AMERICAN LANGUAGE HUB_Level2_Student'sBook_Answerkey.pdf
Small Ruminant Nutrition Problems and Solutions
1. SMALL RUMINANT NUTRITION AND
THE PROBLEMS THAT ARISE FROM
POOR NUTRITIONAL MANAGEMENT
SUSAN SCHOENIAN (Shay-nē-ŭn)
Sheep & Goat Specialist
Western Maryland Research & Education Center
University of Maryland Extension
sschoen@umd.edu - www.sheepandgoat.com
http://www.slideshare.net/schoenian
2. IMPORTANCE OF NUTRITION
Feed accounts for the majority of the cost
of raising sheep and/goats; in fact, as high
as 70% of total production costs.
Nutrition is the foundation of good health.
Animals on a higher plane of nutrition are
more resistant to many diseases.
Nutritional problems are second only to
respiratory problems in frequency of
occurrence.
Nutrition has a large effect on
reproduction.
3. NUTRIENTS REQUIRED BY ALL SHEEP AND GOATS
Energy
Protein
Minerals
Vitamins
Water
4. ENERGY (CALORIES)
Nutrient required in the greatest quantity.
Is usually the most limiting nutrient.
Comes primarily from carbohydrates and
fats in the diet, but also from excess protein.
Excess energy is stored as fat in the body.
Energy is expressed in different ways:
• TDN - total digestible nutrients, %
• DE - digestible energy, kcal
• ME - metabolizable energy, kcal
• NEp - net energy, kcal
• DOM - digestible organic matter, %
5. IMPROPER FEEDING OF ENERGY TO LIVESTOCK
Not enough energy in diet
Reduced fertility
Reduced growth rates
Reduced milk production
Loss of body condition, reserves
Higher critical temperature
Less cold tolerance
Reduced resistance to disease.
Risk of pregnancy toxemia (ketosis)
Reduced nutrient absorption.
Increases protein requirements
(%) of ration.
Too much energy in diet
Wasteful economically
Over-conditioned (BCS >4.5) animals
have impaired reproduction.
Fat females are more prone to pregnancy
toxemia and dystocia.
Feed efficiency decreases once an
animal has reached its optimal finish.
Excess finish is undesirable to most to
consumers.
6. PROTEIN (AMINO ACIDS)
Required in smaller amounts than energy.
More expensive feed ingredient than energy.
Amount more important than quality.
Excess protein is not stored in the body:
excess protein is broken down and used
as energy.
Some excess protein may be beneficial if parasites
burdens, esp. barber pole worm, are excessive.
Protein is expressed in different ways:
• CP – crude protein
• DP – digestible protein
• MP – metabolizable protein
• DIP – rumen degraded intake protein
• UIP – undegraded intake protein
7. CRUDE PROTEIN (CP): N X 6.26
True or “natural” protein
1. Rumen degraded intake protein (DIP)
Digested in rumen
N ammonia microbial protein
2. Undegradable intake protein (UIP)
(bypass or escape protein)
Not digested in rumen.
Digested postruminally (in small intestines) or
excreted in feces.
Non-protein nitrogen (NPN), e.g. urea
8. BY-PASS PROTEIN
A mix of microbial protein and
bypassed dietary protein is desired
for optimal productivity.
In high-producing animals, it is
recommended that 25-35% of
protein be bypass protein.
20-30 % of the protein in fresh, high
quality forage is bypass protein.
Supplementing with bypass protein
may improve productivity and/or
increase resistance to internal
parasites.
Bypass Protein sources
Low
Soybean meal
Medium
Cottonseed meal
Dehydrated alfalfa
Distiller’s grains
High
Corn gluten meal
Fish meal
9. IMPROPER FEEDING OF PROTEIN TO LIVESTOCK
Not enough protein
Protein deficiency is rare;
most effects are sub-clinical
Impaired reproduction
Reduced growth rates
Reduced hoof and horn growth.
Reduced milk production
Decreased fiber production
Reduced resistance to disease,
especially parasites.
Reduced rumen function
Reduced nutrition absorption
Too much protein
Expensive and inefficient source of energy.
Risk of pizzle rot in males
Can cause metabolic harm
Excess N ammonia UREA (BUN)
Reduces performance, as energy is
required for removal.
Excess protein has been shown to cause
reproductive problems in cattle .
Excess N (in manure) can be an environmental
concern (“a resource out-of-place”)
10. MICRONUTRIENTS
REQUIRED IN SMALL AMOUNTS: OUNCES, GRAMS, INTERNATIONAL UNITS (IU), OR PARTS-PER-MILLION
(PPM).
