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TRACHEAL DISORDERS 
Dr.JINO JUSTIN.J 
RADIOLOGY RESIDENT 
RMMCH
TRACHEA 
The trachea is a cylindrical tube that projects onto the spine 
from C6 to the level of T5. 
As it passes downwards, it follows the curvature of the spine, 
and courses slightly backward. 
Near the tracheal bifurcation, it deviates slightly to the right. 
The subglottis ends 2 cm below the level of the vocal cords. 
This corresponds cranially to the inferior margin of the cricoid 
cartilage, which is the inferior margin of the larynx and forms 
the only complete cartilage ring in the airway.
• 18-22 cartilaginous rings 
• There are 2.1 rings/cm 
• Becomes intrathoracic at 
6th cartilaginous ring 
• Length 9 -17 cm 
• Intrathoracic portion: 6- 
15 cm 
• Cross-section area of 
women about 40% 
less than men.
The membranous posterior membrane allows 
esophageal expansion during deglutition 
Contains 
glands, small 
arteries, 
nerves, lymph 
vessels and 
elastic fibers 
Trachealis 
muscle overlies 
esophageal 
muscle and 
epithelium
Tracheal dimensions 
Trachea 
– Average cross-sectional 
area of the male adult 
trachea is approximately 2.8 
cm2 
– Transverse (lateral) diameter is 
of 25 mm and sagittal diameter 
is of 27 mm are the upper limits 
of normal (males) 
– The lower limit is normal for both 
transverse and sagittal diameters 
& it is about 13 mm in men and 
10 mm in women
Some facts about tracheal anatomy 
 The cervical segment (extrathoracic) ends at the 
sternal manubrium and encompasses about the 
first six tracheal rings. 
 The U-shaped trachea is probably the most 
frequent shape found. 
 A man’s cross sectional tracheal area is usually 
about 40 percent larger than a woman’s.
The trachea is lined by ciliated columnar epithelium. 
The trachea in children is very pliable. It may be 
deviated to the right at almost 90° in a normal 
expiratory film. 
It only deviates to the left if the aortic arch is on the 
right side.
•Relations of the trachea 
•CERVICAL 
The anterior relations are as 
follows: 
•Anterior: 
Isthmus of thyroid anterior to 
the second, third and fourth rings 
Inferior thyroid veins 
Strap muscles: Sternohyoid and 
Sternothyroid 
•Posterior: 
Oesophagus 
Recurrent laryngeal nerves 
Lateral: 
Lobes of thyroid gland 
Common carotid artery.
Thoracic: 
The thoracic relations are as follows: 
• Anterior: 
Brachiocephalic and left common carotid arteries 
Left brachiocephalic vein 
• Posterior: Oesophagus and left recurrent 
laryngeal nerve 
• Left lateral: 
 Arch of the aorta 
Left common carotid and left subclavian arteries 
• Right lateral: Right brachiocephalic artery 
Right vagus nerve 
Arch of the azygos vein 
Pleura (in direct contact unlike the other side).
•BLOOD SUPPLY OF THE TRACHEA 
The upper trachea is supplied by the inferior 
thyroid artery and the lower part is supplied 
by branches of the bronchial artery. 
Venous drainage is to the inferior thyroid 
venous plexus.
Mediastinal lymph nodes that drain the lung are named according to their 
position: 
Tracheobronchial nodes above the tracheobronchial junction 
Right and left paratracheal nodes on either side of the trachea.
The trachea is seen as a midline 
translucency with a slight inclination to the 
right in its lower half. 
Its lumen is 1.5-2 cm in diameter. 
The right paratracheal stripe (normally < 3 
mm) is formed by the right wall of the 
trachea and the pleura, outlined on both 
sides by air. 
The left side of the trachea is not seen 
separately from the mediastinal shadows. 
A smooth indentation on the trachea is 
commonly seen just above the bifurcation on 
the left side. This is caused by the arch of the 
aorta.
RADIOLOGICAL FEATURES OF 
TRACHEA ON LATERAL CHEST 
RADIOGRAPH 
On a lateral chest radiograph the trachea is seen to enter the thorax midway 
between the sternum and the vertebrae. 
Owing to some posterior inclination it ends closer to the vertebrae. 
The posterior paratracheal stripe is formed by the posterior wall of the trachea 
and the pleura and is visible if the lung passes behind the trachea. 
The tracheo-oesophageal stripe is formed by the posterior wall of the trachea 
and the anterior wall of the oesophagus. 
It is visible if there is air in the oesophagus.
Disorders 
• CONGENITAL ABNORMALITIES. 
• TRACHEAL NARROWING. 
• TRACHEAL WIDENING. 
• TRACHEAL AGENESIS. 
• TRACHEAL STENOSIS. 
• TRACHEOMALACIA. 
• Tracheobronchopathia Osteochondroplastica. 
• Neoplasm
CONGENITAL ABNORMALITIES 
• TRACHEOESOPHAGEAL FISTULA. 
– USUALLY OCCURS IN ASSOCIATION WITH ESOPHAGEAL ATRESIA,CAN ALSO 
OCCUR AS AN ISOLATED ANOMALY. 
– MAY ALSO BE ASSOCIATED WITH OTHER ABNORMALITIES LIKE 
– DUE TO FAILED FUSION OF TRACHEO ESOPHAGEAL RIDGES during third week 
of embryological development. 
– Commonly associated with vater complex 
– VERTEBRAL. 
– ANAL. 
– TRACHEO-OESOPHAGEAL. 
– RENAL.
TYPES 
• Type 1-esophageal agenesis{very rare}. 
• Type 2-proximal and distal esophageal bud 
present,with a missing mid-segment. 
• Type 3-3A,3B&3C. 
• 3A-proximal esophageal termination on the 
lower trachea with distal esophageal bud. 
• 3B-proximal esophageal atresia with distal 
end arising from the lower trachea or 
carina.(most common upto 90%of cases). 
• 3C-proximal esophageal termination on the 
lower trachea or carina with distal 
esophagus arising from the carina. 
• Type D-if two segments of esophagus 
communicates ,termed as H type 
fistula(resemblane of the letter H).
TRACHEAL AGENESIS 
• Very rare and commonly associated with 
maternal polyhydramniaos. 
 C/F : Acute severe respiratory distress, absent 
cry, inability to intubate.
• Type 1 – Absent upper 
trachea with lower trachea 
connecting to the 
oesophagus 
• Type 2 – Common bronchus 
connecting right and left 
main bronchi to oesophagus 
with absent trachea 
• Type 3 -Right and left main 
bronchi arises 
independentaly from 
oesophagus
TRACHEAL NARROWING 
• Other wise known as sub-glottic stenosis(just below the vocal cord). 
