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Rajesh vardhan
HISTORY Volkmann 1881
Richard von Volkmann published an article in which
he attempted to describe the condition of irreversible
contractures of the flexor muscles of the hand to
ischemic processes occurring in the forearm
Hildebrand 1906
First used the term Volkmann ischemic contracture to
describe the final result of any untreated compartment
syndrome, and was the first to suggest that elevated
tissue pressure may be related to ischemic contracture.
Thomas 1909
Reviewed the 112 published cases of Volkmann ischemic
contracture and found fractures to be the predominant
cause. Also, noted that tight bandages, an arterial
embolus, or arterial insufficiency could also lead to the
problem
Murphy 1914
First to suggest that fasciotomy might prevent the
contracture. Also, suggested that tissue pressure and
fasciotomy were related to the development of
contracture
What is a compartment?
Closed area of muscles
group, nerves & blood
vessels surrounded by
fascia
Pressure: 5-15 mmhg
DEFINITION
Increase in interstitial fluid presuure within an
osseofascial compartment of significant magn
itude to cause compromise in microcirculation
and myoneuralnecrosis
What is a compartment
syndrome?
 intra comp. pressure
(35-40 mmhg)
 capillaries collapse
 Blood flow to muscles
and nerves
 Bl.Vs collapse
Pathophysiology
Increased compartment pressure
leads to increased venous pressure
which decreases A-V gradient
resulting in muscle and nerve
ischemia.
vicious cycle
Consequences –vicious cycle
Why is it dangerous?
Nerves:
neuropraxia: will
regenerate
Ischemia: cell death
Muscles: contracture
(Volkmann's ischemic
contracture)
Gangrene
Normal tissue pressure
 0-4 mm Hg
8-10 with exertion
Absolute pressure theory
30 mm Hg - Mubarak
45 mm Hg - Matsen
Pressure gradient theory
< 20 mm Hg of diastolic
pressure – Whitesides
McQueen, et al
Muscle
 3-4 hours - reversible
changes
6 hours - variable
damage
8 hours - irreversible
changes
Nerve
2 hours - looses nerve
conduction
4 hours - neuropraxia
8 hours - irreversible
Compartment Syndrome- CAUSES
Causes
 Fractures
 Contusions
 Surgery
 Post Ischemic swelling after arterial occlusion
 Major vascular trauma
 Crush injuries
 Burns
 Prolonged limb compression
Causes
Fracture of a long bone
(Supracondylar humerus,
forearm, hand,tibia and
foot)
CAUSES
Drilling &
reaming
Dissection
Tourniquet
CAUSES
Tight cast
swelling
Bluish
discolorationnumbness
CAUSES
Severe bruised muscle
(even if there is no fracture)
Don’t take contusion lightly
Signs and Symptoms
Increased Pressure and Tightness
Progressive pain out of proportion to initial injury
Markedly swollen area
Progressive neurologic deficit
Seven P’s
 Pain
 Pressure
 Pain with passive stretch
 Parethesia
 Paresis/ Paralysis
 Pulses
 Pallor
SYMPTOMS
Severe pain inappropriate
to the injury(not relieved
even with morphia)
SYMPTOMS
Burning of the affected limb
Tight muscle(rigid)tightness feeling
Numbness: bad sign
SIGNS & DIAGNOSIS
Passive stretching of fingers or toes (muscle stretch)will
lead to severe pain (diagnostic sign)
Never wait for signs of ischemia (5 Ps):irreversible
damage
STRECH TEST
It is possible to strech the
affected muscles by
passively moving the
joints in direction
opposite to that of the
damaged muscles,s action
(( e.g. ::: passive
extension of fingers
produces pain in flexor
compartment of forearm
Technique
STRYKER TECHNIQUE
MERCURY MANOMETER
Wick hand held
instrument
Whiteside maneuver Wick hand held instrument
syringe
3 way stopcock
mmhg
mano.
