Tarsal tunnel syndrome is caused by entrapment of the posterior tibial nerve as it passes through the tarsal tunnel behind the ankle. It results in pain, tingling, and numbness in the sole of the foot. Diagnosis is made through physical exam maneuvers like Tinel's test and imaging like MRI or electrodiagnostic testing. Conservative treatment includes splinting, anti-inflammatories, and steroid injections, while surgery to decompress the nerve through tarsal tunnel release is considered if conservative measures fail. Differential diagnoses include other causes of foot pain like Achilles tendinitis, plantar fasciitis, or lumbar nerve impingement.
2. Definition
• Entrapment
neuropathy of
Posterior Tibial Nerve
or its branches as it
passes through tarsal
tunnel deep to the
flexor retinaculum.
• Branches of tibial
nerve may also get
compressed
individually.
3. Anatomy
• Branches of tibial nerve
1. Calcaneal :
Sensory heel in medial & posterior
distribution
2. Medial Plantar :
Sensory medial sole of foot
Motor branch
Abductor hallucis
Flexor Digitorum brevis
3. Lateral Plantar :
Sensory fifth toe
Motor branch
Abductor digiti quinti
Quadratus Plantae
4. Tarsal Tunnel
• Fibro – osseous space
• Postero-medial aspect of ankle
• Passage way for tendons, nerves, vessles
from posterior leg to foot
Borders :
Floor :-
• Concave
• Medial aspect of Tibia, Talus
& Calcaneum.
Roof:-
• Flexor Retinaculum (converts the floor
into a tunnel)
• Obliquely spans between medial malleolus
& medial Tubercle of Calcaneus
• Continous from deep fascia of leg & foot
6. Etiology
EXTRINSIC
• Poor fitting shoes
• Fractures & malunion
• Tarsal coalition
• Post surgical scarring
• Valgus deformity of hindfoot
INTRINSIC
• Inflammotory conditions: tenosynovitis, RA
• Space occupying lesions: lipoma, ganglion, schwanoma, exostosis
• Varicosities
• Neural tumor
• Perineural fibrosis
• Osteophytes
• Double crush syndrome: lumbar spondylosis or PIVD at S1, tibial nerve
more susecptible to compression
Risk Factors:
• DM
• Hypothyroidism
• Gout
• Mucopolysaccharidoses
• Hyperlipidemia
7. Clinical Features
Presenting Symptoms
• Pain in foot & sole – sharp shooting type radiating to distribution of
posterior tibial nerve
• Pain increases on extremes of dorsiflexion & eversion, increses on
walking and relieves on rest.
• Tingling & burning sensation.
• Pain & Paresthesia usually distal to the site of compression, but in
1/3rd population pain may radiate radiate proximally to mid calf.
(Valleix Phenomenon)
8. Examination
Inspection
• Visible foot deformities, scar
• Atrophy of intrinsic foot muscles in late cases
• Long standing cases – neuropathic changes with deformity like pes cavus, atrophic
changes like loss of hair, skin ulceration etc
Palpation
• Tenderness over tarsal tunnel
• Perineural thickening may be palpable in patients with leprosy & amyloid neuropathy.
Percussion
• Tinel’s test : - repeated tapping over posterior tibial nerve cause paresthesisas of the
supplying region
• Light touch & two point discrimination test: may have diminished planter senses in
areas supplioed by either medial or lateral branch
Movements
• Muscle strength and foot range of motion should be checked
• Gait : examining foot deformities
9. Provocative test
• Dorsiflexion- eversion test :
Triple Compression Stress Test
maximal dorsiflexion of ankle and
MTP joint and firm eversion of foot for
sometime produces similar symptoms
• Tourniquet test : increase the
pressure over ankle with tourniquet to
produce similar symptoms
10. Investigations
• X ray – ankle & foot for osteophytes,
anomalies, old trauma.
• MRI – not sensitive for diagnosis, may help
include or exclude other causes like
tendonitis, tenosynovitis, lipoma, ganglion,
varicose veins
• USG – Nerve & its bifurcations, other soft
tissue structures can be evaluated.
• EMG & NCV – best diagnostic modality
Site of compression
Type (sensory/motor)
Cause (axonal/demyelinating)
Neurological changes in muscle
• Sensory nerve conduction studies are more
abnormal than motor.
Hematological:
• HbA1c
• ESR
• BUN
• Creatinine
• Vit B12
13. Treatment
• Medical Management
▫ In absence of demonstrable nerve compression
▫ NSAIDS, oral steriods, neurotropic vitamins
▫ For non responsive patients – local steriod injection in tarsal
tunnel
▫ Correection of plano valgus deformity with physiotherapy or
orthosis
▫ Night splints to maintain plantar flexion and inversion for
symptomatic relief
14. • Surgical Management
Unresponsive patients after 3-6 months or with
demonstrable pathology on MRI or Clinically
deformity is present
Failed conservative treatment :
▫ longer motor nerve conduction latency = 7.4
milliseconds or greater
▫ Greater predominance of foot deformities
15. • Tarsal tunnel release
Incision made posterior to the tibia
Layers that must be released:
➢ flexor retnaculum
➢ Deep investing fascia of lower leg
➢ Superficial & deep fascia of Abductor hallucis
Identify tibial nerve, leave it undisturbed unless there is fibrosis
for which neurolysis is required.
Neuroma or compressing lesion should be identified and removed.
• External neurolysis : of adhesions and scar tissue causing nerve
impingement
• Space occupying masses: specific removal.
• Correction of deformities of foot.