• In babies and young children subglottis is the narrowest part of the airway 
and most stenosis do occur at this level. 
• CAUSES 
• Fibrosing Mediastinitis 
• Post tracheostomy(most common). 
• Wegeners granulomatosis. 
• Idiopathic progressive sub-glottic stenosis. 
• Amyloidosis. 
• Benign tumors e.gCarcinoid. 
• Tracheal trauma/rupture.
FIBROSING MEDIASTINITIS 
• Usually occurs due to tuberculosis 
and histoplasmosis causing tracheal 
narrowing Fibrosing Mediastinitis – 
Coronal CT Image 
• There is airway narrowing of the right 
lower lobe bronchus. There is 
thickening of the right pleura and 
right interlobular septae. There are 
partially calcified right hilar and 
mediastinal lymph nodes
TRACHEOBRONCHOPATHIA 
OSTEOCHONROPLASTICA 
• rare, benign disease 
• characterized by development of cartilaginous and osseous 
nodules within submucosa of the tracheal and bronchial walls. 
• typical in men > 50yrs 
• C/F - incidental finding mostly. 
- dyspnea, cough, hemoptysis&wheezing 
may be present. 
- Nodules tend to be localized to the submucosa directly 
associated with the tracheal cartilage, sparing the posterior 
tracheal membrane .
• The nodules - 3 - 8 mm in diameter 
usually calcified. 
• irregular than normal cartilage calcification. 
• Similar central bronchial calcification also is 
seen in many patients. 
• No significant decrease in tracheal diameter is 
seen with forced expiration.
CT image at two levels shows nodular 
thickening and calcifi cation of the anterior and 
lateral tracheal wall. The posterior tracheal 
mem-brane is normal in thickness and devoid 
of calcifi cations 
Diagrammatic representation of 
the appearance of the trachea in 
tracheobronchopathia 
osteochondroplastica. Tracheal 
cartilages are thickened, with small 
irregular calcifi c nodules along 
their inner aspect, protruding into 
the tracheal lumen.
nodules arising from the anterior and lateral 
tracheal wall, projecting into the lumen. The 
nodules are uncalcified. 
Bronchoscopy shows nodules 
arising from tracheal 
cartilage. The posterior 
trachea is normal.
Tracheal Stenosis 
• Congenital tracheal stenosis may result from a ring-shaped tracheal 
cartilage. 
• Acquired tracheal stenosis usually is due to prior intubation or 
tracheostomy. 
• Progressive dyspnea following extubation typically is present. 
• Infl ammation and pressure necrosis of the tracheal mucosa most 
commonly occur at either the tracheostomy stoma or at the level of the 
tube balloon, 1 to 1.5 cm proximal to the tube tip; the stenosis usually 
involves 1.5 to 2.5 cm of the tracheal wall. 
• The extrathoracic trachea most often is involved. 
• Focal narrowing may be seen if the tube tip presses on one part of the 
tracheal wall, usually the anterior wall. 
• Acute postintubation stenosis results from edema of the tracheal wall or 
intraluminal granulation tissue. 
.
• Plain films - may show an eccentric or hourglass-shaped 
tracheal nar-rowing. 
• On CT, this may be seen as eccentric or 
concentric soft tissue internal to normal-appearing 
tracheal cartilage. The outer tracheal 
wall has a normal appear-ance, without evidence 
of deformity or narrowing. Dynamic expiratory 
images show little change in tracheal dimensions. 
• Acute and chronic stenosis may also result from 
sarcoidosis, histoplasmosis, Wegener’s 
granulomatosis, and ulcerative colitis.
Postintubation tracheal stenosis due to granulation tissue. 
A: Near the thoracic inlet, the trachea appears normal 
B: Below the level shown in (A), focal narrowing of the tracheal lumen is 
associated with increased soft tissue (white arrows) within the tracheal 
lumen. The calcified tracheal cartilage (black arrows) appears normal, 
without evidence of deformity or collapse.
Coronal reconstruction in a patient with an hourglass-shaped tracheal 
stenosis following intubation. The tracheal wall appears normal (large 
arrows), and granulation tissue is seen narrowing the tracheal lumen (small 
arrows).
Postintubation tracheal stenosis due to stricture 
CT image at two levels shows side-to-side 
narrowing of the tracheal lumen resulting from 
deformity of the tracheal cartilage (arrows).
Postintubation tracheal stenosis due to deformity of tracheal cartilage 
CT shows side-to-side narrowing of the tracheal lumen resulting from deformity of the 
tracheal cartilage (arrows). The tracheal wall is outlined by mediastinal fat. 
B: Coronal reconstruction shows an hourglass-shaped stenosis, with inward collapse of 
the tracheal wall (white arrows). Calcifi ed tracheal cartilage (black arrow) is displaced 
inward. 
C: Three-di-mensional reconstruction shows the hourglass-shaped stenosis.
AMYLOIDOSIS 
• rare 
• Symptoms - hoarseness, stridor, dyspnea, cough, hemoptysis, and 
recurrent infections. 
• Primary tracheobronchial amyloidosis - usually confined to the airways, 
with no evidence of concurrent parenchymal disease. 
• Deposits are multifocal or diffuse 
submucosal in location 
involve the length of the trachea 
main bronchi also are commonly affected. 
• plain radiographs and CT 
diffuse tracheobronchial amyloidosis usually leads to concentric or 
nodular thickening of the tracheal wall 
Calcication or ossification is common. 
Malacia is not present. 
Rarely, a single localized submucosal nodule is present, resulting in 
eccentric wall thickening. 
Multiple isolated lesions also may be seen. 
Atelectasis may be associated with bronchial involvement.
• Tracheobronchial amyloidosis. CTs through the proximal trachea using lung 
(A) and soft tissue (B) windows show eccentric thickening of the tracheal 
wall (arrows)
CT shows nodular thickening and calcifi cation of the walls of the right 
main and right upper lobe bronchi (arrows). D: Coronal reforma-tion 
shows tracheal wall thickening (large arrows) and focal thickening and 
calcifi cation of bronchial walls (small arrows). Lymph node calcifi cation 
also is visible
WEGENER’S GRANULOMATOSIS 
• systemic vasculitis. 
• 90% of cases, serum antineutrophil cytoplasmic antibodies 
characterized by a diffuse granular cytoplasmic immunofl 
uorescent staining pattern (cytoplasmic antineutrophil 
cytoplasmic antibody [C-ANCA]) are present.
• tracheobronchial involvement - 15% to 25% of cases; 
• symptoms - hoarseness, cough, and stridor. 