electrode
Direct
reading
Stryker Stic System
Easy to use
Can check multiple compartments
Different areas in one compartment
Complications related to CS
Late Sequelae
 Volkmann’s contracture
 Weak dorsiflexors
 Claw toes
 Sensory loss
 Chronic pain
 Amputation
Management
Non surgical management:
 Remove any tight bandage, tubigrip or soaked dressing
 Cast should be removed completely
 Elevation
•management
Surgical management:
(FASCIOTOMY)
Open skin and fascia
down to a compartment
It is a surgical procedure
where the fascia is cut to
relieve tension or
pressure commonly to
treat the resulting loss of
circulation to the tissue
Fasciotomy Principles
Make early diagnosis
Long extensile incisions
Release all fascial compartments
Preserve neurovascular structures
 Debride necrotic tissues
Coverage within 7-10 days
Compartment Syndrome
Indications for Fasciotomy
Unequivocal clinical findings
Pressure within 15-20 mm hg of DBP
Rising tissue pressure
Significant tissue injury or high risk pt
> 6 hours of total limb ischemia
Injury at high risk of compartment syndrome
CONTRAINDICATION - Missed compartment
syndrome (>24-48 hrs)
TYPES
SINGLE INCISION
DOUBLE INCISION
SINGLE INCISION
Perifibular Fasciotomy
Matsen et al (1980)
Single incision just
posterior to fibula
Common peroneal nerve
DOUBLE INCISION
In most instances it affords better exposure of the
four compartments
2 vertical incisions separated by minimum 8 cm
One incision over anterior and lateral compartments
 Superficial peroneal nerve
One incision located
1-2 cm behind postero
-medial aspect of tibia
 Saphenous nerve and vein
Flexor digitorum
longus
Gastroc-soleus
Superficial peroneal ner
Intermuscular septum
Forearm Fasciotomy
 Volar-Henry approach
Include a carpal tunnel
release
Release lacertus
fibrosus and fascia
Protect median nerve,
brachial artery and
tendons after release
Fasciotomy of Hand
10 separate osteofascial
compartments
dorsal interossei (4)
palmar interossei (3)
thenar and hypothenar (2)
adductor pollicis (1)
Wound Management
Wound is not closed at initial surgery
Second look debridement with consideration for
coverage after 48-72 hrs
Limb should not be at risk for further swelling
Pt should be adequately stabilized
Usually requires skin graft
Primary closure possible if residual swelling is minimal
Flap coverage needed if nerves, vessels, or bone exposed
Goal is to obtain definitive coverage within 7-10 days
Wound Management
After the fasciotomy, a bulky compression dressing and a
splint are applied.
“VAC” (Vacuum Assisted Closure) can be used
 Foot should be placed in neutral to prevent equinus
contracture.
Incision for the fasciotomy usually can be closed after three
to five days
It may need skin graft
Close skin by sutures
after oedema subsides
Complications Related to
Fasciotomies
Altered sensation within the margins of the wound (77%)
Dry, scaly skin (40%)
Pruritus (33%)
Discolored wounds (30%)
Swollen limbs (25%)
Tethered scars (26%)
Recurrent ulceration (13%)
Muscle herniation (13%)
Pain related to the wound (10%)
Tethered tendons (7%)
Fitzgerald, McQueen Br J Plast Surg 2000Fitzgerald, McQueen Br J Plast Surg 2000
Compartment syndrome is a serious syndrome, Which needs
to be diagnosed early.
Palpable pulse doesn’t exclude compartment syndrome
If diagnosis and fasciotomy were done within 24 hrs, the
prognosis is good.
If delayed, complications will develop.
The earlier you diagnose, the safer you are

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Compartment syndrome basics

  • 2. HISTORY Volkmann 1881 Richard von Volkmann published an article in which he attempted to describe the condition of irreversible contractures of the flexor muscles of the hand to ischemic processes occurring in the forearm
  • 3. Hildebrand 1906 First used the term Volkmann ischemic contracture to describe the final result of any untreated compartment syndrome, and was the first to suggest that elevated tissue pressure may be related to ischemic contracture.
