Cardiovascular assessment

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Cardiovascular assessment

  1. 1. CARDIOVASCULAR ASSESSMENT D. Safaa eidJ. Borrero 9/10 1
  2. 2. LECTURE OBJECTIVES1. Review anatomy & physiology of the cardiovascular system.2. Describe physical assessment of cardiovascular status.3. Review diagnostic procedures 2
  3. 3. CARDIOVASCULAR EXAMINATIONPart I: Assessment of cardiovascular functionHealth historyPhysical assessmentInspectionPalpationPercussionAuscultationLaboratory testCardiac enzymeLipid profileCoagulation studies
  4. 4. • Part II: Assessment of cardiovascular structure• Diagnostic studies• ECG• Echocardiography• X- ray• The exercise stress• Cardiac catheterization
  5. 5. Anatomy & PhysiologyFunctions of the heart & CV system• Pumps blood to tissues to supply O2 & nutrients• Remove CO2 & metabolic wastes 5
  6. 6. Circulation in the Heart1. Oxygen-poor blood(shown in blue) flows fromthe body into the right atrium.2..Blood flows through theright atrium into the rightventricle.3. The right ventricle pumpsthe blood to the lungs, wherethe blood releases wastegases and picks up oxygen.
  7. 7. 3. The newly oxygen-rich blood (shown in red) returns to the heart and enters the left atrium.4. Blood flows throughthe left atrium into the leftventricle.5. The left ventriclepumps the oxygen-richblood to all parts of thebody.
  8. 8. Coronary Circulation
  9. 9. Coronary Blood Flow
  10. 10. Valves of the Heart• Tricuspid – Directs the flow of blood from the right atrium to the left ventricle.• Mitral Valve – Directs the flow of blood from the left atrium to the left ventricle.• Pulmonic (semilunar) – Lies between the right ventricle and the pulmonary artery.• Aortic Valve (semilunar) – Lies between the left ventricle and the aortic artery.
  11. 11. Part I: Assessment of cardiovascular function physical assessment
  12. 12. 1. Health history• a- Socio - cultural history: -• Age, sex, occupation, educational level, marital status• b- Patient history:-• Past medical history , Past surgical history• C-Family History• d- Psychosocial Profile
  13. 13. - Symptom Analysis• 1- Chest Pain• - Location: - Substernal, pericardial diffuse, localized• - Radiation: -Radiates to jaw, arm, neck• - Character: - Dull, aching, pressure, burning tightness, crushing• - Intensity: - Mild, moderate, severe• - Onset: - Sudden, gradual• - Duration: - 1 -10, more than 15 min, or continuous• - Precipitating factors: - exercise, motion, eating• - Relieving factors: - rest, walking, warmth, drugs• - Accompanying symptoms : -dyspnea, restlessness, sweating, vomiting, cough, syncope, fatigue
  14. 14. Pain Assessment Techniques• The patients self-reported pain is often measured by using pain scales• Numeric Pain Intensity Scale uses a 0-10 scale to assess the degree of pain. Simple Description Intensity Scale, uses such words as "mild", "moderate", and "severe" to describe the patients pain intensity.
  15. 15. • Visual Analog Scale (VAS) requires patients to mark a point on a 10 cm horizontal or vertical line to indicate their pain intensity, with• 0 indicating "no pain“• and 10 indicating "the worst possible pain".
  16. 16. Substernal or 5-15min Usually related Rest,retrosternal pain to exertion, nitroglycerin,spreading across emotion, eating, oxygenchest; may radiate to coldinside of arm, neck,or Angina Pectoris jaw
  17. 17. MI Substernal pain or pain >15 Occurs Morphine over precordium; may min spontaneo sulfate, spread widely usly but successful throughout chest. Pain may be reperfusion in shoulders and handsMyocardial Infarction sequela to of blockedAngina Pectoris may be present. unstable coronary angina artery
  18. 18. Esophageal Pain Substernal pain; 5–60 Recumbency, Food, antacid. may be projected min cold liquids, Nitro-glycerin around chest toAngina Pectoris exercise. relieves Spasm. shoulders. May occur Spontaneously .