• Subglottic tracheal involvement is most typical 
• variable involvement of the vocal cords, distal trachea, and 
proximal main stem bronchi. 
• Abnormalities may be focal or diffuse. 
• Pathologic findings - 
circumferential airway wall thickening and inflammation, 
concentric narrowing of the tracheal lumen; 
mucosal ulceration and destruction of the cricoid or 
tracheal cartilage are less common. 
• Plain radiographs - tracheal narrowing on both the frontal 
and lateral radiographs; this narrowing may be localized or 
diffuse.
CT findings 
• focal or circumferential thickening of the tracheal wall 
• an increase in the overall tracheal diameter due to wall thickening 
• narrowing of the tracheal lumen 
• Malacia may be present 
• Proximal bronchi may be involved
• Subglottic tracheal stenosis in Wegener’s 
granulomatosis. A: Excessive soft tissue (arrows) is 
visible internal to the cricoid cartilage. B: Coronal 
reconstruction in subglottic stenosis due to Wegener’s 
granulomatosis. The focal narrow-ing (large arrows) is 
just below the level of the vocal cords (small arrows).
• Tracheal and bronchial narrowing in Wegener’s granulomatosis. A: Chest 
radiograph shows bilateral lung nodules (arrows)..B: CT shows tracheal narrowing 
associated with concentric thickening of the tracheal wall (arrows).
Tracheal and bronchial narrowing in Wegener’s 
granulomatosisC: At the level of the carina, 
bronchial wall thickening (arrow) also is seen
• Tracheomalacia in Wegener’s granulomatosis. A: CT image on 
inspiration shows the tracheal lumen to be slightly reduced in 
diameter. B: CT during dynamic forced expiration shows marked 
reduction in the tracheal lumen.
• Bronchial narrowing in Wegener’s granulomatosis. A: CT shows narrowing 
of the proxi-mal left main bronchus, associated with thickening of its wall 
(arrows). B: Three-dimensional recon-struction shows left main bronchus 
narrowing (arrows). The distal trachea is slightly narrowed.
SABER SHEATH TRACHEA 
• almost always is associated with chronic obstructive 
pulmonary disease. 
• characterized by marked decrease in the coronal diameter 
of the intrathoracic trachea associated with an increase in 
its sagittal diameter 
• extrathoracic trachea is normal. 
• may involve the entire intrathoracic trachea 
• earliest stages it is visible only at the thoracic inlet. 
• thought to be due to chronic injury and malacia of tracheal 
cartilage due to coughing or increased intrathoracic 
pressure. 
• The main bronchi are of normal size.
• On frontal radiographs 
a characteristic side-to-side narrowing of the tracheal lumen is visible 
beginning at the thoracic inlet 
The right paratracheal stripe, primarily representing tracheal wall, 
appears normal or slightly increased in thickness. 
• On the lateral radiograph 
the tracheal diameter appears normal or slightly increased. 
tracheal diameter on the lateral fi lm measures 1.5 times that seen on 
frontal fi lm, saber sheath trachea is considered to be present
CT 
• inward displacement of the lateral portions of the tracheal 
wall and cartilage with side-to side narrowing of the lumen 
• tracheal wall usually normal thickness. 
• During forced expiration - further inward bowing of the 
tracheal walls in many patients.
Saber-sheath trachea in a patient with chronic obstructive 
pulmonary disease 
• PA chest radiograph - hourglass-shaped narrowing of the intrathoracic trachea 
(black arrows). The extrathoracic trachea (white arrows) appears normal. 
• lateral projection- the tracheal diameter appears increased (arrows).
SABER SHEATH –TRACHEA ON CT 
• The extrathoracic trachea is normal. 
• tracheal cartilage is calcified
• The intrathoracic trachea at two levels is markedly narrowed from 
side to side (arrows), with deformity of the tracheal cartilage. 
• The sagittal tracheal diameter is increased. 
• The tracheal wall is otherwise normal in appearance.
RELAPSING POLYCHONDRITIS 
• rare systemic disorder 
• characterized by recurrent episodes of cartilage inflammation 
• most commonly affecting the ear, nose, joints, and the 
laryngeal and tracheal cartilage 
• The upper airways are affected >50% of cases 
• recurrent pneumonia is most common cause of death. 
• Diffuse tracheal involvement, characterized by a dense 
inflammatory exudate, is limited to the cartilage and 
perichondrium 
• does not affect the mucosa or submucosa.
• Plain radiographs 
cylindrical narrowing of the extrathoracic and intrathoracic trachea and main 
bronchi 
• CT 
thickening of the anterior and lateral tracheal wall 
the posterior membrane is of normal thickness 
inner and outer margins of the thickened tracheal walls are smooth in contour. 
Collapse of tracheal cartilage may be seen in chronic disease. 
Narrowing of both the tracheal lumen and the main bronchi is often present. 
Tracheomalacia is often present
Relapsing polychondritis. Chest radiograph 
shows cylindrical narrowing of the entire 
trachea (arrows).
Relapsing polychondritis. The anterior and lateral tracheal walls (i.e., the 
cartilaginous portions) are thickened (large arrows). The posterior tracheal 
membrane is of normal thickness (small arrow). This appearance is 
characteristic. B: Narrowing of the main bronchi also is seen. The anterior 
bronchial walls are thickened (large arrows), while the posterior wall of the 
bronchus appears normal (small arrow)
RELAPSING POLYCHONDRITIS 
CT shows marked narrowing of the tracheal 
lumen, with typical thickening of the anterior 
and lateral tracheal walls.
RELAPSING POLYCHONDRITIS 
Coronal reconstruction shows diffuse 
narrowing of the trachea with thickening of its 
lateral walls (arrows). Sagittal reformation 
shows diffuse narrowing of the trachea 
(arrows)
Relapsing polychondritis with 
tracheomalacia. Expiratory CT 
shows sig-nifi cant collapse of 
the tracheal lumen compared 
with an inspiratory scan .
TRACHEAL DIVERTICULUM 
• focal herniation of tracheal mucosa through the tracheal 
wall. 
• may be seen in normal subjects, although it tends to be 
associated with chronic obstructive pulmonary disease. 
• usually is asymptomatic and is detected incidentally. 
• almost always occurs near the thoracic inlet, along the 
posterolateral right trachea, between the cartilaginous and 
muscular portions of the tracheal wall 
• Can appear as an isolated paratracheal air cyst , usually a 
few millimeters in diameter, or as an airfilled structure 
communicating with the tracheal lumen 
• Tracheal diverticulum is easily seen on CT, but is rarely 
visible on plain radiographs.