  • 4. Thomas 1909 Reviewed the 112 published cases of Volkmann ischemic contracture and found fractures to be the predominant cause. Also, noted that tight bandages, an arterial embolus, or arterial insufficiency could also lead to the problem
  • 5. Murphy 1914 First to suggest that fasciotomy might prevent the contracture. Also, suggested that tissue pressure and fasciotomy were related to the development of contracture
  • 6. What is a compartment? Closed area of muscles group, nerves & blood vessels surrounded by fascia Pressure: 5-15 mmhg
  • 7. DEFINITION Increase in interstitial fluid presuure within an osseofascial compartment of significant magn itude to cause compromise in microcirculation and myoneuralnecrosis
  • 8. What is a compartment syndrome?  intra comp. pressure (35-40 mmhg)  capillaries collapse  Blood flow to muscles and nerves  Bl.Vs collapse
  • 9. Pathophysiology Increased compartment pressure leads to increased venous pressure which decreases A-V gradient resulting in muscle and nerve ischemia.
  • 12. Why is it dangerous? Nerves: neuropraxia: will regenerate Ischemia: cell death Muscles: contracture (Volkmann's ischemic contracture) Gangrene
  • 13. Normal tissue pressure  0-4 mm Hg 8-10 with exertion Absolute pressure theory 30 mm Hg - Mubarak 45 mm Hg - Matsen Pressure gradient theory < 20 mm Hg of diastolic pressure – Whitesides McQueen, et al Muscle  3-4 hours - reversible changes 6 hours - variable damage 8 hours - irreversible changes Nerve 2 hours - looses nerve conduction 4 hours - neuropraxia 8 hours - irreversible
  • 14. Compartment Syndrome- CAUSES Causes  Fractures  Contusions  Surgery  Post Ischemic swelling after arterial occlusion  Major vascular trauma  Crush injuries  Burns  Prolonged limb compression
  • 15. Causes Fracture of a long bone (Supracondylar humerus, forearm, hand,tibia and foot)
  • 16.
  • 20. CAUSES Severe bruised muscle (even if there is no fracture) Don’t take contusion lightly
  • 21.
  • 22. Signs and Symptoms Increased Pressure and Tightness Progressive pain out of proportion to initial injury Markedly swollen area Progressive neurologic deficit Seven P’s  Pain  Pressure  Pain with passive stretch  Parethesia  Paresis/ Paralysis  Pulses  Pallor
  • 23. SYMPTOMS Severe pain inappropriate to the injury(not relieved even with morphia)
  • 24. SYMPTOMS Burning of the affected limb Tight muscle(rigid)tightness feeling Numbness: bad sign
  • 25. SIGNS & DIAGNOSIS Passive stretching of fingers or toes (muscle stretch)will lead to severe pain (diagnostic sign) Never wait for signs of ischemia (5 Ps):irreversible damage
  • 26. STRECH TEST It is possible to strech the affected muscles by passively moving the joints in direction opposite to that of the damaged muscles,s action (( e.g. ::: passive extension of fingers produces pain in flexor compartment of forearm
  • 28. Whiteside maneuver Wick hand held instrument syringe 3 way stopcock mmhg mano. electrode Direct reading
  • 29. Stryker Stic System Easy to use Can check multiple compartments Different areas in one compartment
  • 30. Complications related to CS Late Sequelae  Volkmann’s contracture  Weak dorsiflexors  Claw toes  Sensory loss  Chronic pain  Amputation
  • 31.
