Will you measure-up to become Type 2 Diabetic FREE in 2012?


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Will you measure-up to become Type 2 Diabetic FREE in 2012?

  1. 1. ==== ====For great Natural way to treat Type 2 DIABETES, Check this out:www.healthy-inside-out.com==== ===="All truth passes through three stages.First, it is ridiculed.Second, it is violently opposed.Third, it is accepted as being self-evident."Arthur Schopenhauer(1788 - 1860)What is the true cause of heart disease, and how can we truly reduce the risk of death?Atherosclerosis, or Coronary Artery Disease (CAD), is the leading cause of death in both men andwomen. In the U.S. alone, there are more than one million heart attacks every year, one third ofthem resulting in death. The majority of men and women currently have, or are activelydeveloping, atherosclerosis. By age 20, most people already have a 15-25% narrowing of theirarteries due to plaque formation. By age 40, there is a 30-50% clogging of their arteries.In the beginning of the Twentieth Century, congestive heart disease (CHD) was mostly a result ofrheumatic fever, which was a childhood disease. However by the year 1936 there was a dramaticchange in the main cause of heart disease. Cardiovascular disease caused by atherosclerosis, orplaque buildup, took first place as the primary cause of heart disease, making congestive heartfailure a distant second.During the 1950s, the autopsies conducted on men who died of heart disease that revealedplaque-clogged arteries concluded that cholesterol was the cause of hardening of the arteries(atherosclerosis) and coronary artery disease. Cholesterol, not calcium, was considered the"cause" of heart disease, despite plaque consisting of 95% calcium and a relatively smallpercentage of cholesterol. By 1956 there were 600,000 deaths annually from heart disease in theU.S. Of those 600,000, 90% were caused by atherosclerosis, or clogged arteries. In fewer than 25years, the number one cause of death in the U.S. had changed dramatically ...from congestiveheart disease to coronary artery disease.Because cholesterol was dubbed the "cause" of atherosclerosis, the effort to lower cholesterol byany means began in earnest. Both the food industry and the pharmaceutical industry seized uponthis opportunity to cash in on a cholesterol-lowering campaign by creating foods and drugs thatwould supposedly save lives. Diets, such as the Prudent Diet, were established to lower the
  2. 2. amount of cholesterol intake from food. There was no doubt that both polyunsaturated oils anddrugs reduced cholesterol, but by 1966 it was also apparent that lowering cholesterol did nottranslate into a reduced risk of death from heart disease.As there was so much money to be made from pharmaceutical development, the campaign toproduce cholesterol-lowering drugs kicked into high gear, despite the lack of evidence showingthat the lowering cholesterol reduced the risk of untimely death from heart disease.Heart disease kills 725,000 Americans annually, with women accounting for 2/3 or nearly 500,000of those deaths. After thirty years of cholesterol-lowering medications failure to significantly lowerthe death rate from cardiovascular disease, in 1987 a new and more dangerous class of drugswas unleashed upon the world: the "statin" drugs. Cholesterol-lowering statin drugs are now thestandard of care that physicians are indoctrinated into prescribing to reduce cardiovasculardisease. Are statin drugs the best way to prevent heart attacks and death?Before 1936 the most common type of heart disease was congestive heart disease (CHD). Itrarely caused sudden death and could be treated with the drug digitalis. The incidence of CHDremained stable until 1987, after which the incidence of the disease skyrocketed. Interestingly, thetiming of the increased incidence of congestive heart disease coincides with the introduction ofcholesterol-lowering statin drugs. Could cholesterol-lowering statin drugs have something to dowith the weakening of heart muscles and the increased incidence of congestive heart failure? Wewill see that lowering the bodys co-enzyme Q10 levels, a side effect of statin drugs, does indeedincrease the risk of muscle damage, including the muscles of the heart.Atherosclerosis is a disease characterized primarily by inflammation of the arterial lining caused byoxidative damage from homocysteine, a toxic amino acid intermediary found in everyone.Homocsyteine, in combination with other free radicals and toxins, oxidizes arteries, LDLcholesterol, and triglycerides, which in turn releases C Reactive Protein (CRP) from