1. Balantidium coli
SOULSBY, E. J. L. 1982. Helminths, Arthropods and Protozoa of
Domesticated Animals. 7th Edition. Bailliere Tindall. Greycoat House,
19 Greycoat Place, London SW. P. SB.
Prepared by:
NOE P. MENDEZ
CENTRAL MINDANAO UNIVERSITY (CMU)
npolomendez@gmail.com
2. Balantidium coli
A parasitic species of ciliate
protozoan that causes the
disease Balantidiasis.
Pathogenic to humans
3. SCIENTIFIC CLASSIFICATION
Domain: Eukaryota
Phylum: Ciliophora (Doflein, 1901)
Class: Kinetofragminophorea (De Puytorac et al., 1974)
Order: Vestibuliferida
Family: Balantiididae
Genus: Balantidium
Species: B. coli (Malmsten, 1857)
8. TRANSMISSION
Fecal-oral route
Eating meat, fruits, and vegetables that have been
contaminated by an infected person or contaminated
by fecal matter from an infected animal
Drinking and washing food with contaminated water
Having poor hygiene habits
10. DIAGNOSIS
Based on the clinical signs
Post mortem evidence of an ulcerative condition of
the large intestine
Presence of very large numbers of B. coli
12. PREVENTION
Do not use human feces as
fertilizer in agriculture.
Wash your hands after going to
the toilet and before meal.
Only drink pure water.
Wash vegetables and cook meat
properly.
Infective B. coli cysts are killed
by heat.
13. EPIDEMIOLOGY
Balantidiasis
common in the Philippines, but it can be
found anywhere in the world
Considered to be rare and occurs in less than
1% of the human population
the problem mostly in developing countries,
where water sources may be contaminated
with swine or human feces
Oval to ellipsoidal
There is a distinct macronucleus and small micronucleus
The cilia are arranged in longitudinal rows
A mouth, or peristome, is situated near the anterior end of the organism
There is a weakly developed cytopharynx
Infection occurs when a host ingests a cyst, which usually happens during the consumption of contaminated water or food.[1][5] Once the cyst is ingested, it passes through the host’s digestive system.[3] While the cyst receives some protection from degradation by the acidic environment of the stomach through the use of its outer wall, it is likely to be destroyed at a pH lower than 5, allowing it to survive easier in the stomachs of malnourished individuals who have less stomach acid.[3][5] Once the cyst reaches the small intestine, trophozoites are produced.[1][3] The trophozoites then colonize the large intestine, where they live in the lumen and feed on the intestinal flora.[1][3] Some trophozoites invade the wall of the colon using proteolytic enzymes and multiply, and some of them return to the lumen.[1][3][5] In the lumen trophozoites may disintegrate or undergo encystation.[1][3] Encystation is triggered by dehydration of the intestinal contents and usually occurs in the distal large intestine, but may also occur outside of the host in feces.[1][3] Now in its mature cyst form, cysts are released into the environment where they can go on to infect a new host.
They die within 15-30 min above 40 degree C, although in a moist environment they may survive one to three days at room temperature.
Especially among those that are in close contact with swine.