Heal diverticulum in a patient with chronic 
obstructive pulmonary disease. A defect in the 
right posterolateral tracheal wall (small arrow) 
communicates with a small diverticulum (large 
arrow).
A paratracheal air cyst is 
present in the upper 
mediastinum, representing a 
diverticulum (arrow). 
A defect in the right 
posterolateral tracheal wall 
(arrow) communicates with 
the diverticulum.
MOUNIER-KUHN SYNDROME 
(TRACHEOBRONCHOMEGALY) 
• It is Common in men in third and 
fourth decades 
Tracheobronchomegaly refers to 
patients who have marked dilatation 
of the trachea and mainstem bronchi. 
• It is often associated with tracheal 
diverticulosis, recurrent lower 
respiratory tract infection and 
bronchiectasis. 
• Atrophy affects the elastic and 
muscular elements of both the 
cartilaginous and membranous parts 
of the trachea. The diagnosis is based 
on radiological findings. The 
immediate subglottic trachea has a 
normal diameter, but it expands as it 
passes to the carina and this 
dilatation often continues into the 
major bronchi.
Primary malignant Tracheal Tumors 
• Most common (85% of cases) 
Squamous cell carcinoma 
Adenoid cystic carcinoma 
• Rare 
Other types of bronchogenic carcinoma 
Carcinoid tumor 
Sarcoma 
Lymphoma 
Metastatic 
• Direct invasion most common 
Thyroid carcinoma 
Laryngeal cancer 
Lung cancer 
Esophageal cancer 
• Hematogenous metastases 
Melanoma 
Breast carcinoma 
Colon carcinoma 
Kidney carcinoma 
Benign 
Squamous cell papilloma 
Papillomatosis 
Hamartoma 
Mesenchymal tumors
TUMOURS 
• Benign 
-It present as small, well defined intraluminal 
nodules 
-papilloma, 
fibroma,chondroma,haemangioma
• Malignant 
-Usually occurs close to the carina 
-Mostly squamous,adenoid cystic or 
adenocarcinoma 
-Extraluminal best assessed by CT finding
Squamous 
cell 
carcinoma 
• associated with smoking 
• multifocal in 10% of cases 
• often involving the distal trachea; a 
main bronchus also may be involved
Adenoid cystic carcinoma 
• originates from tracheal mucous glands 
• most common in the upper trachea 
• less common than squamous cell carcinoma. 
• often arises from the posterolateral tracheal wall
Adenoid cystic carcinoma of the proximal tra-chea. 
An eccentric narrowing (arrows) of the 
tracheal lumen is caused by a sessile mass 
arising from the right tracheal wall.
• Adenoid cystic carcinoma of the 
trachea. A large mass (large 
arrows) in the upper trachea 
markedly narrows the tracheal 
lumen (small arrow) and invades 
the medi-astinum 
• On a sagittal 
reconstruction, the mass 
(arrows) can be seen to 
arise from the posterior 
tracheal wall. This location 
is typical of adenoid cystic 
carcinoma.
On CT 
• a primary malignant tracheal tumor may appear as polypoid lesion, a focal sessile 
lesion, eccentric narrow-ing of the tracheal lumen, or circumferential wall 
thickening 
• Attachment to the tracheal wall may be either broad based or narrow and 
pedunculated. 
• CT may underestimate the longitudinal extent of the tumor; submucosal spread 
may be difficult to see on CT. 
• However, CT is superior to bronchoscopy in evaluating extraluminal spread and the 
trachea distal to an obstructing lesion.
Adenoid cystic carcinoma results in a sessile 
mass (M) arising from the posterior tracheal 
wall and protruding into the tracheal lumen. 
The mass extends into the adjacent 
mediastinum (arrows). 
CT appearances of primary 
tracheal tumor. A: Tracheal 
malignancies may appear 
polypoid, sessile, or 
circumferential
Metastases 
• Metastases to the trachea may occur via direct extension or by 
hematogenous spread. 
• Direct extension to involve the trachea most often is secondary to a 
primary tumor of the lung, larynx, esophagus, or thyroid. 
• These tumors may compress the trachea, displacing tracheal cartilage 
inward, or may invade the tracheal lumen, with tumor being seen as 
abnormal tissue internal to tracheal cartilage . 
• Hematogenous metastases usually originate from melanoma, or from 
carcinomas of the breast, colon, or kidney. 
• On CT, hematogenous metastases may appear as single or multiple, sessile 
or pedunculated endotracheal lesions
Tracheal metastasis. Tracheal metastases may result in 
tracheal compression with inward displacement of the 
tracheal wall, an endotracheal mass, or a combination of 
these findings. A, B: There is narrowing of the trachea with 
an endoluminal mass (arrow) 
Soft-tissue window at the same 
level as (B) shows a mass 
involving the right tracheal wall 
and mediastinal soft tissues 
(arrows)
Squamous cell papilloma 
• most common benign tracheal tumor. 
• It represents an abnormal proliferation of squamous 
epithelium 
• may appear sessile, papillary, lobulated, or polypoid. 
• Solitary papilloma is associated with smoking and is most 
common in adults. 
• The condition of mul-tiple papillomas (i.e., papillomatosis) 
usually begins in child-hood with laryngeal involvement and is 
associated with human papillomavirus infection.
• On CT, a solitary papilloma appears 
as a well-circumscribed nodule that 
is confined to the tracheal wall and 
projects into the tracheal lumen; 
• it often shows acute angles where it 
contacts the tracheal wall. 
• Tracheal cartilage is unaffected. 
• Papillomatosis is characterized by 
numerous nodules involving the 
entire length of the trachea or 
diffuse thickening of the tracheal 
wall .
• Other benign tracheal tumors include 
hamartoma and tumors of mesenchymal 
origin such as lipoma or chondroma.
Summary 
• CT is currently the primary noninvasive examination to evaluate the 
trachea because it offers multiplanar capabilities, evaluates the 
morphology of the tracheal wall and lumen, and can be acquired in 
seconds. 
• The trachea is supported by C-shaped cartilaginous rings anteriorly; the 
posterior trachea is primarily supported by the thin trachealis muscle. 
• The lunate-shaped trachea on inspiration is highly suggestive of 
tracheomalacia. 
• Traditionally, collapse of greater than 50% of the trachea during expiration 
was defined as tracheomalacia; however, recent evidence suggests that 
greater than 50% dynamic expiratory collapse can be seen in healthy 
patients. 
• In saber-sheath trachea, only the coronal diameter of the intrathoracic 
trachea is narrowed; primarily men with chronic obstructive pulmonary 
disease are affected.
