12. 1.
Dwaram nirudhya shirsa
OVER ENLARGEMENT OF FETAL
HEAD
2.
Jatharen kaschit
ABDOMEN PRESENTATION Or
TRANSVERSE LIE OR CORD
PRESENTATION
3.
Shareera parivartit kubja deha
Body rotated and presenting with hump
back.
4.
Eka bhuja
Hand prolapse in transverse lie or in
vertex presentation
5.
Bhuja dwayena
Presenting with both hands
6.
Tiryaka gata
Transverse lie without flexion of fetal
body
7.
Kaschit aangmukho anyah
Face presentation
8.
Parshavapvrit gati
12
Presentation with flanks or lateral delivery
18. 1.vata prakopaka ahara vihara ati sevana by
garbhini stri
2.ati maithuna
3.ati jagarana
prakopa of
yoni marga
gata and
garbhashy
astha vayu
death of
garbhini
02-02-2014
closes
yoni
marga
dwara
sva avrodha
of vayu-
YONISAM
VARANA
Excessive pressure of
vata in garbhashya and
yoni patha(birth canal)
as well as association
garbha
between garbha’s nabhi
mrityu
nadi and garbhini’sSharma, PG 2nd Year
Dr.Priya
hridaya nadi
avrodha of
garbhashya
dwara
ati
peeda
na of
garbh
a by
this
vayu
nirudha shwasa of
garbha
18
19. •
Yoni Samvaranam
•
Closure of GARBHASHYA MUKHA
•
गर्ा् कऺौ सक्तो र्वनत
ु
02-02-2014
गर्ा संग
Dr.Priya Sharma, PG 2nd Year
19
20. • वभयु् प्रकवऩत् कयभात ् संरुध्य रुधधरं स्रुतम ्
ु
ु
• सतभयभ हृस्त्त्िरोबस्त्रतमऱम ् मक्कऱसंगयकम ् ll
ू
ू
• मक्कऱो रक्तमभरुतज् िऱ वविेष् Madhukosha tika
ू
Although sushruta has considered shoola in prasoota
stri as makkala, but here, prasava poorva shoola or
pain before labour pain is also taken as MAKKALA
02-02-2014
Dr.Priya Sharma, PG 2nd Year
20
22. Garbhakosha Parasanga
Rupture Uterus
Yoni Samvaranam
Cervical Dystocia
Makkala
Intrapartum haemorrhage with severe pain
OR tetanic or spasmodic or irregular
uterine contractions(tetany of uterus)
Yoni Bhramsha
Uterine prolapse
Yoni Sanga/sankocha
Obstruction of fetus in maternal passage
due to contraction of pelvis.
Sheetangta
Improper functioning of circulatory system
Neelodhita sira
Indicative of loss of physical power
Nirpatrapa or lajjaheenata
Indicative of loss of psychological power
22
23. Akshepaka
Due to toxemia
Kasa , shwasa, bhrama
Due to weakness or HTN
Pooti udgara
Due to ketone bodies formation
Mukashto -harita
Slow progress –prolonged labour
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Dr.Priya Sharma, PG 2nd Year
23
24. • Last two Gatis of Mudhagarbha are Asadhya i.e. –
• Hasta-pada-shirodaya (obstructed labour due to
faulty presentation)
• One foot in yoni & other in anus(remote effects of
undiagnosed obstructed labour)
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Dr.Priya Sharma, PG 2nd Year
24
25. Modern comparision of Asadhya
Mudha Garbha lakshanasOBSTRUCTED LABOUR
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25
26. • Defined as the one in which inspite of good uterine
contractions, the progressive descent of presenting part
is arrested due to mechanical obstruction.
• Incidence – 1-2% in developing countries
• Causes• Fault in Passage
• Fault in Passenger
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Dr.Priya Sharma, PG 2nd Year
26
27. • 1.BONY OBSTRUCTIONS
• 2.SOFT TISSUE OBSTRUCTIONS
• BONY-1.CONTRACTED PELVIS AND CPD are main
causes
• 2.SOFT TISSUE OBSTRUCTIONS- includes cervical
dystocia, cervical or broad ligament fibroid, impacted
ovarian tumour or non graavid horn of bicornuate uterus
below the presenting part.
02-02-2014
Dr.Priya Sharma, PG 2nd Year
27
28. • Transverse lie
• Brow presentation
• Congenital malformation of foetus- hydrocephalus, fetal
ascitis
• Big Baby- occipito posterior position
• Compound presentation
• Locked twins
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28
30. •
•
•
•
•
•
•
Caused by a tear in the wall of the uterus, when
the uterus can’t stand the pressure exerted on it.
Predisposing FactorsVertical scar
Multiple Gestation
Prolonged labor
Obstructed labor
Faulty presentation
Traumatic Maneuvers
Faulty use of oxytocin
02-02-2014
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30
31. • 1. Complete — direct communication
between the uterine and peritoneal cavities.
• 2. Incomplete — rupture into the peritoneum,
covering the uterus or into broad ligament
but not in the peritoneal cavity
• 3. Dehiscence — a partial separation of an old
Scar.
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31
32. – Abdominal pain and tenderness
– Chest pain between the scapula or on inspiration
– Hypovolemic shock caused by hemorrhage
– Signs associated with impaired fetal oxygenation
– Absent fetal heart tones , cessation of uterine
contractions
– Palpation of fetus outside the uterus
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32
35. • Cervical dystocia: Difficult labor and delivery caused by
mechanical obstruction at the cervix.
• Dystocia comes from the Greek "dys" meaning "difficult,
painful, disordered, abnormal" + "tokos" meaning "birth."