the liver-amarker of an inflammatory response within the arteries. Inflammation (oxidation) is the beginningof plaque buildup and ultimately, cardiovascular disease. Plaque, combined with the thickening ofarterial smooth muscles, arterial spasms, and clotting, puts a person at a high risk of sufferingheart attack or stroke.For years, doctors have hyper-focused on cholesterol levels. First it was the total cholesterol; laterthe focus became the ratio of "good" HDL cholesterol to "bad" LDL cholesterol. In other words,how much of your cholesterol was good, and how much was bad? Of the two, the importantparameter is the level of HDL cholesterol, not LDL cholesterol. HDL, or high-density lipoproteincholesterol, is responsible for clearing out the LDL cholesterol that sticks to arterial walls.Exercise, vitamins, minerals, and other antioxidants, particularly the bioflavonoid and olivepolyphenol antioxidants, increase HDL cholesterol levels and protect the LDL cholesterol fromoxidative damage, and therefore do more to reduce the risk of heart disease than any medicationever could.There is nothing inherently bad about LDL cholesterol. LDL cholesterol is critical to maintain life.LDL cholesterol only becomes "bad" when it is damaged, or oxidized by free radicals. Only thedamaged, or oxidized form of LDL cholesterol sticks to the arterial walls to initiate the formation ofplaque.
  3. 3. Let us look towards cigarette smoking for a simple example demonstrating that we really need toreduce oxidized LDL cholesterol to prevent atherosclerosis, as opposed to indiscriminatelylowering LDL cholesterol with statin drugs. Everyone knows that cigarette smoking increases therisk of many chronic diseases, such as cancer, heart disease, and stroke. Smokers with normallevels of LDL cholesterol are at an even greater risk of developing heart disease than a non-smoker who has elevated levels of LDL cholesterol. Of course the reason why a smoker withnormal levels of LDL cholesterol is at greater risk of disease is because his LDL gets excessivelyoxidized.Cigarette smoke releases so many toxins and free radicals that the LDL cholesterol, thetriglycerides, and the arterial walls are extensively oxidized. Homocysteine levels are alsoincreased by cigarette smoking which further oxidizes LDL cholesterol and the arterial lining.Oxidation is the initiating cause of atherosclerosis. Therefore, the more and longer one smokes,the more oxidative damage he sustains and the greater his risk of developing heart disease. Thedegree of oxidation directly corresponds to the risk of heart disease.If you are not taking vitamins, minerals, and antioxidants then your LDL cholesterol is beingoxidized, it is sticking to your arterial walls, and you ARE developing heart disease EVEN IFYOUR CHOLESTEROL LEVELS ARE NORMAL! LDL cholesterol starts sticking to arterial wallsbefore the age of 5.Among the many free radicals that damage cholesterol, triglycerides and the arterial lining ishomocysteine, a toxic intermediate biochemical produced during the conversion of the amino acidmethionine into another important amino acid, cysteine. Both methionine and cysteine are non-toxic, but homocysteine is very toxic to the lining of the arterial endothelium. Homocysteineoxidizes LDL cholesterol, triglycerides and the arterial lining.Homocysteine is an amino acid normally produced in small amounts from the amino acidmethionine. The normal role of homocysteine in the body is to control growth and support boneand tissue formation. However a problem arises when homocysteine levels in the body areelevated, causing excessive damage to LDL cholesterol, as well as to arteries. Furthermore,homocysteine actually stimulates growth of arteriosclerotic plaque, which leads to heart disease.Thyroid hormone controls the level of homocysteine, but numerous factors play a role in theelevation of homocysteine. Normal aging, kidney failure, smoking, some medications, andindustrial toxins all elevate homocysteine levels. Interestingly, estrogen helps lower homocysteine.Homocysteine becomes elevated in the blood with a deficiency of the B vitamins-B6, B12 and folicacid. Genetics also play a role. About 12% of the population has an undetected defect requiringhigher levels of folic acid than the rest of population to help maintain homocysteine levels in a saferange (below 6.5). Therefore if you have high homocysteine levels (> 7.0) even though you aretaking supplemental B complex vitamins, then you may be among the 12% who need more than1000 mcg of folic acid per day. In addition, betaine, also known as trimethylglycine (TMG) lowershomocysteine.Homocysteine is second only to cigarette smoking in its oxidative destruction. It causes smallnicks or tears in the arterial lining, while also oxidizing and damaging LDL cholesterol. Thedamaged, or oxidized LDL cholesterol sticks to the homocysteine-damaged areas of the arterial