  19. 19. anxietyPain over chest; may 2–3 min Stress, Removal ofbe variable. Does not emotional stimulus, radiate. Patient may tachypnea relaxation complain of numbness andtingling of hands and mouth.
  20. 20. • 2- Palpitations• 3- Syncope• Syncopal attacks (dizziness) are another symptom that may signal cardiovascular problems.• 4- Edema• Edema may be seen with right-sided CHF and vascular disease.
  21. 21. • Pitting edema is a depression in the skin from pressure.• To demonstrate the presence of pitting edema, the nurse presses firmly with his or her thumb over a bony surface• The severity of edema is described on a five-point scale, from none (0) to very marked (4).
  22. 22. • 1+ Mild pitting, slight indentation, no perceptible swelling of the leg• 2+ Moderate pitting, indentation subsides rapidly• 3+ Deep pitting, indentation remains for a short time, leg looks swollen• 4+ Very deep pitting, indentation lasts a long time, leg is very swollen
  23. 23. • 5- Fatigue• fatigue is associated with cardiovascular disease.• 6- Extremity Changes• Changes in the extremities may provide clues about underlying cardiovascular disease. Symptoms such as Paresthesia (numbness, tingling), coolness, and intermittent claudication (pain in calves during ambulation) may be associated with vascular disease, coronary heart disease, or cerebral vascular disease.
  24. 24. • 7- Dyspnea and Cough• Dyspnea may also occur with cardiac disease such as left-sided CHF.
  25. 25. B- Physical assessment• General Appearance• Vital Signs• Height and Weight
  26. 26. Inspection and palpation1-Skin• Color• Turgor• Temperature and moisture:-
  27. 27. • 2- Nails• Nails should be assessed for color, shape, thickness, symmetry, and adherence.• Normal nail color is some variation of pink• Nail thickness generally is 0.3 to 0.65 mm, but it may be thicker in men
  28. 28. • Nail abnormalities:-• -Peripheral vascular disease can produce nail depression,• Clubbing
  29. 29. • -Clubbing of the fingers is associated with decreased oxygen.• In clubbing, the distal tips of the fingers become bulbous, the nails are thickened hard, and curved at the tip, and the nail bed feels boggy when squeezed.• - Separation from the nail bed produces a white, yellowish, or greenish color on the non-adherent portion of the nail.
  30. 30. • Capillary refill time:• is a quickly test to assess the adequacy of circulation in an individual with poor cardiac output. An area of skin is pressed firmly by (say) a fingertip until it becomes white; the number of seconds for the area to turn pink again indicates capillary refill time. Normal capillary refill takes around 2 seconds.
  31. 31. 2- Inspection and palpation• 1- Inspection of neck
  32. 32. • Inspecting the carotid artery and jugular venous system• With the patient in a supine position, inspect the carotid and jugular venous systems in the neck for pulsations.• To visualize external venous pulsations, look for pulsations in the supraclavicular area.
  33. 33. • To visualize internal venous pulsations, look for pulsations at the suprasternal notch.• Using a penlight to cast a shadow on the neck vessels may help you visualize the pulsations• Carotids have visible pulsation, jugulars have undulated wave.
  34. 34. • Carotids not affected by respirations, jugulars are.• Carotids not affected by position, jugulars normally only visible when client is supine.• Large, bounding visible pulsation in neck of at suprasternal notch: HTN, aortic stenosis,.
  35. 35. Measuring Jugular Venous Pressure• -Position patient with the head of bed at 30 to 45-degree angle.• - Place a ruler vertically, perpendicular to the chest at the angle of Louis (sternal angle).
