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Management in
Multi- ligamentous knee injury
(MLKI)
Dr. Jose Austine
Resident, Dept. of Orthopaedic surgery,
Kasturba Medical College, Mangalore
Moderators
Dr. Atmananda Hegde
Dr. Sunil Murthy
• Knee	dislocation	and	
MLKI
• <0.02%	incidence
• Life	and	limb	
threatening(neuro-
vascular)	complications
• Large-scale	data	for	
comparative	analysis	
and	defining	a	standard	
treatment	protocol	are	
not	available.
• Conservative	vs	operative
• Timing	of	surgery	
• Repair	vs	reconstruction	
• Allograft	vs	autograft	
• Partial	vs	total	repair
• Immediate	vs	delayed	mobilization
Management Controversies
All ‘supported’ by at best Level 3 evidence.
1
2
3
MLKI
Acute Chronic
Acute
MLKI
Popliteal	artery	injury	~	30	% CPN	injury	~	35	%
Acute MLKI- Emergency Mx
• Prompt	reduction	of	dislocation	in	the	ER
• Re-evaluate	the	neurovascular	status
• Splint	the	limb
Ankle brachial index (ABI)
ABI	=	Doppler	systolic	blood	pressure	in	the	injured	limb(A)
Systolic	blood	pressure	in	the	uninjured	upper	limb(B)
ABI	<0.8	or	<0.9	then	angiogram
Revascularisation
of popliteal artery
Temporary
external fixation
Chronic MLKI
• In	chronic	cases,	long-leg	radiographs	
are	recommended	to	evaluate	
alignment.
• Patients	with	chronic	PLC	injuries	and	
varus malalignment	should	be	treated	
with	an	osteotomy	to	correct	
malalignment	prior	to	PLC	
reconstruction.
• Untreated	mal-alignment	will	likely	
cause	the	grafts	to	stretch	over	time	
and	fail.
Surgical vs Non-surgical Mx
• Dedmond et	al	(Meta	analysis	2001)
• Richter	et	al	(Meta	analysis	2002)
• Levy	et	al	(Meta	analysis	2009)
ĂźLiterature	supports	surgical	treatment	and	
postoperative	functional	rehabilitation	of	multi-
ligament	knee	injuries.
ĂźRare	occasions	such	as	advanced	age,	immobility	and	
comorbidities	that	nonsurgical	treatment	can	be	
considered.
Repair vs reconstruction
• Patients	with	repair	of	cruciate	ligaments	had	higher	rates	of	flexion	deficit	>6°,	higher	rates	of	posterior	
instability	and	lower	rates	of	return	to	preinjury	activity	levels.	(Mariani et	al)
• High	reoperation	rates	have	been	reported	in	patients	with	posterolateral	injuries	treated	with	repair.
Ăź Anatomic	reconstruction	of	the	injured	structures	using	biomechanically	validated	techniques	
yield	improved	outcomes.
Ăź In	the	setting	of	multi-ligament	injuries, reconstruction	of	the	torn	ligaments	is	recommended.
Ăź Repair	of	the	collaterals	is	usually	reserved	for	bony	avulsion	injuries.
Timing of surgery
Timing of surgery
• Early	surgery	(<3	week)	has	shown	higher	incidences	of	postoperative	
stiffness	and	a	fixed	flexion	deformity	with	higher	rates	of	manipulation
under	general	anesthesia	as	compared	to	delayed	repair.
• Delayed	repair	has	higher	chances	of	scarring of	soft	tissue	with	more	
difficulty	in	identification	and	navigation	in	the	joint	leading	to	higher	
chances	of	vascular	complications.
• No	conclusive	evidence	is	suggestive	of	an	advantage	offered	by	a	single	
or	a	staged	procedure.
Timing of surgery
• Acute- generally	favoured in	literature
- 3	weeks(	before	scarring	or	necrosis	occurs)
(Engebretsen et	al	,	Mariani et	al	,	Fanelli et	al,	Harner et	al)	
- 6	weeks	regarded	as	acute	by	Levy	et	al,	Laprade et	al
• Chronic – Not	recommended	unless	forced	delay	
(eg- vascular	injury)
Choice of graft- Auto vs Allo
Auto-grafts Allo-grafts
• Tensor	fascia	lata
• Bone	patella	tendon	
bone
• Hamstring
• Quadriceps
• Peroneus	longus
• Tibialis	posterior/	anterior
• Tendoachilles
• Patellar	tendon
• Hamstrings
La Prade
Anatomical mcl
reconstruction
La Prade
Anatomical PLC
reconstruction
Surgical
Principles
Avoiding
tunnel
convergence
Tensioning
sequence
Avoiding tunnel convergence
• Tunnel	convergence	increases	the	risk	of	
reconstruction	graft	failure.
• Potential damage	to	reconstruction	grafts	and	
fixation	devices.	
• Not	having	sufficient	bone	stock	between	the	grafts	
for	fixation	and	graft	incorporation.
Tensioning sequence
Moatshe G,	Laprade et	al
• AL	bundle	of	PCL	at	90°(restore	normal	tibial step	off)
• PM	bundle	of	PCL	in	extension
• FCL	at	20°−30° knee flexion and slight valgus.
• Other PLC	structures at	60°of	flexion and neutral rotation.
• ACL	near full extension
• Finally,	Posteromedial corner.