Minerals
Macrominerals
Calcium (Ca) and phosphorus (P)
Salt: sodium and chloride (NaCl)
Potassium (K)
Magnesium (Mg)
Sulfur (S)
Trace or microminerals
1. Cobalt (Co)
2. Copper (Cu)
3. Fluoride (F)
4. Iron (Fe)
5. Iodine (I)
6. Manganese (Mn)
7. Molybdenum (Mo)
8. Selenium (Se)
9. Zinc (Zn)
Vitamins
Fat-soluble - A – D – E – K
Carotene (green forages) vitamin A
Sunlight Vitamin D
Vitamin E sometimes requires supplementation
Vitamin K synthesized in rumen
Water-soluble - B complex – C
B vitamins synthesized in rumen
Vitamin C synthesized in tissues
11. POSSIBLE MICRONUTRIENT PROBLEMS
Deficiencies
Calcium and phosphorus
rickets
Calcium
Milk fever
Copper
Various, including swayback,
stringy wool,
hair color
Iodine
Goiter
Magnesium
Grass tetany
Excesses
Calcium
milk fever
Copper (esp. sheep)
Chronic or acute toxicity
Selenium toxicosis
Sulfur - polio
Imbalances
Copper, molybdenum, and sulfur
Copper toxicity or deficiency
Calcium and phosphorus
Urinary calculi
Salt
Lower feed and water
consumption
Selenium and Vitamin E
White muscle disease
Various other problems
Thiamin (B2)
Polioencephamalacia
Zinc
Wool picking, skin problems,
hoof problems
12. IMPORTANT NUTRITION INTERACTIONS
Calcium and phosphorus
Ratio should never go below 1:1
2:1 is ideal for most rations, especially for males
and castrates.
Cobalt and vitamin B12
Copper, molybdenum and sulfur
Molybdenum forms insoluble complex with copper
Sulfur binds with molybdenum
Nitrogen and sulfur
10:1 to 12:1 ratio is ideal
Energy and protein
Excess protein (N) reduces nutrient absorption
and causes intestinal imbalances.
13. NUTRIENT REQUIREMENTS OF SHEEP AND GOATS
Vary by
Species
Genetics (type)
Size (weight)
Body condition
Sex
Age
Stage and level of production
Disease
Environment
14. NUTRIENT REQUIREMENTS VARY BY SPECIES
AND GENETICS
0.50
0.70
0.90
1.10
1.30
1.50
1.70
1.90
2.10
2.30
Angora Boer Local Dairy Lamb
Energy (TDN) requirements, lb/day
66 lb. intact male kids/lamb
0.33
0.33
0.33
0.66
0.09
+
fiber
growth
0.50
0.70
0.90
1.10
1.30
1.50
1.70
1.90
Mature ewe Meat doe Dairy doe Angora doe
Energy (TDN) requirements, lb/day
132 lb. female, maintenance
+
fiber
growth
Source: Nutrient requirements of small ruminants, 2007
16. ENERGY REQUIREMENTS VARY BY STAGE OF PRODUCTION
1.00
1.20
1.40
1.60
1.80
2.00
2.20
2.40
Maintenance Breeding Early gestation Late gestation Lactation
Energy requirements, lb. TDN/day
132-lb. mature non-dairy doe (twins)
1.00
1.50
2.00
2.50
3.00
3.50
Maintenance Breeding Early gestation Late gestation Lactation
Energy requirements, lb. TDN/day
176-lb. mature ewe (twins)
Source: Nutrient requirements of small ruminants, 2007
17. PROTEIN REQUIREMENTS VARY BY STAGE OF PRODUCTION
0.10
0.20
0.30
0.40
0.50
0.60
0.70
0.80
Maintenance Breeding Early gestation Late gestation Lactation
Protein requirements, lb/day
176-lb. mature ewe (twins)
0.10
0.15
0.20
0.25
0.30
0.35
0.40
0.45
0.50
Maintenance Breeding Early
gestation
Late gestation Lactation
Protein requirements, lb/day
132-lb. non-dairy doe (twins)
Source: Nutrient requirements of small ruminants, 2007
18. MINERAL REQUIREMENTS VARY BY STAGE AND
LEVEL OF PRODUCTION
1.0
2.0
3.0
4.0
5.0
6.0
7.0
8.0
9.0
10.0
Maintenance Breeding Early gestation Late gestation Early lactation
Mineral requirements, g/d
176-lb. mature ewe (twins)
1.0
2.0
3.0
4.0
5.0
6.0
7.0
8.0
9.0
10.0
Maintenance Breeding Early gestation Late gestation Early lactation
Mineral requirements, g/d
132-lb non-dairy doe (twins)
Calcium
Phosphorus
Source: Nutrient requirements of small ruminants, 2007
19. NUTRIENT REQUIREMENTS VARY BY