  36. 36. • -identify the highest level of the jugular vein pulsation; if unable to see pulsations, use the highest level of jugular vein distension.• - Place another ruler horizontally at the point of the highest level of the venous pulsation.
  37. 37. • - Measure the distance up from the chest wall.• The normal JVP is less than 3 cm. A central venous pressure can be estimated by adding 5 cm to the JVP
  38. 38. • Elevated JVP: Right-sided CHF, constrictive pericarditis, tricuspid stenosis, or superior vena cava obstruction.• Low JVP: Hypovolemia.
  39. 39. Palpation• Palpating the Carotid
  40. 40. • -Lightly palpate each carotid separately.• - Note rate, rhythm, amplitude, contour, symmetry, elasticity, thrills.
  41. 41. Palpating the Jugulars• Palpate jugular veins and check direction of fill.• Occluding under the jaw, the jugular should flatten, but the wave form become more prominent.• Occluding above the clavicle, the jugular normally distends
  42. 42. • Palpating the Precordium• - Identify and palpate each cardiac site for pulsations, and thrills:• - Apex (left ventricular area), or mitral area fifth intercostals space, midclavicular line.
  43. 43. • - Base right (aortic area), second intercostals space right sternal border.
  44. 44. • - LLSB (tricuspid area), fourth to fifth intercostal space at left sternal border.
  45. 45. • - Base left (pulmonic area), second intercostal space left sternal border. - Listen at each site with both the bell and the diaphragm.
  46. 46. - Listen at each site with both the bell and the diaphragm.
  47. 47. PALPATION• Impulses - finger pads• Thrills (vibrations palpated secondary to a murmur—turbulent blood flow through a heart valve) - Bony part of hand, ball of hand
  48. 48. • Thrills are palpable vibrations created by turbulent blood flow.• Lifts or heaves are diffuse, lifting impulses.• A thrust is a rocking movement.
  49. 49. AUSCULTATION• Diaphragm – medium and high frequency sounds• Bell – low frequency sounds• Normally hear closure of valve Sounds from left side of heart louder than equivalent sounds from right side of heart
  50. 50. • S1 – closure of mitral and tricuspid valves• S2 – closure of aortic and pulmonic valves• Low pitched sounds S3, S4, mitral stenosis
  51. 51. • Right 2nd intercostal space Aortic Area• Left 2nd intercostal space Pulmonic Area• Left lower sternal border Tricuspid area• Apex – over apical impulse Mitral area
  52. 52. Landmarks
  53. 53. • the aortic and pulmonic areas are correlated anatomically with the base of the heart.
  54. 54. • S3 (also called a ventricular gallop) may be heard in the tricuspid and mitral areas during the early to mid-diastole following the S2 sound.• S3 is heard well when the client is in the left lateral recumbent position,
  55. 55. • S4 (also called atrial diastolic gallop) may be heard in the tricuspid and mitral areas during the late phase of diastole, before S1 of the next cardiac cycle.• S4 is heard well when the client is in the supine position
  56. 56. Auscultating the Precordium• Auscultate at apex.• - Note rate, rhythm, extra sounds, or murmurs.• - Note S1, S2, extra sounds, or murmurs.• - Listen at each site with both the bell and the diaphragm.
  57. 57. Murmurs and Stenosis…• A valve that does not close efficiently, results in the backflow of blood (i.e., insufficiency or regurgitation).• A valve that does not open wide enough may cause turbulent backflow secondary to obstruction or narrowing (i.e., stenosis).
  58. 58. Abnormal finding• Irregular rhythm: Arrhythmia.• Accentuated S1: High-output states, mitral or tricuspid stenosis.• Diminished S1: First-degree heart block, CHF, CAD.• -Variable S1: Atrial fibrillation.• S3, low-pitched, early diastolic sound: CHF.• S4, low-pitched late-diastolic sound: CAD, HTN, MI.