• The posterior wall of the trachea and the central bronchi are classically 
spared in both relapsing polychondritis and tracheobronchopathia 
osteochondroplastica. However, the presence of focal coarse calcification 
and ossification is highly suggestive of tracheobronchopathia 
osteochondroplastica rather than relapsing polychondritis. 
• Nodular calcification of the trachea is common in tracheobronchopathia 
osteochondroplastica and amyloidosis. However, amyloidosis tends to 
involve the airway concentrically, as opposed to tracheobronchopathia 
osteochondroplastica which spares the posterior wall. 
• Wegener granulomatosis most often affects the subglottic trachea but can 
be diffuse or multifocal. 
• Mounier-Kuhn syndrome is unique among the diffuse tracheal diseases in 
that it results in diffuse airway dilatation. Diverticula project between the 
cartilaginous rings giving the trachea and proximal bronchi a corrugated 
appearance.
Thank You!
BOYs, C U Again in ‘IMAGING IN 
GYNAEC!’

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Tracheal pathologies

  • 1. TRACHEAL DISORDERS Dr.JINO JUSTIN.J RADIOLOGY RESIDENT RMMCH
  • 2. TRACHEA The trachea is a cylindrical tube that projects onto the spine from C6 to the level of T5. As it passes downwards, it follows the curvature of the spine, and courses slightly backward. Near the tracheal bifurcation, it deviates slightly to the right. The subglottis ends 2 cm below the level of the vocal cords. This corresponds cranially to the inferior margin of the cricoid cartilage, which is the inferior margin of the larynx and forms the only complete cartilage ring in the airway.
  • 3. • 18-22 cartilaginous rings • There are 2.1 rings/cm • Becomes intrathoracic at 6th cartilaginous ring • Length 9 -17 cm • Intrathoracic portion: 6- 15 cm • Cross-section area of women about 40% less than men.
  • 4. The membranous posterior membrane allows esophageal expansion during deglutition Contains glands, small arteries, nerves, lymph vessels and elastic fibers Trachealis muscle overlies esophageal muscle and epithelium
  • 5. Tracheal dimensions Trachea – Average cross-sectional area of the male adult trachea is approximately 2.8 cm2 – Transverse (lateral) diameter is of 25 mm and sagittal diameter is of 27 mm are the upper limits of normal (males) – The lower limit is normal for both transverse and sagittal diameters & it is about 13 mm in men and 10 mm in women
  • 6. Some facts about tracheal anatomy  The cervical segment (extrathoracic) ends at the sternal manubrium and encompasses about the first six tracheal rings.  The U-shaped trachea is probably the most frequent shape found.  A man’s cross sectional tracheal area is usually about 40 percent larger than a woman’s.
  • 7. The trachea is lined by ciliated columnar epithelium. The trachea in children is very pliable. It may be deviated to the right at almost 90° in a normal expiratory film. It only deviates to the left if the aortic arch is on the right side.
  • 8. •Relations of the trachea •CERVICAL The anterior relations are as follows: •Anterior: Isthmus of thyroid anterior to the second, third and fourth rings Inferior thyroid veins Strap muscles: Sternohyoid and Sternothyroid •Posterior: Oesophagus Recurrent laryngeal nerves Lateral: Lobes of thyroid gland Common carotid artery.
  • 9. Thoracic: The thoracic relations are as follows: • Anterior: Brachiocephalic and left common carotid arteries Left brachiocephalic vein • Posterior: Oesophagus and left recurrent laryngeal nerve • Left lateral:  Arch of the aorta Left common carotid and left subclavian arteries • Right lateral: Right brachiocephalic artery Right vagus nerve Arch of the azygos vein Pleura (in direct contact unlike the other side).
  • 10. •BLOOD SUPPLY OF THE TRACHEA The upper trachea is supplied by the inferior thyroid artery and the lower part is supplied by branches of the bronchial artery. Venous drainage is to the inferior thyroid venous plexus.
  • 11. Mediastinal lymph nodes that drain the lung are named according to their position: Tracheobronchial nodes above the tracheobronchial junction Right and left paratracheal nodes on either side of the trachea.
  • 12. The trachea is seen as a midline translucency with a slight inclination to the right in its lower half. Its lumen is 1.5-2 cm in diameter. The right paratracheal stripe (normally < 3 mm) is formed by the right wall of the trachea and the pleura, outlined on both sides by air. The left side of the trachea is not seen separately from the mediastinal shadows. A smooth indentation on the trachea is commonly seen just above the bifurcation on the left side. This is caused by the arch of the aorta.
  • 13. RADIOLOGICAL FEATURES OF TRACHEA ON LATERAL CHEST RADIOGRAPH On a lateral chest radiograph the trachea is seen to enter the thorax midway between the sternum and the vertebrae. Owing to some posterior inclination it ends closer to the vertebrae. The posterior paratracheal stripe is formed by the posterior wall of the trachea and the pleura and is visible if the lung passes behind the trachea. The tracheo-oesophageal stripe is formed by the posterior wall of the trachea and the anterior wall of the oesophagus. It is visible if there is air in the oesophagus.
  • 14. Disorders • CONGENITAL ABNORMALITIES. • TRACHEAL NARROWING. • TRACHEAL WIDENING. • TRACHEAL AGENESIS. • TRACHEAL STENOSIS. • TRACHEOMALACIA. • Tracheobronchopathia Osteochondroplastica. • Neoplasm
  • 15. CONGENITAL ABNORMALITIES • TRACHEOESOPHAGEAL FISTULA. – USUALLY OCCURS IN ASSOCIATION WITH ESOPHAGEAL ATRESIA,CAN ALSO OCCUR AS AN ISOLATED ANOMALY. – MAY ALSO BE ASSOCIATED WITH OTHER ABNORMALITIES LIKE – DUE TO FAILED FUSION OF TRACHEO ESOPHAGEAL RIDGES during third week of embryological development. – Commonly associated with vater complex – VERTEBRAL. – ANAL. – TRACHEO-OESOPHAGEAL. – RENAL.
  • 16. TYPES • Type 1-esophageal agenesis{very rare}. • Type 2-proximal and distal esophageal bud present,with a missing mid-segment. • Type 3-3A,3B&3C. • 3A-proximal esophageal termination on the lower trachea with distal esophageal bud. • 3B-proximal esophageal atresia with distal end arising from the lower trachea or carina.(most common upto 90%of cases). • 3C-proximal esophageal termination on the lower trachea or carina with distal esophagus arising from the carina. • Type D-if two segments of esophagus communicates ,termed as H type fistula(resemblane of the letter H).
  • 17.