  • 32. Management Non surgical management:  Remove any tight bandage, tubigrip or soaked dressing  Cast should be removed completely  Elevation
  • 33. •management Surgical management: (FASCIOTOMY) Open skin and fascia down to a compartment It is a surgical procedure where the fascia is cut to relieve tension or pressure commonly to treat the resulting loss of circulation to the tissue
  • 34. Fasciotomy Principles Make early diagnosis Long extensile incisions Release all fascial compartments Preserve neurovascular structures  Debride necrotic tissues Coverage within 7-10 days
  • 35. Compartment Syndrome Indications for Fasciotomy Unequivocal clinical findings Pressure within 15-20 mm hg of DBP Rising tissue pressure Significant tissue injury or high risk pt > 6 hours of total limb ischemia Injury at high risk of compartment syndrome CONTRAINDICATION - Missed compartment syndrome (>24-48 hrs)
  • 37. SINGLE INCISION Perifibular Fasciotomy Matsen et al (1980) Single incision just posterior to fibula Common peroneal nerve
  • 38. DOUBLE INCISION In most instances it affords better exposure of the four compartments 2 vertical incisions separated by minimum 8 cm One incision over anterior and lateral compartments  Superficial peroneal nerve One incision located 1-2 cm behind postero -medial aspect of tibia  Saphenous nerve and vein
  • 41. Forearm Fasciotomy  Volar-Henry approach Include a carpal tunnel release Release lacertus fibrosus and fascia Protect median nerve, brachial artery and tendons after release
  • 42. Fasciotomy of Hand 10 separate osteofascial compartments dorsal interossei (4) palmar interossei (3) thenar and hypothenar (2) adductor pollicis (1)
  • 43.
  • 44. Wound Management Wound is not closed at initial surgery Second look debridement with consideration for coverage after 48-72 hrs Limb should not be at risk for further swelling Pt should be adequately stabilized Usually requires skin graft Primary closure possible if residual swelling is minimal Flap coverage needed if nerves, vessels, or bone exposed Goal is to obtain definitive coverage within 7-10 days
  • 45. Wound Management After the fasciotomy, a bulky compression dressing and a splint are applied. “VAC” (Vacuum Assisted Closure) can be used  Foot should be placed in neutral to prevent equinus contracture. Incision for the fasciotomy usually can be closed after three to five days
  • 46. It may need skin graft
  • 47. Close skin by sutures after oedema subsides
  • 48. Complications Related to Fasciotomies Altered sensation within the margins of the wound (77%) Dry, scaly skin (40%) Pruritus (33%) Discolored wounds (30%) Swollen limbs (25%) Tethered scars (26%) Recurrent ulceration (13%) Muscle herniation (13%) Pain related to the wound (10%) Tethered tendons (7%) Fitzgerald, McQueen Br J Plast Surg 2000Fitzgerald, McQueen Br J Plast Surg 2000
  • 49. Compartment syndrome is a serious syndrome, Which needs to be diagnosed early. Palpable pulse doesn’t exclude compartment syndrome If diagnosis and fasciotomy were done within 24 hrs, the prognosis is good. If delayed, complications will develop. The earlier you diagnose, the safer you are

Editor's Notes

  1. Seven P’s: Pain- pain in excess of the presenting injury Pressure- affected compartment may be tense to palpation Pain with passive stretch- pain increases with passive ROM Paresthesia- Numbness over the cutaneus distribution of the nerve that runs through the affected compartment Paresis/Paralysis- Ischemia or necrosis of the nerves or muscles develops within the affected compartment Pulses- could be absent but frequently palpable Pallor- skin discoloration may be visible due to impaired venous drainage Co-Morbidities include: Mild: ~ mm weakness ~fatigue ~ Myositis ossificans Severe ~ Severe life threatening vascular compromise ~ decreased arteriovenous gradient **To Remember** ~ Do not elevate limb above hear because it decreases the arteriovenous gradient therefore decreasing blood flow ~ Instead limb should be elevated at heart level to maintain arteriovenous gradient and assist with venous drainage ~ Remove any compression casts or dressings
  2. These are indications for surgical decompression. A missed CS &amp;gt; 24-48 hours should not be opened. (see Rockwood and Green 5th edition and Campbell’s 10 th edition) The damage cannot be reversed and there is a significant infection rate when the dead tissue is exposed to the hospital environment. The surgeon must deal with the residual contractures of the ischemic muscle and not risk the chance of infection. Some have suggested that the scarred ant tib muscle can serve as a check rein and limit foot drop sequelae.
  3. Volar approach use Henry Approach to release superficial and deep flexors flexors followed by the pronators and supinator. The dorsal approach is centered over the proximal forearm and can be used to release the Mobile Wad (BR, ECRL,ECRB). It is important to remember when treating and electrical injury with a compartment syndrome, the most damage is deep along the bone (i.e. the Pro and Sup muscles) and may require debridement at the initial surgery…Do not miss this!