  59. 59. Ejection fraction (EF)• The ejection fraction (EF) represents the amount of blood pumped out of the heart (left ventricle) with each beat. In the healthy heart, it is around 70%.• An EF below 55% is considered abnormal.
  60. 60. CARDIAC CYCLEEKG – A 12 lead EKG is a graphic record of the electrical forces produced by the heart 64
  61. 61. Acute Anteroseptal MI
  62. 62. ELECTRODE POSITIONS“LEADS”• Leads measure electrical activity between 2 points• Movement toward ⊕ electrode causes positive deflection• Movement away from ⊕ electrode causes negative deflection 67
  63. 63. ELECTRODE POSITIONSA 12 Lead EKG shows electrical activityfrom 12 different positions in the heart,concentrating on (L) ventricleA 14 Lead EKG includes (R) ventricleactivity 68
  64. 64. Cardiac output• SV-• CO-• Preload-• Afterload-• Ejection fraction• GOAL is to maintain adequate MAP so perfusion of oxygenated blood to vital organs occurs 69
  65. 65. Stroke Volume (Sv) & Cardiac Output (Co)• SV – amount of blood ejected by 1 ventricle in 1 beat• CO – volume ejected in 1 minControl of SV and HR = SV&HR are continually adjusted by the body, and are affected by the return of blood from the tissues (think of exercise)CO = SVxHR 70
  66. 66. 71
  67. 67. Decreased S1:♥ Slowed ventricular ejection rate/volume♥ Mitral insufficiency♥ Increased chest wall thickness♥ Pericardial effusion♥ Hypothyroidism11-09 NR 47
  68. 68. Decreased S1 (cont.):♥ Cardiomyopathy♥ LBBB♥ Shock♥ Aortic insufficiency♥ First degree AV block
  69. 69. Other Abnormal S1 (cont.):♥ Increased S1: − Increased cardiac output − Increased A-V valve flow velocity (acquired mitral stenosis, but not congenital MS)♥ Wide splitting of S1: − RBBB (at tricuspid area) − PVC’s − VT
  70. 70. S2:♥ From closure vibrations of aortic and pulmonary valves♥ Often ignored, but it can tell much♥ Divided into A2 and P2 (aortic and pulmonary closure sounds)♥ Best heard at LMSB/2LICS♥ Higher pitched than S1--better heard with diaphragm11-09 NR 47
  71. 71. S2 splitting (normal):♥ Normally split due to different impedance of systemic and pulmonary vascular beds♥ Audible split with > 20 msec difference♥ Split in 2/3 of newborns by 16 hrs. of age, 80% by 48 hours♥ Harder to discern in heart rates > 100 bpm11-09 NR 47
  72. 72. S2 splitting (normal, cont.):♥ Respiratory variation causes ↑ splitting on inspiration: ↓ pulmonary vascular resistance♥ When supine, slight splitting can occur in expiration♥ When upright, S2 usually becomes single with expiration11-09 NR 47
  73. 73. S2 splitting (abnormal):♥ Persistent expiratory splitting − ASD − RBBB − Mild valvar PS − Idiopathic dilation of the PA − WPW11-09 NR 47
  74. 74. S2 splitting (abnormal, cont.):♥ Widely fixed splitting − ASD − RBBB
  75. 75. S2 splitting (abnormal, cont.):♥ Wide /mobile splitting − Mild PS − RVOTO − Large VSD or PDA − Idiopathic PA dilation − Severe MR − RBBB − PVC’s
  76. 76. S2 splitting (abnormal, cont.):♥ Reversed splitting − LBBB − WPW − Paced beats − PVC’s − AS − PDA − LV failure