  4. 4. lining. The combination of oxidized LDL cholesterol and a damaged arterial lining is what causesLDL cholesterol to stick to the arteries, whether or not the LDL cholesterol level is normal.Cholesterol-lowering statin drugs are the standard for treating high cholesterol. This is dogma, andanyone who states otherwise is committing medical heresy. Many people find it hard to believethat pharmaceutical companies could ever succeed in paying medical researchers, medicalassociations, and doctors to recommend something detrimental to our health.Most people do not know that pharmaceutical companies fund medical institutions, medicaleducation, medical conferences, and still reward doctors and research institutions for providingfavorable results on their drugs. Likewise, pharmaceutical companies often suppress negativeresults from studies done on their drugs. Money has the power to sweep negative results andserious side effects under the rug. Money has the power to influence the FDA to decide whichdrugs make it to market and which drugs become the "standard" of treatment.Former editor of the New England Journal of Medicine (NEJM), Dr. Marcia Angell, warned of theproblem of commercializing scientific research in her outgoing editorial titled "Is AcademicMedicine for Sale?" Angell called for stronger restrictions on pharmaceutical stock ownership andother financial incentives for researchers. She said that growing conflicts of interest were taintingscience, warning "When the boundaries between industry and academic medicine become asblurred as they are now, the business goals of industry influence the mission of medical schools inmultiple ways." She did not discount the benefits of research but said, "a Faustian bargain" nowexisted between medical schools and the pharmaceutical industry. Angell left the NEJM in June2000 and has written a book, "The Truth About the Drug Companies: How They Deceive Us andWhat to Do About It."Two years later, in June 2002, the NEJM announced that it was going to begin accepting articlesthat were written by biased researchers, as there werent enough unbiased researchers left towrite articles. In other words, most research institutions were now funded by one or more of thenumerous pharmaceutical companies.An ABC report noted that a survey of clinical trials revealed that when a drug company did notfund a study, favorable results regarding a drug were found only 50% of the time. In studiesfunded by drug companies favorable results about the drugs were reported an amazing 90% of thetime. Money can and does buy the desired results. This is how most medical research and drugsare now developed and brought to market.In 1977, the internationally-renowned heart surgeon, Dr. Michael DeBakey pointed out that only30-40% of people with blocked arteries and heart disease have elevated blood cholesterol levels,and posed the logical question, "How do you explain the other 60-70%?"Because lowering cholesterol did not reduce the risk of death from heart disease, the CholesterolConsensus Conference in 1984 developed new guidelines to lower the "acceptable level" ofcholesterol. High cholesterol would now be the diagnosis for any man or woman with a cholesterollevel over 200. Doctors had to convince their patients that they had the disease and needed totake one or more expensive drugs for the rest of their lives.However, when lowering total cholesterol levels below 200 did not translate into saving lives from
  5. 5. heart attacks, the focus then turned to LDL cholesterol levels. The "disease" of high cholesterolwas refined to the disease of high LDL cholesterol. The unfortunate patient who had an LDLcholesterol level above 130 was now condemned to a lifetime of expensive drugs. Thoughcompletely illogical, even when a person with normal LDL cholesterol levels suffered a heartattack, he would still be prescribed a cholesterol-lowering drug.As we shall see, statin drugs reduce the risk of death by repeat heart attacks by as much as 30%,but interestingly enough, the mechanism of action in reducing the risk of death after a heart attackis not via statin drugs ability to lower cholesterol! It has been discovered that statin drugs have amodest anti-inflammatory and antioxidant effect. Yet, there are many natural antioxidants thatreduce inflammation and