LEVEL OF PRODUCTION
1.60
1.70
1.80
1.90
2.00
2.10
2.20
2.30
2.40
Single Twins Triplets
Energy requirements, lb. TDN/day
132-doe, late gestation
1.60
1.80
2.00
2.20
2.40
2.60
2.80
3.00
3.20
3.40
Single Twins Triplets
Energy requirements, lb. TDN/d
176-lb. mature ewe, late gestation
Source: Nutrient requirements of small ruminants, 2007
20. NUTRIENT REQUIREMENTS VARY BY DESIRED
LEVEL OF PERFORMANCE, E.G. ADG
0.50
0.70
0.90
1.10
1.30
1.50
1.70
No gain 0.11 0.22 0.33 0.44 0.55
Energy requirements, lb. TDN/d
44-lb. intact Boer buckling
0.50
1.00
1.50
2.00
2.50
3.00
0.44 0.55 0.66 0.88
Energy requirements, lb. TDN/d
66-lb. ram lamb, 4 mos. early maturing
Source: Nutrient requirements of small ruminants, 2007
22. ACIDOSIS
ruminal lactic acidosis, grain overload, grain poisoning, engorgement
Develops as a result of animals consuming too much
carbohydrates; primarily grain, sometimes by-
product feed (often accidental consumption).
Results in lowered rumen pH
Can be life threatening
clinical vs. subclinical (which goes unnoticed).
More common in sheep than goats and cattle.
Symptoms: simple indigestion, discomfort, anorexia,
diarrhea, teeth grinding, muscle twitching, laminitis,
and ruminal stasis (and death).
Treatment: depends upon severity of symptoms,
sodium bicarbonate, antacids, antibiotics, IV fluids,
surgery
Prevention: introduce grains slowly to diet, add
dietary buffers, feed ionophores, feed whole grains,
have roughage in diet, free choice sodium
bicarbonate.
23. BLOAT
rumen tympany
Form of indigestion caused by excessive accumulation of gas in
rumen.
Symptoms
Can be life threatening
Distended rumen (left side)
Pain and discomfort
Anxiety
Labored breathing
Staggering and recumbancy
Death
Treatment (depends upon severity)
Emergency rumenotomy
Insertion of rumen trocar
Passage of stomach tube
Administration of anti-foaming agent
(vegetable oil, mineral oil, polaxalene)
Antacid drench
Force animal to burp
24. TYPES OF BLOAT
Frothy
Pasture
Common in
animals
consuming
legume-dominant
pastures
(especially alfalfa
and clover) and
green chop,
cereal grains, and
wet spring grass.
Frothy
Feedlot
Occurs in animals
consuming high
grain diets,
especially finely
ground rations.
Free gas
or dry
bloat
Animal is unable
to eructate.
Often caused by
an obstruction.
Can also be the
result of posture,
e.g. cast.
Abomasal
Occurs in mostly
in artificially
reared lambs and
kids.
Cause not known:
believed to be
caused by a
build-up of
bacteria in the
stomach.
25. PREVENTION OF BLOAT
Pasture bloat
Gradual introduction to pasture
Restrict intake by feeding hay before
grazing
Mixed swards of grass and legumes
Anti-foaming agents
Ionophores
Abomasal
Small meals
Self-feeding
Cold milk
Add yogurt or
probiotics to milk
Vaccination for
enterotoxemia
Feed lot bloat
Gradual introduction of
concentrates to diet
Don’t feed finely ground feeds
Whole grain feeding
Roughage in diet
Anti-foaming agents
Ionophores
26. COPPER TOXICITY
Sheep are most susceptible.
Goats are more susceptible than cattle and pigs
Can be acute or chronic
Toxicity occurs when copper accumulates in liver to exceed
1000 mg Cu/kg DM
Breed differences exist.
Many factors affect copper metabolism.
Copper aborption more important than concentration in
feed; influenced by type of diet and level of Mo, S, Fe, and
to a lesser extent Ca and Zn.
70-75% aborption rate in newborn ruminants
< 10 perent in adults
Copper has many antagonists
27. SOURCES OF COPPER IN DIET
All feeds and forages contain copper.
Sources which provide supplemental
Cu
Minerals formulated for other species.