  77. 77. Single S2:♥ Single S2 occurs with greater impedance to pulmonary flow, P2 closer to A2♥ Single and loud (A2): TGA, extreme ToF, truncus arteriosus♥ Single and loud (P2): pulmonary HTN!!♥ Single and soft: typical ToF♥ Loud (not single) A2: CoA or AI11-09 NR 47
  78. 78. Extra heart sounds11-09 NR 47
  79. 79. S3 (gallop):♥ Usually physiologic♥ Low pitched sound, occurs with rapid filling of ventricles in early diastole♥ Due to sudden intrinsic limitation of longitudinal expansion of ventricular wall♥ Makes Ken-tuck-y rhythm on auscultation11-09 NR 47
  80. 80. S3 (cont.):♥ Best heard with patient supine or in left lateral decubitus♥ Increased by exercise, abdominal pressure, or lifting legs♥ LV S3 heard at apex and RV S3 heard at LLSB11-09 NR 47
  81. 81. S3 (abnormal):♥ Seen with Kawasaki’s disease-- disappears after treatment♥ If prolonged/high pitched/louder: − can be a diastolic flow rumble indicating increased flow volume from atrium to ventricle
  82. 82. S4 (gallop):♥ Nearly always pathologic♥ Can be normal in elderly or athletes♥ Low pitched sound in late diastole♥ Due to elevated LVEDP (poor compliance) causing vibrations in stiff ventricular myocardium as it fills♥ Makes “Ten-nes-see” rhythm11-09 NR 47
  83. 83. S4 (cont.):♥ Better heard at the apex or LLSB in the supine or left lateral decubitus position♥ Occurs separate from S3 or as summation gallop (single intense diastolic sound) with S311-09 NR 47
  84. 84. S4 Associations:♥ CHF!!!♥ HCM♥ severe systemic HTN♥ pulmonary HTN♥ Ebstein’s anomaly♥ myocarditis
  85. 85. S4 Associations (cont.):♥ Tricuspid atresia♥ CHB♥ TAPVR♥ CoA♥ AS w/ severe LV disease♥ Kawasaki’s disease
  86. 86. Click:♥ Usually pathologic♥ Snappy, high pitched sound usually in early systole♥ Due to vibrations in the artery distal to a stenotic valve11-09 NR 47
  87. 87. Can be associated with:♥ Valvar aortic stenosis or pulmonary stenosis♥ Truncus arteriosus♥ Pulmonary atresia/VSD♥ Bicuspid aortic valve♥ Mitral valve prolapse (mid-systolic click)♥ Ebstein’s anomaly (can have multiple clicks)11-09 NR 47
  88. 88. Does NOT occur w/ supravalvar or subvalvar AS, or calcific valvar AS.11-09 NR 47
  89. 89. Whoop (sometimes called a honk):♥ Loud, variable intensity, musical sound heard at the apex in late systole♥ Classically associated w/ MVP and MR♥ Seen w/ VSD’s closing w/ an aneurysm, subAS, rarely TR♥ Some whoops evolve to become systolic murmurs11-09 NR 47
  90. 90. Friction rub:♥ Creaking sound heard with pericardial inflammation♥ Classically has 3 components; can have fewer than 3 components♥ Changes with position, louder with inspiration11-09 NR 47
  91. 91. Murmur:♥ Sounds made by turbulence in the heart or blood stream♥ Can be benign (innocent, flow, functional) or pathologic♥ Murmurs are the leading cause for referral for further evaluation♥ Don’t let murmurs distract you from the rest of the exam!!11-09 NR 47
  92. 92. Laboratory tests• Creatine kinase (CK) and its isoenzyme CK-MB• Lactic dehydrogenase• Troponin I• as low-density lipoproteins (LDL) and high-density lipoproteins (HDL).
