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Anthar Vidradhi

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Anthar Vidradhi

  1. 2. <ul><li>Definition </li></ul><ul><ul><ul><li>“ Dushta rakta atimatratvat sa vai seeghram vidahyate </li></ul></ul></ul><ul><ul><ul><li>tatah seeghra vidahitvat vidradhi ityabhidheeyate l “ </li></ul></ul></ul><ul><ul><ul><li>( Ca. su 17/ 95) </li></ul></ul></ul><ul><ul><ul><li>Due to an excessive vitiation of rakta, abscess gets suppurated quickly. This is called vidradhi because of vidaha. </li></ul></ul></ul>
  2. 3. <ul><li>Bahya and abhyantara. </li></ul><ul><li>Abhyantara vidradhi is further divided into 4 different types: </li></ul><ul><li>Vatiki, paittiki slaishmiki and sannipatiki. </li></ul>
  3. 4. <ul><li>Excessive intake of food which is seetaka,vidahi, ushna, ruksha, viruddha, ajirna, samklishta,vishama and asatmya. </li></ul><ul><li>Intake of vyapanna madya. </li></ul><ul><li>Excessive intake of madya. </li></ul><ul><li>Vegasandharana, srama, jihma vyayama, shayana, atibhara,atyadhwa, atimaidhuna </li></ul>
  4. 5. <ul><ul><ul><li>Antahshareere mamsaasruk aavishanti yadaa malaa …. </li></ul></ul></ul><ul><li>Malas reaching the mamsa and asrk of antahshareera giving rise to granthi which is fatal. </li></ul>
  5. 6. <ul><li>SITES </li></ul><ul><li>Hrudaya </li></ul><ul><li>Kloma </li></ul><ul><li>Yakrut </li></ul><ul><li>Pleeha </li></ul><ul><li>Kukshi </li></ul><ul><li>Vrkka </li></ul><ul><li>Nabhi </li></ul><ul><li>Vankshana </li></ul><ul><li>Vasti </li></ul>
  6. 7. <ul><li>Vidradhi of kloma results in pipasa, mukhasosha and galagraha. </li></ul><ul><li>Vidradhi of kukshi causes sula in kukshi,parsva and amsa. </li></ul>
  7. 8. <ul><li>Sannipathiki maranaaya. </li></ul><ul><li>Chirothitha vidradhi is like sastra, sarpa, vidyut and agni. </li></ul><ul><li>So it should be treated very quickly. </li></ul>
  8. 10. <ul><li>1. Gallstones (30-60 %) </li></ul><ul><li>2. Alcohol (15-30 %) </li></ul><ul><li>3. Hypertriglyceridemia ( 1.3-3.8 %) </li></ul><ul><li>Serum triglyceride> 11.3 mmol/L </li></ul><ul><li>Derangement in lipid metabolism unrelated to pancreas. </li></ul><ul><li>Diabetes mellitus / who are on certain medications also develop high triglyceride levels. </li></ul>
  9. 11. <ul><li>4. ERCP ( Endoscopic Retrograde Cholangio Pancreatography ) 5-20 % </li></ul><ul><li>5. Drug related 2-5 % </li></ul><ul><li>Either by hypersensitivity rection or by generation of a toxic metabolite. </li></ul><ul><li>6. Sphincter of Oddi dysfunction </li></ul><ul><li>7. Trauma especially blunt abdominal trauma </li></ul><ul><li>UNCOMMON CAUSES </li></ul><ul><li>1. Vascular causes and vasculitis. </li></ul><ul><li>2. Connectivse tissue disorders and thrombotic thrombocytopenic purpura( TTP ) </li></ul>
  10. 12. <ul><li>Cancer of pancreas. </li></ul><ul><li>Hypercalcemia. </li></ul><ul><li>Periampullary diverticulum. </li></ul><ul><li>Pancreas divisum. </li></ul><ul><li>Hereditary pancreatitis. </li></ul><ul><li>Cystic fibrosis </li></ul><ul><li>Renal failure </li></ul><ul><li>RARE CAUSES </li></ul><ul><li>Infection ( Mumps, coxsackie virus, cytomegalovirus, parasites) </li></ul><ul><li>Autoimmune ( eg: Sjögren’s Syndrome ) </li></ul>
  11. 13. <ul><li>PANCREATITIS </li></ul><ul><li>Causes: Infection Improperly treated </li></ul><ul><li>Obstruction acute pancreatitis </li></ul><ul><li>Incompletely treated </li></ul><ul><li>acute pancreatitis </li></ul>ACUTE CHRONIC
  12. 14. <ul><li>Inflammation of pancreas </li></ul><ul><li>hypersecretion of pancreatic juice </li></ul><ul><li>Autodigestion of pancretic tissue </li></ul><ul><li>Collection of serous material in the pancretic duct and peritoneal covering </li></ul><ul><li>Septicemia leading to hypovolemic shock </li></ul><ul><li>Acute circulatory failure Coma andDEATH </li></ul>
  13. 15. <ul><li>Obstruction below the level of pancreatic duct </li></ul><ul><li>Stasis of pancretic juice </li></ul><ul><li>Autodigestion of the pancreas </li></ul><ul><li>Collection of serous fluid in the peritoneal sac </li></ul><ul><li>Hypovolemia </li></ul><ul><li>Shock, coma and Death </li></ul>
  14. 16. <ul><li>Abdominal pain </li></ul><ul><li>Burning and pricking type of pain in the epigastric region </li></ul><ul><li>Fever in case of infection </li></ul><ul><li>Obstructive jaundice, itching, clay coloured stools in case of obstruction </li></ul><ul><li>Nausea, vomitting, abdominal distension </li></ul><ul><li>Increased pulse,decreased BP,Tachypnoea </li></ul><ul><li>Cullen’s sign,Turner’s sign </li></ul>
  15. 17. <ul><li>Haemoglobin </li></ul><ul><li>TC ,DC[increases in infection] </li></ul><ul><li>ESR [increases in infection] </li></ul><ul><li>LFT[increases in obstruction] </li></ul><ul><li>Serum amylase ( Normal- within 200 units/dl ) </li></ul><ul><li>USG,CT,MRI,ERCP[in case of chronic pseudopanceatic cyst can be seen] </li></ul><ul><li>In chronic Fbs,Rbs,PPbs increases </li></ul>
  16. 18. <ul><li>Chronic abdominal pain </li></ul><ul><li>Decreased appetite </li></ul><ul><li>Low protein digestion </li></ul><ul><li>Jaundice </li></ul>
  17. 20. <ul><li>HARRISON’S PRINCIPLES OF INTERNAL MEDICINE </li></ul><ul><li>CHARAKA SAMHITA </li></ul><ul><li>CURRENT SURGERY – Dr. J.C.PAL </li></ul><ul><li>CONCISE MEDICAL PHYSIOLOGY- CHAUDHARI </li></ul><ul><li>INTERACTIVE WORKSHOP ON AYURVEDA (HEPATOLOGICAL DISORDERS) </li></ul><ul><li>30 - 31 JAN. 2007,NEW DELHI </li></ul><ul><li>ESSENTIALS OF MEDICAL PHYSIOLOGY - JAYPEE </li></ul>
  18. 21. <ul><li>Dr.SIVA RAMA PRASAD.K </li></ul><ul><li>Dr. KRISHNA KUMAR </li></ul>

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