  • 18. TRACHEAL AGENESIS • Very rare and commonly associated with maternal polyhydramniaos.  C/F : Acute severe respiratory distress, absent cry, inability to intubate.
  • 19. • Type 1 – Absent upper trachea with lower trachea connecting to the oesophagus • Type 2 – Common bronchus connecting right and left main bronchi to oesophagus with absent trachea • Type 3 -Right and left main bronchi arises independentaly from oesophagus
  • 20. TRACHEAL NARROWING • Other wise known as sub-glottic stenosis(just below the vocal cord). • In babies and young children subglottis is the narrowest part of the airway and most stenosis do occur at this level. • CAUSES • Fibrosing Mediastinitis • Post tracheostomy(most common). • Wegeners granulomatosis. • Idiopathic progressive sub-glottic stenosis. • Amyloidosis. • Benign tumors e.gCarcinoid. • Tracheal trauma/rupture.
  • 21. FIBROSING MEDIASTINITIS • Usually occurs due to tuberculosis and histoplasmosis causing tracheal narrowing Fibrosing Mediastinitis – Coronal CT Image • There is airway narrowing of the right lower lobe bronchus. There is thickening of the right pleura and right interlobular septae. There are partially calcified right hilar and mediastinal lymph nodes
  • 22. TRACHEOBRONCHOPATHIA OSTEOCHONROPLASTICA • rare, benign disease • characterized by development of cartilaginous and osseous nodules within submucosa of the tracheal and bronchial walls. • typical in men > 50yrs • C/F - incidental finding mostly. - dyspnea, cough, hemoptysis&wheezing may be present. - Nodules tend to be localized to the submucosa directly associated with the tracheal cartilage, sparing the posterior tracheal membrane .
  • 23. • The nodules - 3 - 8 mm in diameter usually calcified. • irregular than normal cartilage calcification. • Similar central bronchial calcification also is seen in many patients. • No significant decrease in tracheal diameter is seen with forced expiration.
  • 24. CT image at two levels shows nodular thickening and calcifi cation of the anterior and lateral tracheal wall. The posterior tracheal mem-brane is normal in thickness and devoid of calcifi cations Diagrammatic representation of the appearance of the trachea in tracheobronchopathia osteochondroplastica. Tracheal cartilages are thickened, with small irregular calcifi c nodules along their inner aspect, protruding into the tracheal lumen.
  • 25. nodules arising from the anterior and lateral tracheal wall, projecting into the lumen. The nodules are uncalcified. Bronchoscopy shows nodules arising from tracheal cartilage. The posterior trachea is normal.
  • 26. Tracheal Stenosis • Congenital tracheal stenosis may result from a ring-shaped tracheal cartilage. • Acquired tracheal stenosis usually is due to prior intubation or tracheostomy. • Progressive dyspnea following extubation typically is present. • Infl ammation and pressure necrosis of the tracheal mucosa most commonly occur at either the tracheostomy stoma or at the level of the tube balloon, 1 to 1.5 cm proximal to the tube tip; the stenosis usually involves 1.5 to 2.5 cm of the tracheal wall. • The extrathoracic trachea most often is involved. • Focal narrowing may be seen if the tube tip presses on one part of the tracheal wall, usually the anterior wall. • Acute postintubation stenosis results from edema of the tracheal wall or intraluminal granulation tissue. .
  • 27. • Plain films - may show an eccentric or hourglass-shaped tracheal nar-rowing. • On CT, this may be seen as eccentric or concentric soft tissue internal to normal-appearing tracheal cartilage. The outer tracheal wall has a normal appear-ance, without evidence of deformity or narrowing. Dynamic expiratory images show little change in tracheal dimensions. • Acute and chronic stenosis may also result from sarcoidosis, histoplasmosis, Wegener’s granulomatosis, and ulcerative colitis.
  • 28. Postintubation tracheal stenosis due to granulation tissue. A: Near the thoracic inlet, the trachea appears normal B: Below the level shown in (A), focal narrowing of the tracheal lumen is associated with increased soft tissue (white arrows) within the tracheal lumen. The calcified tracheal cartilage (black arrows) appears normal, without evidence of deformity or collapse.
  • 29. Coronal reconstruction in a patient with an hourglass-shaped tracheal stenosis following intubation. The tracheal wall appears normal (large arrows), and granulation tissue is seen narrowing the tracheal lumen (small arrows).
  • 30. Postintubation tracheal stenosis due to stricture CT image at two levels shows side-to-side narrowing of the tracheal lumen resulting from deformity of the tracheal cartilage (arrows).
  • 31. Postintubation tracheal stenosis due to deformity of tracheal cartilage CT shows side-to-side narrowing of the tracheal lumen resulting from deformity of the tracheal cartilage (arrows). The tracheal wall is outlined by mediastinal fat. B: Coronal reconstruction shows an hourglass-shaped stenosis, with inward collapse of the tracheal wall (white arrows). Calcifi ed tracheal cartilage (black arrow) is displaced inward. C: Three-di-mensional reconstruction shows the hourglass-shaped stenosis.
  • 32. AMYLOIDOSIS • rare • Symptoms - hoarseness, stridor, dyspnea, cough, hemoptysis, and recurrent infections. • Primary tracheobronchial amyloidosis - usually confined to the airways, with no evidence of concurrent parenchymal disease. • Deposits are multifocal or diffuse submucosal in location involve the length of the trachea main bronchi also are commonly affected. • plain radiographs and CT diffuse tracheobronchial amyloidosis usually leads to concentric or nodular thickening of the tracheal wall Calcication or ossification is common. Malacia is not present. Rarely, a single localized submucosal nodule is present, resulting in eccentric wall thickening. Multiple isolated lesions also may be seen. Atelectasis may be associated with bronchial involvement.
  • 33. • Tracheobronchial amyloidosis. CTs through the proximal trachea using lung (A) and soft tissue (B) windows show eccentric thickening of the tracheal wall (arrows)
  • 34. CT shows nodular thickening and calcifi cation of the walls of the right main and right upper lobe bronchi (arrows). D: Coronal reforma-tion shows tracheal wall thickening (large arrows) and focal thickening and calcifi cation of bronchial walls (small arrows). Lymph node calcifi cation also is visible
  • 35. WEGENER’S GRANULOMATOSIS • systemic vasculitis. • 90% of cases, serum antineutrophil cytoplasmic antibodies characterized by a diffuse granular cytoplasmic immunofl uorescent staining pattern (cytoplasmic antineutrophil cytoplasmic antibody [C-ANCA]) are present.