  93. 93. • Cholesterol (normal level, less than 200 mg/dL)• LDL (normal level, less than 130 mg/dL) • HDL (normal range in men, 35 to 65 mg/dL; in women, 35to 85 mg/dL) have a protective action• Triglycerides (normal range, 40 to 150 mg/dL), composed of free fatty acids and glycerol, are stored in the adipose tissue and are a source of energy
  94. 94. • Coagulation Studies• Partial thromboplastin time (PTT)• Prothrombin time (PT)
  95. 95. • Chest x-ray and fluoroscopy• Electrocardiography
  96. 96. Diagnostic Procedures1. EKG 12 Lead continuous cardiac monitoring holter monitor2. Chest x-ray – detects enlargement of heart & pulmonary congestion 101
  97. 97. Diagnostic procedures3. Echocardiography – ultrasound that reveals size, shape and motion of cardiac structures Evaluates heart wall thickness, valve structure, differentiates murmurs4. TEE – transesophageal echocardiography provides a clearer image because less tissue for sound waves to pass through 102
  98. 98. Diagnostic procedures5. Angiography / cardiac catherization determines coronary lesion size, location, evaluate (L) ventricular function, measures heart pressures6. Exercise tolerance test7. Radionuclide Imaging 103
  99. 99. Lab StudiesCardiac enzymes = enzymes are released when cells are damaged (MI). Enzymes are found in many tissues/muscles, and some are specific to cardiac tissue. 104
  100. 100. Cardiac enzymes = CPK – MB (CK-MB),myoglobin, Troponin In general, the greater the rise in the serum level of an enzyme, the greater the degree or extent of damage to the muscle.LDH
  101. 101. LAB studies2. Electrolytes3. Lipid panel4. CBC5. C – Reactive Protein6. BNP- Human B-Natriuretic Peptide7. Blood coags-PT/PTT/INR 106
  102. 102. Cholesterol Level : AHA Recommendation• Total Cholesterol – < 200 mg/dL • best – 200 – 239 • borderline high – 240 mg/dL and above • 2X risk of CAD
  103. 103. Cholesterol Level : AHA Recommendation• HDL Cholesterol – < 40 mg/dL (men) – < 50 mg/dL (women) – > 60 mg/dL • cardioprotective
  104. 104. Cholesterol Level : AHA Recommendation• LDL Cholesterol – < 100 mg/dL • Optimal – 100 – 129 mg/dL • Near or above optimal – 130 – 159 mg/dL • Borderline – 160 – 189 mg/dL • High – 190 mg/dL • Very high
  105. 105. Cholesterol Level : AHA Recommendation• Triglyceride – < 150 mg/dL • Normal – 150 – 199 mg/dL • Borderline high – 200 – 499mg/dL • High – 500 mg/dL and above • Very high
  106. 106. NCLEX TIMEMary is attending a sophomore level nursing class on anatomy and physiology. Which statement, if made by Mary, demonstrates a good understanding of the anatomy and physiology of the heart?A."The heart is encapsulated by a protective coating called the endocardium.“B."The SA node is considered the main regulator of heart rate.“C."The left atrium receives deoxygenated venous blood from all peripheral tissues.“D."Stroke volume is the amount of blood ejected by the right ventricle during each diastole 111
  107. 107. NCLEX TIMEKirsten is completing her graduate clinical rotation in a large urban teaching hospital in a medical coronary care unit (CCU). Which observation demonstrates a good understanding of completing a thorough cardiac examination?• A. In an obese client, an adult cuff size of 12 to 14 cm is preferable.• B.The carotid artery on the neck is auscultated to assess for the presence of a bruit.• C.The apical impulse is auscultated over the fifth intercostal space in the midclavicular line.• D.Palpation is used to determine cardiac size. 112
  108. 108. NCLEX TIMEEdward is a 40-year-old white male. He is an accountant who works on average 11 hours per day. He reports feeling stressed each day, even with mundane things such as a traffic jam. His father had a massive myocardial infarction at the age of 48. His mother has a history of congestive heart failure. He seldom has time to exercise, but does eat balanced meals when possible, although he does not get to eat three meals a day. Select all factors that place Edward at risk for heart disease.• A.Family history• B.Age• C.Coping-stress tolerance• D.Race• E.Occupation 113

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