide diminish inflammation and improve neurorehabilitation in aged rats by simultaneously targeting multiple points of intervention. This technique could have a higher probability of success in treating the illness, as the Pr. Popa-Wagner affirms. The next step in post-stroke therapy, according to the speaker, might be the combination of nanotherapy with cell therapy.Multi-stage, multimodal Approaches for regenerative stroke therapies
Multi-stage, multimodal Approaches for regenerative stroke therapiesVall d'Hebron Institute of Research (VHIR)
La infección activa (IA) por el CMV es frecuente en el paciente crítico sin inmunosupresión canónica (0-55% de los pacientes seropositivos para el CMV). Esta es comúnmente el resultado de la reactivación viral en el tracto respiratorio inferior, sin que pueda descartarse la reinfección como origen, en algunos casos. La IA por el CMV se ha asociado consistentemente con una mayor mortalidad, especialmente relacionada con el desarrollo de ALI/ARDS, una estancia en UCI más prolongada, una mayor duración del período de ventilación mecánica y un riesgo mayor de “superinfección” bacteriana y fúngica. No existen, sin embargo, vínculos incontestables de causalidad. Solo un ensayo clínico controlado puede precisar el papel del CMV como agente patogénico en este grupo de pacientes. En este seminario se tratarán los siguientes temas: (i) posible patogenia de la infección por el CMV en el paciente crítico; (ii) ensayos clínicos de tratamiento antiviral en marcha; (iii) factores biológicos (genómicos e inmunológicos) de riesgo para el desarrollo de IA en estos pacientes.Cytomegalovirus (CMV) in Intensive Care Unit (ICU) patients (David Navarro, PhD)
Cytomegalovirus (CMV) in Intensive Care Unit (ICU) patients (David Navarro, PhD)Vall d'Hebron Institute of Research (VHIR)
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School quiz(the first has hyperlinks ) and yeah its for juniors
School quiz(the first has hyperlinks ) and yeah its for juniors
Multi-stage, multimodal Approaches for regenerative stroke therapies
Multi-stage, multimodal Approaches for regenerative stroke therapies
Cytomegalovirus (CMV) in Intensive Care Unit (ICU) patients (David Navarro, PhD)
Cytomegalovirus (CMV) in Intensive Care Unit (ICU) patients (David Navarro, PhD)