  • 36. • tracheobronchial involvement - 15% to 25% of cases; • symptoms - hoarseness, cough, and stridor. • Subglottic tracheal involvement is most typical • variable involvement of the vocal cords, distal trachea, and proximal main stem bronchi. • Abnormalities may be focal or diffuse. • Pathologic findings - circumferential airway wall thickening and inflammation, concentric narrowing of the tracheal lumen; mucosal ulceration and destruction of the cricoid or tracheal cartilage are less common. • Plain radiographs - tracheal narrowing on both the frontal and lateral radiographs; this narrowing may be localized or diffuse.
  • 37. CT findings • focal or circumferential thickening of the tracheal wall • an increase in the overall tracheal diameter due to wall thickening • narrowing of the tracheal lumen • Malacia may be present • Proximal bronchi may be involved
  • 38. • Subglottic tracheal stenosis in Wegener’s granulomatosis. A: Excessive soft tissue (arrows) is visible internal to the cricoid cartilage. B: Coronal reconstruction in subglottic stenosis due to Wegener’s granulomatosis. The focal narrow-ing (large arrows) is just below the level of the vocal cords (small arrows).
  • 39. • Tracheal and bronchial narrowing in Wegener’s granulomatosis. A: Chest radiograph shows bilateral lung nodules (arrows)..B: CT shows tracheal narrowing associated with concentric thickening of the tracheal wall (arrows).
  • 40. Tracheal and bronchial narrowing in Wegener’s granulomatosisC: At the level of the carina, bronchial wall thickening (arrow) also is seen
  • 41. • Tracheomalacia in Wegener’s granulomatosis. A: CT image on inspiration shows the tracheal lumen to be slightly reduced in diameter. B: CT during dynamic forced expiration shows marked reduction in the tracheal lumen.
  • 42. • Bronchial narrowing in Wegener’s granulomatosis. A: CT shows narrowing of the proxi-mal left main bronchus, associated with thickening of its wall (arrows). B: Three-dimensional recon-struction shows left main bronchus narrowing (arrows). The distal trachea is slightly narrowed.
  • 43. SABER SHEATH TRACHEA • almost always is associated with chronic obstructive pulmonary disease. • characterized by marked decrease in the coronal diameter of the intrathoracic trachea associated with an increase in its sagittal diameter • extrathoracic trachea is normal. • may involve the entire intrathoracic trachea • earliest stages it is visible only at the thoracic inlet. • thought to be due to chronic injury and malacia of tracheal cartilage due to coughing or increased intrathoracic pressure. • The main bronchi are of normal size.
  • 44. • On frontal radiographs a characteristic side-to-side narrowing of the tracheal lumen is visible beginning at the thoracic inlet The right paratracheal stripe, primarily representing tracheal wall, appears normal or slightly increased in thickness. • On the lateral radiograph the tracheal diameter appears normal or slightly increased. tracheal diameter on the lateral fi lm measures 1.5 times that seen on frontal fi lm, saber sheath trachea is considered to be present
  • 45. CT • inward displacement of the lateral portions of the tracheal wall and cartilage with side-to side narrowing of the lumen • tracheal wall usually normal thickness. • During forced expiration - further inward bowing of the tracheal walls in many patients.
  • 46. Saber-sheath trachea in a patient with chronic obstructive pulmonary disease • PA chest radiograph - hourglass-shaped narrowing of the intrathoracic trachea (black arrows). The extrathoracic trachea (white arrows) appears normal. • lateral projection- the tracheal diameter appears increased (arrows).
  • 47. SABER SHEATH –TRACHEA ON CT • The extrathoracic trachea is normal. • tracheal cartilage is calcified
  • 48. • The intrathoracic trachea at two levels is markedly narrowed from side to side (arrows), with deformity of the tracheal cartilage. • The sagittal tracheal diameter is increased. • The tracheal wall is otherwise normal in appearance.
  • 49. RELAPSING POLYCHONDRITIS • rare systemic disorder • characterized by recurrent episodes of cartilage inflammation • most commonly affecting the ear, nose, joints, and the laryngeal and tracheal cartilage • The upper airways are affected >50% of cases • recurrent pneumonia is most common cause of death. • Diffuse tracheal involvement, characterized by a dense inflammatory exudate, is limited to the cartilage and perichondrium • does not affect the mucosa or submucosa.
  • 50. • Plain radiographs cylindrical narrowing of the extrathoracic and intrathoracic trachea and main bronchi • CT thickening of the anterior and lateral tracheal wall the posterior membrane is of normal thickness inner and outer margins of the thickened tracheal walls are smooth in contour. Collapse of tracheal cartilage may be seen in chronic disease. Narrowing of both the tracheal lumen and the main bronchi is often present. Tracheomalacia is often present
  • 51. Relapsing polychondritis. Chest radiograph shows cylindrical narrowing of the entire trachea (arrows).
  • 52. Relapsing polychondritis. The anterior and lateral tracheal walls (i.e., the cartilaginous portions) are thickened (large arrows). The posterior tracheal membrane is of normal thickness (small arrow). This appearance is characteristic. B: Narrowing of the main bronchi also is seen. The anterior bronchial walls are thickened (large arrows), while the posterior wall of the bronchus appears normal (small arrow)
  • 53. RELAPSING POLYCHONDRITIS CT shows marked narrowing of the tracheal lumen, with typical thickening of the anterior and lateral tracheal walls.
  • 54. RELAPSING POLYCHONDRITIS Coronal reconstruction shows diffuse narrowing of the trachea with thickening of its lateral walls (arrows). Sagittal reformation shows diffuse narrowing of the trachea (arrows)
  • 55. Relapsing polychondritis with tracheomalacia. Expiratory CT shows sig-nifi cant collapse of the tracheal lumen compared with an inspiratory scan .
  • 56. TRACHEAL DIVERTICULUM • focal herniation of tracheal mucosa through the tracheal wall. • may be seen in normal subjects, although it tends to be associated with chronic obstructive pulmonary disease. • usually is asymptomatic and is detected incidentally. • almost always occurs near the thoracic inlet, along the posterolateral right trachea, between the cartilaginous and muscular portions of the tracheal wall • Can appear as an isolated paratracheal air cyst , usually a few millimeters in diameter, or as an airfilled structure communicating with the tracheal lumen • Tracheal diverticulum is easily seen on CT, but is rarely visible on plain radiographs.
  • 57. Heal diverticulum in a patient with chronic obstructive pulmonary disease. A defect in the right posterolateral tracheal wall (small arrow) communicates with a small diverticulum (large arrow).
  • 58. A paratracheal air cyst is present in the upper mediastinum, representing a diverticulum (arrow). A defect in the right posterolateral tracheal wall (arrow) communicates with the diverticulum.