oxidation of LDL cholesterol and the lining of the arteries, which maysoon be discovered to be more effective in reducing the risk of death than "antioxidant drugs,"without toxic side effects.The myth that high LDL cholesterol is the primary cause of heart disease, and that we must be ondrugs to protect ourselves is dispelled by the evidence. If the premise were true that people withhigh levels of LDL cholesterol get heart disease, then we could assume that people with normallevels of LDL should not get heart disease, or at least very few should get it. However, as Dr.DeBakey observed, approximately 60% of those who die from heart disease have normal LDLcholesterol levels!Furthermore, after over 45 years of doctors prescribing cholesterol-lowering drugs, heart diseaseand stroke still remain the number one cause of death in both women and men. This says thatregardless of whether you have a high or a normal level of cholesterol, you have a 50% chance ofdying from heart disease. If this is so, and it is, then why take a dangerous drug to attempt to loweryour cholesterol in the first place?In 2001, the target level of LDL cholesterol was lowered from 130 to 100, and overnight thenumber of people considered to be candidates for cholesterol statin drugs doubled. Many peoplesuch as myself bristled at the news, because we knew the effectiveness of vitamins, minerals, andantioxidants in preventing and reversing heart disease. Many of us could see the conspiracy forwhat it was.The level at which LDL cholesterol is considered normal has continually been influenced bypharmaceutical companies, who pull the financial strings of research grants that keep medicalschools and medical organizations in business. The lower they can establish the level at whichLDL cholesterol is considered to be normal, the more people automatically become victims of thedreaded disease of "high cholesterol." Therefore, more people will be persuaded that they need tobe taking a statin drug, and voilà, more profit for the manufacturers. When you considerthe size of the profits already received, let alone the potential profit from statin drugs over the nextseveral years, the cholesterol conspiracy is one of the largest money making schemes everperpetrated on the world.In July 2004, the level of LDL cholesterol considered normal underwent another change. The newnorm plunged from 100 to 70, virtually doubling again the number of people who are "infected"with the plague of high cholesterol. Why, its the epidemic of our time! Many enlightened peoplehowled at this news, wondering if the masses would ever wake up and see who is behind this, andwhy. Why is the medical establishment ignoring the thousands of published medical studies that
  6. 6. show the beneficial effects of nutritional supplements against heart disease? Why is the medicalestablishment down-playing the dangerous and deadly side effects of statin drugs?The "updated" LDL cholesterol recommendations were published in the July 2004 issue of theAmerican Heart Associations publication, Circulation. A panel from the National Heart, Lung andBlood Institute, a division of the National Institutes of Health, which is endorsed by the AmericanCollege of Cardiology, and the American Heart Association, were the ones who actuallypronounced the new cholesterol level at which drugs should be prescribed. Sounds pretty officialand reliable if these powerful medical institutions are backing up these recommendations, right?The fact is eight of the nine panel members making the new LDL cholesterol recommendationswere being paid by the statin-producing pharmaceutical companies. The panelists did not disclosetheir financial conflict of interest. This information was uncovered by Newsday, a Long Island, NewYorknewspaper (D. Ricks and R. Robins, Newsday, July 15, 2004). Seven of the nine panelists havefinancial connections to Pfizer, the makers of Lipitor®. Five of the nine served as "consultants"to Pfizer. So, what did the other two panelists do to deserve their money? Seven of the ninepanelists also received money from Merck, the producers of Zocor®, with four of them servingas "consultants" to the company. Eight of the panelists who made the recommendations thatwould increase the prescribing of statin drugs have received either research grants or honorariafrom Pfizer, Merck, AstraZeneca, Novartis, Glaxo Smith Kline, Johnson & Johnson, Bayer,and many other drug companies that produce statin drugs.You would think that with all the advertising and recommendations from medical experts on thebenefits of statin drugs, the medical community would possess