Complete feeds or supplements formulated for
other species.
Some by-product feeds contain higher levels
of copper or copper antagonists.
Pastures that have been fertilized with poultry
or pig manure.
Footbaths containing copper (copper sulfate)
Water (especially copper pipes)
28. WHAT ABOUT COPPER DEFICIENCY?
More common in goats; can occur in sheep.
Non-specific signs: ill thrift, scouring, rough, dull
hair coat, anemia, and poor performance (milk,
growth, and reproductive)
More specific symptoms: swayback (enzootic
ataxia), poor wool quality (steely or stringy wool),
and loss of hair color.
Copper deficiency should be confirmed with
diagnostic testing.
Copasure® (Animax) and UltraCruz® (Santa
Cruz) boluses are a means to provide
supplemental copper to small ruminants. They
may also have an inhibitory affect on the barber
pole worm.
Producers should consult with a nutritionist and/or
veterinarian before providing supplemental copper
to their animals, especially sheep.
29. ENTEROTOXEMIA
OVEREATING DISEASE, PULPY KIDNEY DISEASE
There are several enterotoxemias (clostridium perfringins)
that can affect small ruminants. Type C and especially D
are most important.
Bacteria are normal inhabitant of gut. They bacteria
proliferate, they produce a toxin.
Disease is usually triggered by change in diet, usually an
increase in the amount of grain, supplement, milk, milk
replacer, and/or grass (feeds that are rich in starch, sugar
and/or protein).
Heavy infestations of tapeworms can predispose lambs to
enterotoxemia.
Death is usually sudden.
Treatment (rarely effective) includes anti-toxin and
antibiotics,
Prevention includes vaccination of females (in late
gestation) and offspring and good feeding management.
30. VACCINATION FOR ENTEROTOXEMIA
It is recommended that all sheep and goats be vaccinated
for clostridial diseases with either the 3, 7, or 8 way
vaccine.
Pregnant females should be vaccinated approximately one
month prior to parturition with the toxoid.
If females have never been vaccinated or their vaccination
status is unknown, they will require two injections,
approximately 3-4 weeks apart, followed by an annual
booster.
Lambs/kids that consume sufficient colostrum will acquire
passive immunity, which will begin to wane at approximately 6
weeks.
Lambs and kids should be vaccinated at approximately 6-8
and 10-12 weeks of age; earlier vaccinations can be given to
offspring from unvaccinated dams or who did not consume
adequate colostrum.
Feeder lambs/kids should receive two CDT vaccinations.
The antitoxin can be given in the event of an outbreak and/or
during periods of high risk.
31. FLOPPY KID SYNDROME (FKS)
fading kid syndrome
First reported in 1987. Cause unknown
Affects kids 3-10 days of age (normal at birth)
They exhibit metabolic acidosis (low blood pH) and have
elevated d-lactate.
Symptoms: depression, weakness, flaccid paralysis, and
distension of abdomen.
Differential diagnosis: enterotoxemia, white muscle disease,
abomasal bloat, and septicemia
Treatment: early detection, remove milk from diet, IV or oral
administration of sodium bicarbonate, supportive care
No prevention
Research showed that milk ingestion plays a central role.
Hand-rearing and feeding bovine colostrum/milk or milk
replacer can prevent disease (Germany, Italy).
32. LISTERIOSIS
circling disease
Central nervous system and digestive system
infection caused by bacteria Listeria
monocytogenes.
Commonly associated with feeding of spoiled
silage; otherwise occurs sporadically.
Small ruminants more susceptible.
Causes neurological symptoms: encephalitis,
depression, anorexia, disorientation, head tilt,
circling, and facial paralysis; can cause abortion
in females.
Differential diagnoses: polio, rabies, pregnancy
toxemia, brain abscess, ear infection, meningeal
worm.
Treatment: early intervention with high doses of
antibiotics (Penicillin G) and supportive care. http://www.shesafarmer.com/blog/sick-goat-saga-listerios
33. MILK FEVER
parturient paresis, hypocalcaemia
Deficiency of calcium (Ca) in bloodstream
Can occur before or after parturition.
Caused by inadequate (or excess intake) of calcium
during late pregnancy
Symptoms: stiff uncoordinated gait, muscling
trembling, weakness, bloat, depressed rumen
motility, recumbancy, hind legs bent behind, and
death.
Differential diagnosis: pregnancy toxemia
Treatment: depends upon progression (severity),
usually IV injection of calcium borogluconate, with
oral or SQ administration of calcium to prevent
relapse. Also treat for pregnancy toxemia.