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  • 60. MOUNIER-KUHN SYNDROME (TRACHEOBRONCHOMEGALY) • It is Common in men in third and fourth decades Tracheobronchomegaly refers to patients who have marked dilatation of the trachea and mainstem bronchi. • It is often associated with tracheal diverticulosis, recurrent lower respiratory tract infection and bronchiectasis. • Atrophy affects the elastic and muscular elements of both the cartilaginous and membranous parts of the trachea. The diagnosis is based on radiological findings. The immediate subglottic trachea has a normal diameter, but it expands as it passes to the carina and this dilatation often continues into the major bronchi.
  • 61. Primary malignant Tracheal Tumors • Most common (85% of cases) Squamous cell carcinoma Adenoid cystic carcinoma • Rare Other types of bronchogenic carcinoma Carcinoid tumor Sarcoma Lymphoma Metastatic • Direct invasion most common Thyroid carcinoma Laryngeal cancer Lung cancer Esophageal cancer • Hematogenous metastases Melanoma Breast carcinoma Colon carcinoma Kidney carcinoma Benign Squamous cell papilloma Papillomatosis Hamartoma Mesenchymal tumors
  • 62. TUMOURS • Benign -It present as small, well defined intraluminal nodules -papilloma, fibroma,chondroma,haemangioma
  • 63. • Malignant -Usually occurs close to the carina -Mostly squamous,adenoid cystic or adenocarcinoma -Extraluminal best assessed by CT finding
  • 64. Squamous cell carcinoma • associated with smoking • multifocal in 10% of cases • often involving the distal trachea; a main bronchus also may be involved
  • 65. Adenoid cystic carcinoma • originates from tracheal mucous glands • most common in the upper trachea • less common than squamous cell carcinoma. • often arises from the posterolateral tracheal wall
  • 66. Adenoid cystic carcinoma of the proximal tra-chea. An eccentric narrowing (arrows) of the tracheal lumen is caused by a sessile mass arising from the right tracheal wall.
  • 67. • Adenoid cystic carcinoma of the trachea. A large mass (large arrows) in the upper trachea markedly narrows the tracheal lumen (small arrow) and invades the medi-astinum • On a sagittal reconstruction, the mass (arrows) can be seen to arise from the posterior tracheal wall. This location is typical of adenoid cystic carcinoma.
  • 68. On CT • a primary malignant tracheal tumor may appear as polypoid lesion, a focal sessile lesion, eccentric narrow-ing of the tracheal lumen, or circumferential wall thickening • Attachment to the tracheal wall may be either broad based or narrow and pedunculated. • CT may underestimate the longitudinal extent of the tumor; submucosal spread may be difficult to see on CT. • However, CT is superior to bronchoscopy in evaluating extraluminal spread and the trachea distal to an obstructing lesion.
  • 69. Adenoid cystic carcinoma results in a sessile mass (M) arising from the posterior tracheal wall and protruding into the tracheal lumen. The mass extends into the adjacent mediastinum (arrows). CT appearances of primary tracheal tumor. A: Tracheal malignancies may appear polypoid, sessile, or circumferential
  • 70. Metastases • Metastases to the trachea may occur via direct extension or by hematogenous spread. • Direct extension to involve the trachea most often is secondary to a primary tumor of the lung, larynx, esophagus, or thyroid. • These tumors may compress the trachea, displacing tracheal cartilage inward, or may invade the tracheal lumen, with tumor being seen as abnormal tissue internal to tracheal cartilage . • Hematogenous metastases usually originate from melanoma, or from carcinomas of the breast, colon, or kidney. • On CT, hematogenous metastases may appear as single or multiple, sessile or pedunculated endotracheal lesions
  • 71. Tracheal metastasis. Tracheal metastases may result in tracheal compression with inward displacement of the tracheal wall, an endotracheal mass, or a combination of these findings. A, B: There is narrowing of the trachea with an endoluminal mass (arrow) Soft-tissue window at the same level as (B) shows a mass involving the right tracheal wall and mediastinal soft tissues (arrows)
  • 72. Squamous cell papilloma • most common benign tracheal tumor. • It represents an abnormal proliferation of squamous epithelium • may appear sessile, papillary, lobulated, or polypoid. • Solitary papilloma is associated with smoking and is most common in adults. • The condition of mul-tiple papillomas (i.e., papillomatosis) usually begins in child-hood with laryngeal involvement and is associated with human papillomavirus infection.
  • 73. • On CT, a solitary papilloma appears as a well-circumscribed nodule that is confined to the tracheal wall and projects into the tracheal lumen; • it often shows acute angles where it contacts the tracheal wall. • Tracheal cartilage is unaffected. • Papillomatosis is characterized by numerous nodules involving the entire length of the trachea or diffuse thickening of the tracheal wall .
  • 74. • Other benign tracheal tumors include hamartoma and tumors of mesenchymal origin such as lipoma or chondroma.
  • 75. Summary • CT is currently the primary noninvasive examination to evaluate the trachea because it offers multiplanar capabilities, evaluates the morphology of the tracheal wall and lumen, and can be acquired in seconds. • The trachea is supported by C-shaped cartilaginous rings anteriorly; the posterior trachea is primarily supported by the thin trachealis muscle. • The lunate-shaped trachea on inspiration is highly suggestive of tracheomalacia. • Traditionally, collapse of greater than 50% of the trachea during expiration was defined as tracheomalacia; however, recent evidence suggests that greater than 50% dynamic expiratory collapse can be seen in healthy patients. • In saber-sheath trachea, only the coronal diameter of the intrathoracic trachea is narrowed; primarily men with chronic obstructive pulmonary disease are affected.
  • 76. • The posterior wall of the trachea and the central bronchi are classically spared in both relapsing polychondritis and tracheobronchopathia osteochondroplastica. However, the presence of focal coarse calcification and ossification is highly suggestive of tracheobronchopathia osteochondroplastica rather than relapsing polychondritis. • Nodular calcification of the trachea is common in tracheobronchopathia osteochondroplastica and amyloidosis. However, amyloidosis tends to involve the airway concentrically, as opposed to tracheobronchopathia osteochondroplastica which spares the posterior wall. • Wegener granulomatosis most often affects the subglottic trachea but can be diffuse or multifocal. • Mounier-Kuhn syndrome is unique among the diffuse tracheal diseases in that it results in diffuse airway dilatation. Diverticula project between the cartilaginous rings giving the trachea and proximal bronchi a corrugated appearance.
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  • 80. BOYs, C U Again in ‘IMAGING IN GYNAEC!’