overwhelming evidence that thedrugs reduce the risk of death from cardiovascular disease. A hint of some of the smoke andmirrors in the pharmaceutical companies advertising can be seen in their TV commercials. Readcarefully the small print on some of Crestors® commercial advertising. Their commercialstates how much it lowers LDL cholesterol. However, in the same ad you can read,"...Crestor® has not been shown to reduce the risk of heart disease or heart attack." If so, thenwhy take it? Isnt the bottom line to prevent death?The system for reporting adverse effects from medications is tremendously flawed, so much sothat many people are seriously harmed or killed by some medications before they are finallyremoved from the market. Most doctors do not know what symptoms or effects are due to thedrug, what should be reported, or even to whom to report adverse effects. They assume that theresearch that went into developing the drug has already identified all the effects and that a drugbrought to market is "safe." However, only one in twenty side effects is ever reported to eitherhospital administrators or the FDA.Statin drugs block cholesterol production in the body by inhibiting the enzyme called HMG-CoAreductase in the early steps of its synthesis in the mevalonate pathway. Cholesterol is one of threeend products in the mevalonate chain. This same biosynthetic pathway is also used to create co-enzyme Q10, or co-Q10, as well as dilochol. Therefore, one unfortunate consequence of statindrugs is the unintentional inhibition of both Co-Q10 and dilochol synthesis.The drug information insert of a statin drug states that it lowers co-enzyme Q10 levels. Mostdoctors have forgotten their biochemistry class in medical school, and forgotten about the
  7. 7. importance of Co-Q10. Therefore they apparently are not concerned about such a statement onthe drug labeling information sheet. They may even reassure their patients that lowering Co-Q10is nothing to worry about, but at the same time warn them that the drug may cause liver damageand to have their liver enzymes checked every three to six months to make sure the drug isntkilling them. They do not realize that it is the depletion of Co-Q10 that leads to liver damage anddeath.Ubiquinone, or co-enzyme Q10, is a critical cellular nutrient created in the cells mitochondria, the"engines" that produce energy for the cell. Mitochondria use sugar, oxygen, and water to produceenergy molecules known as ATP. Without ATP cells could do nothing. Damaged tissues could notbe repaired. Cells could not divide or produce or utilize proteins, enzymes, or hormones. Death ofcells, and indeed of the human body would occur if ATP could no longer be produced and utilized.Co-Q10 functions within the mitochondria as an electron carrier to cytochrome oxidase, our mainrespitory enzyme, which helps turn oxygen and sugar into energy. The heart requires high levelsof oxygen, sugar, and Co-Q10 since it utilizes a lot of energy. A form of Co-Q10 called ubiquinoneis found in all cell membranes, where it plays a role in maintaining membrane integrity, so criticalto nerve conduction and muscle contraction. Co-Q10 is also vital for the formation of elastin andcollagen, which make up the connective tissues of the skin, musculature, and the cardiovascularsystem.The most common side effect of statin drugs is muscle pain and weakness. In fact, many patientswho start on the statin drugs almost immediately notice generalized fatigue and muscle weakness.This is due to the depletion of Co-Q10 needed to support muscle function. Dr. Beatrice Golomb ofSan Diego, California, is currently conducting a series of studies on statin side effects. Thepharmaceutical industry insists that only 2-3% of patients get muscle aches and cramps, when infact in one study, Golomb found that 98% of patients taking Lipitor®, and one-third of thepatients taking Mevacor® (a lower dose statin), suffered noticeable to significant muscleproblems.Some people on statin drugs lose coordination of their muscles. Some develop pain in theirmuscles, some are not able to write due to loss of fine motor skills. Many lose the strength toexercise. Others are falling more frequently as their muscles give out, still others have troublesleeping due to muscle cramping and twitching. Even worse, many people are experiencing mostof these side effects. The problems are so numerous, it is difficult to list all the symptoms peoplemight experience. These problems do not come from the "disease" of high cholesterol, but thedisease of ignorance in prescribing these drugs.As we age, Co-Q10 levels decline naturally. From