Major Pitfalls
Ø Patient	positioning
Ø Graft	preparation- Prevent	oversizing,	ensure	availability	
Ø Tunnel	convergence
Ø Meniscal	root	injuries- Malposition	of	PCL	and	ACL	tunnel
Ø Neurovascular	complications- when	creating	tibial tunnels
Ø Fixation
Summary
Ø Complex	problem
Ø Assessment	of	vascular	and	neurological	injury	paramount.	
Ø Data	lacking	for	definitive	management	protocols.
Ø Any	intervention	needs	to	be	individualized	by	the	presence	of	any	life- or	limb-threatening	
complication
Ø Early	operative	treatment	yields	improved	functional	and	clinical	outcomes	compared	with	non-
operativemanagement or	delayed	surgery.
Multi ligamentous knee injury

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Multi ligamentous knee injury

  • 1. Management in Multi- ligamentous knee injury (MLKI) Dr. Jose Austine Resident, Dept. of Orthopaedic surgery, Kasturba Medical College, Mangalore Moderators Dr. Atmananda Hegde Dr. Sunil Murthy
  • 2.
  • 3. • Knee dislocation and MLKI • <0.02% incidence • Life and limb threatening(neuro- vascular) complications • Large-scale data for comparative analysis and defining a standard treatment protocol are not available.
  • 4. • Conservative vs operative • Timing of surgery • Repair vs reconstruction • Allograft vs autograft • Partial vs total repair • Immediate vs delayed mobilization Management Controversies All ‘supported’ by at best Level 3 evidence.
  • 5.
  • 10. Acute MLKI- Emergency Mx • Prompt reduction of dislocation in the ER • Re-evaluate the neurovascular status • Splint the limb
  • 11.
  • 12. Ankle brachial index (ABI) ABI = Doppler systolic blood pressure in the injured limb(A) Systolic blood pressure in the uninjured upper limb(B) ABI <0.8 or <0.9 then angiogram
  • 15.
  • 16.
  • 17. • In chronic cases, long-leg radiographs are recommended to evaluate alignment. • Patients with chronic PLC injuries and varus malalignment should be treated with an osteotomy to correct malalignment prior to PLC reconstruction. • Untreated mal-alignment will likely cause the grafts to stretch over time and fail.
  • 18. Surgical vs Non-surgical Mx • Dedmond et al (Meta analysis 2001) • Richter et al (Meta analysis 2002) • Levy et al (Meta analysis 2009) ĂźLiterature supports surgical treatment and postoperative functional rehabilitation of multi- ligament knee injuries. ĂźRare occasions such as advanced age, immobility and comorbidities that nonsurgical treatment can be considered.
  • 19. Repair vs reconstruction • Patients with repair of cruciate ligaments had higher rates of flexion deficit >6°, higher rates of posterior instability and lower rates of return to preinjury activity levels. (Mariani et al) • High reoperation rates have been reported in patients with posterolateral injuries treated with repair. Ăź Anatomic reconstruction of the injured structures using biomechanically validated techniques yield improved outcomes. Ăź In the setting of multi-ligament injuries, reconstruction of the torn ligaments is recommended. Ăź Repair of the collaterals is usually reserved for bony avulsion injuries.
  • 21. Timing of surgery • Early surgery (<3 week) has shown higher incidences of postoperative stiffness and a fixed flexion deformity with higher rates of manipulation under general anesthesia as compared to delayed repair. • Delayed repair has higher chances of scarring of soft tissue with more difficulty in identification and navigation in the joint leading to higher chances of vascular complications. • No conclusive evidence is suggestive of an advantage offered by a single or a staged procedure.
  • 22. Timing of surgery • Acute- generally favoured in literature - 3 weeks( before scarring or necrosis occurs) (Engebretsen et al , Mariani et al , Fanelli et al, Harner et al) - 6 weeks regarded as acute by Levy et al, Laprade et al • Chronic – Not recommended unless forced delay (eg- vascular injury)
  • 23. Choice of graft- Auto vs Allo Auto-grafts Allo-grafts • Tensor fascia lata • Bone patella tendon bone • Hamstring • Quadriceps • Peroneus longus • Tibialis posterior/ anterior • Tendoachilles • Patellar tendon • Hamstrings
  • 24.
  • 25. La Prade Anatomical mcl reconstruction La Prade Anatomical PLC reconstruction
  • 27. Avoiding tunnel convergence • Tunnel convergence increases the risk of reconstruction graft failure. • Potential damage to reconstruction grafts and fixation devices. • Not having sufficient bone stock between the grafts for fixation and graft incorporation.
  • 28.
  • 29.
  • 30.
  • 31. Tensioning sequence Moatshe G, Laprade et al • AL bundle of PCL at 90°(restore normal tibial step off) • PM bundle of PCL in extension • FCL at 20°−30° knee flexion and slight valgus. • Other PLC structures at 60°of flexion and neutral rotation. • ACL near full extension • Finally, Posteromedial corner.
  • 32.
  • 33.
  • 34. Major Pitfalls Ø Patient positioning Ø Graft preparation- Prevent oversizing, ensure availability Ø Tunnel convergence Ø Meniscal root injuries- Malposition of PCL and ACL tunnel Ø Neurovascular complications- when creating tibial tunnels Ø Fixation
  • 35.
  • 36. Summary Ø Complex problem Ø Assessment of vascular and neurological injury paramount. Ø Data lacking for definitive management protocols. Ø Any intervention needs to be individualized by the presence of any life- or limb-threatening complication Ø Early operative treatment yields improved functional and clinical outcomes compared with non- operativemanagement or delayed surgery.