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Dr.Priya Sharma, PG 2nd Year
35
36. • Cervical dystocia is nothing but a complication arising
during labor that causes difficulty in delivery because the
cervix is obstructed.
• This abnormal condition of labor is a result of the
ineffectual dilation of the cervix ,though quite a rare
condition, it can lead to serious difficulties to the
mother and the baby.
• A cervical dystocia basically happens at the external os.
The complete cervical canal is consumed, and then often
thinned out. The external os however, remains
incompletely dilated or even closed at times.
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Dr.Priya Sharma, PG 2nd Year
36
37. • 1.Inefficient Uterine Contractions
• 2.Malpresentation, malposition
• 3.Spasm of cervix
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37
38. • Primary
• Secondary
• PRIMARY- commonly observed during the first birth
where the external os fails to dilate.
• Uterine contractions are often ineffective
• Edema of cervix also might occur and delivery may be
accomplished with version of anterior lip.
• SECONDARY Cervical Dystocia- results usually due to
excess scarring or rigidity of cervix from effect of previous
operation or disease.
• Treatment- delivery by cessarian section preferred
02-02-2014
Dr.Priya Sharma, PG 2nd Year
38
39. • Pronounced retraction occurs involving whole of uterus
upto level of internal os.
• So, the physiological differentiation between active upper
segment and passive lower uterine segment of uterus is
lost.
• No thinnig of lower segment of uterus occurs.
• The uterine contraction ceases and the whole uterus
undergoes a sort of tonic muscular spasm holding the
foetus inside.
• Treatment- cs section preferred
02-02-2014
Dr.Priya Sharma, PG 2nd Year
39
40. • Failure to overcome obstruction by powerful contractions
of uterus
• Injudicious use of oxytocics
• CLINICAL FEATURES• Severe continuous pain
• Uterus appears smaller in size , tense and tender on
examination.
• FHS is not audible
• Vaginal examination reveals jammed head with big caput
as well as dry and oedematous vagina.
02-02-2014
Dr.Priya Sharma, PG 2nd Year
40
41. • There is marked hypertrophy and oedema of cervix and
first degree becomes second degree, cystocele and
rectocele become pronounced and there is aggravation
of stress incontinence.
• Vaginal discharge may be copious and decubitus ulcer
may develop when the cervix remains outside the
interoitus.
• Incarceration might occur if uterus fails to rise above the
pelvis by 16th weak of pregnancy.
02-02-2014
Dr.Priya Sharma, PG 2nd Year
41
42. •
•
•
•
•
•
•
•
There are increased chances of1. abortion
2.PROM
3.Intrauterine infection
EFFECTS ON LABOUREarly Rupture of membranes
Cervical dystocia
Prolonged labour due to non dilatation of cervix and
obstruction due to sagging cystocele aand rectocele
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Dr.Priya Sharma, PG 2nd Year
42
43. • Bed rest complete
• Intravaginal plugging soaked with glycerine and
acriflavine
• Prophylactic antibiotics
• Manual stretching of cervix or pushing up of cystocele or
rectocele
• Duhrssen’s incision at 2 and 10 O’ clock positions
followed by ventouse or forceps extraction
• Cessarian section – if cx.is undilated, thick or edematous
and/ or head is high up.
02-02-2014
Dr.Priya Sharma, PG 2nd Year
43
44. • Alteration in size and /or shape of pelvis of sufficient
degree so as to alter the normal mechanism of labour in
an average size baby.
• Causes• Nutritional and environmental defects
• Diseases or injuries affecting bones of pelvis like
fractures, tumours, kyphosis of spine, scoliosis,
coccygeal deformities etc.
• Developmenatl defects- robert’s pelvis, Naegele’s pelvis
02-02-2014
Dr.Priya Sharma, PG 2nd Year
44
45. •
•
•
•
Increased incidence of EROM
Increased chances of cord prolapse
Cervical dilatation slowed
Increased tendency of prolonged labour and obstructed
labour with features like exhausation, dehydration, keto
acidosis and sepsis
• Increased incidence of operative interfarence, shock ,
PPH and sepsis.
• Increased maternal morbidity and mortality
• Increased fetal mortality and morbidity
02-02-2014
Dr.Priya Sharma, PG 2nd Year
45
47. • Maternal• Immediate- exhaustion
•
•
•
•
•
Neelodhita sira
Dehydration- (Sheetangata)
Metabolic acidosis- (pooti udgara)
Genital sepsis
Injury to genital tract
PPH and shock
• Death occurs due to rupture uterus and sepsis with
metabloic changes
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Dr.Priya Sharma, PG 2nd Year
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48. If patient survives-genito urinary fistula or recto vaginal
fistula
• Variable degree of vaginal atresia
• Secondary amenorrhoea following hysterectomy due to
rupture or Sheehan’s syndrome.
02-02-2014
Dr.Priya Sharma, PG 2nd Year
48
54. • Indications :
• Maternal death in Mudhagarbha Avastha to save the fetus.
• Pre-requisites :
• Fetus has attained full maturity --eÉlqÉMüÉsÉã lÉuÉqÉqÉÉxÉÉÌSMüÉsÉã | (Qû)}
• Maximum Time of Udarapatana :
• Upto 2 ghatis i.e.; 48 mins.
• Site of Incision : Bastidwara
02-02-2014
Dr.Priya Sharma, PG 2nd Year
54
55. •
•
•
•
•
•
•
Apara Patana
Abhyanga
Yoni Sneha, pichu
Vataghna Yogas for 10 days
Sneha pana for 3, 5 or 7 days (depending on Prakruti)
Asava or Arishta pana at night
Pathya-for 4 months
02-02-2014
Dr.Priya Sharma, PG 2nd Year
55