the age of 20 to 80, Co-Q10 levels fall by nearly50%. Along with the natural decline of Co-Q10, comes a natural decrease in energy and anincrease in the risk of heart disease, stroke, and cancer. If the natural decline of Co-Q10 levelsincreases the risk of fatigue, cancer, heart disease, and stroke, would it not make sense thataccelerating the decline of Co-Q10 levels with statin drugs would have the same effect? They doindeed!Demonstrating the importance of Co-Q10 to cardiovascular health, in a randomized, double blind,placebo-controlled study of people either taking or not taking statin drugs, supplementation withCo-Q10 reduced the risk of heart attacks and death in those with heart disease and prior heartattacks by 50%, regardless of whether they were on a statin drug or not. (Singh R, Neki N,
  8. 8. Kartikey K, et al. Effect of coenzyme Q10 on risk of atherosclerosis in patients with recentmyocardial infarction. Mol Cell Biochem. 2003 Apr; 246(1-2):75-82.)Additionally, Co-Q10 was shown to increase blood levels of vitamin E and significantly increasethe levels of protective HDL. As low HDL is a major risk factor for heart disease, increasing it is adefinite benefit. Statin drugs were shown not to provide any benefit beyond that of supplementingwith Co-Q10. Let me make this clear - in this study only the co-enzyme Q10 provided any benefit,not the drugs!Cardiologist Dr. Peter Langsjoen of East Texas University reported the effects of Lipitor®among 20 patients who started with completely normal hearts. After six months on a low dose of20 mg of Lipitor® per day, two thirds of the patients started to show signs of heart failure, asseen by abnormalities in the hearts filling phase. According to Dr. Langsjoen, this malfunction isdue to Co-Q10 depletion. Nine controlled trials using statin drugs in humans have been conductedthus far. Eight of these showed significant statin-induced Co-Q10 depletion leading to a decline inleft ventricular function and other biochemical imbalances.In the United States, the incidence of heart attacks over the past ten to fifteen years has declinedslightly. But congestive heart failure and cardiomyopathy have risen alarmingly. Is it a coincidencethat statin drugs were first marketed in 1987, and then from 1989 to 1997, deaths from congestiveheart failure more than doubled? 38 It scares me that virtually all patients with heart failure are puton statin drugs, even if their cholesterol is already low. In my opinion, the worst thing to do for afailing heart is take a statin drug. The best thing is to take is a full range of quality nutritionalsupplements, ...vitamins, minerals, fish oil, and other antioxidants, including Co-Q10.Various antioxidants work synergistically, each contributing to the fight against free radicals indifferent areas and in different ways. In the blood stream, water-soluble antioxidants, such asvitamin C, and grape seed extract come in contact with and neutralize free radicals before theydamage LDL-cholesterol. Other antioxidants saturate arterial walls and other tissues, and protectcollagen and elastic fibers from free radical damage, reducing inflammation and plaque formation.The fat-soluble antioxidants, vitamin E, beta carotene, and co-enzyme Q10 ride along in the bloodfat (triglycerides) and LDL cholesterol, protecting them and the endothelium from oxidation.Vitamin E sits on the surface of LDL cholesterol, protecting it from free radical damage. Betacarotene, grape seed extract and olive extract penetrate deeper inside the LDL cholesterol andarterial walls, adding more protection from oxidation. Quercetin and alpha lipoic acid work throughnitrous oxide pathways to reduce high blood pressure, a major risk factor for heart disease.A report published in the Archives of Internal Medicine in 2005 looked at 97 double-blind controlledstudies comparing the efficacy of cholesterol-lowering statin drugs to fish oil. They found thatcholesterol-lowering statin drugs reduced the risk of death from heart disease by only 13%, andinteresting enough it was NOT due to the effect of lowering cholesterol. The benefits, althoughsmall, were derived from the fact that statin drugs have a slight antioxidant effect.Even more interesting, the salmon oil was shown to reduce the risk of death from heart disease by23%, nearly double the benefit of statin drugs. Salmon oil is an omega-3 fatty acid that getsincorporated into cholesterol and triglycerides and prevents the oxidation of LDL cholesterol. SinceLDL cholesterol is protected from excessive oxidation there is less plaque buildup and less risk ofheart disease.