Prevention: proper amount of calcium in diet (green
leafy legume hay, limestone are good sources of
calcium) minimal handling of pregnant females
34. POLIOENCEPHALOMALACIA
PEM, polio, cerebrocortical necrosis, thiamine deficiency Metabolic disorder with neurological symptoms.
Associated with thiamine status and/or high sulfur
intake.
Thiamine deficiency caused by inadequate
production by rumen or factors that interfere with
action of thiamine.
Sulfur-related PEM due to high sulfur intake
Can occur on pasture, but animals on concentrate
diets
(↓ rumen pH) are most susceptible.
Can also result from prolonged treatment with
Corid® (thiamine inhibitor).
Acute: blindness, star gazing, followed by
recumbency,.
Subacute: separation, stop eating, twitching of ears
and face, head held upright.
Differential diagnosis: pregnancy toxemia,
enterotoxaemia, and listeriosis, rabies, tetanus, CAE,
and plant poisoning.
35. PREGNANCY TOXEMIA
lambing paralysis, twin lamb disease, pregnancy ketosis
Affects females during late pregnancy (1-3 weeks)
Caused by a deficiency of energy (TDN) during late
pregnancy (often indicative of a flock/herd nutrition
problem).
Females carrying multiple fetuses are most prone; fat
females also susceptible.
During early stages of disease, can treat with oral propylene
glycol; it may also be prudent to induce parturition (Rx).
Treatment of advanced cases is usually less rewarding and
may require an emergency caesarian section.
Differential diagnosis: milk fever
Prevent with good feeding management
BCS’s of >2.5 during late gestation
Grain feeding during late gestation to meet increased
energy requirements, especially for multi-bearing females.
36. DIARRHEA (SCOURS)
Increased frequency, fluidity, or volume of fecal excretion
Many causes: bacterial, viral, parasites, diet, and
stress.
Accounted for 46% of lamb losses in study at US
Sheep Experiment Station.
In younger lambs/kids, e. coli is most common
cause of scours.
In older lambs/kids, GI parasites, especially
coccidia, are most common causes of diarrhea.
In adults, most scours are self-limiting.
Often a symptom of other illness, e.g. acidosis,
enterotoxemia, Johne’s disease, and plant toxins.
Treatment depends upon cause: anti-diarrheal
medications, anti-coccidial medications, antibiotics,
anthelmintics, probiotics, and electrolytes
(hydration).
37. URINARY CALCULI
urolithiasis, water belly, stones
Common metabolic disease of male sheep
and goats, especially wethers.
Calculi stones, usually phosphate salts,
lodge in urinary tract and prevent urination
Primary cause of urinary calculi is high
grain-low roughage diets, with an imbalance
of calcium to phosphorus.
Rations high in phosphorus and magnesium
Stones can also be composed of other
minerals, depending upon diet (e.g. calcium
rich diets).
Affected animals strain to pass urine and
stand with arched back.
Treatment depends upon location of
obstruction and progression of symptoms:
ammonium chloride drench, smooth muscle
relaxants, surgery.
38. PREVENTION OF URINARY CALCULI
Calcium to phosphorus ratio of at least 2:1;
up to 4:1, not less than 1:1
Roughages as a part of all small ruminant diets.
- to increase saliva production
+ alfalfa is a good source of calcium
Adequate water intake
- clean, constant source of water
Feed additives
Limestone is a good source of calcium
Salt to increase water intake and urine production
Ammonium chloride to acidify urine
Castration alternatives
Production: do you really need to castrate?
Pets: later castration (by vet, under anesthesia)
39. WHITE MUSCLE DISEASE
WMD, stiff lamb disease, nutritional muscular hypertrophy
Degenerative muscle disease of large animals.
Caused by a deficiency of selenium and/or Vitamin E
Can affect skeletal or cardiac muscles (sudden death, poor
response to Tx); can also cause symptoms of ill thrift and
reproductive losses.
Most common in young lambs/kids: newborns and fast-
growing, kids more than lambs
Causes
Inadequate dietary supply of selenium and/or vitamin E
Feeding of poor quality hay; lack of access to pasture
Treatment
Supplemental selenium and/or vitamin E
Prevention
Adequate selenium and vitamin E in diet, especially pregnant
females, as selenium crosses placenta (vitamin E does not)
Feed balanced rations, force feed minerals
Injections for at-risk animals (poor alternative to proper diet)
40. RECOMMENDED RESOURCES
Nutrient Requirements of Small ruminants:
Sheep, Goats, Cervids, and New World
Camelids, National Research Council 2007