  9. 9. Inflammation is a well-known component in the formation of atherosclerosis. To keep it simple,think of inflammation and oxidation as the same process. The immune systems response toinflammation is torelease peroxides that act like acid to break down damaged tissues, so that cells from the immunesystem, macrophages, can consume the molecules and clean up the site. But peroxides escalatethe oxidation/inflammation process, thus damaging more tissue. The arterial walls become moreinflamed, escalating the formation of plaque and scarring. The downward cycle continues untilatherosclerosis is so advanced that the occurrence of a heart attack or stroke becomes imminent.The livers response to inflammation is to release C reactive protein (CRP) into the blood. Otherinflammatory causes can cause elevated CRP levels, including cigarette smoking, obesity, insulininsensitivity, diabetes, rheumatoid arthritis, infections, dementia, colorectal cancer, high bloodpressure, and aging. Accordingly, elevated CRP levels are a direct indication of inflammation inthe body and that atherosclerosis, including heart disease, is actively developing.Homocysteine and high sensitivity CRP levels can and should be tested. Dr. Jialal, of theUniverstity of Texas Southwestern Medical School at Dallas, is well known for his researchcorrelating oxidized LDL cholesterol as the true cause of atherosclerosis, has also identified highsensitivity C reactive protein as a predictive risk factor for inflammation of arterial walls and plaqueformation. Your doctor may not test for these routinely, but you should insist on getting these testsdone. Both of these predictive values can be kept at "safe" levels. Vitamins, minerals, antioxidants,and omega-3 fatty acids can lower the levels of homocysteine and CRP. The B vitamins, alongwith betaine, or tri-methyl-glycine (TMG), change homocysteine into safer amino acids and reduceinflammation of the LDL cholesterol and the arterial lining.When you receive the results of your homocysteine test, do not accept the answer, "Your test wasnormal." Ask for the actual number. The doctor and nurse usually know what is normal by what thelab slip states as the "normal range." Most lab results report a normal homocysteine level as beingbelow 10.4, when in fact, since the early 1990s, researchers have known that a homocysteinecount above 6.5 signals a rapid linear rise in the risk for heart disease.Furthermore, with every 3 point elevation of homocysteine above 6.5, e.g., when homocysteinelevels are 9.5, the risk of coronary artery disease (CAD) rises by an additional 35%! Yet you maybe told that 9.5 is "normal and not to worry." With a homocysteine level of 12.5, the increase in therisk for heart disease exceeds 70%. The greater the homocysteine level, the greater the oxidationof both LDL cholesterol and the arterial lining. The greater the inflammation, the higher the CRP. Isit any wonder that homocysteine and CRP levels are more predictive for risk of heart disease thancholesterol levels and ratios?I need to emphasize that anyone whether they have a medical problem or not, should discuss thisinformation with their physician before acting upon anything written here. The information providedis not meant to diagnose or treat any disease. It is for informational purposes only; and no oneshould make decisions about their medications without consulting with their physician. No oneshould come off a cholesterol-lowering statin drug in lieu of nutritional supplements without athorough discussion with their physician who is keenly aware of all the pros and cons of bothtreatment modalities.
  10. 10. In summary, I recommend a full spectrum of quality nutritional supplements, along with a healthydiet and exercise, to help obtain and maintain optimal heart and arterial health. I believe all wouldagree that lifestyle changes are the most important factor for optimal health, ...and many believethat quality nutritional supplements are key in protecting against the process that leads to, andaccelerates the development of almost all chronic degenerative diseases, that of oxidation. Tocombat oxidation we need a full range of quality antioxidants.http://comparativeguide.comhttp://customer.usana.comhttp://cholesterolconspiracy.blogspot.comhttp://mcnamaraupdates.blogspot.comArticle Source:http://EzineArticles.com/?expert=Ladd_McNamara,_M.D.==== ====For great Natural way to treat Type 2 DIABETES, Check this out:www.healthy-inside-out.com==== ====